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1 milar context until the link crystallizes in long term memory.
2 0 in the MB of adult flies causes defects in long-term memory.
3 , synaptic markers and volume, and protected long-term memory.
4  into late LTP and of short-term memory into long-term memory.
5 memory and in spatial reference learning and long-term memory.
6 some, but not all, learning experiences into long-term memory.
7 ter, which we suspected of being involved in long-term memory.
8  schema congruence has a strong influence on long-term memory.
9 ck into working memory during retrieval from long-term memory.
10 esponsive genes, including those involved in long-term memory.
11 ) it is involved in spontaneous retrieval of long-term memory.
12 ich are the cellular analogs of two forms of long-term memory.
13 c plasticity, a cellular mechanism mediating long-term memory.
14 protein synthesis at activated synapses, and long-term memory.
15 nding and intron removal selectively impairs long-term memory.
16 plasticity contributes to the maintenance of long-term memory.
17 consolidating the learned association into a long-term memory.
18 that forms functional amyloids necessary for long-term memory.
19 ole in binding temporally adjacent events in long-term memory.
20 res synapse number, synaptic plasticity, and long-term memory.
21  differed markedly on subsequent measures of long-term memory.
22 wn to be a key component in the formation of long-term memory.
23  JJJ2 enhances the animal's capacity to form long-term memory.
24 vation mediated IEG expression implicated in long-term memory.
25 er, this is at the expense of forming stable long-term memory.
26 N-terminal targeting domain, does not affect long-term memory.
27 genously boost memory consolidation and thus long-term memory.
28 own to be important for Orb2 aggregation and long-term memory.
29 to be crucial for converting experience into long-term memory.
30 KC isoform proposed to maintain late-LTP and long-term memory.
31 involved in the formation and maintenance of long-term memory.
32 crucial for expression and likely storage of long-term memory.
33 imental to the maintenance of information in long-term memory.
34 identified as relating to the maintenance of long-term memory.
35 estored spine density prevents the rescue of long-term memory.
36 engram cells restores both spine density and long-term memory.
37 resulting in chronic deficits in working and long-term memory.
38 g and short-term memory while also impairing long-term memory.
39 s is thought to be essential for maintaining long-term memory.
40 between physical activity during the day and long-term memory.
41 f, generally ineffective training to produce long-term memory.
42  neprilysins are involved in middle-term and long-term memory.
43 tic regulation of gene expression underlying long-term memory.
44 eported to be involved in the maintenance of long-term memory.
45 and unstable memory transforms into a stable long-term memory.
46 yonic axis specification to the formation of long-term memory.
47 rophysiological signal hypothesized to index long-term memory.
48  T cell response, promoting the formation of long-term memory.
49 nship between perception, working memory and long-term memory.
50 eurons in the lateral amygdala (LA) impaired long-term memory.
51 in synthesis rates, impedes the formation of long-term memory.
52 ierarchical organization of information into long-term memory.
53 ssociated with dopamine, game enjoyment, and long-term memory.
54 ent, thus predictable from schemas stored in long-term memory.
55 is a key player in the epigenetic control of long-term memory.
56 2 and overlapping roles for AKT1 and AKT3 in long-term memory.
57 mplex dynamical networks (CDNs) that exhibit long-term memory.
58 ecular processes for synaptic plasticity and long-term memory.
59 , bolstered synaptic plasticity and enhanced long-term memory.
60 vent to promote its successful encoding into long-term memory.
61 REB target genes involved in the encoding of long-term memory.
62 ibutes both to the encoding and retrieval of long-term memories.
63 uates the formation of hippocampus-dependent long-term memories.
64 or, consistent with impaired ability to form long-term memories.
65 ng the synaptic changes required for storing long-term memories.
66 experience is necessary for the formation of long-term memories.
67 architecture that supports it and shapes our long-term memories.
68 lateral amygdala during the consolidation of long-term memories.
69 expression are required for the formation of long-term memories.
70  HPC is not obligatory for these features of long-term memories.
71 t with trends towards reduced short-term and long-term memories.
72 ritical for the formation and maintenance of long-term memories.
73                        How do neurons encode long-term memories?
74 ecular switch that promotes the formation of long-term memory(1-4).
75 r LTP generation, which may be implicated in long-term memory acquisition and in its deterioration in
76 gle-strand DNA damage, was also required for long-term memory acquisition in a variety of learning pa
77 involvement in the encoding of experience to long-term memory across time and biological levels of or
78 An enzyme called PKM zeta may have a role in long-term memory after all.
79  areas and mark those synapses for forming a long-term memory after the prion form is established.
80 on of adaptive immune responses endowed with long-term memory against mycobacterial antigens.
81  critical for understanding the evolution of long-term memories and for enabling the development of n
82 s) are important for neuronal functions like long-term memory and are well-characterized in mammals b
83 election for complex dynamical networks with long-term memory and by leveraging the structure of the
84 ural and functional plasticity, formation of long-term memory and cognitive function.
85 rocess during the formation and retrieval of long-term memory and define general memory-implicated ge
86 priately timed physical exercise can improve long-term memory and highlight the potential of exercise
87 e abstract representations consolidated into long-term memory and how are they integrated with pre-ex
88 rucial for translational control involved in long-term memory and in late long-term potentiation (LTP
89 fore, in this paper, we study the effects of long-term memory and of the topological properties on th
90 igosaccharide analogs of ST-5 CPS RU induced long-term memory and protective immune responses in rabb
91 mation is buffered when being retrieved from long-term memory and reconcile current theories of memor
92 odic memories are gradually assimilated into long-term memory and this process is strongly influenced
93  lesions may selectively affect orthographic long-term memory and working memory processes, relativel
94 ritical illness in early life are at risk of long-term-memory and attention impairments.
95 s a major role in encoding and consolidating long-term memories, and undergoes plastic changes during
96 es in working memory and target templates in long-term memory, and consider how searches are terminat
97 roles for dTau in regulation of translation, long-term memory, and footshock habituation are also rev
98 sed seizures, rescued ASD-like behaviors and long-term memory, and normalized metabolic changes in th
99 reduced brain inflammation, restored LTP and long-term memory, and reversed social and communication
100                                              Long-term memories are believed to be stored in the syna
101  PNNs may be an important substrate by which long-term memories are stored in the central nervous sys
102        Binding information in short-term and long-term memory are functions sensitive to Alzheimer's
103 and consolidation were prevented by blocking long-term memory arising from the new experiences.
104 bout the word's letters and their order from long-term memory as well as the maintenance and processi
105 ontaining neurons are necessary to establish long-term memories associating predictive stimuli with r
106  increase the precision of hippocampal-based long-term memory associations by assessing the salience
107      Stroked animals developed impairment in long-term memory at 4-weeks post-stroke despite recovery
108 f antagonists, and coexistence of short- and long-term memory at loci with weak and strong constituti
109    Here, assuming that CRISPR functions as a long-term memory-based defense against a diverse landsca
110                                Some forms of long-term memories become labile and can be reversed wit
111                                          The long-term memory binding task was a Paired Associates Le
112  quantify changes in the brain mechanisms of long-term memory (BM-LTM).
113 ptic remodeling associated with formation of long-term memories, but the mechanisms that regulate thi
114  binding protein Orb2 has been implicated in long-term memory, but how conformational conversion of O
115  to associate different stimuli is vital for long-term memory, but how neural ensembles encode associ
116                           Prior knowledge in long-term memory can aid in efficient encoding of inform
117                   On retrieval, consolidated long-term memory can be neutralized by extinction if the
118 ll generation or in postmitotic cells, while long-term memory can survive multiple rounds of cell div
119  rapidly forgets, or is not yet able to form long-term memories, can exert such a long-lasting and im
120 ither their short-term effector functions or long-term memory capacity.
121 mpal synaptic plasticity, and impairments in long-term memory caused by sleep deprivation.
122 ntiation of memory precursor WP T cells into long-term memory cells.
123 m for the discovery of orthologous mammalian long-term memory components.
124  before fear learning significantly impaired long term memory consolidation, whereas short-term memor
125 evidence in rodents and humans suggests that long-term memory consolidation can be enhanced by the ex
126 REM) sleep is supposed to play a key role in long-term memory consolidation transferring information
127 ) did not affect working memory but impaired long-term memory consolidation.
128  help guide the prioritization of events for long-term memory consolidation.
129 uring sleep and seizures specifically impair long-term memory consolidation.
130 hippocampal spikes during sleep would impair long-term memory consolidation.
131  that the hippocampus, known for its role in long-term memory, contributes to these flexible aspects
132 loss results in late-onset spatial reference long-term memory deficit.
133  working memory processes, with orthographic long-term memory deficits centred in either the left pos
134 sion and rescued the synaptic plasticity and long-term memory deficits in DS mice.
135                Finally, synaptic tagging and long-term memory deficits in mice lacking translin/trax
136 erexpression in aged animals ameliorated the long-term memory deficits observed in control animals.
137 place cell representations could explain the long-term memory deficits observed in previous behaviora
138                             Consolidation of long-term memories depends on de novo protein synthesis.
139                                   Persistent long-term memory depends on successful stabilization and
140 TEMENT Awake experience is consolidated into long-term memories during sleep.
141 o internal representations, whether semantic long-term memory (e.g., letters, digits, words), sensory
142          In this work, we leverage the short/long term memory effects due to the electron trapping ev
143 gy switch in the mushroom body that controls long-term memory encoding.
144 re of well-organized relevant information in long-term memory, enhanced attitude extremity and access
145 g-term memory formation and are required for long-term memory enhancement by a class of broad-acting
146 pamine modulation and reward on two forms of long-term memory: episodic memory for neutral objects an
147 n the unfolding therapeutic process generate long-term memories for reconsolidated emotional material
148 mid and phage targets and a second providing long-term memory for access to chromosomal sites upon en
149 lus, juv ELE and juv-adol ELE formed lasting long-term memory for an object location memory task, whe
150 used a novel auditory paradigm to assess how long-term memory for auditory scenes facilitates detecti
151   In addition, we found selectively enhanced long-term memory for contextual but not cued fear memory
152 has long been known that schemas can enhance long-term memory for related information.
153  and ends by categorizing the sound based on long-term memory for word meaning.
154         Nr4a nuclear receptors contribute to long-term memory formation and are required for long-ter
155 es histone deacetylase 4, and is involved in long-term memory formation and behavior.
156 tate from astrocytes, which is necessary for long-term memory formation and for underlying molecular
157  of its major ligands, IGF2, is critical for long-term memory formation and strengthening.
158 the main function of dentate neurogenesis is long-term memory formation because we assumed that a new
159 he integrated stress response (ISR), impairs long-term memory formation in rodents and birds(11-13).
160 ngle isoform of creb1/crh-1 shows defects in long-term memory formation in the animal and expression
161 le their sucrose intake at an early stage of long-term memory formation initiated the investigation o
162 tically contribute to hippocampus-dependent, long-term memory formation using a combination of transc
163 ement-binding protein, a crucial mediator in long-term memory formation, correlated anatomically and
164 a suggest key roles for Stk11 and Fos in CTA long-term memory formation, dependent at least partly th
165 ranslation-dependent processes necessary for long-term memory formation, entrained by dopamine in CA1
166 extracted Orb2 fibrils, that are involved in long-term memory formation, from Drosophila brains, char
167 tus contain loci annotated with functions in long-term memory formation, mushroom body development, a
168 sting, providing an ideal model for studying long-term memory formation, storage, and retrieval.
169  To evaluate what determines the efficacy of long-term memory formation, we developed an extensive se
170 genetic suppressor of hippocampus-dependent, long-term memory formation.
171 state to control Orb2A protein abundance and long-term memory formation.
172 slation initiation is a central regulator of long-term memory formation.
173 ux is both necessary and sufficient to drive long-term memory formation.
174 ich directly promotes network plasticity and long-term memory formation.
175  amyloid that plays a key role in Drosophila long-term memory formation.
176 wn-regulation may be implicated in decreased long-term memory formation.
177  the interaction between prior knowledge and long-term memory formation.
178 cognitive deficits, including impairments in long-term memory formation.
179 table and having unique predictive value for long-term memory function, hippocampal volume and traini
180 val in a murine orthotopic PDAC model with a long-term memory immune response.
181 rogenesis participates in the development of long-term memory impairment.
182 arliest initiator of synaptic plasticity and long-term memory impairment.
183                               In particular, long-term memories in such paradigms have been extensive
184  show a severe impairment of both short- and long-term memory in a number of behavioral tasks.
185 tice is a strategy to optimize encoding into long-term memory in both young and elderly humans.
186 ere, we show that consolidation of courtship long-term memory in Drosophila is mediated by reactivati
187 n anti-amyloidogenic peptide interferes with long-term memory in Drosophila.
188              We developed tests that measure long-term memory in hamsters.
189 a with the ISR inhibitor ISRIB(11), enhances long-term memory in health and disease(18).
190  Decreasing the levels of caspase-2 restored long-term memory in mice that had existing deficits.
191 n of astrocytic Gs-coupled signaling reduced long-term memory in mice without affecting learning.
192                                              Long-term memory in the brain is thought to arise from a
193 2(-/-) mice had robust and sustained loss of long-term memory in two separate behavioral tasks, Morri
194 the formation of amyloid-like aggregates and long-term memory in vivo.
195 econd, PKMzeta is essential for late-LTP and long-term memory in wild-type mice, and PKMzeta-null mic
196 e in hippocampus blocks late-LTP and spatial long-term memory in wild-type mice, but not in PKMzeta-n
197 s2(-/-) mice displayed impaired formation of long-term memory, increased risk taking and stimulus see
198                      The observed merging of long-term memories is thus similar to the memory conjunc
199 been assumed that information retrieved from long-term memory is represented in working memory, we la
200  vivo, and identify the microRNA miR-21 as a long-term memory keeper of the fibrogenic program in MSC
201                                 We also show long-term memory-like cytokine responses upon subsequent
202 l subsets that have been suggested to play a long-term memory-like response to HCMV infection.
203  studies suggested that NK cells may display long-term, memory-like responses to murine cytomegalovir
204 on a comparable match-to-sample task with no long-term memory load, suggesting that the effect is not
205 changes using aversive olfactory associative long-term memory (LTAM) and identified three major gene
206 hodology to target the mechanisms supporting long term memory (LTM) access/retrieval in language and
207 ositive biological processes that facilitate long-term memories (LTM) but also the suppression of inh
208                        Both the formation of long-term memory (LTM) and dendritic spine growth that s
209 lly involved in middle-term memory (MTM) and long-term memory (LTM) and that their expression is requ
210 initially require the hippocampus for recent long-term memory (LTM) but then become increasingly inde
211 the general field of molecular processing in long-term memory (LTM) by describing a novel form of pre
212 uced performance in Morris Water Maze (MWM), long-term memory (LTM) contextual fear testing, and rota
213 le that activates mTORC2 (A-443654) reverses long-term memory (LTM) deficits in both aged mice and fl
214  the fact that PDE11A is required for social long-term memory (LTM) formation during adolescence and
215                                              Long-term memory (LTM) formation is a critical survival
216 1 expression does not affect contextual fear long-term memory (LTM) formation.
217                 Previously, we reported that long-term memory (LTM) in Aplysia can be reinstated by t
218 brain are required for middle-term (MTM) and long-term memory (LTM) in the dorsal paired medial (DPM)
219 l synaptic plasticity.SIGNIFICANCE STATEMENT Long-term memory (LTM) induced by repeated trials spaced
220 ; short-term memory (STM) is intact, whereas long-term memory (LTM) is significantly impaired.
221                                      Forming long-term memory (LTM) often requires repetitive experie
222                                     Creating long-term memory (LTM) requires new protein synthesis to
223                     The formation of spatial long-term memory (LTM) requires the de novo synthesis of
224                   Short-term memory (STM) or long-term memory (LTM) was evaluated in rutabaga (rut) a
225 transform a subthreshold learning event into long-term memory (LTM), and hSyn-HM4D completely impaire
226 nduce a more persistent memory identified as long-term memory (LTM).
227 ntenance of long-term potentiation (LTP) and long-term memory (LTM).
228 ryonic development, synaptic plasticity, and long-term memory (LTM).
229 ronal excitability (LTEE), two correlates of long-term memory (LTM).
230 soforms on the ability of C. elegans to form long-term memory (LTM).
231 ngs provide initial evidence to suggest that long-term memory may be enhanced by more carefully atten
232 ese findings demonstrate that people rely on long-term memory not only for remembering familiar items
233  with a cGMP-dependent switch from short- to long-term memory observed in vivo after intrahippocampal
234                                              Long-term memories of fear have been notoriously difficu
235 ster prefers D- over L- arabinose, but forms long-term memories of L-arabinose more reliably.
236 ced memory reactivation during sleep renders long-term memories of negative experiences more negative
237 tory sensory input with a social cue induces long-term memory of a food odor.
238 both synaptic plasticity and the encoding of long-term memory of a novel, complex environment.
239 mission of food preference (STFP), mice form long-term memory of food odors presented by a social par
240  unclear whether this inheritance can direct long-term memory of individual gene expression states (c
241 rrent light directionality, rather than by a long-term memory of previous conditions.
242  tend to reside in genomic clusters and that long-term memory of these genes is locally restricted by
243 be explained easily by hippocampal-dependent long-term memory or spatial cognition.
244 animals are awakened may produce maladaptive long-term memory, or may interrupt consolidation.
245 dren undergoing epilepsy surgery may enhance long-term memory outcome.
246 arily provides rich associated material from long-term memory, permitting rapid chunking.
247  in selective attention, working memory, and long-term memory persist in remission from a major depre
248 s PKMzeta is essential for wild-type LTP and long-term memory, persistent PKCiota/lambda activation c
249 dence that human hippocampal activity during long-term memory processing is sensitive to temporal dur
250  medial temporal lobe, which is critical for long-term memory processing.
251 fically enhances protein synthesis-dependent long-term memory (PSD-LTM), but not anesthesia-resistant
252 reflex, we compared acquisition learning and long-term memory recall of uninfected (control) honey be
253  synthesis is crucial for the maintenance of long-term memory-related synaptic plasticity.
254 ENT How new experiences are transformed into long-term memories remains a fundamental question for ne
255 lity and concentration-features relevant for long-term memory-remains unknown.
256 central role as the orthographic lexicon-the long-term memory representations of visual word forms.
257  into nested events, which form the basis of long-term memory representations.
258  both by their current environment and their long-term memory representations.
259 lies, formation and expression of artificial long-term memory require flies to be hungry.
260                    After effective training, long term memory required subsequent transcription and t
261          It is not clear whether the altered long-term memory resides within the T-cell or the B-cell
262 red to induce antibody isotype switching and long-term memory responses.
263 ent GR-phosphorylation (PO(4)) sites impairs long-term memory retention and maintenance of newly form
264 g the stopping of action and the stopping of long-term memory retrieval (stopping thoughts), where in
265     We found that male mice exhibit stronger long-term memory retrieval than do female mice, and this
266 in generating explanations (e.g., attention, long-term memory) shape the outputs of this process.
267 ereas long-term inhibition of GluT4 impaired long-term memory, short-term memory was enhanced.
268 ive memory stabilization was associated with long-term memory specificity.
269 ion mechanisms have been proposed to mediate long-term memory stabilization and transformation.
270 hought to be a critical mechanism supporting long-term memory stabilization.
271   The hippocampal complex (HC) is central to long-term memory storage and retrieval as well as spatia
272 icant insights into the molecular biology of long-term memory storage at the level of the synapse.
273 use its inhibition disrupts LTP in vitro and long-term memory storage in vivo.
274 trong neurobiological evidence suggests that long-term memory storage involves formation of new synap
275 rieved and must undergo reconsolidation into long-term memory storage to be maintained.
276 l node of the neocortical network supporting long-term memory storage) undergoes rapid modifications
277 on retrieval and must be reconsolidated into long-term memory stores to persist.
278 of GABAergic cells, is important to modulate long-term memory strength and precision.
279 ed to reflect the use of a separate episodic long-term memory system, rather than working memory.
280 t solely due to the use of separate episodic long-term memory systems.
281               We trained mice in an auditory long-term memory task and measured learning-related dyna
282 pite the fact that these mice are amnesic in long-term memory tests when natural recall cues are used
283 d stimulus was found in both short-term- and long-term-memory tests.
284 emories that last for seconds to minutes and long-term memories that last for hours or longer [1, 2].
285 rd a possible molecular mechanism underlying long-term memory that involves CPEB3's binding to actin,
286 ynaptic substrate for a socially conditioned long-term memory that operates at the level of the initi
287  promote the storage of new information into long-term memory through the activation of the SN/VTA-Hi
288 apid working memory during ongoing tasks and long-term memory to guide future action, respectively).
289 ding of the cellular architecture supporting long-term memory traces has also substantially improved.
290 -organized hierarchical peak structure has a long-term memory transmitted across several waves.
291  primates is especially complex and requires long-term memory, value comparison, strategic planning,
292 or medium (22 [29%]) in size, although three long-term memory variables showed large deficits: g=-0.8
293                                    Moreover, long-term memory was formed immediately after training r
294 found that the retrieval of information from long-term memory was limited to just a few simple object
295 h-frequency stimulation, both the short- and long-term memories were robustly improved in the novel-o
296  presents both classic diauxic behaviors and long-term memory, where the bacteria can pause for >11 h
297  enhances CA1 neuronal excitability, LTP and long-term memory whereas its overexpression weakens memo
298           In addition, when the data exhibit long-term memory (which is the case in most climate reco
299 tumor regression, endogenous T cells secured long-term memory with a broad repertoire of antigen spec
300 sticity and attenuates hippocampus-dependent long-term memories without affecting anxiety.

 
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