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1 t them to the site of infection and to local lymphatic tissue.
2  tumor infiltration into the bone marrow and lymphatic tissue.
3  CD86 or CCR7, which directs DC migration to lymphatic tissue.
4 l immunological status comparable to that of lymphatic tissue.
5  within the allograft as well as in draining lymphatic tissue.
6 distinct lipidomic fragment distributions in lymphatic tissue.
7  with Ag-specific activation as it occurs in lymphatic tissue.
8 trointestinal tissues, peripheral blood, and lymphatic tissues.
9 hat will fully suppress viral replication in lymphatic tissues.
10 vated (CD86(+)) dendritic cells in secondary lymphatic tissues.
11 e inoculum traffic from the vaginal lumen to lymphatic tissues.
12 es at the site of inoculation, in liver, and lymphatic tissues.
13 meostasis by inducing collagen deposition in lymphatic tissues.
14 s also highly expressed in hematopoietic and lymphatic tissues.
15 troviral treatment remain T-cell depleted in lymphatic tissues.
16  could decrease virus-producing cells in the lymphatic tissues.
17 mphoma/leukemia 10 (Bcl10)-mucosa-associated lymphatic tissue 1(MALT1) (CBM) complex, which appears t
18 at, even if it is largely free of neural and lymphatic tissue, a comprehensive effort to map the dist
19  the upstream Bcl10-MALT1 (mucosa-associated lymphatic tissue) adapter complex.
20 nograft and persistence of microchimerism in lymphatic tissue after graft removal.
21 munodeficiency virus type 1 (HIV-1)-infected lymphatic tissues after treatment, and we undertook mapp
22 on based on its association with substantial lymphatic tissue, although the specific nature of that p
23 e epithelial barrier, the mucosal-associated lymphatic tissues and microbiota.
24 ficiency virus type 1 (HIV-1) replication in lymphatic tissues and partially reverses the pathologica
25            This is also effective in sealing lymphatic tissues and thereby facilitating dissection.
26 o BALB/c mice, but the mutants colonized the lymphatic tissues and were sufficiently immunogenic to p
27 of IFN-alpha/beta at peak acute infection in lymphatic tissues, and (iii) natural SIV hosts downregul
28 and CpG DNA are elevated in microbe-draining lymphatic tissues, and unraveling the basis for IL-15-dr
29 in collagen deposition and disruption of the lymphatic tissue architecture, and this damage contribut
30  fibrosis within the T cell zone disrupt the lymphatic tissue architecture, contributing to depletion
31 ith preservation of CD4(+) T cell counts and lymphatic tissue architecture.
32  center T follicular helper cells (GCTfh) in lymphatic tissue are critical for B cell differentiation
33 tissues, such as adipose, muscle, heart, and lymphatic tissues, are outlined in this review.
34  promotes CLL cell survival and expansion in lymphatic tissue areas designated proliferation centers.
35                      Virus was isolated from lymphatic tissue as early as 2 days post-IN inoculation;
36 nt in smooth muscle and heart, but scarce in lymphatic tissues, brain, and liver.
37 f encephalitogenic T cells in the peripheral lymphatic tissue, but Nlrx1(-/-) mice are more susceptib
38                         In vivo infection of lymphatic tissues by the human immunodeficiency virus ty
39                                           In lymphatic tissues, chronic lymphocytic leukemia (CLL) ce
40 ociated with progressive CD4(+) T-cell loss, lymphatic tissue destruction and premature death.
41 , viral replication and immune activation in lymphatic tissue drive a premature immunosuppressive res
42 nent of the inflammatory response induced by lymphatic tissue-dwelling filariae.
43 cells first settle in secondary (2 degrees ) lymphatic tissues (e.g., the spleen) where, in the absen
44 inducible T(reg) cell stimulation of primary lymphatic tissue fibroblasts to produce collagen type I
45 ALT or CLN to address the necessity of these lymphatic tissues for the development of a local protect
46                                  Analysis of lymphatic tissues from 12/15-LO-deficient mice confirmed
47 d depletion of CD4 T cells in gut-associated lymphatic tissue (GALT), spleen, and lymph nodes during
48 y virus-1 (HIV-1) infections, gut-associated lymphatic tissue (GALT), the largest component of the ly
49 tion of CD4+ T lymphocytes in gut-associated lymphatic tissue (GALT).
50 d incomplete, particularly in gut-associated lymphatic tissues (GALT).
51 erminal centers (GCs) in patients' secondary lymphatic tissues has been reported.
52 ized that systemically targeting antigens to lymphatic tissue in vivo might modulate immunity.
53 e follicular dendritic cell (FDC) network in lymphatic tissues in HIV-1 and simian immunodeficiency v
54 aging and immunohistochemical examination of lymphatic tissues in mice heterozygous (+/-) for a targe
55 ive studies evaluating targeted therapies to lymphatic tissues in mucosal immune system responsible f
56 eripheral blood mononuclear cells (PBMC) and lymphatic tissues in the acutely infected baboons were i
57 al blood mononuclear cells (PBMCs) and other lymphatic tissues in vivo.
58 sive early replication in the gut-associated lymphatic tissue, including intestinal Peyer's patches,
59 d the transcriptional profiles of intestinal lymphatic tissue infected with Y. enterocolitica.
60 rains the egress of CCR7(+) lymphocytes from lymphatic tissues into the blood, thus resulting in redu
61 ical processes occurring in human pancreatic lymphatic tissues is lacking.
62  in microarray studies of HIV-1 infection in lymphatic tissue (LT) and show in this study that increa
63 ection establishes a persistent infection in lymphatic tissues (LT).
64                                              Lymphatic tissues (LTs) are structurally organized to pr
65 emination, and establishment of infection in lymphatic tissues (LTs) during the acute stage of infect
66 trol but do not eradicate infection from the lymphatic tissues (LTs) or prevent the particularly mass
67 tic ulcer disease, gastric mucosa-associated lymphatic tissue lymphoma, or gastric adenocarcinoma.
68 llectomy as one of targeted interventions to lymphatic tissues may provide additional improvement to
69                     In this study, we report lymphatic tissue microarray analyses, revealing for the
70 lication of M. avium and SIV was analyzed in lymphatic tissues obtained from rhesus macaques experime
71 xpression as well as in vivo accumulation in lymphatic tissue of migratory DCs.
72                                       In the lymphatic tissue of simian immunodeficiency virus (SIV)-
73                         The cells within the lymphatic tissue of the gut are likely to be central for
74  cells and mouse macrophages in the skin and lymphatic tissues of humanized mice.
75 lls from pancreatic, mesenteric, and splenic lymphatic tissues of non-diabetic control (ND), beta cel
76  we confirm depletion of conventional DC2 in lymphatic tissues of SPPL2a(-/-) mice and demonstrate de
77  microbes and restricted bacterial access to lymphatic tissues of the mice, thereby reducing microbio
78 adily colonize and induce disease within the lymphatic tissues of the small intestine.
79 of CD4 memory T cells in blood and secondary lymphatic tissues of these patients.
80                         We hypothesized that lymphatic tissues play a critical role in HO formation.
81 ristic of eTreg cells residing in particular lymphatic tissues, regardless of the challenge.
82 increase ARV distribution and persistence in lymphatic tissue reservoirs of HIV-1.
83        Emerging research suggests that local lymphatic tissue such as tonsils have important role in
84  AIDS-related disease progression within the lymphatic tissues, such as vascular proliferation and ly
85 higher absolute numbers of CD8(+) T cells in lymphatic tissues than mice controlling tumor developmen
86          To identify genetic determinants in lymphatic tissue that critically affect HIV-1 replicatio
87  abnormal proliferation and sequestration of lymphatic tissues that are disconnected from the rest of
88 ors in vivo that impact viral replication in lymphatic tissue, the primary anatomical site of virus-h
89 ain "specified offals," including neural and lymphatic tissues, thought to contain high titers of pri
90 s study was to determine the contribution of lymphatic tissue to heterotopic ossification (HO).
91 rformance of an extended PLND, including all lymphatic tissue to the level of the aortic bifurcation.
92                         Enrichment of mature lymphatic tissues was noted 2 weeks after injury at the
93 ool for Yersinia pseudotuberculosis-infected lymphatic tissues, we revealed numerous alterations of h
94  from peripheral blood to the gut-associated lymphatic tissue, where they may contribute to immune ac
95 ls proliferate in pseudofollicles within the lymphatic tissues, where signals from the microenvironme