ライフサイエンスコーパス: low cardiac output

1 assist in the detection of low-flow states (low cardiac output).

2 ated with underfilling of the left heart and low cardiac output.

3 gement of patients with ADHF presenting with low cardiac output.

4 wer limb SCDs because they produce a falsely low cardiac output.

5 iliary circulatory support in the setting of low cardiac output.

6 severe arterial unsaturation, and usually a low cardiac output.

7 ospital mortality attributable to persisting low-cardiac output.

8 idence of perioperative infarction was 3.3%, low cardiac output 2.7%, stroke 2.2%, reoperation for bl

9 after tetralogy of Fallot repair results in low cardiac output and a prolonged stay in the intensive

10 erosclerosis, and the combined end points of low cardiac output and atrial fibrillation.

11 Low cardiac output and cardiac arrest, inflammation-rela

12 of sepsis and was only found in models with low cardiac output and decreased renal blood flow (p < 0

13 recognized that these patients present with low cardiac output and high peripheral resistance and th

14 use severe LA and LV enlargement, leading to low cardiac output and hydrops.

15 of age, P=0.06), with a parallel decline in low cardiac output and length of hospital stay.

16 Fluid overload, renal dysfunction, low cardiac output and neurological dysfunction remain m

17 in principle by hypervolaemia, salt avidity, low cardiac output and normal left ventricular function.

18 Two patients developed low cardiac output, and 3 patients had pleural effusions

19 both ventricles accompanied by hypotension, low cardiac output, and high filling pressures occurring

20 erative support techniques and postoperative low cardiac output are associated with cerebral hypoperf

21 ion of the right ventricle (RV); hydrops and low cardiac output are often associated.

22 ocusing on the importance of the syndrome of low cardiac output as a key feature of advanced heart fa

23 ocardiography is a useful tool for assessing low cardiac output AS.

24 During states of low cardiac output, cerebral blood flow and thus cerebra

25 on of mitral regurgitation (MR) results in a low cardiac output (CO) state because of an acute increa

26 In addition to "abnormal flow" through low cardiac output, dilated cardiac chambers and poor co

27 ysfunction is a commonly considered cause of low cardiac output in critically ill patients.

28 0.55), as well as persistent hypotension and low cardiac output (in 83 percent of the patients, vs. 1

29 rculation, which can be impaired either by a low cardiac output or arterial vasodilation, is an impor

30 lobal or regional hypoxia or ischemia due to low cardiac output or cardiac arrest.

31 ent, reflected the presence of an associated low cardiac output or low renal blood flow syndrome.

32 CI 1.1 to 1.7], p = 0.02), and postoperative low cardiac output (OR 3.0 [95% CI 1.7 to 5.2], p = 0.00

33 ed by elevated cardiac filling pressures and low cardiac output, or b) ongoing signs of hypoperfusion

34 acity in people with severe TR is related to low cardiac output reserve relative to metabolic needs,

35 ons, and (3) physiologic phenotypes, such as low cardiac output, right HF, cardiorenal syndromes, con

36 common use of inotropes and vasodilators for low cardiac output septic shock associated with elevated

37 a significant reduction in the incidence of low cardiac output state (odds ratio, 0.22; 95% confiden

38 a significant reduction in the incidence of low cardiac output state and the need for inotropic supp

39 A low cardiac output state can complicate the postoperativ

40 ally challenging scenarios including AS with low cardiac output state or other structural heart disea

41 etry may be more accurate in patients with a low cardiac output state.

42 tant therapeutic option in children with the low cardiac output state.

43 l in origin, including small aortic calibre, low cardiac output states, high vasopressor requirements

44 of infection (12.9% vs 29.7%; p = 0.002) and low cardiac output syndrome (6.5% vs 26.6%; p = 0.002).

45 Low cardiac output syndrome (LCOS), affecting up to 25%

46 f congenital heart disease, that can develop low cardiac output syndrome (LCOS).

47 Low cardiac output syndrome after cardiac surgery is ass

48 own in small studies to prevent or treat the low cardiac output syndrome after cardiac surgery.

49 Low cardiac output syndrome and multiorgan failure were

50 ped relationship was observed for stroke and low cardiac output syndrome but not for renal replacemen

51 entification and aggressive treatment of the low cardiac output syndrome peculiar to these patients.

52 Composite end point reflecting low cardiac output syndrome with need for a catecholamin

53 d 20.9% of the standard care group developed low cardiac output syndrome within 48 hours, needed extr

54 ailure were the most common causes of death (low cardiac output syndrome, 70 [36.5%]; multiorgan fail

55 econdary end points including a composite of low cardiac output syndrome, extracorporeal life support

56 rge as a composite of myocardial infarction, low cardiac output syndrome, infection, stroke, or in-ho

57 y bypass oxygenator may reduce postoperative low cardiac output syndrome, leading to improved recover

58 Complications such as low cardiac output syndrome, prolonged ventilation and r

59 utcome (death within 30 days or in-hospital, low cardiac output syndrome, stroke, or Fontan takedown)

60 atients after cardiac surgery because of the low cardiac output syndrome.

61 The primary outcome was the incidence of low cardiac output syndrome.

62 rative levosimendan to prevent postoperative low cardiac output syndrome.

63 ive stroke rate unless it was accompanied by low cardiac output syndrome.

64 eplacement remained high, largely because of low cardiac output syndrome.

65 y of intervention, conversion to sternotomy, low cardiac output that required mechanical support, aor

66 tion died 7 weeks after surgery secondary to low cardiac output; the other 3 had resolution of effusi

67 maceuticals clinically used for treatment of low cardiac output with cardiogenic shock.

68 y may potentially lead to an inappropriately low cardiac output, with a subsequent compromise of micr