ライフサイエンスコーパス: long terminal repeat

1 prevent downstream transcription from the 3' long terminal repeat.

2 romoter sequences in the U3 region of the 3' long terminal repeat.

3 docking of Tat at the TAR region of the HIV long terminal repeat.

4 s and differential DNA methylation at its 5'-long terminal repeat.

5 ngle-stranded RNA40 (ssRNA40) from the HIV-1 long terminal repeat.

6 of NF-kappaB and its recruitment to the HIV-long terminal repeat.

7 ription through HIF-1 association with HIV-1 long terminal repeat.

8 al cells using the mouse mammary tumor virus long terminal repeat.

9 the HIV Tat protein to transactivate the HIV long terminal repeat.

10 s and histone-modifying enzymes to the HIV-1 long terminal repeat.

11 thers had acquired dual mutations within the long terminal repeat.

12 ta-globin locus within the self-inactivating long-terminal repeat.

13 omplex that is associated with repressed HIV long terminal repeats.

14 mating the age of the elements, if they have long terminal repeats.

15 most common hybrid genomes had heterologous long terminal repeats.

16 anscripts is promoted by endogenous intronic long terminal repeats.

17 l vector genomes that consisted of linear, 1-long-terminal-repeat (1-LTR), and 2-LTR circular DNAs.

18 verse transcripts, a moderate reduction of 2-long terminal repeat (2-LTR) circles, and a relatively l

19 HIV-1 DNA, including 2-long terminal repeat (2-LTR) circles, and multiply splic

20 Two-long terminal repeat (2-LTR) circles, which are formed i

21 g total HIV DNA, integrated HIV DNA, and two long terminal repeat (2-LTR) circles.

22 as defined by an increase in the level of 2-long terminal repeat (2-LTR) circles.

23 iral DNA to circularization in the form of 2-long terminal repeat (2-LTR) circles.

24 seminal plasma CMV (P = 0.04), detectable 2-long terminal repeat (2-LTR), and lower nadir CD4(+) (P

25 An increase in 2-long-terminal-repeat (2-LTR) circles in the depleted FAC

26 onintegrated viral DNA, particularly the two-long-terminal-repeat (2-LTR) circles.

27 episomes containing two copies of the viral long terminal repeat (2LTR circles) were analyzed in usi

28 6-bp extension at the viral U5 end of the 3' long terminal repeat (3'-LTR), which is a poor substrate

29 eIF3f and eIF3f specifically targeted the 3' long terminal repeat (3'LTR) region in the viral mRNA.

30 node tissue from axilla was positive for the long-terminal repeat (33 copies per 10(6) cells) and env

31 ic drugs and tested these compounds on HIV-1 long terminal repeat-activated transcription.

32 ransactivated gene expression from the HIV-1 long terminal repeat and COT Nef mediated downregulation

33 Both P-TEFb recruitment to the HIV long terminal repeat and enhanced HIV processivity were

34 ve and cryptic splice sites in the HIV 5iota long terminal repeat and gag gene as well as in the beta

35 restrictive chromatin structures at the HIV long terminal repeat and limiting P-TEFb levels contribu

36 encoded protein Tax transactivates the viral long terminal repeat and plays a critical role in virus

37 -1(JR-CSF) regulated by the endogenous HIV-1 long terminal repeat and the hu-cycT1 gene under the con

38 ications in the regulatory elements in their long terminal repeats and differed in a helical segment

39 The DNA of the element is marked by long terminal repeats and encodes a single large protein

40 and DNMT3a/3b in suppressing retrotransposon long terminal repeats and long interspersed elements, re

41 HIV-1 subgenomic DNA fragments, spanning the long terminal repeats and the Gag gene, are excised in v

42 ersed repetitive elements such as Alu, LINE, long-terminal repeats and simple tandem repeats are freq

43 efines the end of the U5 region in the right long terminal repeat, and the subsequent removal of this

44 s, mutational analysis of the HERV-K (HML-2) long terminal repeat, and treatments with agents that in

45 E. granulatus genome, especially Gypsy-like long terminal repeats, and there has also been an expans

46 detected at satellite, telomeric and active long-terminal repeats, and can spread into proximal uniq

47 wild type enzyme in assembling on the viral long terminal repeat, as each variant required more prot

48 ecific gene family expansions, the number of long-terminal-repeat-based retrotransposons, and mechani

49 ity function analysis indicates that several long-terminal-repeat bursts that occurred from 5.7 milli

50 rase II (Pol II) binding to the HIV provirus long terminal repeat but did not prevent the induction o

51 iral load was associated with undetectable 2-long terminal repeat circles (P < .001) and HIV-negative

52 d in a smaller increase in the number of two-long terminal repeat circles than for virus specifically

53 V production (ie, HIV-specific antibodies, 2-long terminal repeat circles) and markers of immune acti

54 virus type 1 (HIV-1) late RT products and 2-long terminal repeat circles.

55 r HIV RNA transcription or more detectable 2-long terminal repeat circles.

56 ion-competent reservoirs, proviral DNA, or 2-long-terminal repeat circles, although APOBEC3G, TRIM5al

57 mutants synthesized normal quantities of two-long-terminal-repeat circles in arrested HeLa cells, ind

58 rectly processed viral ends and abortive two-long-terminal-repeat circles.

59 (P=.013), and frequency of detectable HIV 2-long terminal repeat circular DNA (P=.013) were signific

60 eeds normally in MX2-expressing cells, but 2-long terminal repeat circular forms of HIV-1 DNA are les

61 We demonstrate that the foamy virus long terminal repeats contain an insulator element that

62 he (+)-RNA strand genome of retroviruses and long terminal repeat-containing retrotransposons reflect

63 that many 2C transcripts are initiated from long terminal repeats derived from endogenous retrovirus

64 ds, and recognize an unexpected abundance of long terminal repeat-derived and LINE1-mobilized transpo

65 However, during HIV-1 infection, two-long terminal repeat DNA circles (2-LTRcs) are also gene

66 cultures, hCMV IE-driven, but not the viral long terminal repeat-driven, silent GFP reporter express

67 the effects of different plasmid-based HIV-1 long-terminal-repeat-driven constructs expressing antise

68 ents and chimeric transcripts initiated from long terminal repeats during zygotic genome activation.

69 We show that a long terminal repeat element inserted into intron 35 exp

70 e we demonstrate that DNA hypomethylation at long terminal repeat elements representing the most rece

71 Self-inactivating vectors devoid of viral long-terminal-repeat enhancers have proven safe; however

72 ession dominance, temporally associated with long-terminal-repeat expansion in the A subgenome that a

73 and HDAC3 contribute to repression of HIV-1 long terminal repeat expression in the HeLa P4/R5 cell l

74 ongation rate included the density of exons, long terminal repeats, GC content of the gene, and DNA m

75 ng lentiviral vectors with self-inactivating long terminal repeats, have been shown to have improved

76 fied Tat was unable to transactivate the HIV long terminal repeat in U937 human macrophages.

77 heat shock factor binding HREs within their long terminal repeats in seven Brassicaceae species.

78 Insertions of long terminal repeats in the past 5 million years are re

79 (Lamc2(jeb)) due to a murine leukemia virus long terminal repeat insertion in Lamc2 (laminin gamma2

80 human immunodeficiency virus type 1 (HIV-1) long terminal repeat is present on both ends of the inte

81 es show that this retrotransposon with LTRs (Long Terminal Repeats) is widely distributed among the R

82 indicated that sequences at the envelope-3' long terminal repeat junction are required for proper ex

83 A 476-bp fragment that spans the envelope-long terminal repeat junction had activity equivalent to

84 Using the analysis of two long terminal repeat junctions in HIV-infected cells, we

85 on similar to a repeat associated with a non-long terminal repeat-like element and is often found acc

86 ncomitant inhibition of NF-kappaB and HTLV-1 long terminal repeat (LTR) activation.

87 actors have been reported to stimulate HIV-1 long terminal repeat (LTR) activity.

88 : MGEScan-long terminal repeat (LTR) and MGEScan-non-LTR are succe

89 ergent, the two orthologs similarly restrict long terminal repeat (LTR) and non-LTR retrotransposons

90 two NF-kappaB sites in the U3 region of the long terminal repeat (LTR) are critical for Cav-1-mediat

91 in (C/EBP) beta and C/EBP sites in the HIV-1 long terminal repeat (LTR) are crucial for HIV-1 replica

92 mulate RNA polymerase II (RNAP II) on the 5' long terminal repeat (LTR) but not on the 3' LTR.

93 f histone proteins at the HIV type 1 (HIV-1) long terminal repeat (LTR) by histone deactylases (HDACs

94 e ART were assayed for total HIV-1 DNA and 2-long terminal repeat (LTR) circles by quantitative polym

95 l-signaling pathways and to characterize the long terminal repeat (LTR) cis-acting elements involved

96 en domestic cat cell susceptibility and FeLV long terminal repeat (LTR) copy number, similar to obser

97 Domestic cat enFeLV env and long terminal repeat (LTR) copy numbers were determined

98 , we found that repressive histone marks and long terminal repeat (LTR) DNA methylation could be dete

99 eve targeted insertion of a gamma-retroviral long terminal repeat (LTR) driving a GFP expression cass

100 ing pattern of RelA recruitment to the HIV-1 long terminal repeat (LTR) during continuous tumor necro

101 erged to the Repbase collection of known ERV/long terminal repeat (LTR) elements to annotate the retr

102 ramer or octamer complex with the viral cDNA long terminal repeat (LTR) ends termed an intasome.

103 myc reporter plasmid indicated that the TBLV long terminal repeat (LTR) enhancer is necessary for T-c

104 r, the transcriptional activity of the viral long terminal repeat (LTR) from Vpr-deficient proviruses

105 cts but 11.7% with intact genomes and normal long terminal repeat (LTR) function.

106 status of the elongation complex on the HIV long terminal repeat (LTR) in a repressed state is not k

107 enetic reprogramming effect of ZL0580 on HIV long terminal repeat (LTR) in microglia.

108 strong transcriptional repressor of the HIV long terminal repeat (LTR) in resting alveolar macrophag

109 on of wild-type WRN transactivates the HIV-1 long terminal repeat (LTR) in the absence of Tat, and WR

110 Here, we show that methylation of the HIV 5' long terminal repeat (LTR) in the latent viral reservoir

111 indings suggest that integration of the XMRV long terminal repeat (LTR) into host DNA could impart an

112 ested, but rather transcription of the HIV-1 long terminal repeat (LTR) is increased in IL-4-producin

113 ndel formation at several targets within the long terminal repeat (LTR) of HIV.

114 so identified a transcript that contains the long terminal repeat (LTR) of lambda-olt 2-1 and shows a

115 rther demonstrate that HspBP1 inhibits HIV-1 long terminal repeat (LTR) promoter activity.

116 HDAC1 are coordinately resident at the HIV-1 long terminal repeat (LTR) promoter and absent from the

117 In addition, we detected Tip110 at the HIV-1 long terminal repeat (LTR) promoter and found that Tip11

118 -1 Tat-mediated transactivation of the viral long terminal repeat (LTR) promoter is essential for HIV

119 lencing of viral transcription driven by the long terminal repeat (LTR) promoter of HIV-1.

120 of the G-quadruplex structures in the HIV-1 long terminal repeat (LTR) promoter suppresses viral tra

121 ence of NucDHS in the proximal region of the long terminal repeat (LTR) promoter was associated with

122 iven by the mouse mammary tumor virus (MMTV) long terminal repeat (LTR) promoter were morphologically

123 t in this cell line was specific for the MLV long terminal repeat (LTR) promoter, as normal levels of

124 complex and recruit P-TEFb to the HIV type 1 long terminal repeat (LTR) promoter.

125 uces expression of the HIV-1 promoter in the long terminal repeat (LTR) region in a Tat-independent m

126 olled by the transcriptional activity of its long terminal repeat (LTR) region.

127 NA encoding the viral proteins is flanked by long terminal repeat (LTR) regions from the retrovirus.

128 module for structural discovery of complete long terminal repeat (LTR) retroelements, which are wide

129 Es), long interspersed elements (LINEs), and long terminal repeat (LTR) retroelements, which include

130 long interspersed nuclear element (LINE) and long terminal repeat (LTR) retroposons.

131 Interestingly, we find that a long terminal repeat (LTR) retrotransposon insertion ups

132 The long terminal repeat (LTR) retrotransposon Tf1 of Schizo

133 nvestigated the phylogenetic distribution of long terminal repeat (LTR) retrotransposon, long intersp

134 silencing of all 13 intact copies of the Tf2 long terminal repeat (LTR) retrotransposon.

135 esents known repeats including a majority of long terminal repeat (LTR) retrotransposons (13.67%).

136 rough recombination with specific classes of long terminal repeat (LTR) retrotransposons and organize

137 rated from these isoforms appeared to target long terminal repeat (LTR) retrotransposons and other un

138 Long terminal repeat (LTR) retrotransposons are an abund

139 Long terminal repeat (LTR) retrotransposons are closely

140 Endogenous retroviruses and long terminal repeat (LTR) retrotransposons are mobile g

141 Retroviruses evolved from long terminal repeat (LTR) retrotransposons by acquisiti

142 CENP-B homologues have been shown to silence long terminal repeat (LTR) retrotransposons by recruitin

143 Long terminal repeat (LTR) retrotransposons constitute a

144 st and mouse intracisternal A particle (IAP) long terminal repeat (LTR) retrotransposons in cultivate

145 ed from either insertions of low-copy number long terminal repeat (LTR) retrotransposons or deletions

146 a nearly twofold increase in the deletion of long terminal repeat (LTR) retrotransposons via solo LTR

147 verted-repeat transposable elements (MITEs), long terminal repeat (LTR) retrotransposons, and non-LTR

148 endogenous retroviruses (ERVs), also called long terminal repeat (LTR) retrotransposons, begins with

149 aluate the performance of methods annotating long terminal repeat (LTR) retrotransposons, terminal in

150 Long terminal repeat (LTR) retrotransposons, the most ab

151 genome surveillance role by controlling Tf2 long terminal repeat (LTR) retrotransposons.

152 81% transposable elements, 77% of which are long terminal repeat (LTR) retrotransposons.

153 abundant elements in eukaryotes are the non long terminal repeat (LTR) retrotransposons.

154 ces within the Ty3/gypsy-like superfamily of long terminal repeat (LTR) retrotransposons.

155 ic transcripts that are initiated within ERV long terminal repeat (LTR) sequences and read-through in

156 We detail here the contribution of different long terminal repeat (LTR) sequences for the establishme

157 promoter phenotypically resembles endogenous long terminal repeat (LTR) sequences, pointing to a sele

158 (HIV-1) integrase (IN) with DNA representing long terminal repeat (LTR) termini was previously assemb

159 a 36-bp insulator located in the foamy virus long terminal repeat (LTR) that has high-affinity bindin

160 t Tax activates transcription from the viral long terminal repeat (LTR) through recruitment of cellul

161 genes; (ii) the 5' end extending from the 5' long terminal repeat (LTR) to the beginning of the capsi

162 er junctions in the viral genome from the 5' long terminal repeat (LTR) to the end of pol.

163 In these species the CMTs silence long terminal repeat (LTR) transposons in the distal chr

164 s mapped to the NF-kappaB sites in the HIV-1 long terminal repeat (LTR) U3 and could be transferred t

165 The transcriptional activity of the XMRV long terminal repeat (LTR) was found to be higher than t

166 BRG1 associates with Tax at the HTLV-1 long terminal repeat (LTR), and coexpression of BRG1 and

167 Elk-1 activated transcription of the HTLV-1 long terminal repeat (LTR), and mutations within either

168 alyses of SIV polymerase (pol), STLV tax and long terminal repeat (LTR), and SFV pol and LTR sequence

169 HIV-1 RNA by recruiting P-TEFb to the HIV-1 long terminal repeat (LTR), and we show that inhibition

170 following the reduction in H3K27 at the HIV long terminal repeat (LTR), subsequent exposure to the H

171 he intrinsic toggling of HIV's promoter, the long terminal repeat (LTR), to generate bimodal ON-OFF e

172 mobility group protein A1 (HMGA1) and viral long terminal repeat (LTR), which led to higher levels o

173 an intrinsic NF-kappaB activator, increased long terminal repeat (LTR)-dependent XMRV transcription.

174 We show that long terminal repeat (LTR)-derived transcripts contribut

175 We found two instances of long terminal repeat (LTR)-driven provirus transcription

176 D), increases HIV infection by enhancing HIV long terminal repeat (LTR)-driven transcription via the

177 ocytomas transiently transfected with an HIV long terminal repeat (LTR)-luciferase reporter that cont

178 ional elongation complex essential for HIV-1 long terminal repeat (LTR)-mediated and general cellular

179 In the mouse, long terminal repeat (LTR)-retrotransposons, or endogeno

180 ericentromeric sequence, uncovered 45 intact long terminal repeat (LTR)-retrotransposons.

181 by activating the HIV-1 promoter, termed the long terminal repeat (LTR).

182 DNA sequences in the U3 region of the viral long terminal repeat (LTR).

183 -A/61E, in the surface glycoprotein (SU) and long terminal repeat (LTR).

184 the transcription initiation site on the HIV long terminal repeat (LTR).

185 methylation of histones located at the viral long terminal repeat (LTR).

186 (H3K4me2) that colocalizes with a retroviral long terminal repeat (LTR).

187 e element (HRE) localized in the proviral 5' long terminal repeat (LTR).

188 ts, as well as the coactivator CBP, with the long terminal repeat (LTR).

189 P/HEXIM1 complex for activation of the viral long terminal repeat (LTR).

190 n, FoxP3 enhances gene expression from HIV-1 long terminal repeat (LTR).

191 integrase and bases at the end of the viral long terminal repeat (LTR).

192 in the CD28-responsive element of the HIV-1 long terminal repeat (LTR).

193 ements (TE), including endogenous retroviral long terminal repeats (LTR), short and long interspersed

194 rspersed nuclear elements (LINE), but not in long terminal repeats (LTR).

195 ously annotated transcripts overlapping with long-terminal repeat (LTR) elements, several thousand of

196 ls resulted in decreased Tat-dependent HIV-1 long-terminal repeat (LTR) promoter transactivation as w

197 BEL/Pao-like long-terminal repeat (LTR) retrotransposons were annotat

198 ions to provide full-length repeats and, for long-terminal repeat (LTR) retrotransposons, calculates

199 tic mobilization of specific low-copy number long-terminal repeat (LTR) retrotransposons, which diffe

200 n of a variety of retroelements, such as the long-terminal repeat (LTR)-containing MusD and Ty1 eleme

201 'Copy-and-paste' long-terminal-repeat (LTR) retrotransposons have been pa

202 d effects on nuclear histone acetylation and long-terminal-repeat (LTR) transcription.

203 nct from their well-characterized effects of long-terminal-repeat (LTR)-driven gene expression.

204 aqMan HIV-1 test v2.0 (targeting gag and the long terminal repeat [LTR]).

205 thylation of, the integrated viral promoter (long terminal repeat [LTR]).

206 We tag hPSCs by GFP, expressed by the long terminal repeat (LTR7) of HERVH endogenous retrovir

207 rs (gammaRV/LV) with self-inactivating (SIN) long terminal repeats (LTRs) and internal moderate cellu

208 CENP-Bs also repress solo long terminal repeats (LTRs) and LTR-associated genes.

209 g endogenous retroviruses and their solitary long terminal repeats (LTRs) compose >40% of the human g

210 irus contains identical 5' and 3' peripheral long terminal repeats (LTRs) containing bidirectional pr

211 from intrachromosomal recombination between long terminal repeats (LTRs) flanking GAP1.

212 Retrotransposons often carry long terminal repeats (LTRs) for retrovirus-like reverse

213 Retrotransposons containing long terminal repeats (LTRs) form a substantial fraction

214 atin insulators, and self-inactivating (SIN) long terminal repeats (LTRs) may have significantly redu

215 omologous recombination between the flanking long terminal repeats (LTRs) of a full-length element, l

216 er Oryza genomes, and based on the dating of long terminal repeats (LTRs) of FRetro3 it was amplified

217 rates of nucleotide substitution between two long terminal repeats (LTRs) of individual orthologous L

218 the strong promoter/enhancer elements in the long terminal repeats (LTRs) of murine leukemia virus (M

219 astrocyte harboring active versus restricted long terminal repeats (LTRs) revealed that the gene expr

220 ing enabled the accurate prediction of 20.5% long terminal repeats (LTRs) that doubled the previous e

221 d enrichment in transposable elements (TEs): long terminal repeats (LTRs) were randomly located acros

222 clustering of telomeres and retrotransposon long terminal repeats (LTRs)) were observed throughout t

223 genous retroviral elements (ERVs) containing long terminal repeats (LTRs), are silenced through trime

224 ) contains identical DNA sequences, known as long terminal repeats (LTRs), at its 5' and 3' ends.

225 localized to endogenous retrovirus-K (ERVK) long terminal repeats (LTRs), which act as imprinted pro

226 YC; this is flanked by two loxP sites in its long terminal repeats (LTRs).

227 young transposable elements (TEs), including Long-Terminal-Repeats (LTRs) and SINE-VNTR-Alus (SVAs),

228 hat Tax-mediated activation of luciferase in long terminal repeat-luciferase (LTR-LUC) mice serves as

229 red with transgenic NF-kappaB reporters (HIV-long terminal repeat/luciferase [HLL]), we found exagger

230 trol of the mouse mammary tumor virus (MMTV) long-terminal repeat (MMT mice).

231 promoter, and the mouse mammary tumor virus long terminal repeat (MMTV-LTR).

232 The non-long terminal repeat (non-LTR) retrotransposon R2 is ins

233 Non-long terminal repeat (non-LTR) retrotransposons are a cl

234 Non-long terminal repeat (non-LTR) retrotransposons are high

235 Many non-long terminal repeat (non-LTR) retrotransposons lack int

236 R2 elements are non-long terminal repeat (non-LTR) retrotransposons with a s

237 ill have the hallmarks that characterize non-long terminal repeat (non-LTR) retrotransposons; they ha

238 he retrotransposition activity of the L1 non-long-terminal-repeat (non-LTR) retrotransposon in both H

239 irus promoter is not diminished, whereas the long terminal repeat of a retrovirus, like the ICP0 prom

240 The long terminal repeats of lymphomagenic P-MLVs are differ

241 as we found TINATs to be encoded in solitary long terminal repeats of the ERV9/LTR12 family, which ar

242 the control of the mouse mammary tumor virus long terminal repeat promoter, develop multifocal hyperp

243 RP1 protein enhances Tat-mediated HIV-1 LTR (long terminal repeat) promoter activity.

244 the binding of C/EBPbeta and p65 to the SIV long terminal repeat region in colonic lamina propria ce

245 eotide-long, noncoding RNA segment in the 5' long terminal repeat region of viral transcripts.

246 y by associating with proviral DNA at the 5' long terminal repeat region, recruiting KAP1 and HP1, an

247 F/EYFP mice, which are transgenic for both a long terminal repeat-regulated full-length infectious HI

248 trate that this model can be useful to study long terminal repeat regulation, as previously character

249 families of long interspersed element 1 and long terminal repeat retroelements, which are disparatel

250 ften be explained by the presence of similar long terminal repeat retroelements, which were enriched

251 Large long terminal repeat retrotransposon clusters occupy sig

252 domolecules, including coverage of the major Long Terminal Repeat retrotransposon families.

253 host-silencing pathways, particularly copia long terminal repeat retrotransposon in Drosophila melan

254 Previous studies of Tf1, a long terminal repeat retrotransposon in Schizosaccharomy

255 Ty1, a long terminal repeat retrotransposon of Saccharomyces, i

256 on of the GP(Y/F) domain in the IN of Tf1, a long terminal repeat retrotransposon of Schizosaccharomy

257 obase gene duplication event mediated by the long terminal repeat retrotransposon Rider.

258 This reduced capacity for long terminal repeat retrotransposon silencing and remov

259 Like its retroviral relatives, the long terminal repeat retrotransposon Ty1 in the yeast Sa

260 The Saccharomyces cerevisiae long terminal repeat retrotransposon Ty3 assembles its G

261 The Saccharomyces cerevisiae long terminal repeat retrotransposon Ty3 integrates with

262 In the Saccharomyces cerevisiae genome, the long terminal repeat retrotransposon Ty3 is found at RNA

263 ement is not a DNA transposon but instead is long terminal repeat retrotransposon-like with human end

264 Tf1, a long-terminal repeat retrotransposon in Schizosaccharomy

265 hese findings echo an earlier study with the long-terminal-repeat retrotransposon of Saccharomyces ce

266 We initially investigated 510 long terminal repeat-retrotransposon (LTR-RT) families c

267 Analyzing the long terminal repeat-retrotransposon (LTR-RT) type of TE

268 Long terminal repeat retrotransposons (LTR-RTs) are prev

269 of transposable elements (TEs), particularly long terminal repeat retrotransposons (LTR-RTs), in reco

270 d primarily by the periodic amplification of long terminal repeat retrotransposons (LTR-RTs).

271 mologous end joining, mediates clustering of long terminal repeat retrotransposons at centromeres in

272 tion and evolutionary and genomic studies of long terminal repeat retrotransposons in other genomes.

273 Integrases (INs) of retroviruses and long terminal repeat retrotransposons possess a C-termin

274 iniature (TRIMs) are a unique group of small long terminal repeat retrotransposons that are difficult

275 ensis genome due to a rapid amplification of long terminal repeat retrotransposons that occurred 38 m

276 s in T. halophila were found to contain five Long Terminal Repeat retrotransposons, MuDR DNA transpos

277 These repeat clusters are almost exclusively long terminal repeat retrotransposons, of which the pale

278 Unlike other long terminal repeat retrotransposons, TRIMs are enriche

279 ement activity predominately driven by Gypsy long terminal repeat retrotransposons, which extended th

280 ersity of the replication strategies used by long terminal repeat retrotransposons.

281 ely derived from internal deletions of large long terminal repeat retrotransposons.

282 Finally, analysis of KERV long terminal repeat sequences using massively parallel

283 o the consensus sequence in both protein and long terminal repeat sequences.

284 endogenous retroviruses [ERVs] and 488 solo long terminal repeats [sLTRs]) within the C57BL/6J mouse

285 l replication-competent avian leukosis virus long terminal repeat, splice acceptor (RCAS)/TVA system

286 of uridine-rich ssRNA derived from the HIV-1 long terminal repeat (ssRNA40) on activation of NK cells

287 ete proviruses and proviruses devoid of a 5' long terminal repeat, suggesting that the expression of

288 Here we tested a set of long terminal repeat TE families for roles as enhancers

289 t least five nucleosomes, is found at the 5' long terminal repeat, the location and modification stat

290 ween the 3' end of the coding region and the long terminal repeat, this retrotransposon family contai

291 s) that repressed NEERV by binding the NEERV long terminal repeat to recruit the transcriptional regu

292 r contains additional NF-kappaB sites in the long terminal repeats to enhance viral replicative capac

293 otide (nt) pri-miRNA, encoded within the BFV long terminal repeat U3 region, that is subsequently cle

294 of transcription factors to the HIV provirus long terminal repeat using chromatin immunoprecipitation

295 of functional annotations (e.g. centromeres, long terminal repeats) using 3D genome reconstructions f

296 transcription; M. minutoides cells produce 2-long-terminal-repeat viral DNA circles and linear viral

297 sponsive MMTV-LTR (mouse mammary tumor virus-long terminal repeat), we show that BRG1 and BRM are rec

298 rtions of the viral envelope gene and the 3' long terminal repeat were tested in the presence and abs

299 ial cells and the replication-competent ASLV long terminal repeat with a splice acceptor/tv-a glioma

300 Given the shift toward self-inactivating long terminal repeats with weaker promoters to control t