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1 s that are required for the development of a lupus-like syndrome.
2 reat Graves' disease, occasionally induces a lupus-like syndrome.
3 f a stimulatory graft-vs-host disease with a lupus-like syndrome.
4 ular disease, thrombocytopenia, and systemic lupus-like syndrome.
6 s the first evidence that the induction of a lupus-like syndrome by pristane is not restricted to BAL
8 ainamide and numerous other medications is a lupus-like syndrome characterized by autoantibodies dire
9 as (lpr) or Fas ligand (gld) cause a limited lupus-like syndrome in B6 mice by interfering with the d
10 s, in double-knock-out (DKO) mice leads to a lupus-like syndrome in females marked by accumulation of
12 the development of anti-dsDNA antibodies and lupus-like syndrome in patients undergoing anti-TNF ther
14 function might contribute to the etiology of lupus-like syndromes in the context of an autoimmune-pro
15 nti-nRNP/Sm (+) sera from BALB/c mice with a lupus-like syndrome induced by pristane recognized Ul-C.
16 tment of NZB/WF1 mice, which develop a fatal lupus-like syndrome, inhibited NF-kappaB2 processing and
18 raperitoneal injection of pristane induces a lupus-like syndrome whose pathogenesis implicates the se
19 subjects with trisomy of chromosome 9p had a lupus-like syndrome with joint manifestations and antinu