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1 of transplant rejection and the treatment of lymphangioleiomyomatosis.
2 peutic approach for the treatment of TSC and lymphangioleiomyomatosis.
3 F-D dysregulation and its cellular origin in lymphangioleiomyomatosis.
4 an immunodeficient mouse xenograft model of lymphangioleiomyomatosis.
5 uberous sclerosis complex (TSC) and sporadic lymphangioleiomyomatosis.
6 uberous sclerosis complex (TSC) and sporadic lymphangioleiomyomatosis.
7 with tuberous sclerosis complex and sporadic lymphangioleiomyomatosis.
8 e individuals to both tuberous sclerosis and lymphangioleiomyomatosis.
9 sorders tuberous sclerosis complex (TSC) and lymphangioleiomyomatosis.
10 h the tuberous sclerosis complex or sporadic lymphangioleiomyomatosis.
11 h the tuberous sclerosis complex or sporadic lymphangioleiomyomatosis.
12 ut not in normal volunteers or patients with lymphangioleiomyomatosis.
13 rapy for the treatment of angiomyolipoma and lymphangioleiomyomatosis.
14 regulated in TSC-related angiomyolipomas and lymphangioleiomyomatosis.
15 factor VEGF-D are elevated significantly in lymphangioleiomyomatosis.
16 l manifestations of TSC, including pulmonary lymphangioleiomyomatosis.
17 hangioleiomyomas are common in patients with lymphangioleiomyomatosis.
18 ties are seen in a majority of patients with lymphangioleiomyomatosis.
20 associated closely with the pathogenesis of lymphangioleiomyomatosis, a rare and progressive neoplas
26 interstitial lung diseases, with a focus on lymphangioleiomyomatosis and pulmonary Langerhans cell h
27 d lesions in multiple organs including lung (lymphangioleiomyomatosis) and kidney (angiomyolipoma and
28 y that was enriched for those with pulmonary lymphangioleiomyomatosis, and was therefore composed mos
29 h the tuberous sclerosis complex or sporadic lymphangioleiomyomatosis are associated with mutations i
31 ly always present in patients with pulmonary lymphangioleiomyomatosis, but the mechanisms underlying
32 plore the feasibility of targeting tumors in lymphangioleiomyomatosis by melanoma immunotherapy, we t
33 ma as well as the susceptibility of cultured lymphangioleiomyomatosis cells to melanoma reactive cyto
35 yomatosis with serum levels in patients with lymphangioleiomyomatosis correlating with impaired lung
36 requency and volume, and in a mouse model of lymphangioleiomyomatosis, genetic downregulation of DGKA
39 of clinical characteristics of subjects with lymphangioleiomyomatosis have been based on a limited nu
42 age range of women afflicted with pulmonary lymphangioleiomyomatosis is broader than previously appr
43 ed in TSC-associated renal tumors, pulmonary lymphangioleiomyomatosis, kidneys from Tsc2(+/-) mice, a
44 and neural stem-like cell characteristics of lymphangioleiomyomatosis (LAM) and angiomyolipoma cells.
50 involved in the growth and proliferation of lymphangioleiomyomatosis (LAM) cells, abnormal smooth mu
84 were made on lung tissue from 10 women with lymphangioleiomyomatosis (LAM) to evaluate the distribut
85 e diagnosis and pharmacological treatment of lymphangioleiomyomatosis (LAM) were recently published.
87 -based RNA therapy in a preclinical model of lymphangioleiomyomatosis (LAM), a destructive lung disea
94 e as a potential therapeutic target for TSC, lymphangioleiomyomatosis (LAM), and other mTORC1-hyperac
95 ECM in diseases such as atherosclerosis and lymphangioleiomyomatosis (LAM), both characterized by ex
96 cluding tuberous sclerosis complex (TSC) and lymphangioleiomyomatosis (LAM), drives B7-H3 expression
97 notably renal angiomyolipomas and pulmonary lymphangioleiomyomatosis (LAM), emerge later, placing ad
98 TSC1 or TSC2, linked to a rare lung disease, lymphangioleiomyomatosis (LAM), manifests as neoplastic
107 oliferation of abnormal smooth muscle cells (lymphangioleiomyomatosis [LAM] cells) in LAM, we perform
108 ypsin deficiency, tuberous sclerosis complex/lymphangioleiomyomatosis, Loeys-Dietz syndrome, cystic f
109 efore, boosting immune responses to gp100 in lymphangioleiomyomatosis may offer a highly desirable tr
110 table gp100 expression; thus, tumor cells in lymphangioleiomyomatosis may process melanosomal antigen
112 2-deficient cells and pulmonary nodules from lymphangioleiomyomatosis patients contributes to tumor g
114 tumor-promoting roles in the pathogenesis of lymphangioleiomyomatosis, perhaps acting as both autocri
115 od of sirolimus therapy, among patients with lymphangioleiomyomatosis, the mean forced expiratory vol
116 compared with melanoma, cells cultured from lymphangioleiomyomatosis tissue were susceptible to cyto
118 om 1998 to 2001, 243 subjects with pulmonary lymphangioleiomyomatosis were enrolled into a national r
119 diagnosis of tuberous sclerosis or sporadic lymphangioleiomyomatosis were randomly assigned, in a 2:
122 SC-related skin tumors, angiomyolipomas, and lymphangioleiomyomatosis with serum levels in patients w