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1 its precursors) were associated with shorter luteal phase.
2 ity (P = 0.09) during menses than during the luteal phase.
3 id-follicular phase to the symptomatic, late luteal phase.
4 ncreased from the mid-follicular to the late luteal phase.
5 ic environment during the follicular vs. the luteal phase.
6 cination in the follicular phase but not the luteal phase.
7 the development of mood symptoms during the luteal phase.
8 vels as follicular cells transition into the luteal phase.
9 domised order: late follicular phase and mid-luteal phase.
10 r for half of the menstrual cycle during the luteal phase.
11 teins (P = 5.62E-4) were elevated during the luteal phase.
12 e of 18 proteins that best distinguished the luteal phase.
13 n </= 5 ng/mL and no LH peak in the mid/late luteal phase.
14 wer endometrial expression of FST during the luteal phase.
15 rgery during the follicular phase versus the luteal phase.
16 eft fronto-polar cortex more than during the luteal phase.
17 ating hormone and luteinizing hormone in the luteal phase.
18 d a 0.06-log(PdG) decrease (p = 0.03) in the luteal phase.
19 ficantly associated with length of the prior luteal phase.
20 icular phase and then decreased again in the luteal phase.
21 ls the entire night in the luteal and pseudo luteal phase.
22 se, and 0.76 +/- 0.07 kJ/min during the late luteal phase.
23 -cycle compared with both the follicular and luteal phases.
24 een the early follicular, ovulatory, and mid-luteal phases.
25 ed twice, during the late-follicular and mid-luteal phases.
26 in females (n = 12) in their follicular and luteal phases.
27 E1 were determined during the follicular and luteal phases.
28 follicular phase to the mid luteal and late luteal phases.
29 levels during the follicular, ovulatory, and luteal phases.
30 easured in the early and late follicular and luteal phases.
31 sion showed that decreases in follicular and luteal phase 17beta-estradiol levels were positively ass
32 hthalate (MCOP) were associated with shorter luteal phase [2nd tertile vs. 1st tertile: -0.5 days (95
34 men comprised the study cohort: 230 (28%) in luteal phase; 363 (44%) in follicular phase; and 241 gro
35 ase (59 [17]) compared with women during the luteal phase (53 [14]) and compared with men (46 [16]; P
36 tal cortex and amygdala more than during the luteal phase (6-10 days after luteinizing hormone surge)
38 t driven by the follicular compared with the luteal phase and directly related to craving and fluctua
40 transformation of the endometrium during the luteal phase and evaluate markers of endometrial recepti
41 ition of uILCs in the endometrium during the luteal phase and in the decidua during early pregnancy.
42 onth, once during their active pill phase or luteal phase and once during their pill pause or menses.
43 traception could mimic the high-progesterone luteal phase and predispose women to human immunodeficie
44 cular phase, 0.70 +/- 0.10 kJ/min during the luteal phase, and 0.76 +/- 0.07 kJ/min during the late l
45 e onset of melatonin levels for women in the luteal phase, but it had little effect on melatonin leve
48 poradic anovulation, irregular cycle length, luteal phase deficiency, long menses, and heavy blood lo
49 e the estradiol during the follicular versus luteal phase (Delta), the higher the Deltadrug cue react
50 mones measured either midcycle or during the luteal phase, despite good statistical power to detect m
52 e anxiety and dysphoria associated with late luteal phase dysphoria disorder and major unipolar depre
53 trajectory showed that its left shift in the luteal phase (e.g., earlier rise in progesterone) expose
54 lection of gene expression profiles from mid-luteal phase endometrial biopsies (n = 115) from women e
55 o [OR] 1.11, 95% CI 1.03-1.20; p=0.0063) and luteal phase endometrial thickness lower (0.90, 0.83-0.9
58 5-1988) of a prospective study, midcycle and luteal-phase estrogens and progestins were measured in 1
64 articular susceptibility observed during the luteal phase in nonhuman primate models and ex vivo huma
65 ereas drug reappraisal was higher during the luteal phase in the anterior PFC/orbitofrontal cortex.
66 arker to distinguish the follicular from the luteal phase in univariate and multivariate analyses and
67 th, 16.0 (standard deviation, 4.4) days; and luteal phase length, 12.9 (standard deviation, 1.7) days
69 and increased follicular phase and decreased luteal phase lengths; Hispanic ethnicity with anovulatio
70 sing linear mixed models for follicular- and luteal-phase lengths, discrete-time fecundability models
71 ese chemicals in relation to follicular- and luteal-phase lengths, time to pregnancy, and early pregn
73 any of the following cycle endpoints: short luteal phase (< or = 10 days), long follicular phase (>
74 to an increased risk for anovulation, short luteal phase (< or =10 days), long follicular phase (> o
75 ion of Oocyte Retrieval performed during the Luteal Phase (LuPOR) in poor responders, as defined by t
80 l magnetic resonance imaging during the late luteal phase of fifty-one women diagnosed with PMDD, com
82 ons comparable to levels observed during the luteal phase of premenopausal women and were significant
84 a1-induced COL1 protein abundance in the mid-luteal phase of the estrous cycle after 48 h (p < 0.05).
85 ats mimics the progesterone component of the luteal phase of the human menstrual cycle, these finding
86 ature levels in women were tested during the luteal phase of the menstrual cycle (n=30) or the pseudo
87 omen using no long-term contraceptive in the luteal phase of the menstrual cycle also had a 3.25 time
88 discrimination of facial emotions during the luteal phase of the menstrual cycle and altered reactivi
89 h levels comparable to those observed in the luteal phase of the menstrual cycle and modestly increas
91 egnanolone levels from the follicular to the luteal phase of the menstrual cycle by blocking the conv
92 s were significantly (P=0.0078) lower in the luteal phase of the menstrual cycle compared to the foll
93 Changes in neurosteroid levels during the luteal phase of the menstrual cycle may precipitate affe
94 onadotropin secretion was blocked during the luteal phase of the menstrual cycle with a gonadotropin-
95 istered to female rhesus macaques during the luteal phase of the menstrual cycle, 40 min before admin
96 T cell populations were detected during the luteal phase of the menstrual cycle, and longitudinal an
98 often occur during pregnancy and during the luteal phase of the menstrual cycle, when levels of prog
109 rovided) were measured in the follicular and luteal phases of 2 menstrual cycles before a single inje
110 of the first menstrual cycle and during the luteal phases of both the first and third menstrual cycl
113 l females, and females in the follicular and luteal phases of the menstrual cycle (FDR-adjusted p-val
115 days apart to distinguish the follicular and luteal phases of the menstrual cycle, and phases were co
116 e glucuronide (E1G) in the periovulatory and luteal phases of the menstrual cycle, and to assess the
119 ding with the follicular, periovulatory, and luteal phases of their menstrual cycle were studied.
125 s increased sixfold to eightfold in the late luteal phase (P < 0.001) and those of swelling or bloati
127 re higher in the late follicular than in the luteal phase (P = 0.02 and P = 0.04, respectively).
128 re higher in the late follicular than in the luteal phase (P = 0.03 and P = 0.02, respectively).
129 nsive pregnancies were tested during the mid-luteal phase (PRE) and early pregnancy (EARLY; 6.2 +/- 1
130 romedial prefrontal cortex and, in the early luteal phase, reduced central and corticomedial amygdala
131 previously, we showed more inhibition in the luteal phase relative to the midfollicular menstrual pha
132 rinking probability and odds ratios vs. late luteal phase, respectively: 17%, odds ratio=1.340, 95% C
134 nd increases in progesterone associated with luteal phases, resulting in safe and potentially lifetim
137 inary sodium loss, not retention, during the luteal phase; severity of menstrual symptoms was unchang
140 ed in women with PMDD from follicular to the luteal phases, suggesting the absence of effect of the l
142 Despite the proven superiority of various luteal phase support protocols (LPS) over placebo in vie
144 s used to optimize pregnancy rates; however, luteal phase support remains largely 'black-box' with in
146 Consequently, hormonal supplementation with luteal phase support, principally exogenous progesterone
148 t is greater during the early follicular and luteal phases than in the late follicular (periovulatory
149 ocytes during IVF results in a dysfunctional luteal phase that can be insufficient to support implant
152 re collected, characterized as follicular or luteal phase using days since last menstrual period, and
153 evel, in the default mode network during the luteal phase when passively viewing negative emotional s
154 ng postovulation (average of luteal and late luteal phases), when it was 0.73 +/- 0.07 kJ/min, compar
155 antly higher than those measured in the late luteal phase, whereas aging and cigarette smoking reduce
156 mmunocytochemically detectable GAL-R1 in the luteal phase, whereas only a twentieth expressed GAL-R1
157 offer candidate mechanisms through which the luteal phase, wherein progesterone is dominant relative
158 Specifically, we outline the physiological luteal phase, which is regulated by progesterone from th
159 modeling and leukocyte infiltration with the luteal phase, which may represent potential hormone-asso