ライフサイエンスコーパス: lung epithelial cell

1 sed the presentation of HLA-B, -C, and -E on lung epithelial cells.

2 V-induced lysis of mammalian fibroblasts and lung epithelial cells.

3 ated in allergen-challenged human and murine lung epithelial cells.

4 of human lung carcinoma and CNT-transformed lung epithelial cells.

5 ar lavage fluid (BALF) and culture medium of lung epithelial cells.

6 hibits reactive oxygen species production in lung epithelial cells.

7 ls for normal and oncogene-transformed human lung epithelial cells.

8 ed to catalase, reversed oxidative stress in lung epithelial cells.

9 n enhanced IL-33-induced cytokine release in lung epithelial cells.

10 KRAS-induced transformation of immortalized lung epithelial cells.

11 d gain of function studies were performed in lung epithelial cells.

12 apoptosis or interferon expression in human lung epithelial cells.

13 th factor beta 1 (TGF-beta1) mediated EMT in lung epithelial cells.

14 cells, but not other NSCLC cells and normal lung epithelial cells.

15 sion and also attenuated mucus production in lung epithelial cells.

16 nt differentiation into predominantly distal lung epithelial cells.

17 attach to and invade both decidual cells and lung epithelial cells.

18 ycytidylic acid [poly(I:C)] in primary human lung epithelial cells.

19 ertain anti-inflammatory genes in human A549 lung epithelial cells.

20 induces IL-8/CXCL8 expression and release in lung epithelial cells.

21 ells, whereas there were no differences with lung epithelial cells.

22 none of the strains effectively invaded the lung epithelial cells.

23 antitative measurement of cytolysis of human lung epithelial cells.

24 ximo-distal differentiation pattern of human lung epithelial cells.

25 iverse staphylococci, to induce apoptosis of lung epithelial cells.

26 n, and gain- and loss-of-function studies in lung epithelial cells.

27 cherichia coli-induced IL-8 release in human lung epithelial cells.

28 in-driven feed-forward signaling loop in the lung epithelial cells.

29 adation, which increased cytokine release in lung epithelial cells.

30 gradation and increasing cytokine release in lung epithelial cells.

31 ssential and sufficient for proliferation of lung epithelial cells.

32 n the ESAT-6 induction of IL-8 expression in lung epithelial cells.

33 AT-6 on the regulation of IL-8 expression in lung epithelial cells.

34 and transient expression strategies in human lung epithelial cells.

35 pendium of gene expression profiles of human lung epithelial cells.

36 ishing lung adenocarcinoma cells from normal lung epithelial cells.

37 nduced Fn14 expression when expressed in rat lung epithelial cells.

38 a A viral infection on ER stress pathways in lung epithelial cells.

39 ulating an anti-viral state in human-derived lung epithelial cells.

40 on in APCs, but had no significant impact in lung epithelial cells.

41 otypic cultures leads to reduced cohesion of lung epithelial cells.

42 5 was selectively knocked out in fetal mouse lung epithelial cells.

43 promote the clonal expansion of premalignant lung epithelial cells.

44 of unrepaired DNA lesions in differentiated lung epithelial cells.

45 ich triggers detachment-induced apoptosis in lung epithelial cells.

46 target gene of Smad1 in the developing mouse lung epithelial cells.

47 contain higher PIC levels than untransformed lung epithelial cells.

48 ssion of ligands for inhibitory receptors on lung epithelial cells.

49 es and live influenza A virus (IAV) in human lung epithelial cells.

50 stream effectors in both BEAS-2B and primary lung epithelial cells.

51 nd an inflammatory response in monocytes and lung epithelial cells.

52 tivities of cGAS, STING, and PYHINs in human lung epithelial cells.

53 asing the number of Epcam(+) Sca-1(+) distal lung epithelial cells.

54 terized by the loss and aberrant function of lung epithelial cells.

55 phils, and preventing bacterial adherence to lung epithelial cells.

56 rnalization of Staphylococcus aureus by A549 lung epithelial cells.

57 the micro RNA (miRNA) transcriptome in human lung epithelial cells.

58 p open and active chromatin respectively, in lung epithelial cells.

59 -8 and induces apoptotic cell death in human lung epithelial cells.

60 lammation via induced expression of IL-33 by lung epithelial cells.

61 reduced stretch-induced CD147 expression on lung epithelial cells.

62 A expression and protein secretion from A549 lung epithelial cells.

63 cytosis, transformation and tumorigenesis of lung epithelial cells.

64 ng adenocarcinomas when compared with normal lung epithelial cells.

65 Lung epithelial cells (16-HBE, BEAS-2B) and monocytes (U

66 oduction of reactive oxygen species (ROS) in lung epithelial cells (A549 and primary normal human bro

67 ion of diesel exhaust aerosol (DEA) on human lung epithelial cells (A549) in a prototype exposure cha

68 Here, lung epithelial cells (A549) residing at the air-liquid

69 ne, in mouse lung tissue as well as in human lung epithelial cells (A549).

70 ombin-induced IL-8/CXCL8 expression in human lung epithelial cells (A549).

71 ibution of oxide nanoparticles inside living lung epithelial cells (A549).

72 in patients' BALF and in EVs of IAV-infected lung epithelial cells (A549).

73 In ex vivo assays, lung epithelial cells acquire a cancer-associated parenc

74 In addition, transfection of ORMDL3 in lung epithelial cells activated ATF6alpha and induced SE

75 MiR-150 conferred a cytoprotective role in lung epithelial cells after oxidative stress, whereas FO

76 uman MG53 (rhMG53) protein protects cultured lung epithelial cells against anoxia/reoxygenation-induc

77 tions of CuO NPs induced IL-8 release in the lung epithelial cells already at subtoxic concentrations

78 erent species (rat and human), two different lung epithelial cells (alveolar type II and bronchial ep

79 affects the expression of selected genes in lung epithelial cells and airway tissue.

80 Intercellular communications between lung epithelial cells and alveolar macrophages play an e

81 lly, miR-155 was also highly expressed in CF lung epithelial cells and circulating CF neutrophils bio

82 into the interaction of this virus with host lung epithelial cells and endothelial cells, which resul

83 ral effects of acetate involving IFN-beta in lung epithelial cells and engagement of GPR43 and IFNAR.

84 ate PcCbk1 after the organism interacts with lung epithelial cells and extracellular matrix component

85 stic fungal pathogen Pneumocystis carinii to lung epithelial cells and extracellular matrix proteins

86 rved both in adherence and invasion of human lung epithelial cells and in cytotoxicity of these cells

87 We examined EMT-competent lung epithelial cells and lung fibroblasts from control

88 on of interleukin 8 by wood smoke extract in lung epithelial cells and lung inflammation induced by w

89 tory protein 2 (an interleukin-8 homolog) in lung epithelial cells and lungs and lung inflammation, a

90 ide levels, remarkably, higher expression in lung epithelial cells and macrophages in vitro and in vi

91 important opportunistic fungal pathogen with lung epithelial cells and matrix.

92 that it enhances viral replication in human lung epithelial cells and primary human airway tissues b

93 duced in breast cancer cells by signals from lung epithelial cells and promotes fibronectin fibril fo

94 n of the two-pore-domain K channel TREK-1 in lung epithelial cells and proposed a role for this chann

95 ne transfer, MD-2s was strongly expressed in lung epithelial cells and readily detected in bronchoalv

96 te vapor aberrantly alters the physiology of lung epithelial cells and resident immune cells and prom

97 nhibits USA-WA1/2020 SARS-CoV-2 infection in lung epithelial cells and viral entry in human lung orga

98 ted upon S. pneumoniae infection of cultured lung epithelial cells and was critical for AA release fr

99 killing was inducible directly from isolated lung epithelial cells and was not abrogated by the prese

100 that phospho-Akt levels were elevated in CF lung epithelial cells and were specifically lowered by e

101 TC-conjugated dextran through a monolayer of lung epithelial cells, and assessment of bacterial outgr

102 VAMP8 mRNA was present in human airway and lung epithelial cells, and deep-sequencing and expressio

103 sults for primary human keratinocytes, human lung epithelial cells, and murine macrophages.

104 DC-SIGN (SIGN-R1) is present on mouse lung epithelial cells, and SAP and the aminothiazole pot

105 romotes malignant transformation by limiting lung epithelial cell apoptosis and promoting immune esca

106 Here we present evidence that lung epithelial cells are connected by TNTs, and in spit

107 Lung epithelial cells are considered important sources o

108 nesis is a multistep process in which normal lung epithelial cells are converted to cancer cells thro

109 e factor of S. aureus clinical isolates, and lung epithelial cells are highly sensitive to alpha-toxi

110 ed that the majority of EHF binding sites in lung epithelial cells are intergenic or intronic and coi

111 ivator or a repressor, though its targets in lung epithelial cells are largely uncharacterized.

112 PH-dependent redox cycling activity in mouse lung epithelial cells as sepiapterin reductase (SPR), an

113 induced early apoptosis (P < 0.05, n = 6) in lung epithelial cells, as measured by annexin/propidium

114 croscopy (FM) investigation of exocytosis in lung epithelial cells (ATII cells) allows the detection

115 cal stress on lung fibrotic formation, human lung epithelial cells (BEAS-2B) were exposed to mechanic

116 Human lung epithelial cells (BEAS-2B) were used to study the e

117 tional deletion of beclin 1 (Becn1) in mouse lung epithelial cells (Becn1Epi-KO), either at early (E1

118 iously demonstrated that upregulation of the lung epithelial cell beta6 integrin during influenza vir

119 Although the ST-23 strain attached to lung epithelial cells better than ST-17 and -19 strains,

120 In cultured lung epithelial cells, BMP-4 induced VEGF expression thr

121 increase in interleukin 8 release from A549 lung epithelial cells but decreased interleukin 1beta, i

122 nstrated similar levels of adherence to A549 lung epithelial cells, but the mutant strains were defec

123 ect evidence of telomerase activity in adult lung epithelial cells, but typical culture conditions on

124 the proinflammatory expression of IL-8 in CF lung epithelial cells by lowering SHIP1 expression and t

125 2 expression was induced in rodent and human lung epithelial cells by Th2 cytokines, which was mediat

126 ell, Liu et al. show that TLR3 activation in lung epithelial cells by tumor exosomal RNAs triggers ne

127 Mice in which lung epithelial cells can be induced to express an oncog

128 Lung epithelial cells can influence immune responses to

129 ne genetics to determine how the identity of lung epithelial cells changes upon loss of their master

130 , to the neoplastic-like properties of human lung epithelial cells chronically exposed to a low-dose

131 enetration than control, improve uptake into lung epithelial cells compared to uncoated or PEGylated-

132 findings suggest that a high viral load from lung epithelial cells coupled with induction of host res

133 Lung epithelial cell damage and dysfunctional repair pla

134 ction of proinflammatory cytokines, and less lung-epithelial cell damage.

135 by SARS-CoV-2-activated neutrophils promote lung epithelial cell death in vitro.

136 demonstrated that autophagy proteins promote lung epithelial cell death, airway dysfunction, and emph

137 FOXD3-AS1 promoted oxidative stress-induced lung epithelial cell death.

138 h and thereby exaggerating hyperoxia-induced lung epithelial cell death.

139 been shown to stimulate immune responses in lung epithelial cells, dendritic cells, and alveolar mac

140 We found that lung epithelial cell-derived collagen I activates fibrob

141 These data demonstrate that, within lung epithelial cells, different FGFRs function independ

142 in mesenchyme perturbed trachea development, lung epithelial cell differentiation and lung growth.

143 In human COPD, lung epithelial cells displayed increased expression of

144 than circumference as a large proportion of lung epithelial cells divide parallel to the airway long

145 These findings suggest that cyclic MS of lung epithelial cells down-regulates LPS-mediated inflam

146 scription factor that is highly expressed by lung epithelial cells during development and has been sh

147 that beta-catenin signaling is activated in lung epithelial cells during neutrophil transmigration,

148 on of the ferric or ferryl HbS with cultured lung epithelial cells (E10) induced a drop in mitochondr

149 e Ser/Thr and Tyr phosphoproteomes of murine lung epithelial cells early and late after infection wit

150 HDM and the innate immune response driven by lung epithelial cells (ECs) and dendritic cells (DCs) th

151 In A549 lung epithelial cells, ectopically expressed LtpD locali

152 ltured lung adenocarcinoma but not in normal lung epithelial cells elevated H2S stimulates mitochondr

153 Polarized lung epithelial cells express IL-17RA and IL-17RC basola

154 TGF-beta1 secretion is not observed in lung epithelial cells expressing the common SP-A2 varian

155 or potential ankyrin-1 (TRPA1) expression by lung epithelial cells expressing the TRPV1-I585V variant

156 Rat lung epithelial cells expressing v-K-ras or murine lung

157 Apc deficiency altered lung epithelial cell fate by inhibiting Clara and ciliat

158 nterest, expression of Sonic Hh increased in lung epithelial cells following the induction of allergi

159 tes and macrophages (LysM/STAT1) or ciliated lung epithelial cells (FoxJ1/STAT1) deleted were created

160 We show that IFNalpha protects human lung epithelial cells from alpha-toxin-induced intracell

161 so protects Caenorhabditis elegans and human lung epithelial cells from killing by P. aeruginosa.

162 d enriched LTbetaR target gene expression in lung epithelial cells from patients with smoking-associa

163 We determined that miR-31 regulates lung epithelial cell growth and identified 6 negative re

164 In human lung epithelial cells, GSK3008348 induces rapid internal

165 Additionally, VHR-knockout fibroblast and lung epithelial cells had elevated ligand-induced epider

166 st, transgenic mice that overexpress Grx1 in lung epithelial cells had significantly higher lung bact

167 Injury to lung epithelial cells has a role in multiple lung diseas

168 WT and usher deletion strains to A549 human lung epithelial cells, HEp-2 human cervical epithelial c

169 cluster is critical for lung development and lung epithelial cell homeostasis and is predicted to tar

170 s essential for buffering Nkx2.1 expression, lung epithelial cell identity, and tissue homeostasis.

171 us of a novel regulatory network, connecting lung epithelial cell identity, migration, and cell-cell

172 Abundantly expressed in lung epithelial cells, IL-33 plays critical roles in bot

173 tribution of cytokines generated by stressed lung epithelial cells (IL25, IL33, TSLP) to DEP-induced

174 o-inflammatory cytokines and enzymes in A549 lung epithelial cells in a manner devoid of S1P agonism.

175 of the ubiquitin ligase Nedd4-2 (Nedd4l) in lung epithelial cells in adult mice produces chronic lun

176 55 was more than 5-fold elevated in CF IB3-1 lung epithelial cells in culture, compared with control

177 , and pathogen killing could be induced from lung epithelial cells in isolation.

178 zo(a)pyrene (BaP) induce PD-L1 expression on lung epithelial cells in vitro and in vivo, which is med

179 ntified OMVs, which were then used to infect lung epithelial cells in vitro as well as C57BL/6J mice

180 Six1 overexpression in MLE15 lung epithelial cells in vitro inhibited cell differenti

181 In A549 lung epithelial cells in vitro we show that inhibition o

182 improved viability, and protection of human lung epithelial cells in vitro.

183 Loss of Pten in the lung epithelial cells in vivo activated adipokine signal

184 most efficient agent for the transfection of lung epithelial cells in vivo following intra-nasal admi

185 densation and functional delivery of pDNA to lung epithelial cells in vivo.

186 of human lung and skin fibroblasts, but not lung epithelial cells, in vitro and is present in sclero

187 r level of apoptosis upon infection of human lung epithelial cells, indicating that a T4SS effector(s

188 ng to host extracellular matrix proteins and lung epithelial cells induced the phosphorylation (activ

189 nflammation by causing barrier disruption in lung epithelial cells inducing the elevation of interleu

190 s and activated oncogenic signaling in AT II lung epithelial cells, inducing emphysema and adenocarci

191 vivo were compared with those made by human lung epithelial cells infected in vitro with RV16.

192 m on host expression networks by using human lung epithelial cells infected with either wild-type vir

193 Both transformed and primary human lung epithelial cells infected with P. aeruginosa genera

194 Compared with wild-type primary lung epithelial cells, infection of Glrx1(-/-) cells wit

195 In conclusion, lung epithelial cells influence the efficacy of most ant

196 siRNA knockdown of ATF-6alpha in lung epithelial cells inhibited expression of SERCA2b, w

197 itional deletion of either TBK1 or TBKBP1 in lung epithelial cells inhibits tumourigenesis in a mouse

198 Lung epithelial cells interconnected by tight junctions

199 rleukin-8 induction by wood smoke extract in lung epithelial cells is mediated by novel NADPH oxidase

200 of unrepaired DNA lesions in differentiated lung epithelial cells is unclear.

201 ction of TGFbeta1-mim was evaluated in human lung epithelial cells, Jurkat cells, and rat basophilic

202 In LPS-stimulated A549 human lung epithelial cells, kallistatin attenuated apoptosis,

203 nstrate that water transport across NCI-H441 lung epithelial cell layers and apical surface liquid (A

204 Expression of mutant SP-A2 in lung epithelial cells leads to secretion of latent TGF-b

205 To determine whether IKK2-dependent lung epithelial cell (LEC) responses contribute to the a

206 tides transiently disrupted TJs in the human lung epithelial cell line 16HBE and delayed TJ formation

207 angement, detachment, and death in the human lung epithelial cell line A549.

208 A human normal lung epithelial cell line and four lung cancer cell line

209 ng and detachment of cells of the A549 human lung epithelial cell line as well as the Xps-mediated de

210 In the human lung epithelial cell line BEAS-2B, LL-37, but not mCRAMP

211 Human postmortem lung tissue and human lung epithelial cell line BEAS-2B.

212 ormed with unlabeled BMP on the human distal lung epithelial cell line NCI-H441.

213 e shown to be cytocompatible with A549 human lung epithelial cell line.

214 detachment, and death of A549 cells, a human lung epithelial cell line.

215 A similar result was obtained with infected lung epithelial cell lines and the lungs of infected A/J

216 Reduction of Grhl2 in lung epithelial cell lines results in lower expression l

217 synthesizing hepoxilin A(3) are expressed in lung epithelial cell lines, primary small airway epithel

218 nt transforming growth factor (TGF)-beta1 in lung epithelial cell lines.

219 genetic inactivation of KRas(G12D) in mouse lung epithelial cells markedly impairs the progression o

220 w of fetal susceptibility for BPA effects on lung epithelial cell maturation in the third trimester.

221 cted lungs, suggesting that association with lung epithelial cells may provide an advantage to spore

222 ism by which bacterial factors modulate host lung epithelial cell mitochondrial function and to inves

223 Mouse lung epithelial cells (MLE12) transfected with human Mmp

224 Mouse lung epithelial cells (MLE12) with small interfering RNA

225 asing the I-kappaB degradation rate in mouse lung epithelial cells (MLE12).

226 ic surfaces, using a three-dimensional (3-D) lung epithelial cell model.

227 xperimental wound healing in human and mouse lung epithelial cell monolayers and stimulated epithelia

228 tely reduced intracellular VZV yield in A549 lung epithelial cells, MRC-5 lung fibroblasts, and ARPE-

229 hLAL expression in lung epithelial cells not only reduced tumor-promoting m

230 1-encoding gene (Becn1) specifically within lung epithelial cells of embryonic mice resulted in neon

231 l line (BEAS-2B) and specifically deleted in lung epithelial cells of mice (Stard7(epi/)).

232 To assess how LAL in lung epithelial cells plays a role in this inflammation-

233 del it is identified that 24.6% of the total lung epithelial cell population is transfected after a s

234 Using lung epithelial cells, primary human monocyte-derived ma

235 Lung epithelial cells produce increased hepoxilin A(3) a

236 ter Ad-CMVcre infection in a wide variety of lung epithelial cells produces LCNEC.

237 Factors that affect lung epithelial cell proliferation and activation may be

238 Increases in lung epithelial cell proliferation and bronchoalveolar l

239 d sacculation, accompanied by altered distal lung epithelial cell proliferation and differentiation a

240 Lamin B1 silencing in lung epithelial cells promoted epithelial-mesenchymal tr

241 RNA knockdown of Suv39H1 or Suv39H2 in fetal lung epithelial cells repressed H3K9 methylation and gre

242 Knockdown of Nur77 in human lung epithelial cells resulted in a marked increase in I

243 Knock-down of FHL2 in human lung epithelial cells resulted in a striking decrease in

244 the CA/09-483HA virus through primary human lung epithelial cells resulted in increased pathogenicit

245 Knockdown of PINK1 expression in lung epithelial cells resulted in mitochondria depolariz

246 KLF6 gene silencing in human lung epithelial cells resulted in the drastic loss of NO

247 reported that IL-8 mRNA is stabilized in CF lung epithelial cells, resulting in concomitant hyperexp

248 We show that distinct lung epithelial cells secrete acidic mammalian chitinase

249 In the lung, epithelial cells serve as the initial barrier to a

250 Six1(-/-) lung epithelial cells show increased expression of diffe

251 Normal lung epithelial cells show increased rates of migration

252 In vitro studies in lung epithelial cells showed that p52 overexpression red

253 ofound effects upon gene expression in human lung epithelial cells, some of which are epigenetically

254 se and insulin tolerance tests revealed that lung epithelial cell-specific IKK2 deficiency resulted i

255 [Formula: see text] (lung epithelial cell-specific IKK2 knockout, KO) and [Fo

256 In comparison with tgWT mice, lung epithelial cell-specific IKK2-deficient mice had fe

257 Lung epithelial cell-specific IKK2-deficient mice had si

258 In the present study, we exploited a lung epithelial cell-specific inhibitor [Formula: see te

259 mitochondrial CYP1B1 was assessed using A549 lung epithelial cells stably expressing shRNA against NA

260 lly, deletion of Smo in Rb1 and Trp53-mutant lung epithelial cells strongly suppressed SCLC initiatio

261 the protective effect of ACSL4 knockdown on lung epithelial cells subjected to hypoxia/reoxygenation

262 that induction of ER stress leads to EMT in lung epithelial cells, suggesting possible cross-talk be

263 In human fetal lung epithelial cells, Suv39H1 and Suv39H2 mRNA levels d

264 nels expressed by airway sensory neurons and lung epithelial cells that may also be activated by CFA1

265 t that LAL is a critical metabolic enzyme in lung epithelial cells that regulates lung homeostasis, i

266 In human lung epithelial cells, the microRNA-150 (miR-150) was id

267 We have previously reported that in mouse lung epithelial cells, the pro-EGF ligands TGFalpha, neu

268 inflammation and canonical Wnt signaling in lung epithelial cells, thereby restricting lung tumorige

269 ernalization with a H2-T6SS mutant) and into lung epithelial cells through a phosphatidylinositol 3-k

270 y the ubiquitin-proteasome pathway in murine lung epithelial cells through lysine 183 as an acceptor

271 duces bacterial adhesion and uptake by human lung epithelial cells, thus protecting M. catarrhalis fr

272 stent Nkx2.1 deficiency and reprogramming of lung epithelial cells to a posterior endoderm fate.

273 LIP gene in IPF lungs may predispose injured lung epithelial cells to apoptosis and to the developmen

274 sults demonstrate that tobacco smoke enables lung epithelial cells to escape from adaptive immunity t

275 Exposure of human lung epithelial cells to H2O2 induces apoptosis concomit

276 human induced pluripotent stem cell-derived lung epithelial cells to model early-stage lung adenocar

277 t to investigate the responsiveness of human lung epithelial cells to the TNF family molecules LIGHT

278 AV) 6 capsid that demonstrates efficiency in lung epithelial cell transduction based on imaging and f

279 A549 lung epithelial cells transfected with plasmid encoding

280 PP2A was silenced in lung epithelial cells treated with A1AT and matrix metal

281 review, we focus on the regionally distinct lung epithelial cell types that contribute to repair aft

282 xpression of full-length IL-33 is induced in lung epithelial cells under conditional control.

283 nsing (ECIS) platform to monitor behavior of lung epithelial cells upon exposure to a library of SWCN

284 In accordance, RV16 infection of human lung epithelial cells upregulated TSLP and IL-33 express

285 m miR-200b targets were studied in bronchial lung epithelial cells using a SMAD luciferase assay, Wes

286 ression of the leptin/leptin receptor in the lung epithelial cells via activation of MEK/ERK, PI3K/AK

287 independent induction of oxidative stress in lung epithelial cells via the release of bacterial facto

288 an lung cancer, is restrictedly expressed in lung epithelial cells [via Clara cell secretory protein

289 mulator of type II innate lymphoid cells, in lung epithelial cells was associated with suppression of

290 Hypoxia-inducible factor-1alpha in lung epithelial cells was shown to regulate interleukin-

291 tand the interactions between MSC and distal lung epithelial cells, we added MSC to lung progenitor 3

292 By using human breast and lung epithelial cells, we have further characterized Del

293 vivo in transgenic mice and human PTEN-null lung epithelial cells, we identify the leptin-driven fee

294 analyte, air-liquid interface cultured A549 lung epithelial cells were exposed to critical concentra

295 Although lung epithelial cells were initially merely regarded as

296 Lung epithelial cells were the main source of CXCL5 upon

297 Rat lung epithelial cells were treated with 52 stimuli, and

298 ver, its expression transformed immortalized lung epithelial cells while a transgenic model featuring

299 Here, we show that infection of human lung epithelial cells with influenza A virus (IAV) also

300 tion induces the early expression of TSLP by lung epithelial cells with multiple consequences.