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2 mbined systematic mutagenesis studies at the luteinizing hormone receptor and the thyroid-stimulating
3 utant can inhibit signaling to G(s) from the luteinizing hormone receptor by 97% and from the calcito
5 s in the HinRs and the interhelical loops of luteinizing hormone receptor/follicle stimulating hormon
6 R is suggested by the common position of the luteinizing hormone receptor Form D alternate acceptor s
8 t study investigated regulation of the human luteinizing hormone receptor (hLHR) gene by histone deac
9 d within the TATA-less promoter of the human luteinizing hormone receptor (hLHR), was identified as a
13 n hormones to their cognate human receptors (luteinizing hormone receptor (LHR) and thyroid-stimulati
15 n A (TSA), induces derepression of the human luteinizing hormone receptor (LHR) gene by de-recruitmen
17 ously demonstrated that transcription of the luteinizing hormone receptor (LHR) gene is subject to re
19 the past decade, however, the expression of luteinizing hormone receptor (LHR) has also been reporte
23 ollicular differentiation markers, including luteinizing-hormone receptor (LHR), inhibin-alpha, micro
24 PD-PALM of two functionally defined mutant luteinizing hormone receptors (LHRs), a ligand-binding d
25 as well as human chorionic gonadotropin and luteinizing hormone receptors on male breast tissue, may
27 erize with its closely related receptor, the luteinizing hormone receptor; this association may have
28 ressin, and the catecholamine isoproterenol (luteinizing hormone receptor, type 2 vasopressin recepto