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1 laborate network of airways from the initial lung bud.
2 5 in the primitive gut and in the developing lung bud.
3 , which gives rise to the future trachea and lung buds.
4 w local expression of Fgf10 and induction of lung buds.
5 eginning on E11 in a few cells of the distal lung buds.
6 itions poorly restore FGF10 signaling in the lung buds.
7  the epithelial cells in the tips of growing lung buds.
8 ng and growth after formation of the primary lung buds.
9 eficient foreguts show a capacity to undergo lung budding and early branching in the presence of RA o
10 shed well before formation of the definitive lung buds at E9-9.5.
11 Foxd1-expressing embryonic progenitors enter lung buds before 13.5 days post-conception, expand, and
12        They were smaller and had rudimentary lung bud branches, collapsed conducting airways, and loo
13 rsal-ventral patterning, lung specification, lung budding, branching morphogenesis, and, finally, mat
14 n Bmpr1a;b mutants does not suppress ectopic lung budding but does rescue trachea formation and NKX2-
15 e throughout the intestine and in the distal lung buds, correlating with tolerance to centriole loss.
16                                          The lung buds develop as an outgrowth of the foregut, which
17  NCC that leave the gut and migrate into the lung buds during early development.
18 and profound defect in lung development with lung buds failing to undergo branching morphogenesis and
19                    Here we report that early lung bud formation and subsequent branching morphogenesi
20 n the distal mesenchyme adjacent to sites of lung bud formation has long been thought to drive stereo
21 ignaling activation in the epithelium during lung bud formation, and that the effect of FGF10 in patt
22 fication from the foregut endoderm, prior to lung bud formation, are poorly understood.
23                                       As the lung buds grow out, proximal epithelial cells become fur
24 mediated lung specification and RA-regulated lung bud growth.
25 l NCC that migrate from the foregut into the lung buds; (ii) like ENS precursors, these NCC express t
26                  Furthermore, examination of lung bud in E9.5 Mgat1-/- mutant embryos showed complete
27 repressing Tgfbeta allowed induction of both lung buds in RA-deficient foreguts.
28 for the classically reported failure to form lung buds in vitamin A deficiency.
29 of RAR alpha and beta is critical for proper lung bud initiation and endodermal differentiation.
30 f Sox2; and second, it restricts the site of lung bud initiation.
31 te that Notch signaling is not necessary for lung bud initiation; however, Notch is required to maint
32                                    A primary lung bud is specified in the RA-deficient embryos, which
33 ion, we studied primary fibroblasts from the lung buds of wild-type (RB+/+) and null-mutant (RB-/-) m
34 t when other endodermal tissues, such as the lung bud or stomach, were implanted.
35 endoderm, ventral anterior foregut endoderm, lung bud organoids and finally lung organoids.
36 le of Pea3/Erm during the dynamic process of lung bud outgrowth and proximal-distal differentiation,
37 sal groove and in the first branchial pouch, lung buds, precartilagenous condensations, and mesenchym
38           However, in vitro organ culture of lung buds removed from 11.5-day-old LKLF(-/-) embryos sh
39 expressed in the foregut endoderm before the lung buds, T1alpha mRNA and protein levels increase subs