ライフサイエンスコーパス: long-term latency

1 t sustained US28 expression is necessary for long-term latency.

2 cess to memory B cells, a major reservoir of long-term latency.

3 IL-10 is associated with decreased gammaHV68 long-term latency.

4 is possible to effectively vaccinate against long-term latency.

5 in germinal center and memory B cells during long-term latency.

6 after infection but was undetectable during long-term latency.

7 does not prevent gammaHV68 from establishing long-term latency.

8 ng early latency and immature B cells during long-term latency.

9 ed in a variety of cell populations but that long-term latency (6 months postinfection) in the spleen

10 According to recent research, 70-90% of long-term latency and chronic human disease incidence is

11 suppression of cytokine storm: both aimed at long-term latency and host-pathogen stand-off, suggestin

12 activation of ERK-MAPK signaling impacts on long-term latency and reactivation in hematopoietic cell

13 response against gammaherpesviruses to alter long-term latency and suggest that limiting long-term la

14 ual evoked potential (mfVEP) technique after long-term latency changes in optic neuritis (ON)/multipl

15 , our work shows how tumor recurrences after long-term latency evolve toward T-cell resistance by ind

16 tion and short-term latency but cannot alter long-term latency further call into question the possibi

17 s during the acute infection and establishes long-term latency in B cells and lung epithelial cells.

18 Normal long-term latency in lymphoid tissue was established non

19 o PCs after infection by EBV, thus favouring long-term latency in MBC and asymptomatic persistence.

20 , the germinal center response, to establish long-term latency in memory B cells.

21 nipulate B cell differentiation to establish long-term latency in memory B cells.

22 is established in a variety of cell subsets, long-term latency in the lung is only maintained in B ce

23 Papillomaviruses establish a long-term latency in vivo by maintaining their genomes a

24 Finally, we demonstrate that long-term latency is accompanied by a low level of infec

25 Our data show that long-term latency is maintained in a variety of anatomic

26 ablished in germinal center B cells and that long-term latency is preferentially maintained in two di

27 g the EBNA-1 expression necessary to sustain long-term latency is presented.

28 characterized by proliferative indolence and long-term latency relapses.

29 wnregulation of viral gene expression in the long-term latency reservoir is likely to facilitate evas

30 ce proteins is turned off in the predominant long-term latency reservoir of resting memory B cells, s

31 BV infection, including the establishment of long-term latency within B lymphocytes, and is therefore