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1 (total workload in RT exercises of upper and lower limbs).
2  different body sites (face/arms, trunk, and lower limbs).
3  sunburns at other body sites (face/arms and lower limbs).
4        It also exhibits a humanlike foot and lower limb.
5  remote ischemia was induced to the upper or lower limb.
6 rings and gastrocnemius are extensors of the lower limb.
7 quantify collateral arterial function of the lower limb.
8 e performed due to critical ischemia of left lower limb.
9 n both the superficial and deep veins in the lower limb.
10 (31%); these changes were more common in the lower limb.
11 patients with metastatic bone disease of the lower limb.
12 monoarthritis that affects the joints of the lower limb.
13 d in patients with metastatic disease in the lower limb.
14 active hand vector was co-ordinated with the lower limbs.
15  to severe sensory and motor deficits in the lower limbs.
16 or neurons and progressive spasticity of the lower limbs.
17 ne oxygenation with neurologic injury of the lower limbs.
18 me with symptoms predominantly affecting the lower limbs.
19 : the intentional and systematic breaking of lower limbs.
20 r who have deep vein thrombosis (DVT) of the lower limbs.
21 f complex natural movements in the upper and lower limbs.
22 rized by abnormal gait and spasticity of the lower limbs.
23  18-29 years and for counts of the trunk and lower limbs.
24 itation have been described in the upper and lower limbs.
25 ion after nonmajor orthopedic surgery of the lower limbs.
26  disorder characterized by spasticity in the lower limbs.
27 aracterised by progressive spasticity of the lower limbs.
28 ure, heart attacks, stroke and amputation of lower limbs.
29 logical abnormalities were restricted to the lower limbs.
30 phoedema that causes massive swelling of the lower limbs.
31 with a minimum length of 10 cm in 1 of their lower limbs.
32  undergone previous revascularization of the lower limbs.
33 -section multidetector CT angiography of the lower limb (0.625-mm collimation, intravenous administra
34 8 patients with vascular malformation of the lower limbs, 19 patients (5%) had documented venous rect
35  [36%]) ulcerative (805 [66%]) lesion of the lower limb (733 [60%]).
36                     Somatotopy (upper versus lower limb: 93% accuracy for both conditions) was also d
37 tures extracted to characterize balance, the lower limb accelerometry-based metrics proved to be most
38 e objective was to determine whether reduced lower limb activity alters IMAT in healthy young adults
39 althy proximal muscle of patients undergoing lower limb amputation (n = 3), were analyzed for capilla
40                Nine "painless" patients with lower limb amputation and nine control subjects (sex- an
41  the extent of cortical reorganization after lower limb amputation in patients with nonpainful phanto
42    We excluded participants with known prior lower limb amputation.
43                     It is a leading cause of lower-limb amputation and disabling neuropathic pain.
44 s associated with high risk of infection and lower-limb amputation.
45 critical limb ischemia patients undergoing a lower-limb amputation.
46 ife-threatening disease that often result in lower limb amputations and a shortened lifespan.
47  receptor alpha (PPARalpha) agonist, reduces lower limb amputations in patients with type 2 diabetes.
48         Secondary outcomes included rates of lower limb amputations, bypass surgical procedures, and
49             In this study of 7 patients with lower limb amputations, inclusion of EMG signals and tem
50      In the United States, approximately 280 lower-limb amputations for ischemic disease are performe
51 ions of diabetes resulting in >70,000 annual lower-limb amputations in the United States alone.
52                          In individuals with lower-limb amputations, robotic prostheses can increase
53 imination and reaction time) on 12 upper and lower limb amputees and found that consistently reported
54  and humans become mechanically coupled when lower limb amputees walk with powered prostheses, but th
55  demonstrate a functional remapping of S1 in lower limb amputees.
56   To maintain postural stability, unilateral lower-limb amputees (LLAs) heavily rely on visual and ve
57                         Here we describe the lower limb anatomy of Au. sediba and hypothesize that th
58 ombosis DVT, pain, oedema and rubor of right lower limb and drug abuse.
59  peripheral gangrene, and 1 patient required lower limb and finger amputations.
60 ultrasonography, including 20 (57.1%) in the lower limbs and 24 (68.6%) related to central venous cat
61 erized by a predominance of paralysis at the lower limbs and an early adulthood onset.
62 lar veins are subdermal veins located in the lower limbs and are mainly associated with aesthetic com
63 que isometric and isokinetic strength of the lower limbs and handgrip strength.
64              DVT affects most frequently the lower limbs and hence DVT of the leg will be the focus o
65 the right hand, progressive weakness in both lower limbs and weight loss.
66 shortening of proximal segments of upper and lower limbs) and delayed collagen folding.
67 ived RIPost (induced by ischemia to upper or lower limb), and a third was the control group.
68 per limb or distal deep-vein thrombosis in a lower limb, and death from venous thromboembolism and wa
69 cle mass, (iii) redistributed muscle mass to lower limbs, and (iv) decreased relative mass of skin du
70 ildhood hypotonia, progressive spasticity of lower limbs, and abnormal craniofacial features in adult
71 ess, atherosclerotic plaques in the aorta or lower limbs, and diabetes mellitus.
72 y progressive spasticity and weakness of the lower limbs, and pathologically by retrograde axonal deg
73 mproved our understanding of consequences of lower limb angular deformities on knees.
74  calf contraction even when the veins of the lower limb are distended due to the presence of a physio
75 rwent treatment for stenosis or occlusion of lower limb arteries at the Department of Radiology of th
76 ision to perform angiographic examination of lower limb arteries was made.
77 1 ABI value < or =0.9, indicating obstructed lower limb arteries, increased from 18 (9.2%) of 191 pat
78 d atherosclerosis, particularly extensive in lower limb arteries, was admitted to the Department of A
79 vealed that patients assimilated the virtual lower limbs as if they were their own legs.
80 trasound-guided treatments of varices in the lower limbs, as well as to provide a brief overview of t
81 ight patients with inflammatory nodules on a lower limb associated with locoregional anatomical chang
82 y, intellectual impairment, weakness in both lower limbs, ataxia and abnormal facies and diagnosed on
83 r 3 generations displayed mild and quiescent lower-limb axonal sensorimotor neuropathy.
84 importance of a comprehensive description of lower-limb biomechanics that includes consideration of j
85 ines in mediolateral bending strength of the lower limb bones and strength of the humerus are much sm
86 trends in relative strength of the upper and lower limb bones in a sample of 1,842 individuals from a
87 n upper right third molar (RM(3)); and EQH3, lower limb bones of a young Neandertal male.
88  with an anencephalic head, spine, upper and lower limb buds.
89 LA when controlled loads were applied to the lower limb by a linear actuator.
90           Fifty-eight patients scheduled for lower limb bypass or open abdominal aortic aneurysm repa
91 e aim of this study was to determine whether lower limb (calf) sequential compression devices (SCDs)
92 erves as a result of damage to the upper and lower limbs caused by gunshot wounds and fragments of ar
93 erotic occlusions decrease blood flow to the lower limbs, causing ischemia and tissue loss in patient
94  balance control and also that the upper and lower limbs co-ordinate for an appropriate whole-body sw
95  trabecular density throughout the upper and lower limbs compared with other primate taxa and (ii) th
96             Fat fraction correlated with the lower limb components of the inclusion body myositis fun
97 at spike-timing-dependent protocols modulate lower limb cortical circuitry during walking in a phasic
98      Although well-established for suspected lower limb deep venous thrombosis, an algorithm combinin
99 The pulmonary embolism risk was increased by lower-limb deep venous thrombosis (odds ratio 4.0; 95% c
100  a significantly lower incidence of proximal lower-limb deep-vein thrombosis than pharmacologic throm
101 s thromboembolism (pulmonary embolism or any lower-limb deep-vein thrombosis) occurred in 103 of 991
102 ry outcome was incident (i.e., new) proximal lower-limb deep-vein thrombosis, as detected on twice-we
103 d with severe complications such as rickets, lower limb deformities, pain, poor mineralization of the
104 e with DNA from 253 patients with congenital lower limb disorders.
105                  Compared to the upper limb, lower limb distal nerve transfer (DNT) outcomes are poor
106                The reference test was a full lower limb duplex ultrasound.
107 r unit recruitment in specific phases in the lower limbs during walking.
108                              Gait ataxia and lower limb dysaesthesiae were the most frequent presenti
109  13-63), beginning with chorea in 50%, focal lower limb dystonia in 42.5% and parkinsonism in 7.5%.
110 early-onset pyramidal tract signs and marked lower limb dystonia.
111              The main symptoms are bilateral lower limb edema, serosal effusions, and vitamin D malab
112 el-coated angioplasty balloons and stents in lower-limb endovascular interventions for symptomatic pe
113 r different orthopaedic interventions of the lower limbs, endovascular embolisations have proven to b
114 botic actuators, including a custom-designed lower limb exoskeleton capable of delivering tactile fee
115 f the quadriceps musculature in closed chain lower limb extension may be more important than is typic
116 o the motor control strategies available for lower limb extension.
117  unknown mechanisms and this in turn acts to lower limb flow velocity.
118 on; 3.RIPC+IR: 6 cycles of 4x4 min IR of the lower limb followed by IR group procedure; (4) 1H-[1,2,4
119 ing with iatrogenic vascular injuries of the lower limbs following orthopaedic interventions.
120                   HuRANKL-Tg+ mice displayed lower limb force and maximal speed, while their leg musc
121 Furthermore, they demonstrate that upper and lower-limb forces are co-ordinated to produce an appropr
122 6 years or older who underwent surgery for a lower limb fracture caused by major trauma from July 7,
123                             Upper and distal lower limb fracture rates were increased mainly in women
124 o underwent surgery for major trauma-related lower limb fractures, use of incisional negative pressur
125  minimal shoes, and may protect the feet and lower limbs from some of the impact-related injuries now
126 th spinal cord injury had impaired upper and lower limb function bilaterally, a 30% reduced cord area
127  Thoracic cord GM areas were correlated with lower limb function.
128  than RMS and correlates with disability and lower limb function.
129  specifically required for balance and upper/lower limb functional integrity.
130 s lymphadenitis about 40 days after onset of lower limb gangrene.
131 including Neuropathy Impairment Score of the Lower Limbs, hand grip strength, and evaluation of veget
132 onance (BOLD-CMR) to assess perfusion in the lower limb has been hampered by poor reproducibility and
133 uscular electrical stimulation (NMES) of the lower limb has been shown to reverse muscle wasting in t
134 es with distinctly preserved strength of the lower limbs), hearing loss, optic atrophy and respirator
135 hology and strength during 7 d of unilateral lower-limb immobilization, and the role of myofibrillar
136 age: 23 +/- 1 y) underwent 7 d of unilateral lower-limb immobilization, with thrice-daily leucine (LE
137           Lesions predominantly affected the lower limbs in an asymmetric manner and were characteriz
138  lordosis stabilizes the upper body over the lower limbs in bipeds by positioning the trunk's centre
139 pindle afferents are absent in the upper and lower limbs in HSAN III, and we have argued that this ma
140 ecrease tissue perfusion causing ischemia to lower limbs in patients with peripheral arterial disease
141 foot deformity, decreased muscle mass of the lower limbs, inability to walk, and growth retardation.
142  dynamic simulations were performed on seven lower limb (including superficial femoral, deep femoral
143                                              Lower-limb infections in diabetic patients are most ofte
144                     Neurological deficits or lower-limb injuries can lead to slower walking speeds, a
145                                    Upper and lower limb intermuscular coherence was measured during a
146 er than decreased planter flexor strength in lower-limb involved ALS (77.2%vs 38.3%).
147                                              Lower-limb involved ALS patients exhibited "split leg" p
148 cal and electrophysiological features in 141 lower-limb involved ALS patients, 218 normal controls, 6
149 linical items as asymmetry and predominantly lower limb involvement.
150               Ischemia of the myocardium and lower limbs is a common consequence of arterial disease
151 y used imaging tests for symptomatic chronic lower limb ischemia, for which the administration of iod
152  walking economy, stride characteristics and lower limb joint angles were measured.
153 ies aimed at determining the biomechanics of lower limb joint force actions and postural kinematics.
154 ominins, in having an enlarged body size and lower limb joint surfaces in combination with a relative
155 ctivity in the liver, kidneys, and upper and lower limb joints.
156                                       In the lower limb, knee flexors were relatively weaker than ext
157 l aging process and traumatic events such as lower-limb loss can alter the human ability to control s
158 ts underwent full clinical characterization, lower limb magnetic resonance imaging (MRI), muscle biop
159  S1pr2(-/-) mice, whereas family DEM4154 has lower limb malformations in addition to hearing loss.
160                                        Other lower-limb malformations, including patellar hypoplasia,
161 he upper limb (mean = 64 days, p = 0.02) and lower limb (mean = 89 days, p < 0.01).
162 gnificance of the PIMs, we compared foot and lower limb mechanics with and without a tibial nerve blo
163  left-sided weakness involving the upper and lower limbs (medical research council graded muscle stre
164 ed of palpable pulsatile mass, pain, reduced lower limb motion, or visible haematoma; 11 patients had
165 senting single-limb or combined upper and/or lower limb motions.
166 sted the hypothesis that the excitability of lower limb motor cortex assessed during walking is incre
167 scranial magnetic stimulation (TMS) of human lower limb motor cortex paired with common peroneal nerv
168                                          Can lower limb motor function be improved after a spinal cor
169   This allows a much greater preservation of lower limb motor function in the parturient, with a subs
170 ecovery was paralleled by the reemergence of lower limb motor imagery at cortical level.
171 teristics and phase dependent differences in lower limb movement pattern between the two conditions w
172 d the feasibility of decoding kinematics for lower limb movements during walking.
173 on's disease as they executed cued upper and lower limb movements.
174                                              Lower limb MRI scans of patients with LGMD2C-2F, ranging
175                     The extent of lesions on lower limb MRI showed a high correlation with overall cl
176 tive rest" postures require higher levels of lower limb muscle activity than chair sitting.
177 d with lumbar intrathecal fentanyl impairing lower limb muscle afferent feedback (FENT).
178 e investigated the influence of group III/IV lower limb muscle afferents on the development of supras
179 g feedback from mu-opioid receptor-sensitive lower limb muscle afferents.
180 readmission at 1 year, including measures of lower limb muscle as part of a larger clinical trial.
181          We hypothesized that signaling from lower limb muscle group III/IV sensory afferents to the
182                                              Lower limb muscle mass and the stimulated limb to non-st
183                                   pelvic and lower limb muscle MRI scans of 269 symptomatic individua
184             Corticospinal drive to upper and lower limb muscle shows developmental changes with an in
185 r extremity-predominant, is characterized by lower limb muscle weakness and wasting, associated with
186 arly old age, statin use was associated with lower limb muscle-related outcomes, and some were only a
187              This study aimed to investigate lower-limb muscle activities in gait phases and co-contr
188                              The dissociated lower-limb muscle impairment was quantified by plantar f
189  differential diagnosis value of dissociated lower-limb muscle impairment, we performed a retrospecti
190        Previous descriptions have focused on lower limb muscles in small cohorts of patients with OPM
191             We applied this technique to the lower limb muscles of 4 patients with confirmed amyotrop
192 milarity between various distal upper versus lower limb muscles of tetrapod adults may be derived.
193 nucleotide injections into single peripheral lower limb muscles, whereas a viable therapy will need a
194 over, none of them examined the behaviour of lower limb muscles.
195  modulate motor system excitability of other lower limb muscles.
196 ospinal-motor neuronal synapses of upper- or lower-limb muscles (depending on the injury level), 1-2
197 reliable tool for assessing perfusion in the lower limb musculature and merits further investigation
198  MRI magnetisation transfer ratios (MTRs) in lower-limb musculature as markers of pathology in periph
199                                       MRI of lower-limb musculature can be used to detect minimal sig
200          Magnetic resonance imaging (MRI) of lower-limb musculature systematically showed fatty atrop
201                                    Traumatic lower-limb musculoskeletal injuries are pervasive amongs
202  muscle contraction with validated models of lower limb musculotendon paths and segment dynamics.
203 large insertions in the upper limb (n = 21), lower limb (n = 27), and spine (n = 4) from 60 cadavers.
204       Patients had inflammatory nodules on a lower limb (n = 6) that were unilateral (n = 6) and had
205 bined locoregional anatomical changes in the lower limbs (n = 5).
206 type, and Neuropathy Impairment Score in the Lower Limbs (NIS-LL) were evaluated.
207 eatment of choice for reticular veins in the lower limbs, no consensus has been reached regarding to
208  randomly assigned adult patients undergoing lower-limb nonmajor orthopedic surgery who were consider
209  past 12 weeks for any reason (p=0.003), and lower limb oedema (p=0.009) independently predicted deep
210 IE-large and small fibres polyneuropathy and lower limbs oedema.
211           Subsequent remineralization in the lower limb of the AMOC, between subpolar and subtropical
212 ough the Faroe Bank Channel and supplies the lower limb of the Atlantic Meridional Overturning Circul
213   Antarctic Bottom Water (AABW) supplies the lower limb of the global overturning circulation and ven
214 tern subpolar North Atlantic, both upper and lower limbs of the AMOC transport high-DOC waters.
215  sensory loss that predominantly affects the lower limbs, often preceded by hyperpathia and spontaneo
216 system region defined as bulbar, upper limb, lower limb or diaphragmatic), diagnosis, functional invo
217 ls developed spasticity predominantly of the lower limbs over the course of the disease.
218     High heels are a major source of chronic lower limb pain.
219 hetic devices is essential to restore a full lower limb perceptual experience in spinal cord injury (
220              However, the effect of ischemic lower limb peripheral arteries on hemodynamics remains u
221                                              Lower limb peripheral artery disease is a prevalent chro
222 ibre density (thigh), computerised myometry (lower limbs), plasma 1-deoxysphingolipid levels, calf-le
223  The primary disease presentation was a pure lower limb predominant spastic paraplegia.
224 ffected individuals, weakness and wasting is lower-limb predominant, and typically involves both prox
225 -years-old girl reported a lump in the right lower limb present for approximately 2 years, which was
226 h bilateral, asymmetrical lymphoedema of the lower limb present for more than 1 year, who tested nega
227 -knee amputations for ischemic ulcers of the lower limbs, presented at our clinic due to a sudden los
228 resiensis is that of a biped, but the unique lower-limb proportions and surprising combination of der
229 ctional electrical stimulation (sFES) of the lower-limbs, proprioceptive and tactile feedback, balanc
230                                      Robotic lower limb prostheses can improve the quality of life fo
231 es, ESA therapy did not increase the risk of lower limb proximal deep venous thrombosis (RR 0.97, 95%
232 mortality without an increase in the rate of lower limb proximal deep venous thrombosis.
233 confirmed proximal deep-vein thrombosis in a lower limb, pulmonary embolism, symptomatic deep-vein th
234 ar signs, and the majority had myoclonus and lower limb pyramidal signs, with relatively preserved co
235 pective cohort study, 20 patients undergoing lower limb reconstruction and 10 age-matched controls we
236 nclude that the sensory symptoms and loss of lower limb reflexes in Gerstmann-Straussler-Scheinker sy
237  reduced tendon reflexes, dysarthria, absent lower limb reflexes, and loss of position and vibration
238 der of muscle appearance in the upper versus lower limbs, reinforcing the idea that the similarity be
239    Remote sub-lethal ischemic injury to both lower limbs results in cerebral protection from subseque
240 mmendations for antithrombotic therapy after lower limb revascularization are inconsistent and not al
241 d 3-D rotational imaging of five fresh human lower limbs revealed calcification in all main arteries.
242 ired for mediating the protective effects of lower limb RIPC on hepatic MBF in liver IR injury.
243 ed to assess the role of hepatic sGC-cGMP in lower limb RIPC-induced protection against liver IR inju
244 s protection is nitric oxide (NO) induced by lower limb RIPC.
245 using the Neuropathy Impairment Score of the Lower Limbs (rs = -0.58 [95% CI, -0.84 to -0.11]; P = .0
246  may lead to errors higher than 5 degrees on lower limb sagittal kinematics and should thus be taken
247 study aims to assess a method for predicting lower limb sagittal kinematics using multiple regression
248 ld not be done during the inflation cycle of lower limb SCDs because they produce a falsely low cardi
249 muscular atrophy predominantly of the distal lower limbs, scoliosis, and mild distal sensory involvem
250 iated through the inhibition of group III/IV lower limb sensory muscle afferents.
251 nscriptional profiles in sets of human adult lower limb skeletal muscles.
252 s with incomplete or complete SCI (affecting lower limbs' somatosensation), with respect to controls.
253 e demonstrate a new approach for reproducing lower limb somatosensory feedback in paraplegics by rema
254 somal-recessive neurodegenerative subtype of lower limb spastic paraparesis with additional diffuse s
255 sts with early childhood onset predominantly lower limb spastic tetraparesis and truncal instability,
256 egia (HSP) is a genetic disorder that causes lower limb spasticity and weakness and intellectual disa
257 inantly, but not exclusively, by progressive lower limb spasticity and weakness resulting from distal
258 hy of the corticospinal tracts, resulting in lower limb spasticity and weakness.
259 y classified as either pure with predominant lower limb spasticity, or complex where spastic parapleg
260  are clinically characterised by progressive lower limb spasticity.
261 s in the corticospinal tract and progressive lower limb spasticity.
262 erative disease characterized by progressive lower-limb spasticity and weakness, as well as frequent
263 gical disorders characterized by progressive lower-limb spasticity and weakness.
264 xtension of spontaneous, acute, symptomatic, lower-limb superficial-vein thrombosis (SVT) is debated.
265 nesthesia and postoperative analgesia of the lower limb surgery.
266 muscle fibres decreased following unilateral lower limb suspension (ULLS) and returned to normal afte
267                                   Unilateral lower limb suspension (ULLS) has been suggested as an al
268 ects were subjected to 3 weeks of unilateral lower limb suspension (ULLS), a widely used human model
269 e hypothesis that, in response to unilateral lower limb suspension (ULLS), the magnitude of tendon me
270 od, which was followed by 4 wk of unilateral lower limb suspension.
271 ientated trabeculae were more obvious in the lower limb than the upper limb (59% versus 29%).
272           Nonmajor orthopedic surgery of the lower limbs that results in transient reduced mobility p
273                                              Lower limb thermal thresholds were at floor/ceiling in s
274 pansion of the area representing pain in the lower limb to include face and lip regions.
275 s well as how they are co-ordinated with the lower limbs to maintain balance.
276 ttenuate group III/IV afferent feedback from lower limbs to modify the EPR, while breathing either am
277 e if subjecting a distant organ, such as the lower limb, to a similar priming ischemic insult would r
278 (AIH) in remission, presented with bilateral lower limb ulcers 4 years after the diagnosis of AIH.
279 vein thrombosis, as detected on twice-weekly lower-limb ultrasonography after the third calendar day
280 c troponin I, brain natriuretic peptide, and lower limb ultrasound testing.
281 sitive pulmonary angiogram and four positive lower-limb US scans.
282 mptomatic pulmonary embolism associated with lower-limb vein thrombosis and at least 1 criterion for
283                          Angiogenesis in the lower limb was also increased and was associated with in
284 educed motor function in the right upper and lower limb was associated with lower gamma-aminobutyric
285 ermittent pneumatic compression (IPC) of the lower limbs was effective in reducing venous thromboembo
286 d trunk with more-modest amelioration in the lower limbs was observed in all patients.
287  complete compression ultrasonography of the lower limbs was performed after 8 +/- 2 days of antithro
288  a motor-neurological disorder manifested by lower limb weakness and spasticity and length-dependent
289 ch less common but a milder degree of distal lower limb weakness is often observed.
290 motoneuron disorder responsible for proximal lower limb weakness that subsequently expanded to the up
291           Patients show adult onset proximal lower limb weakness with highly raised serum creatine ki
292 wly growing VT in a patient with progressing lower limb weakness without any history or imaging findi
293 adult-onset myopathy characterized by distal lower limb weakness, calf hypertrophy and progressive de
294 ic distribution of deep venous reflux in the lower limb were determined across the clinical classes o
295  and vessel maturation (angiogenesis) in the lower limb were enhanced in TNFR1-KO but were reduced in
296 er incidence of inattention than those whose lower limbs were most affected.
297 ested in eight patients in a setup where the lower limbs were simulated using immersive virtual reali
298 as the first and most affected muscle in the lower limbs, whereas the triceps and interosseous were p
299           Mild atrophy in the left upper and lower limbs without pain, swelling, or skin lesions was
300 ssover study enrolled 42 patients undergoing lower limb x-ray angiography between February and June 2

 
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