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1 discovered more than 340 yr ago in the human louse.
2 egg production and reproduction in the book louse.
3 be greatest in the cooler temperature of the louse.
4 of cement glands in the adult female salmon louse.
5 poE is significantly upregulated at the body louse (28 degrees C) versus the human host (37 degrees C
6 ere, we present genome sequences of the body louse and its primary bacterial endosymbiont Candidatus
9 Relapsing fever (RF) is caused by tick- and louse-borne Borrelia spp., is characterized by recurrent
13 odies will allow serological confirmation of louse-borne relapsing fever and determination of disease
14 The hallmark of disease caused by tick- and louse-borne relapsing fever due to Borrelia infection is
16 serum samples obtained from 42 patients with louse-borne relapsing fever were tested with an indirect
17 t Borrelia recurrentis genomes, the agent of louse-borne relapsing fever, dating from 2300 to 600 yea
19 thogens have transitioned from tick-borne to louse-borne transmission, which often involves genome re
26 (hbpC and hbpB) predominates in a simulated louse environment (high heme), and subgroup II (hbpA, hb
28 is study, we report that the Atlantic salmon louse expresses three full-length MVP paralogues (LsMVP1
31 ize matters, we measured three components of louse fitness: attachment, feeding, and escape from host
33 nsistent with previous work, we propose that louse flies could serve as valuable sentinels for monito
36 n the intention-to-treat population who were louse-free 1 day after treatment (day 2) and remained so
37 than patients receiving vehicle control were louse-free on day 2 (94.9% vs. 31.3%), day 8 (85.2% vs.
38 Compared with other insect genomes, the body louse genome contains significantly fewer genes associat
43 bacterial community associated with the sea louse, however, it is unknown if these microorganisms sh
45 matic review first identified trials on head-louse infestation; 49 were selected and their methodolog
46 omplicates the public health problem of head-louse infestations and drives the need for continuing de
47 ive than vehicle control in eliminating head-louse infestations at 1, 7, and 14 days after treatment.
48 e efficient than placebo at eliminating body louse infestations by day 14; however, this difference w
53 Furthermore, when this strain is grown at a "louse-like" temperature, an inversion of the transcript
54 nscription of irr (a >5-fold decrease) at a "louse-like" temperature, suggesting that Irr may functio
55 rcular mitochondrial chromosomes of the body louse may be linked to the loss of the gene encoding the
57 s gambiae and Culex quinquefasciatus, a body louse Pediculus humanus and a tick species Ixodes scapul
58 ction of the mycetomic bacterium of the head louse Pediculus humanus capitis and the body louse P. h.
60 s an obligatory parasite of humans, the body louse (Pediculus humanus humanus) is an important vector
62 uefasciatus), tick (Ixodes scapularis), body louse (Pediculus humanus), kissing bug (Rhodnius prolixu
63 ganisms, and another compared the human body louse, Pediculus humanus humanus, against itself and sel
64 d Pediculus humanus corporis (Ph; human body louse) PINK1 resolves an N-terminal helix, revealing the
67 tracted RNA and protein from two of the body louse's morphologically distinct sets of salivary glands
70 ss the potential physiological importance of louse salivary proteins, and consider possible explanati
71 proteomic characterization of the human body louse sialome, discuss the potential physiological impor
72 he complex nature of GABA receptors in human louse that could help in understanding the resistance pa
73 mimetabolous insects, the genome of the body louse thus provides a reference for studies of holometab
74 d optimal designs to standardize future head-louse treatment trials, thereby obtaining valid conclusi
75 o stressors that are encountered in the body louse vector environment, a decreased temperature and an
76 has adapted to both the human host and body louse vector niches, producing persistent infection with
79 the pathogen in the human host and the body louse vector; e.g., we observed a dramatic (>100-fold) i