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1 vity, phenotypes not observed with the eIF4G loss of function mutant.
2 activated by NaCl are absent from the Atann1 loss-of-function mutant.
3 tion mutant of PLN, whereas R14del is a mild loss-of-function mutant.
4  truncated rhBARF1 from clone 16 rhLCV was a loss-of-function mutant.
5 (BL)-insensitive phenotype similar to a bri1 loss-of-function mutant.
6  an avirulent bacterial pathogen in the bap1 loss-of-function mutant.
7 utant than to either wild-type I domain or a loss-of-function mutant.
8  to photoperiod in a manner similar to a cdf loss-of-function mutant.
9 l in vivo, as they complemented a yeast GLXI loss-of-function mutant.
10 ity phenotype was similar to that of the AS2 loss-of-function mutant.
11 he striking abnormalities of lin-4 and let-7 loss of function mutants.
12 naling, and this effect is blocked in usp-46 loss-of-function mutants.
13  show an increase in Lys acetylation in srt2 loss-of-function mutants.
14 e Dlx1/2 mutant phenotypes, we made compound loss-of-function mutants.
15 enient and effective approach for generating loss-of-function mutants.
16 altered in MPK6 overexpression lines or mpk6 loss-of-function mutants.
17 ctase were increased in rfd1 and the AUX/IAA loss-of-function mutants.
18 ly predicts root growth, as observed in bri1 loss-of-function mutants.
19  and root apical meristems observed in fbl17 loss-of-function mutants.
20 redundancy was confirmed through analysis of loss-of-function mutants.
21 ted nutations were also eliminated in etr1-7 loss-of-function mutants.
22 s a permissive temperature for wild-type and loss-of-function mutants.
23 ethality that often occurs in both gain- and loss-of-function mutants.
24  what E2F actually does based on analyses of loss-of-function mutants.
25 ina can often complicate the analysis of CBP loss-of-function mutants.
26 f and floral organ production in stm partial loss-of-function mutants.
27 erous breed-specific non-synonymous SNPs and loss-of-function mutants.
28 fect of ethanol, equivalent to that of slo-1 loss-of-function mutants.
29 d trunk mesoderm, and closely resemble nodal loss-of-function mutants.
30 e may be of interest to restore transport in loss-of-function mutants.
31 nted the observation of strong phenotypes in loss-of-function mutants.
32 ns, R205X and E239X, were shown to be Dyrk1a loss-of-function mutants.
33 th pathways was confirmed by the analysis of loss-of-function mutants.
34 creased cell migration beyond canonical PTEN loss-of-function mutants.
35 ark respiration was observed in the eIFiso4G loss of function mutant, a reduction in chlorophyll leve
36 as a promoter, may be mutagenized to isolate loss-of-function mutants able to survive under selection
37 ality of AtCDA in vivo was demonstrated with loss-of-function mutants accumulating high amounts of cy
38                       A previously described loss-of-function mutant affecting both isoforms, sr45-1,
39 Arabidopsis (Arabidopsis thaliana) quadruple loss-of-function mutant affecting p24 genes from the del
40 r show that removing MUL1 in PINK1 or parkin loss-of-function mutant aggravates phenotypes caused by
41 istant locomotory behavior, resembling slo-1 loss-of-function mutants, albeit to a lesser extent.
42                     We show that the partial loss-of-function mutant alix-1 displays reduced vacuolar
43 entifying EMB genes and characterizing their loss-of-function mutant alleles are needed to understand
44                                    Biallelic loss-of-function mutant alleles underlie several differe
45                         However, the NAD286G loss-of-function mutant also associated with the host fa
46                     Through a combination of loss-of-function mutant analyses, genetic mapping, and t
47                                            A loss-of-function mutant analysis also revealed that sing
48                             A combination of loss-of-function mutant analysis and protein interaction
49 of violaxanthin de-epoxidase in the eIFiso4G loss of function mutant and an increase in its xanthophy
50            Experiments using a C5a peptidase loss-of-function mutant and an intranasal infection mode
51 catula development by isolating the lfl/wox3 loss-of-function mutant and performing genetic crosses w
52       Accordingly, cells behave like a rad53 loss-of-function mutant and show reduced NHEJ efficiency
53 ogen insensitivity syndrome (AIS)-associated loss-of-function mutants and 168 prostate cancer-associa
54                                              Loss-of-function mutants and ectopic expression assays r
55 n of Pm5e was validated by transgenic assay, loss-of-function mutants and haplotype association analy
56                                     In car-1 loss-of-function mutants and in micu-1 overexpressing an
57                         The stabilization of loss-of-function mutants and insensitivity of gain-of-fu
58 GR3, named CGR2, and evaluated the effect of loss-of-function mutants and over-expression lines of CG
59                          Analysis of atprx71 loss-of-function mutants and plants overexpressing AtPRX
60      The combined use of cytochrome c (CYTc) loss-of-function mutants and respiratory complex III inh
61  validate genetic experiments performed with loss-of-function mutants and RNA interference (RNAi) lin
62 l fate and polarity, through the analysis of loss-of-function mutants and transgenic plants that ecto
63 riants conferring resistance to malaria are 'loss-of-function' mutants and appear to be recent polymo
64  isolated dominant-negative mutants, unc-108 loss-of-function mutant animals are defective in locomot
65                                      In Lrrk loss-of-function mutants, anterograde movement of GOPs w
66 n oncogene and neuroprotective protein whose loss-of-function mutants are associated with certain typ
67  propose that the pleiotropic effects of dbf loss-of-function mutants are associated with the require
68 nd phenotypes of blh6, knat7, and blh6 knat7 loss-of-function mutants are consistent with the existen
69                                 Because BOB1 loss-of-function mutants are embryo lethal, we used a pa
70 r in vitro and, therefore, the corresponding loss-of-function mutants are expected to be lethal.
71 d as having highly branched phenotypes, ccd1 loss-of-function mutants are indistinguishable from wild
72                                     Atga2ox9 loss-of-function mutants are more sensitive to freezing
73                                        imb-2 loss-of-function mutants are not viable; however, we ide
74 pment of the trophectoderm lineage and Eomes loss-of-function mutants arrest at implantation.
75  present study, several gain-of-function and loss-of-function mutants as well as engineered mutants o
76                                         CYTc loss-of-function mutants, as well as the d-LDH mutants,
77                                       Strong loss-of-function mutants assemble apolar intersecting mi
78 strates accelerated amyloidogenesis in Trem2 loss-of-function mutants at early stages, which progress
79    The method involves generation of partial loss-of-function mutants, at either buried or functional
80 eritability of Cas9 activity in heterozygous loss-of-function mutant backgrounds, to rapidly evaluate
81 er Myc mutants, MycDeltaMBIV is not a simple loss-of-function mutant because it is hyperactive for G2
82                         We find that in dfer loss-of-function mutants beta-catenin is hypophosphoryla
83  axotomy, which is absent in ced-4 and ced-3 loss-of-function mutants but present in the ced-4(gf) mu
84 n of an AWC(ON) marker is initiated in nsy-7 loss-of-function mutants, but subsequently lost, so that
85 n wild-type maize and in the isogenic mop1-1 loss-of-function mutant by using Illumina's sequencing-b
86 action, we circumvented the lethality of keg loss-of-function mutants by silencing KEG using an artif
87                                    In DSAS-6 loss-of-function mutants, centrioles failed to close and
88  We report here characterization of a C-tail loss-of-function mutant, CF327A, and a related suppresso
89 have reduced FLC expression, but, unlike clf loss-of-function mutants, clf-59 mutants do not display
90 , although stomata of the Arabidopsis syp121 loss-of-function mutant close normally in ABA and high C
91 rs1-2 ers2-3 and triple etr2-3 ers2-3 ein4-4 loss-of-function mutants constitutively nutated in air.
92 ucose-fed conditions, the Arabidopsis sr45-1 loss-of-function mutant contains higher amounts of the e
93                                         Ddok loss-of-function mutant (Ddok(PG155)) germ-line clones p
94 ci appear to be "Myddosomes." The MyD88 S34Y loss-of-function mutant demonstrates how proper cellular
95                Here, we show that single luh loss-of-function mutants develop normal flowers, but lug
96                              Zebrafish rad51 loss-of-function mutants developed key features of FA, i
97                      We determined that Dh31 loss-of-function mutants (Dh31(#51)) showed normal rhyth
98                                        dip-2 loss-of-function mutants display a progressive increase
99                           Intriguingly, gvs1 loss-of-function mutants display allele- and accession-d
100                         We show that partial loss-of-function mutants display temperature-sensitive (
101                                      A cdf-2 loss-of-function mutant displayed impaired growth and re
102                                          bt2 loss-of-function mutants displayed a hypersensitive resp
103                                        jmj17 loss-of-function mutants displayed dehydration stress to
104                                      mtstmir loss-of-function mutants displayed impaired induction of
105                                      bhlh121 loss-of-function mutants displayed severe defects in Fe
106                                     The weak loss-of-function mutant dse1 exhibits pleiotropic phenot
107 activity, but they all appear to manifest as loss-of-function mutants due to defects in solubility, a
108           Here we show that in mef2ca(b1086) loss of function mutant embryos and early larvae, develo
109          Biochemical analysis of single FPGS loss-of-function mutants established that folate polyglu
110 -function ET receptor mutant, etr1-3, or the loss-of-function mutants etr1-7 and ers1-3 and the wild
111                Conversely, wdr-20 and wdr-48 loss-of-function mutants exhibit changes in locomotion b
112 associated with vascular tissues, while vup1 loss-of-function mutants exhibit collapsed morphology of
113 ted deletion of the cmpy locus and find that loss-of-function mutants exhibit excessive NMJ growth.
114                At elevated temperatures, dao loss-of-function mutants exhibit seizures associated wit
115                                     Frazzled loss of function mutants exhibited similar defects in sy
116                                     eIFiso4G loss of function mutants exhibited smaller cell, leaf, p
117                              sleepless (sss) loss-of-function mutants exhibited altered Shaker locali
118 3 (GH43) family in Arabidopsis thaliana GH43 loss-of-function mutants exhibited root cell expansion d
119  the dominant act-5 mutation, or a recessive loss of function mutant, exhibited normal morphology and
120 similar to glh-1/glh-4(RNAi), the kgb-1(um3) loss-of-function mutant exhibits germline over-prolifera
121  root growth after Al exposure, because alt2 loss-of-function mutants fail to halt root growth after
122                                  at4, an At4 loss-of-function mutant fails to redistribute Pi to the
123                                         In a loss of function mutant for genes encoding PHR1 and PHL1
124                        Here, the Arabidopsis loss-of-function mutant for annexin1 (Atann1) was found
125                       In this study, we used loss-of-function mutants for each receptor isoform to de
126 This method can provide homozygous mammalian loss-of-function mutants for forward genetic application
127                                 In gain- and loss-of-function mutants for Gsx1 and Olig1/2, we observ
128 planta by feeding [(3)H]serine to the WT and loss-of-function mutants for OAS-TLs in the cytosol, pla
129 onfers increased Al tolerance similar to the loss-of-function mutants for the cell cycle checkpoint g
130 n the North American monarch butterfly using loss-of-function mutants for the circadian activators CL
131 dendrites mimic ALA axon guidance defects in loss-of-function mutants for the extracellular matrix mo
132 te this issue, we produced a complete set of loss-of-function mutants for the three annotated Arabido
133                  Ultrastructural analyses of loss of function mutants further show that IFC-2 mutants
134 n297, Ala301, Phe307, and Tyr308 represented loss-of-function mutants; furthermore, the measurable re
135                           l(2)tn, along with loss-of-function mutants generated for tn, showed no rel
136 d endoreplicating tissues in Drosophila gatA loss-of-function mutants grow slowly and never achieve w
137                                          UPR loss-of-function mutants, hac1Delta and ire1Delta, are a
138                          Consistently, zat12 loss-of-function mutants had higher Fe content than the
139                         The cpr1-2 (cpr30-1) loss-of-function mutant has constitutive defense respons
140 ment from fertilization, the cyp78a8 cyp78a9 loss-of-function mutant has reduced seed set due to oute
141              Chloroplasts prepared from fro7 loss-of-function mutants have 75% less Fe(III) chelate r
142                                              Loss-of-function mutants have a pleiotropic phenotype in
143                          Both NUA and AtMAD2 loss-of-function mutants have a shorter primary root and
144                                         rte1 loss-of-function mutants have an enhanced ethylene respo
145 tory molecules is wide-ranging, however, few loss-of-function mutants have been identified in miRNA g
146                                   Because no loss-of-function mutants have been identified, the impor
147 ate earlier than the wild type, whereas etr2 loss-of-function mutants have increased sensitivity to A
148                                        msca1 loss-of-function mutants have reduced meristem size and
149                                Instead, etr1 loss-of-function mutants have reduced sensitivity to abs
150 eproductive and social behaviors, yet rodent loss-of-function mutants have relatively subtle behavior
151  recordings identified the R215H mutant as a loss-of-function mutant in inducing GABAergic synaptogen
152                                            A loss-of-function mutant in Kif20b, magoo, was found in a
153                             By screening for loss-of-function mutants in a Caenorhabditis elegans kil
154 natural ELF3 polyQ variants phenocopied elf3 loss-of-function mutants in a common reference backgroun
155                                    All three loss-of-function mutants in Arabidopsis (Arabidopsis tha
156 toperiod-dependent flowering in plants, with loss-of-function mutants in barley (Hordeum vulgare), le
157                                              Loss-of-function mutants in both homeologs of AP2L2 (hen
158        Generation of all major OAS-TL double loss-of-function mutants in combination with radiolabele
159                                              Loss-of-function mutants in dPIP4K show reduced body wei
160              In particular, pink1 and parkin loss-of-function mutants in Drosophila show similar phen
161 rgan defects and indeterminacy that resemble loss-of-function mutants in E-function floral organ spec
162 ies converge on similar causes for olfactory loss-of-function mutants in evolutionary transitions to
163 te that the phenotypes associated with novel loss-of-function mutants in EXO70, are entirely consiste
164      Here, we report single and higher-order loss-of-function mutants in members of the cation/proton
165                                              Loss-of-function mutants in one of the genes, designated
166                                              Loss-of-function mutants in PRC2 subunits initially deve
167                   In this study, we isolated loss-of-function mutants in the Arabidopsis MIPS1 gene f
168 dent in the embryonic epidermis of zebrafish loss-of-function mutants in the cognate Matriptase inhib
169                                              Loss-of-function mutants in the corresponding genes had
170            Here, by generating and analyzing loss-of-function mutants in the liquid facets-Related (l
171 ole of tomosyn in neurosecretion we analysed loss-of-function mutants in the single Caenorhabditis el
172 utant hearts of heartstrings (hst), the tbx5 loss-of-function mutants in zebrafish.
173 rate that the gonadogenesis defects of gon-2 loss-of-function mutants (including a null allele) can b
174 psis resistance to Pst DC3000, whereas ADPG2 loss-of-function mutants increase the resistance to the
175           Importantly, genetic rescue by the loss-of-function mutant indicates that barbed-end bindin
176 tantly, the BIG signature was reduced in Bmp loss-of-function mutants, indicating Bmp-regulated targe
177 sion was drastically reduced in beta-catenin loss-of-function mutants, indicating that Wnt signaling
178                                  The AtSAP18 loss- of-function mutant is more sensitive to NaCl, and
179     The ABA hypersensitive phenotype of FyPP loss-of-function mutants is ABI5 dependent, and the amou
180 osmotic stress, while the growth of erf5erf6 loss-of-function mutants is less affected by stress.
181 h and show that their derepression in miR164 loss-of-function mutants is likely to account for most o
182                           The ability of two loss-of-function mutants, L31A and L31C, of phospholamba
183 n wild-type (WT) STAT1 cells, as well as the loss-of-function mutants L706S and Y701C.
184                                  While elt-7 loss-of-function mutants lack a discernible phenotype, s
185 phenotype of the etr1-6;etr2-3;ein4-4 triple loss-of-function mutant line was examined.
186             Genomic profiling of H3K27me3 in loss-of-function mutant lines for Maize Enhancer of zest
187 importance of Nisch, here we generated Nisch loss-of-function mutant mice and analyzed their metaboli
188                               In contrast to loss-of-function mutants, moderate overexpression of BLF
189                                      Using a loss-of-function mutant mouse lacking the guanylate kina
190 al prefrontal cortex (n = 21); and 4) a Nrg3 loss-of-function mutant (n = 59) to functionally implica
191 d-type FIT2, whereas FIT1 and a FIT2 partial loss-of-function mutant, N80A, had significantly lower t
192 ll three alleles are similar to those of the loss-of-function mutants obtained by RNA interference or
193         Leaf reticulation in the re and rer3 loss-of-function mutants occurred, along with accumulati
194 etion mutant of FOF2 (FOF2DeltaF), or double loss of function mutant of FOF2 and FOL1 (FOF2-LIKE 1) p
195                                              Loss of function mutants of BGD2 are acyanogenic in leav
196                              Although single loss of function mutants of ELF3 and GI have been well s
197  (GBP) interact with the N terminus of GluTR Loss-of function mutants of ClpR2 and ClpC1 proteins sho
198 ants of AGL15, alone or when combined with a loss-of-function mutant of a closely related family memb
199                               We generated a loss-of-function mutant of a coiled-coil domain containi
200 ressing bcl-xL, ced-9, and bcl-xL (G138A), a loss-of-function mutant of bcl-xL.
201                                          The loss-of-function mutant of BIN2 and its homologs, bin2-3
202 rel map development using barrelless mice, a loss-of-function mutant of calcium/calmodulin-activated
203                            R9C is a complete loss-of-function mutant of PLN, whereas R14del is a mild
204                           Co-expression of a loss-of-function mutant of polycystin-2 in CHO cells doe
205          We also studied the phenotypes of a loss-of-function mutant of Prosalpha6T generated by targ
206                                          The loss-of-function mutant of SR1IP1 is more susceptible to
207                         We also found that a loss-of-function mutant of the CPD33 homolog in Arabidop
208  expression phenotypes in overexpression and loss-of-function mutants of 15 Arabidopsis A-type heat-s
209                          Here we report that loss-of-function mutants of AGL15, alone or when combine
210                We also construct conditional loss-of-function mutants of AGO1 to allow transcript pro
211 , we generated double, triple, and quadruple loss-of-function mutants of all four members of the RWA
212 n content and vein density were increased in loss-of-function mutants of Arabidopsis MYC2, a suppress
213 scriptional machinery during leaf senescence Loss-of-function mutants of Arabidopsis thaliana were us
214                                              Loss-of-function mutants of AtPTPN were hyposensitive to
215                                 In midkine-a loss-of-function mutants of both sexes, Muller glia init
216                                      Because loss-of-function mutants of ftsZ(Bbu) might be lethal, t
217 we investigated the binding of A1 to GOF and loss-of-function mutants of GP Ibalpha with mutations in
218                           Here, we generated loss-of-function mutants of Iduna to investigate its phy
219                                We found that loss-of-function mutants of LecRKA4.2 and LecRKA4.3 exhi
220                                              Loss-of-function mutants of LOX6 were more attractive to
221 imilar to the rgi1/2/3/4/5 quintuple mutant, loss-of-function mutants of MPK3 and MPK6, MKK4 and MKK5
222 or P. patens and facilitates the creation of loss-of-function mutants of multiple genes from differen
223                                              Loss-of-function mutants of PALM1 develop dissected leav
224                                        Thus, loss-of-function mutants of PptH displayed antimicrobial
225                                              Loss-of-function mutants of rtp replicate this axonal pr
226                              Using gain- and loss-of-function mutants of seven transcription factors
227 wed precocious flowering, whereas the triple loss-of-function mutants of SPL10 and its two homologous
228 cal neurons and impaired root hair growth in loss-of-function mutants of the ATL1 ortholog rhd3 in th
229                                          The loss-of-function mutants of the BON1 gene family, bon1bo
230                                              Loss-of-function mutants of the NOP2A candidate gene loc
231                            Here, we analyzed loss-of-function mutants of the unicellular cyanobacteri
232                                              Loss-of-function mutants of these selected differentiall
233                                     Although loss-of-function mutants of UBP27 do not show obvious ph
234         Here we report the impact of an LCI1 loss-of-function mutant on growth and photosynthesis in
235 ysis of root phenotypes induced by gain- and loss-of-function mutants or in treatments with GLV-deriv
236                          In addition to this loss of function, mutant p53 can have a dominant negativ
237                                     In mrg-1 loss-of-function mutants, pairing is compromised specifi
238                                          The loss of function mutant phenotype and localization of an
239  comprehensive dataset of 2,400 genes with a loss-of-function mutant phenotype in Arabidopsis.
240  silencing in Drosophila embryos resulted in loss-of-function mutant phenotypes for 43 genes, which i
241 or neurons - where it recapitulates expected loss-of-function mutant phenotypes.
242  GATA17, and GATA17L (GATA17-LIKE), based on loss-of-function mutant phenotypes.
243 tor mutant, myb57-2, closely phenocopied the loss-of-function mutant pin6-2.
244 an phenotypes reported in the literature for loss-of-function mutant plants.
245                    In unc-6/netrin and unc-5 loss-of-function mutants, presynaptic components misloca
246                                       In the loss-of-function mutant pro(DeltaGRAS), all examined GA
247  to freezing temperatures, whereas Atga2ox10 loss-of-function mutants produce considerably more seeds
248 es as either a recessive gain-of-function or loss-of-function mutant protein, depending on signaling
249 NA (mtDNA) in Arabidopsis thaliana Gain- and loss-of-function mutants provided evidence for a role of
250                                    The RCAR7 loss-of-function mutant revealed no changes in ABA respo
251                             Analysis of bhlh loss-of-function mutants revealed that the single bhlh m
252 fted onto PC synthase cad1-3 atpcs2-1 double loss-of-function mutant root tissues.
253 e intestine and interneurons and that ehbp-1 loss-of-function mutants share with rab-10 mutants speci
254                                              Loss-of-function mutants show an elongated hypocotyl und
255 AtSWEET2 sequesters sugars in root vacuoles; loss-of-function mutants show increased susceptibility t
256                                              Loss-of-function mutants show irregular, uncoordinated c
257 plants with defective CIA components, grxs17 loss-of-function mutants showed some degree of hypersens
258 scopy of isolated lipid droplets from cgi-58 loss-of-function mutants showed they contain triacylglyc
259 istent enhanced collagen binding whereas the loss-of-function mutants showed variable degrees of func
260        With the help of a mitochondrial SOD2 loss-of-function mutant, Sod2(n283), we measured the sen
261           Similar to unc-43, tir-1 and nsy-1 loss-of-function mutants, specific disruption of microtu
262                                   In sprouty loss-of-function mutants, splitting of gene expression d
263 on of a phoPR promoter-lacZ fusion in a scoC loss-of-function mutant strain grown in low-phosphate de
264 ant for Pan1p inhibition, and a pan1 partial loss-of-function mutant suppressed the temperature sensi
265                        AURKC p.L49Wfs22 is a loss-of-function mutant that perturbs localization of th
266                             In a tni partial loss-of-function mutant that was originally isolated bas
267 transferred into etr1-6;etr2-3;ein4-4 triple loss-of-function mutants that have constitutive growth i
268                                    In unc-89 loss-of-function mutants that lack the SH3 domain, param
269                    We identified 18 presumed loss-of-function mutants that reduced the growth of the
270                            Moreover, in such loss-of-function mutants the levels of acetylated H4K5,
271                          We used the soc1ful loss of function mutant - the woodiest genotype known in
272 fies cytosolic pH and NO(3) (-) In an AtCLCa loss of function mutant, the cytosolic acidification tri
273                                     In syd-2 loss of function mutants, the normal polarized localizat
274                               alt2-1 and atr loss-of-function mutants, the latter of which affects th
275                             In SAC gain- and loss-of-function mutants, the levels of PtdIns monophosp
276            Furthermore, we show that in cftr loss-of-function mutants, there is a deficiency of larva
277 ction of secondary roots are impaired in the loss-of-function mutant, thus identifying AtANN1 as a ke
278  as in vivo physiological studies in an LCI1 loss-of-function mutant to reveal the C(i) species prefe
279                                 Here, we use loss-of-function mutants to show that RNA polymerase II
280                                     The TPC1-loss-of-function mutant tpc1-2 does not respond to elect
281                                         BRG1 loss-of-function mutant tumours respond to EZH2 inhibiti
282 ues the late-flowering phenotype of the gi-2 loss-of-function mutant under both short-day and long-da
283 erences in germination between etr1 and etr2 loss-of-function mutants under salt stress could not be
284                         By analyzing an rdr2 loss-of-function mutant using two different parallel seq
285  Consistent with these results, a Col-0 rfo1 loss-of-function mutant was more susceptible to f. matth
286                                    An atfs-1 loss-of-function mutant was partially resistant to the e
287               The enhanced resistance in the loss-of-function mutants was associated with increased i
288            Using the EXORIBONUCLEASE4 (XRN4) loss-of-function mutant, we showed that XRN4 poly(A(+))
289                              Using gain- and loss-of-function mutants, we demonstrate that acetylatio
290             Through an extensive analysis of loss-of-function mutants, we demonstrate that the Arabid
291 ting them in the respective four Arabidopsis loss-of-function mutants, we experimentally proved that
292    Using RNA interference (RNAi) to generate loss-of-function mutants, we show dramatic defects in ce
293 pressed AtLTPI-4 Crown galls of the atltpI-4 loss-of-function mutant were much smaller compared with
294 cumulation in wild-type roots, whereas Atao1 loss-of-function mutants were unresponsive to the hormon
295 psis plants expressing HaRxL44 and in med19a loss-of-function mutants, whereas SATI is elevated in pl
296 s found in the arr1-3 arr10-5 arr12-1 triple loss-of-function mutant, which showed almost complete in
297                           In contrast to the loss-of-function mutant with large embryo and small endo
298                             The isolation of loss-of-function mutants with enhanced disease susceptib
299  this function was only apparent when double loss-of-function mutants with pp2ca-1/ahg3 were generate
300 evelopmental processes in situ by generating loss-of-function mutants within otherwise wildtype tissu

 
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