戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 that the resulting phasic contractions drive lymph flow.
2 cessary for the generation and regulation of lymph flow.
3 hese hypertrophic lymphatic sinuses increase lymph flow.
4  are initiated primarily in the direction of lymph flow.
5 ining CLNs was used as a measure of afferent lymph flow.
6 geries while preserving lymph node blood and lymph flow.
7 ction between the contracting vessel and the lymph flow.
8 n, and the consequences of the inadequacy of lymph flow.
9  a physiological stimulus caused by blood or lymph flow.
10 ay address excess, inadequate, or obstructed lymph flow.
11 c endothelial cells and increased mesenteric lymph flow.
12 s within the sinus, preventing their loss in lymph flow.
13                  To do this we measured lung lymph flow after raising left atrial pressure (by inflat
14                We hypothesize that increased lymph flow along with disrupted lymphatics in the affect
15 oparticle adjuvant termed SMNP, which alters lymph flow and antigen entry into lymph nodes.
16 lycerides (LCTs), which stimulate mesenteric lymph flow and are absorbed in chylomicrons through mese
17  accompanied by a threefold increase in lung lymph flow and dramatic increases in plasma and lung lym
18                                              Lymph flow and hemodynamics were unaffected by this hype
19 combined in sheep at rest, and the resulting lymph flow and protein content were the same as seen wit
20 sure, net fluid balance, lung and prefemoral lymph flow and protein content, lung water content, abdo
21 sive capillary hypertension, the increase in lymph flow and reduction in lymph protein content develo
22 mphovenous hemostasis is required for normal lymph flow, and mice deficient in lymphovenous hemostasi
23 ntal vascular and intraperitoneal pressures, lymph flow, and peritoneal space compliance.
24 ressures, cardiac output by ultrasound, lung lymph flow, and ventilation.
25 d in vivo to chronically increased pulmonary lymph flow are hyperproliferative, have enhanced express
26 luminal leaflet valves that function to bias lymph flow back towards the heart.
27         Pulmonary and cardiac function, lung lymph flow, bronchial obstruction score, and wet/dry lun
28 ation during dextran infusion increased lung lymph flow by a factor of 2.4 +/- 0.4, compared with a f
29 otein concentrations by 30%, and raised lung lymph flows by 55%.
30                      The natural patterns of lymph flow can also generate spatial gradients of antige
31 in Kf, leaving open the question of how lung lymph flow can rise in protein depletion with little cha
32 e (Kf) is calculated as the quotient of lung lymph flow divided by net filtration pressure (Pnf), whe
33 atic contractile function and, consequently, lymph flow has been the subject of intense study.
34   Finally, we review how efforts to increase lymph flow have demonstrated potential as a viable thera
35                                              Lymph flow imaging revealed that nanoparticle transit wa
36 phatic network displaying a dynamic range of lymph flow in physiology.
37 ur of fasting and during the lipid infusion, lymph flow in the Nephrotic group averaged 0.6 mL/h high
38 d backfills the lymphatic network and blocks lymph flow in these animals.
39                    Unexpectedly, the loss of lymph flow in these mice causes defects in maturation of
40 exposed to prolonged pathologically elevated lymph flow in vivo within the anatomic and physiologic c
41     During strenuous exercise in sheep, lung lymph flow increases within seconds and rises to levels
42                    These studies reveal that lymph flow initiates and regulates many of the key steps
43                                    Efficient lymph flow is ensured by lymphatic valves (LVs).
44                Sweet et al. demonstrate that lymph flow is essential for the remodeling of primary ly
45 scle cells entirely, suggesting that forward lymph flow is highly dependent on movement and changes i
46  studies using CLEC2-deficient mice in which lymph flow is impaired due to loss of lympho-venous hemo
47                      In addition, during SAH lymph flow is increased but without significant lymphang
48                    Direct in vivo imaging of lymph flow is key to understanding lymphatic system func
49 us, left atrial pressure elevation increased lymph flow less in dextran-treated animals than in contr
50         Hemodynamics, oxygenation, pulmonary lymph flow, lymph protein clearance, and total body weig
51 esses contractile function, which influences lymph flow needed for fluid regulation, humoral immunity
52                       Patients with deranged lymph flow often require critical care, and those who re
53 onolayers and the interruption of mesenteric lymph flow on shock-induced lung injury.
54 tment of thoracic duct pumping to changes in lymph flow pattern.
55 lmonary artery pressure (Ppa), PVR, and lung lymph flow (Q L).
56 ut existing optical techniques for measuring lymph flow require complex protocols and provide limited
57 lanchnic vasculature and deficient abdominal lymph flow resulting in interstitial edema might both be
58 al stimuli generated by chronically elevated lymph flow results in lymphatic dysfunction.
59                        In protein depletion, lymph flow rises with little change in Pnf, suggesting t
60                                              Lymph flow rose and protein content decreased slowly and
61    This is puzzling because it suggests that lymph flow rose with little or no change in the forces a
62                     The model indicates that lymph flow shapes intranodal CCL21 gradients, and that C
63  care may concomitantly demonstrate deranged lymph flow that adversely impacts care.
64                                   Retrograde lymph flow through apparently incompetent interlymphangi
65   Lymph node lymphangiogenesis and increased lymph flow through tumor-draining lymph nodes may active
66 required for lymphangiogenesis and increased lymph flow through tumor-draining lymph nodes, as these
67 cordingly, DCLHb raised lung and soft tissue lymph flows to peak values of 12.2 +/- 3.8 and 1.6 +/- 0
68 ively, while HSA raised lung and soft tissue lymph flows to peak values of 7.5 +/- 4.8 and 4.6 +/- 1.
69          LTalpha(-/-) mice exhibited reduced lymph flow velocities and increased interstitial fluid p
70 r stress (0.07 dynes/cm(2)), consistent with lymph flow via beta1 integrins, including alpha2beta1, a
71                                              Lymph flow was increased after CPB but significantly mor
72             Lymph node lymphatic sinuses and lymph flow were increased in mice implanted with unmarke
73 l sinuses in the entire LN and the extent of lymph flow within them has been unclear.