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1 bsorption in the thick ascending limb of the loop of Henle.
2 e collecting duct predominantly but also the loop of Henle.
3 rtical collecting ducts, distal tubules, and loop of Henle.
4 op diuretics can curtail their action in the loop of Henle.
5 orption in the proximal convoluted tubule or loop of Henle.
6 e to increased reabsorption in the PT and/or loop of Henle.
7 ases in sodium exit from the proximal tubule/loop of Henle.
8 l tubule and the thick ascending limb of the loop of Henle.
9 mental link between proximal tubules and the loop of Henle.
10 s primarily expressed in the thick ascending loop of Henle.
11 give rise to the thin descending limb of the loop of Henle.
12 to identify the thin descending limb of the loop of Henle.
13 ominantly in the thick ascending limb of the loop of Henle.
14 by cells of the thick ascending limb of the loop of Henle.
15 renal progenitor cells and formation of the loop of Henle.
16 in proximal tubules and descending limbs of loops of Henle.
17 expressed by the thick ascending limb of the loops of Henle.
18 essential for the coordinated growth of the loop of Henle, a medullary extension of the nephron that
20 n the kidney by thick ascending limbs of the loop of Henle and by distal convoluted tubules (DCTs).
22 model showed greater Umod expression in the loop of Henle and correspondingly less fibrosis.CONCLUSI
23 r unchanged or upregulated, and those in the loop of Henle and distal tubule lineages were downregula
25 cal Ang II, to stimulate sodium transport in loop of Henle and the distal nephron, and to induce hype
26 ffered by increased reabsorption in both the loop of Henle and the distal nephron, resulting in the o
27 potassium-chloride transporter in the kidney loop of Henle and the KCC2 potassium-chloride transporte
28 inates in the basement membranes of the thin loops of Henle and spreads from there through the inters
29 in urine, such as podocyte, proximal tubule, loop of Henle, and collecting duct, in addition to macro
30 ive cells of the thin descending limb of the loop of Henle, and principal cells of the collecting sys
32 transport in the thick ascending limb of the loop of Henle; and claudin-4, -7, and -8 as determinants
33 iously reabsorbed in the proximal tubule and loop of Henle; and, second, a stimulus to sodium-cation
34 the convoluted tubules, collecting ducts and loops of Henle as well as within the cytoplasm of tubule
36 ow a distinct distribution pattern along the loop of Henle, but the functional significance of this o
37 ocated in the kidney within the lumen of the loops of Henle, but no intracellular storage sites were
38 es targeting the thick ascending limb of the loop of Henle can cause either acquired Bartter syndrome
40 concentration was significantly elevated and loop of Henle Cl absorption was reduced in microperfused
41 a4-specific transcripts in proximal tubule, loop of Henle, distal convoluted tubule, and cortical an
42 strate a4 expression in the proximal tubule, loop of Henle, distal tubule, and collecting duct and su
43 renal lineages, such as the proximal tubule, loop of Henle, distal tubules, and podocytes, using extr
44 e HBD-1 mRNA in the epithelial layers of the loops of Henle, distal tubules, and the collecting ducts
47 the glomerulotubular balance response in the loop of Henle is accompanied by increased Na,K-ATPase ac
50 h is important in sodium reabsorption in the loop of Henle, is maintained or even increased in Foxa1-
52 ctions in proximal stop flow pressure during loop of Henle (LH) perfusion at 40 nl/min with artificia
54 tive only for Tamm-Horsfall protein (THP), a loop of Henle marker, while Tsc1 deficiency in Osx linea
55 n clinical medicine because they inhibit the loop of Henle Na-K-2Cl cotransporter with much higher af
56 t decrease in THP-, NKCC2- and AQP1-positive loop of Henle nephron segments in mutant DeltaSRM kidney
57 n, and activation of ion transporters in the loop of Henle (NKCC2) and distal nephron (NCC, ENaC, and
58 Cl and water absorption along microperfused loops of Henle of NKCC2A-/- mice were unchanged at norma
60 examined in anaesthetized rats by perfusing loops of Henle of superficial nephrons with solutions co
61 id not change significantly during prolonged loop of Henle perfusion in e-5'NT/CD73(+/+) mice, a comp
62 -flow pressure in response to an increase in loop of Henle perfusion rate from 0 to 30 nl/min was com
64 hibit more aging-related gene alterations in loops of Henle, proximal tubules and collecting ducts in
65 CHAC1 RNA and protein were expressed in the loop of Henle region in mouse and human kidneys, and CHA
66 r in vivo assessment of proximal tubular and loop of Henle sodium handling, to assess sodium exit aft
67 by cells of the thick ascending limb of the loop of HENLE: Subsequent aggregation of these proteins
69 the specific contribution of thick ascending loop of Henle (TALH) -derived HO-1, we generated a trans
70 m and volume delivery to the thick ascending loop of Henle (TALH) and macula densa, providing the err
71 bsorption in the thick ascending limb of the loop of Henle (TALH) in vivo was examined in anaesthetiz
73 of the proximal tubule (S1/S2 segment), the loop of Henle, the intercalated cells of the distal conv
74 previously reabsorbed in proximal tubule and loop of Henle) to the distal nephron in quantities equal
75 ial cells, endothelial cells, or proximal or loop of Henle tubular cells, while Osx is known to label
76 ubule and in the thick ascending limb of the loop of Henle, whereas it is transcellular in the distal
77 a and produced kidney cysts primarily in the loops of Henle, whereas inactivation in adult mice did n