ライフサイエンスコーパス: long latency

1 plasia in vivo that progress to tumors after long latency.

2 induced SCC and develop spontaneous SCC with long latency.

3 rred in a cluster after 9 months, indicating long latency.

4 developed one or more mammary tumors after a long latency.

5 cells of mGluR6-null mice, but they occur at long latency.

6 rs non-lethal invasive prostate cancer after long latency.

7 ltered myelopoiesis with severe anemia after long latency.

8 and induces pre-T-cell tumors in mice after long latency.

9 ch, results in mammary tumor formation after long latency.

10 sistent viral replication and maintains life-long latency.

11 s, including breast cancer, appeared after a long latency.

12 cancer is low and occurs after a relatively long latency.

13 rs in mutant glands at low frequency after a long latency.

14 uently causes epilepsy that develops after a long latency.

15 age and, therefore, often after a relatively long latency.

16 velopment of metastatic mammary tumors after long latency.

17 and causes high-grade adenocarcinoma with a long latency.

18 ed glioma but with incomplete penetrance and long latency.

19 rare population of the starting cells with a long latency.

20 which progressed to secondary leukemia after long latency.

21 ctivity in ITC were weak and occurred with a long latency.

22 K14-Cre led to mammary tumor formation with long latency.

23 ent of invasive IHCC with low penetrance and long latency.

24 development of myeloid leukemia, it is with long latency.

25 EC) mice developed intestinal tumors after a long latency.

26 on their own induce T-cell malignancies with long latency.

27 r cells exhibited the late peak with similar long latencies.

28 ulation evokes electromyograms at abnormally long latencies.

29 us myeloid, T- and B-cell malignancies after long latencies.

30 .55; 0.33-0.93), particularly persons with a long latency (10+ years) between the 2 conditions.

31 The long latency (234-299 days) and clonal chromosomal abnor

32 After long latency, 30% of the MN1-TEL-positive mice developed

33 Reach adaptation produced an increase in the long-latency (45-100 ms delay) feedback gains with respe

34 The long latency 5-HT-evoked increase in BAT SNA was prevent

35 with features of human APL, but only after a long latency (8.5 months in MRP8 PML-RARA mice).

36 /abl transgenic mice known to develop, after long latency, a myeloproliferative disorder resembling h

37 We conclude that short as well as long latency acoustic transmissions to click change in t

38 tency activation (<25 msec; 55.1% of cells), long latency activation (>65 msec; 56% of cells), and in

39 ectrode significantly reduced both short and long latency activations evoked in DA neurons by vBNST s

40 play a significant role in short as well as long latency, adaptive acoustic transmission that can en

41 The absence of long-latency AEPs should not, therefore, be considered i

42 Short latency afferent inhibition (SAI) and long latency afferent inhibition (LAI) measured with tra

43 ficantly increase survival; however, after a long latency, all tumors subsequently became resistant.

44 culture, and generate a partially penetrant, long-latency AML in bone marrow transplant recipients.

45 riod and were found to develop lymphoma with long latency and at high probability (more than 85% over

46 rcoma-associated herpesvirus, establish life-long latency and can reactivate in immunocompromised ind

47 of the transfected terminals, along with the long latency and complex physiological responses of thal

48 Given the long latency and incomplete penetrance of AD dementia wi

49 Mutant p53-R334H mice developed tumors with long latency and incomplete penetrance, consistent with

50 Mammary tumour formation occurs after long latency and is associated with genetic instability

51 er associated with asbestos exposure, with a long latency and poor overall survival.

52 f Lmo1 and Lmo2 cause T cell leukaemias with long latency and that Lmo2 expression leads to an inhibi

53 At low levels, spikes occurred with long latency and the firing frequency increased througho

54 d that Mcph1(-/-) mice developed tumors with long latency, and that primary lymphoma developed signif

55 terozygous strain also formed tumors after a long latency, and the cells from those tumors lacked p53

56 Older age and short or long latencies are not associated with DILI mortality.

57 ly variable escape responses with relatively long latencies as well as the unilateral recruitment of

58 Mammary (and other) tumors occur at long latency as compared to oncogene-induced mouse tumor

59 The long latency associated with the development of colorect

60 ess higher auditory processing capabilities, long-latency auditory evoked potentials (AEPs) were reco

61 der lines develop mammary tumors, but with a long latency (average age of 18 months).

62 In the first IGL, a long "latency B-hairpin" protrudes ~30 A from the surfac

63 Pten(fl/fl)) develops prostate cancer with a long latency, because disease initiation in this model r

64 of the early M20, as well as the strength of long latency beta oscillations.

65 The high burden of morbidity, coupled with a long latency between BMT and the development of chronic

66 f its ubiquity, treatment-related morbidity, long latency between premalignant lesions and clinically

67 In diabetes there is a long latency between the onset of hyperglycemia and the

68 No wind-up of either long latency C-fibre or short latency Adelta responses w

69 for discrimination between short-latency and long-latency C-starts (SLCs vs. LLCs) in larval zebrafis

70 for discrimination between short-latency and long-latency C-starts (SLCs vs. LLCs) in larval zebrafis

71 During a long latency, cancer-related genes were disrupted or amp

72 Longer follow-up for typically long-latency cancers and attention to specific cancer si

73 we used dsTMS to systematically investigate long-latency causal interactions between right-hemispher

74 ed in AM and perirhinal cortex by a distinct long-latency code, explaining how the same cell populati

75 chimeric mice, and these tumours occur with long latency compared with those found in MLL-Af9 chimer

76 Whereas the long-latency component seemed to occur with the eye move

77 ions supported the hypothesis that short and long latency components of blink responses represented c

78 In these subjects, the long latency components of the reflex response could, in

79 COMT activity specifically alters long-latency components of the event-related response.

80 from non-DS simple cells for both short- and long-latency components, with temporal phase differences

81 se was composed of two movements: short- and long-latency components.

82 ere that shape-from-shading stimuli evoked a long-latency contextual pop-out response in V1 and V2 ne

83 These results indicate that long-latency contributions to reciprocal inhibition of s

84 se of the study was to assess whether or not long-latency contributions to reciprocal inhibition of s

85 Findings indicate that long-latency contributions to reciprocal inhibition of t

86 n the left eye, whereas the long CS produced long-latency CRs in the right eye.

87 Within this variation, we found robust long-latency decreases (300 and 2000 ms after stimulus p

88 uced leukemias in irradiated recipients with long latencies, demonstrating both a requirement for sup

89 A long-latency depolarization interrupts the IPSP with a p

90 hyperpolarization and before the peak of the long-latency depolarization yields an augmenting respons

91 Long-latency DRPs were also present and superimposed on

92 vocal performance and that HVo neurons show long latency electrophysiologic auditory responses.

93 r stimulation triggers synaptically mediated long-latency epileptiform burst discharges.

94 in <60 ms after perturbation, during the R2 long-latency epoch (~45-75 ms).

95 rved ~75 ms after perturbation during the R3 long-latency epoch (~75-105 ms).

96 ce of a nonpathogenic helper virus developed long-latency erythroblastosis, and subsequent viral pass

97 Prion infections can have long latencies even though there is no protective immune

98 ibition (61.8% of cells, usually followed by long latency excitation).

99 rate that mPFC stimulation evokes short- and long-latency excitation and inhibition in DA neurons.

100 luded a short-latency post-CS inhibition and long-latency excitations before and after the CS.

101 cervical interneurons tested also exhibited long latency excitatory responses to lumbar dorsal root

102 o interneurons was strong enough to generate long-latency feedforward GABAergic input onto pyramidal

103 Given burdensome side-effects and long latency for efficacy with conventional agents, ther

104 ishabituation of the electrically stimulated long-latency giant fiber pathway response were shown in

105 yed short-latency <180 ms (DSLF) and delayed long-latency >180 ms (DLLF).

106 A subset responded to taste with long latencies (>1.0 s), suggesting the activation of ex

107 These connections were weak, had long latencies (>4 msec), and therefore were probably po

108 Long-latency (> 10 ms) EPSPs were seen in both cell type

109 Both short-latency (< 50 ms) and long-latency (> 50 ms) excitatory responses were seen.

110 rus that infects humans and establishes life-long latency, has evolved numerous mechanisms to evade h

111 l frequency, and speed, whereas neurons with long latencies have high spatial acuity, low sensitiviti

112 However, mid- and long-latency homonymous and crossed responses in both hi

113 ice develop lung tumours at high rates after long latencies, implicating defects in the mitotic check

114 ro and induces acute myeloid leukemias after long latencies in syngeneic recipient mice.

115 nduces acute myelogenous leukemia (AML) with long latency in bone marrow transplantation assays.

116 1), a significant pathogen, establishes life-long latency in certain neurons.

117 68 (MHV68, gammaHV68, MuHV-4) establish life-long latency in circulating B cells.

118 uman alpha-herpesviruses that establish life-long latency in neural ganglia after initial primary inf

119 rpesvirinae subfamily members establish life-long latency in neurons within the trigeminal ganglia an

120 roinvasive dsDNA viruses that establish life-long latency in peripheral nervous system (PNS) neurons

121 Later evolution of long latency in Plasmodium vivax was a necessary adaptat

122 Human herpesviruses establish life-long latency in the host, and it is plausible that a lat

123 ll lymphotrophic viruses that establish life-long latency in the host.

124 e other herpesviruses, KSHV establishes life-long latency in the human host with intermittent periods

125 embles the APL-like disease that occurs with long latency in the PML/RARalpha transgenics, suggesting

126 ted relative to that for unfamiliar faces at long latency; in TP this memory-related rotation was muc

127 othalamic terminals induced small-amplitude, long-latency increases and/or decreases of activity in t

128 o2 in T cells results in clonal tumours with long latency indicating that mutations in other genes ar

129 rs appear stochastically and with relatively long latency, indicating an additional requirement for o

130 althcare providers unfamiliar with acute and long latency infections and diseases common in this popu

131 rotrophic factor (BDNF), have both rapid and long-latency influences on synaptic strength.

132 Subjects (n=14) had long-latency inhibition at rest (x = -35.0 +/- 18.7%).

133 n, the latter suggesting that N2 may reflect long-latency inhibition of irrelevant stimuli.

134 l electrical stimulation produced bilateral, long latency inhibitory postsynaptic potentials (IPSPs).

135 correspond to the properties of the second, long-latency inhibitory component of type IV responses.

136 ese excitatory inputs were often followed by long-latency inhibitory postsynaptic potentials.

137 The long-latency inputs could potentially arise from lagged

138 Thus, long-latency interhemispheric interactions, likely refle

139 y to sensory and autonomic nerves where life-long latency is established(1).

140 ubset of neurons survive infection, and life-long latency is established.

141 These pathways are likely to convey long-latency jaw-muscle stretch reflexes and may contrib

142 fferents induced short-latency (SAI) but not long-latency (LAI) afferent inhibition of face M1, while

143 ons responded weakly to sensory stimuli, had long latencies, large receptive fields, and poorly devel

144 dent modulation of monosynaptic (middle) and long-latency (late) stimulation-evoked EMG responses was

145 inct MLL fusion proteins to promote short or long latency leukemogenesis.

146 , short latency sustained (SLS, n = 12), and long-latency (LL, n = 6) to CRD.

147 Both short-latency (SL; 3-5 ms) and long-latency (LL; >/=9 ms) responses were observed after

148 latency for a switch from the short- to the long-latency location.

149 short-duration activation of neurons and (2) long-latency, long-duration activation of neurons.

150 ked in Golgi cells by the same stimuli was a long-latency, long-lasting depression of firing, found i

151 The most common response was a modest, long-latency, long-lasting increase in simple spike outp

152 Meanwhile, long latency-long duration responses potentially reflect

153 rtical neurons interposed in a transcortical long-latency loop leading to pre-programmed reactions to

154 Seizures activate structures via short and long latency loops, and anatomical connections of the se

155 ce expressing PML-RAR alpha develop APL with long latency, low penetrance, and acquired cytogenetic a

156 e retinal ganglion cells (ipRGCs), including long latencies, marked poststimulus persistence, and a p

157 rly stages of colorectal carcinogenesis, but long latencies may be needed to observe a reduction in c

158 The long latency (mean = 157 days), reduced penetrance, and

159 reased BAT SNA (peak: +342% of control) at a long latency (mean onset: 23 min).

160 Om neurons responded to whisker stimuli with long-latency (median, 27 msec) and low-magnitude respons

161 late responses as likely mediated by either long-latency monosynaptic (n = 108) or non-monosynaptic

162 Both old and new M1 generated putative long-latency monosynaptic and non-monosynaptic effects;

163 Long-latency muscle responses paralleled changes in the

164 The long-latency neurons had a mean onset latency of 36.0 +/

165 ma oscillations, infants display a distinct, long latency, noxious evoked 18-fold energy increase in

166 Given the relatively long latency of conditioned responses we observed in PL

167 rences in outcomes of patients with short vs long latency of DILI.

168 The long latency of HD after transplant and lack of associat

169 rvation consistent with the inefficiency and long latency of iPSC reprogramming.

170 The long latency of mammary tumors in WAP-h-Int3sh mice coul

171 eriods of cancer cell dormancy can result in long latency of metastasis development.

172 The long latency of prostate cancer development provides an

173 its answer remains controversial due to the long latency of the auditory feedback pathway and techni

174 However, owing to the long latency of tumor formation and the sporadic occurre

175 clonal Ikaros-mutant pre-B cells resulted in long-latency oligoclonal pre-B-ALL, which demonstrates t

176 The long-latency ON responses are not blocked by metabotropi

177 In mGluR6-null mice, long-latency ON responses are observed in the visual cor

178 y tumors escaped immune surveillance after a long latency or equilibrium period.

179 l pancreas, with tumors forming only after a long latency or pancreatitis induction.

180 ious in providing radical cure of short- and long-latency P. vivax malaria in Nepal.

181 thesis that inaccurate delay compensation in long-latency pathways could be the origin of the tremor.

182 f the animals develop acute leukemia after a long latency period (6-13 months).

183 The long latency period and incomplete penetrance suggest th

184 al degradation is widely prevalent and has a long latency period between exposure and health outcome,

185 However, the long latency period between infection and development of

186 w causes a myeloproliferative disease with a long latency period but with high penetrance.

187 d with caution given the short follow-up and long latency period for most cancers, the intensive medi

188 even decades, which may explain the observed long latency period for neurological disease onset among

189 n infected with HPV clear their lesions, the long latency period from infection to resolution indicat

190 ive cancer progression after a comparatively long latency period is primarily driven by the mobilizat

191 The molecular mechanisms underlying the long latency period of mesothelioma and driving carcinog

192 ular changes drive carcinogenesis during the long latency period of mesothelioma development and show

193 jury, inflammation, and proliferation in the long latency period of MM development that may be perpet

194 Given the long latency period of pancreatic cancer, exploring the

195 potentially contributes to the indolence and long latency period of this disease.

196 The long latency period suggests that additional genetic alt

197 of leukemia and neurological disease after a long latency period, and the mechanism by which the viru

198 imals develop B and T cell lymphomas after a long latency period, but the incidence is dramatically e

199 After a long latency period, we find epithelial-specific PTEN de

200 t (ANV) of MMC (tumor evasion model) after a long latency period.

201 nal noise exposure even after allowing for a long latency period.

202 focal hepatocellular carcinoma (HCC) after a long latency period.

203 duce colorectal cancer risk but only after a long latency period.

204 ce developed mature T cell leukemias after a long latency period.

205 reduced risk of proximal colon cancer with a long latency period.

206 Thus, in spite of long latency periods during which secondary alterations

207 ouse ltk(-) cells confirm a channel that has long latency periods to opening (1.67 +/- 0.073 s at +60

208 Many cancers have long latency periods, and dietary factors in adolescence

209 nesis, by reduced penetrance or by extremely long latency periods.

210 iated with a lower risk of overall CRC after long latency periods.

211 t effect on prostate cancer risk except with long latency periods.

212 ynaptic (n=2), di-or tri-synaptic (n=18) and long-latency polysynaptic (n=16) responses were recorded

213 C5-C7) suggest that much of the delay in the long-latency polysynaptic responses require a bilaterall

214 appears during response preparation, as in a long-latency positive SOA.

215 Selective attention strongly modulated long-latency potentials evoked by words.

216 nsplant (BMT) model, whereas T/T(F) causes a long-latency, pre-B-cell lymphoblastic lymphoma.

217 The long latency raises the possibility that Golgi cells rec

218 neurophysiological measures (i.e. short and long-latency reflex and shortening reaction) were synchr

219 s demonstrate how distinct components of the long-latency reflex can work independently and together

220 By contrast, long-latency reflex components are typically assumed to

221 is essentially ipsilateral, and in whom the long-latency reflex components following digital nerve s

222 for the perturbation but also increases the long-latency reflex gain associated with leftward displa

223 avior is mediated by volitional and possibly long-latency reflex pathways with delays of at least 120

224 fects in the early (50-75 ms) portion of the long-latency reflex, indicating that these components of

225 ondly, that such responses do not use those 'long-latency' reflex pathways probed by cutaneomuscular

226 1 Hz reduced the amplitude of both MEPs and long latency reflexes by 20-30 % for about 10 min after

227 Long latency reflexes were facilitated with the same tim

228 Long-latency reflexes (LLRs) are critical precursors to

229 ensory evoked potential cortical components, long-latency reflexes and decreased short-interval intra

230 s underwent somatosensory evoked potentials, long-latency reflexes and short-interval intracortical i

231 lude that spinal circuits also contribute to long-latency reflexes in distal and forearm muscles, alo

232 Long-latency reflexes progressively increased according

233 or these rapid motor responses, often called long-latency reflexes.

234 tible with a contribution to both short- and long-latency reflexes.

235 Cardiac T2* changes have a long latency relative to liver iron accumulation.

236 le speech) and positive (VBM-pos; relatively long latency), respectively.

237 esulted in a significant wind-up response of long latency response in six of ten cells studied.

238 Furthermore, failures of the long-latency response during habituation, which normally

239 in the interior of the fly brain and (ii) a long-latency response in which electrical stimulation tr

240 The long-latency response is obliterated at concentrations s

241 b muscles, homolateral and diagonal mid- and long-latency response occurrence significantly decreased

242 sponse to whisker deflection, those having a long-latency response, and neurons whose firing is suppr

243 ring learning correlated with changes in the long-latency response, showing subjects who adapted more

244 tudies suggest the involvement of the RST in long latency responses (LLRs).

245 The TTM muscles of cpo mutants exhibit long latency responses coupled with decreased following

246 ere unresponsive and twenty-eight (24%) gave long latency responses following SCN stimulation.

247 Long-latency responses (>25 msec delay; "weakly coupled"

248 Furthermore, long-latency responses at the edge of the receptive fiel

249 Adapted long-latency responses expressed (de-) adaptation simila

250 mbs, however, when present, short-, mid- and long-latency responses maintained their phase-dependent

251 y that maintained consistent activity of the long-latency responses ranged from 40 to 60 Hz, whereas

252 The long-latency responses to epidural stimulation are corre

253 expectedly, many V1 neurons gave significant long-latency responses to texture stimuli located entire

254 At higher intensities, long-latency responses were recruited in a highly nonlin

255 atients' early muscular responses (short and long-latency responses, 20-50 and 50-100 ms, respectivel

256 of the quadrature model because of a lack of long-latency responses.

257 es, although middle (monosynaptic) and late (long latency) responses were more prominent on the non-d

258 value information is taken into account for long-latency saccades.

259 No long-latency spike responses were evoked in response to

260 al level difference sensitivity contained in long-latency spikes.

261 tion between primary SLCs and less frequent, long-latency startle responses (LLCs).

262 The long-latency stretch reflex (LLSR) in human elbow muscle

263 s report is the first demonstration that the long-latency stretch reflex can be modified by repeated,

264 The amplitude of the long-latency stretch reflex of the contralesional hand d

265 ulated sequences, the second goal influenced long-latency stretch responses to external loads applied

266 associated with this clinical sign (i.e. the long-latency stretch-induced reflex).

267 ouse model; however, tumors developed with a long latency, suggesting a second event is needed to tri

268 nd a low percentage of mammary tumors with a long latency, suggesting that the resulting tumors were

269 Such long-latency suppressions also included monotonically in

270 est frequency (BF) of a neuron, we uncovered long-latency suppressions caused by single-tone stimulat

271 Most cells showed long-latency sustained responses.

272 horns of normal spinal cord slices revealed long-latency synaptic responses in lamina II and short-l

273 HSV-1 establishes life-long latency that can result in clinical relapses or in

274 n the intestine can been observed only after long latencies, they result in rapid carcinogenesis in t

275 omatosensory and visual modulations occur in long-latency time windows previously associated with tac

276 le and developed in one or more glands after long latency (time for median tumor-free survival of app

277 second cancers involves modeling: because of long latency times, available data is usually for older,

278 litation (LTF) of phrenic motor output via a long-latency TLR4-dependent mechanism.

279 Reflexes occurred unreliably and at long latency to 44.0 or 0.3 degrees C and were not appro

280 Responding with an unusually long latency to light stimulation, OND RGCs respond earl

281 ped in Mll-AF4 mice after prolonged latency; long latency to malignancy indicates that Mll-AF4-induce

282 he other half of socially defeated rats show long-latencies to defeat (LL/resilient) and are similar

283 The switch to long latency-to-onset occurred abruptly as a function of

284 western Pacific suggests the possibility of long-latency toxins, but pinning down a specific causati

285 hese cerebral events reflect components of a long-latency transcerebral reflex pathway that is affect

286 oneurons and/or interneurons, rather than by long-latency transcortical reflex responses.

287 the increase in force is similar to that of 'long-latency' transcortical reflexes recorded from muscl

288 or cells expressing JAK3 mutants developed a long-latency transplantable T-ALL-like disease, characte

289 ammary tumors from double transgenic mice to long latency tumors from single transgenic mice and obse

290 tumors is changing with increasing latency, long latency tumors in other organs could occur in the f

291 After a long latency, tumors also develop in animals never expos

292 l short-latency units (< 12 ms) but never in long-latency units (> or = 12 ms).

293 The long-latency units were recorded at an average depth of

294 as NMDAR blockade was much more effective in long-latency units.

295 ade reduced onset spikes more effectively in long-latency units.

296 its but reduced spikes substantially for all long-latency units.

297 and the juveniles lacked the characteristic long latency UP state currents in middle layers.

298 that the amygdala enables the development of long-latency (US anticipatory) responses and prevents th

299 models, leukemia developed after a variably long latency, with variable penetrance.

300 M1 with complex dynamics, as well as evoking long-latency, wM1-dependent whisking.