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1 nd expand upon the clinical phenotype of the logopenic variant.
2 found in nfvPPA, but not in the semantic or logopenic variants.
3 -fluent/agrammatic non-fluent/agrammatic and logopenic variants.
4 n the distinction of the agrammatic from the logopenic variant and left considerable latitude to clin
5 ia with 3 subtypes, semantic, nonfluent, and logopenic variants; and movement disorders including pro
6 mical profile, the non-fluent/agrammatic and logopenic variants are difficult to discriminate from ne
7 non-fluent/agrammatic variant, and four with logopenic variant), as well as 28 age-matched healthy co
8 be classified into non-fluent, semantic and logopenic variants based on motor speech, linguistic and
11 omes (non-fluent/agrammatic variant, nfvPPA; logopenic variant, lvPPA; semantic variant, svPPA) and b
12 s with primary progressive aphasia including logopenic variant (n = 14, age = 61 +/- 9 years), non-fl
15 The aim is to explore the evolution of the logopenic variant of primary progressive aphasia as a di
16 ural FTD to FTD with memory onset and to the logopenic variant of primary progressive aphasia in one
18 terior cortical atrophy, 4 subjects with the logopenic variant of primary progressive aphasia, 6 age-
19 including posterior cortical atrophy and the logopenic variant of primary progressive aphasia, differ
21 variant PPA (agPPA) with tau deposition and logopenic variant PPA (lvPPA) with Alzheimer's disease (
22 role of short-term memory in a discussion of logopenic variant PPA, and components of language associ
24 nset, <65 y; memory and executive deficits), logopenic variant primary progressive aphasia (language
25 otypes: Posterior Cortical Atrophy (n = 16); logopenic variant Primary Progressive Aphasia (n = 15);
27 e (age at onset <65 years), 12 patients with logopenic variant primary progressive aphasia and 13 pat
29 pathology, although some phenotypes such as logopenic variant primary progressive aphasia are more c
30 iew aimed at broadening the understanding of logopenic variant primary progressive aphasia beyond the
32 Alzheimer's disease, non-fluent variant and logopenic variant primary progressive aphasia patients v
33 interest, the left language network for the logopenic variant primary progressive aphasia region of
34 nificantly higher in Alzheimer's disease and logopenic variant primary progressive aphasia than seman
35 disease and posterior cortical atrophy than logopenic variant primary progressive aphasia) and highe
36 emantic variant primary progressive aphasia; logopenic variant primary progressive aphasia), compared
37 than in early-onset Alzheimer's disease and logopenic variant primary progressive aphasia), with a t
38 ypical variants (posterior cortical atrophy, logopenic variant primary progressive aphasia, and corti
39 and non-linguistic cognitive difficulties in logopenic variant primary progressive aphasia, and predi
40 ase syndromes (Alzheimer's disease dementia, logopenic variant primary progressive aphasia, or poster
41 ates of non-linguistic cognitive deficits in logopenic variant primary progressive aphasia, suggestin
42 ng research on non-linguistic dysfunction in logopenic variant primary progressive aphasia, we propos
46 9 posterior cortical atrophy/PCA-AD; n = 103 logopenic variant primary progressive aphasia/lvPPA-AD;
47 ad in three non-amnestic variants, including logopenic-variant primary progressive aphasia (n = 25),
49 and occipital cortices in patients with the logopenic variant, within the left inferior frontal cort