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1 od-related cell types (CD3 and CD4+ T cells, lymphoblastoid cell lines).
2 RRP12 in LNCaP, USP14 in DU-145 and SMIN3 in lymphoblastoid cell line.
3 m log-phase GM06990, a karyotypically normal lymphoblastoid cell line.
4 tibility complex class I-deficient 721.221 B-lymphoblastoid cell line.
5 ed the genotype of an HPS1 patient-derived B-lymphoblastoid cell line.
6 astoid cell lines, and EBV genome number per lymphoblastoid cell line.
7    Whole-genome sequencing reads were from a lymphoblastoid cell-line.
8 tal mRNA fractions of 52 HapMap Yoruba human lymphoblastoid cell lines.
9 oning patterns are observed in two different lymphoblastoid cell lines.
10  the expression level of both transcripts in lymphoblastoid cell lines.
11 es less stable or leakier in EBV-transformed lymphoblastoid cell lines.
12 or each of the four genes in the region in B lymphoblastoid cell lines.
13 igene assays and, when available, in patient lymphoblastoid cell lines.
14 rase reporter, and EBV genome maintenance in lymphoblastoid cell lines.
15 n human adipose tissue, skeletal muscle, and lymphoblastoid cell lines.
16 f virus gene expression and the outgrowth of lymphoblastoid cell lines.
17  available RNA-seq expression data sets from lymphoblastoid cell lines.
18 ed IRF7 is detected in latently infected EBV lymphoblastoid cell lines.
19 oci (QTLs) of m(6)A peaks in 60 Yoruba (YRI) lymphoblastoid cell lines.
20 ype and reduced BAK1 expression was shown in lymphoblastoid cell lines.
21 phase arrest and apoptosis in leukemic and B-lymphoblastoid cell lines.
22  normal tissue pairs and 17 matched SCLC and lymphoblastoid cell lines.
23 sing whole-genome expression array data from lymphoblastoid cell lines.
24 lded aberrant results in the majority of SLE lymphoblastoid cell lines.
25 esses that prevent LTIII BHRF1 expression in lymphoblastoid cell lines.
26 hosphorylation was also noted in 2 of 16 SLE lymphoblastoid cell lines.
27 antly higher proliferation in DS than non-DS lymphoblastoid cell lines.
28 er of studies have explored this area, using lymphoblastoid cell lines.
29    Gene expression of SLC1A1 is heritable in lymphoblastoid cell lines.
30 ion 2 (hPMS2)-deficient and proficient human lymphoblastoid cell lines.
31 mary B lymphocytes into continuously growing lymphoblastoid cell lines.
32 for a subset of genes in a separate group of lymphoblastoid cell lines.
33  and vaccinia virus (expressing gB)-infected lymphoblastoid cell lines.
34 197 ethnically defined Human Variation Panel lymphoblastoid cell lines.
35 ivation of EBV-positive Burkitt lymphoma and lymphoblastoid cell lines.
36 ,599 genes in Epstein-Barr virus-transformed lymphoblastoid cell lines.
37 ession in human aortic endothelial cells and lymphoblastoid cell lines.
38 (ChIP-exo) genome-wide analysis of 27 HapMap lymphoblastoid cell lines.
39 the TwinsUK microarray and RNA-Seq cohort in lymphoblastoid cell lines.
40  with daunorubicin IC50 values in a panel of lymphoblastoid cell lines.
41 ed in HL, diffuse large B-cell lymphoma, and lymphoblastoid cell lines.
42 d in proband versus control EBV-immortalized lymphoblastoid cell lines.
43 and RP1-10D13.2 expression levels in the CAP lymphoblastoid cell lines.
44 gkm-SVM models of chromatin accessibility in lymphoblastoid cell-lines.
45 ys were performed on patient and control EBV lymphoblastoids cell lines.
46  appear to regulate gene expression in human lymphoblastoid cell lines, a tightly controlled, largely
47 e method was first applied to RNA-Seq from a lymphoblastoid cell-line, achieving 99.7% precision and
48                                   Using 1220 lymphoblastoid cell lines across platforms and independe
49 eQTLs each affect hundreds of transcripts in lymphoblastoid cell lines across three African populatio
50 ong chromosomes from primary lymphocytes and lymphoblastoid cell lines adapted to long-term growth in
51 ells, HeLa cells, HEK293 cells, and 16 human lymphoblastoid cell lines, all genotyped for the 9p21.3
52 edish patients, and to KMO expression in 717 lymphoblastoid cell lines and 138 hippocampal biopsies.
53 d the structure of HLA-F on the surface of B lymphoblastoid cell lines and activated lymphocytes by d
54 s from human, chimpanzee, and rhesus macaque lymphoblastoid cell lines and compared them to transcrip
55  stimulation with autologous EBV-transformed lymphoblastoid cell lines and correlated with EBV load i
56 rm that CYFIP1 is upregulated in transformed lymphoblastoid cell lines and demonstrate its upregulati
57 R-181c, were significantly down-regulated in lymphoblastoid cell lines and fresh peripheral blood cel
58 990620, that differentially recruits CTCF in lymphoblastoid cell lines and human brain to influence C
59  and association with TMEM106B expression in lymphoblastoid cell lines and human brain.
60 hromatin signature to infer MAE for genes in lymphoblastoid cell lines and human fetal brain tissue.
61 ructure of BMPR1A, we performed 5' RACE from lymphoblastoid cell lines and normal colon tissue, which
62 sociated with lower CTSH expression in human lymphoblastoid cell lines and pancreatic tissue.
63  with a decline in CLDN14 expression in both lymphoblastoid cell lines and T cells (Padj = 0.003 and
64 ic variation in TF binding affinity in human lymphoblastoid cell lines and test their association wit
65 med histone acetylation ChIP-seq on 57 human lymphoblastoid cell lines and used the resulting reads t
66 e associated with lower FBXO33 expression in lymphoblastoid cell lines and with reduced frontal gray
67 etal muscle, subcutaneous adipose tissue and lymphoblastoid cell lines) and observed a tissue-specifi
68  from GM06990, a near-normal EBV-transformed lymphoblastoid cell line, and have compared origin distr
69 clear factor kappaB (p65), were mapped in 10 lymphoblastoid cell lines, and 25 and 7.5% of the respec
70 iP luciferase reporter, EBNA1 DNA binding in lymphoblastoid cell lines, and EBV genome number per lym
71 evel was performed using patient and control lymphoblastoid cell lines, and established experimental
72  large-effect eQTLs identified in the HapMap lymphoblastoid cell lines, and examined the association
73  DLX6 in mouse x human somatic cell hybrids, lymphoblastoid cell lines, and frontal cortex from contr
74  lymphoma and Epstein-Barr virus-transformed lymphoblastoid cell lines, and in T cells activated via
75 ed BZLF1 expression in latently EBV-infected lymphoblastoid cell lines, and knockdown of BGLF2 reduce
76 n both peripheral blood leukocytes (PBL) and lymphoblastoid cell lines; and a study of postoperative
77 als on whom both haplotypes and DH status in lymphoblastoid cell lines are publicly available.
78 d this effect is substantially reversed when lymphoblastoid cell lines are stably infected with a ret
79 ng Salmonella typhimurium infection of human lymphoblastoid cell lines as a means of dissecting the g
80 n transcriptional response in fibroblast and lymphoblastoid cell lines, as well as circulating monocy
81 ce the death of established, EBV-transformed lymphoblastoid cell lines at doses nontoxic to normal ce
82         Glycosylation of mAb-Ds from human B-lymphoblastoid cell lines (B) was similar to anti-D Ig a
83                              Patient-derived lymphoblastoid cell lines bearing a range of expanded al
84 ized endogenously presented targets on EBV B lymphoblastoid cell lines (BLCLs), but not peripheral bl
85 different HLA-typed, human EBV-transformed B lymphoblastoid cell lines (BLCLs).
86 nomic and transcriptomic data from 445 human lymphoblastoid cell lines by combining an RNA editing QT
87 ne expression levels generated for 373 human lymphoblastoid cell lines by the Geuvadis consortium and
88  virus (EBV) to transform human B cells into lymphoblastoid cell lines compared to that of type 2 EBV
89 us (EBV)-encoded RNAs (EBERs) were tested in lymphoblastoid cell lines containing EBER mutants of EBV
90                               In this study, lymphoblastoid cell line cultures (LCLs) from women with
91 nome RNA-seq analysis between mouse vHpc and lymphoblastoid cell line cultures from control women and
92      Application of the methods to the human lymphoblastoid cell line data on chromosomes 14 and 22 f
93 study the response to 23 treatments in three lymphoblastoid cell lines demonstrating that it should a
94 ical cross-correction in an enzyme-deficient lymphoblastoid cell line derived from patients with muco
95  associations with gene expression levels in lymphoblastoid cell lines derived from 480 participants
96 onstructed by transferring mitochondria from lymphoblastoid cell lines derived from a Chinese family
97  generated by transferring mitochondria from lymphoblastoid cell lines derived from a Chinese family
98 icative impairments could be demonstrated in lymphoblastoid cell lines derived from affected individu
99  quantitative immunoblotting of lysates from lymphoblastoid cell lines derived from affected individu
100                                              Lymphoblastoid cell lines derived from carriers of misse
101 iated (P = .01) with FPGS gene expression in lymphoblastoid cell lines derived from combined HapMap A
102 , cytosine modification levels in 133 HapMap lymphoblastoid cell lines derived from individuals of Eu
103 ed gene expression in the full set of HapMap lymphoblastoid cell lines derived from individuals of Eu
104                     In this study, utilizing lymphoblastoid cell lines derived from International Hap
105 ne expression in response to high glucose in lymphoblastoid cell lines derived from matched individua
106      We performed functional assays by using lymphoblastoid cell lines derived from members of Chines
107 ofiling by CpG island microarray analysis of lymphoblastoid cell lines derived from monozygotic twins
108 ition, signaling, and repair mechanisms in B lymphoblastoid cell lines derived from patients with ped
109 used an unbiased whole-genome approach using lymphoblastoid cell lines derived from persons of Europe
110 d expression information for EBV-transformed lymphoblastoid cell lines derived from populations acros
111 expression by affecting the binding to GR in lymphoblastoid cell lines derived from the same patients
112       In MDV-induced T-cell lymphomas and in lymphoblastoid cell lines derived from them, MDV-miR-M4
113                     We sequenced RNA from 69 lymphoblastoid cell lines derived from unrelated Nigeria
114 ar susceptibility to cisplatin in 176 HapMap lymphoblastoid cell lines derived from Yoruba individual
115                  Nonleukemic EBV-transformed lymphoblastoid cell lines displayed highly stable replic
116  in retroviral infection using the chicken B lymphoblastoid cell line DT40.
117 cific quantification assays to a panel of HD lymphoblastoid cell lines, each carrying the major Europ
118                             Surprisingly, in lymphoblastoid cell lines, EBNA3C is extremely stable, a
119 a demonstration, Epstein-Barr virus-infected lymphoblastoid cell lines (EBV-LCL) were isolated based
120                                  We show for lymphoblastoid cell lines established from individuals o
121                                        The B-lymphoblastoid cell lines exhibited a unimodal distribut
122 otein from V362I and R381Q variants in human lymphoblastoid cell lines exhibited lower expression lev
123 with reduced survival in a panel of 24 human lymphoblastoid cell lines exposed to the alkylating agen
124     Here, we show that EBV-immortalized FSHD lymphoblastoid cell lines express DUX4 and both early an
125                                   Thus, FSHD lymphoblastoid cell lines express DUX4 and early and lat
126  C1 ligand group are activated in vitro by B lymphoblastoid cell lines expressing the C2 group.
127 udies, we collected RNA-sequencing data from lymphoblastoid cell lines for 431 Hutterite individuals.
128 red the cytotoxicity of 156 compounds in 884 lymphoblastoid cell lines for which genotype and transcr
129 measure chromatin accessibility in 70 Yoruba lymphoblastoid cell lines, for which genome-wide genotyp
130                                            A lymphoblastoid cell line from one affected individual sh
131 ed the genomes of a malignant melanoma and a lymphoblastoid cell line from the same person, providing
132  address these questions, we perform Hi-C on lymphoblastoid cell lines from 20 individuals.
133 s to evaluate transcriptional profiles using lymphoblastoid cell lines from 62 prostate cancer patien
134 ne relative protein levels of 5,953 genes in lymphoblastoid cell lines from 95 diverse individuals ge
135 ormed a genome-wide gene expression study in lymphoblastoid cell lines from 96 Hutterites.
136 rometry to perform an integrated analysis in lymphoblastoid cell lines from a diverse group of indivi
137                               We established lymphoblastoid cell lines from a G319S homozygote and co
138  Research design and methods: We established lymphoblastoid cell lines from a G319S homozygote and co
139 itation, we derived normal and DNMT3A mutant lymphoblastoid cell lines from a germline mosaic individ
140                           Using immortalized lymphoblastoid cell lines from a healthy study populatio
141                  Protein blot analysis using lymphoblastoid cell lines from affected individuals show
142  OGT protein levels was observed in isolated lymphoblastoid cell lines from affected individuals, con
143 - to 3-fold overexpression of DIAPH3 mRNA in lymphoblastoid cell lines from affected individuals.
144 This miniATM variant was also highlighted in lymphoblastoid cell lines from AT patients and was shown
145 f NK cells to kill freshly established human lymphoblastoid cell lines from autologous or allogeneic
146  and extended in three human EBV-transformed lymphoblastoid cell lines from individuals with MSS, lea
147 rigin effect, except for differentiating the lymphoblastoid cell lines from other cell types.
148         Using simvastatin and sham incubated lymphoblastoid cell lines from participants of the Chole
149 tein (FMRP), its upregulation in transformed lymphoblastoid cell lines from patients with duplication
150 to interrogate DSB repair and recognition in lymphoblastoid cell lines from patients with pediatric S
151 suggest that DSB repair is defective in some lymphoblastoid cell lines from pediatric patients with S
152                                              Lymphoblastoid cell lines from subjects with the p.V228F
153                                We used 1,086 lymphoblastoid cell lines from the 1000 Genomes Project,
154                                              Lymphoblastoid cell lines from the index family in compa
155 , we present a genome-wide model using human lymphoblastoid cell lines from the International HapMap
156                                           In lymphoblastoid cell lines from two translocation subject
157 we utilized gene expression association from lymphoblastoid cells lines from 754 p.Phe508del CF-affec
158 r virus-transformed B-cell cultures (human B-lymphoblastoid cell lines) from 19 healthy donors.
159                                              Lymphoblastoid cell lines generated from affected childr
160            Integration with ENCODE data from lymphoblastoid cell line GM12878, demonstrates that IRF4
161 cell RNA-seq protocol to study the reference lymphoblastoid cell line GM12878.
162 lic occupancy of 24 TFs and EP300 in a human lymphoblastoid cell line GM12878.
163  depletion and gene activation necessary for lymphoblastoid cell line growth and survival.
164 BNA-2 confers a type 1 growth phenotype in a lymphoblastoid cell line growth maintenance assay.
165             Moreover, NCL silencing impaired lymphoblastoid cell line growth.
166           DNA methylation (DNAm) measured in lymphoblastoid cell lines has been repeatedly demonstrat
167 icular, studies using FRDA patient blood and lymphoblastoid cell lines have detected increased DNA me
168    Applied to high-resolution Hi-C data in a lymphoblastoid cell line, HiC-DC detects significant int
169 k haplotype show no binding of STAT1, and in lymphoblastoid cell lines homozygous for the CAD non-ris
170                                              Lymphoblastoid cell lines homozygous for the CAD risk ha
171 C data of chromosome 5 from GM12878 (a human lymphoblastoid cell line), (ii) 40-kb resolution whole-g
172  in nonsynonymous substitutions in all three lymphoblastoid cell lines in our study, unlike RNA editi
173 nd transform B lymphocytes into immortalized lymphoblastoid cell lines in vitro.
174  using >300 expression microarrays (from 117 lymphoblastoid cell lines) in corticosteroid (dexamethas
175 h resulted in the generation of EBV-positive lymphoblastoid cell lines, indicating that the virus in
176       Here, we show that growth of a human B lymphoblastoid cell line infected with Epstein-Barr viru
177  of 237 up-regulated genes derived from FSHD lymphoblastoid cell lines is elevated in FSHD muscle bio
178 rom the exosome fractions derived from human lymphoblastoid cell line (LCL) culture media.
179 o compare skin eQTLs to a published panel of lymphoblastoid cell line (LCL) eQTLs.
180  expression of most cell genes essential for lymphoblastoid cell line (LCL) growth and survival.
181 p16(INK4A) is essential for immortal human B-lymphoblastoid cell line (LCL) growth.
182                   Perturbation of ESEs stops lymphoblastoid cell line (LCL) growth.
183 his study, integrative analyses of published lymphoblastoid cell line (LCL) Hi-C data and our 4C-seq
184 ytes with the autologous virus-transformed B-lymphoblastoid cell line (LCL) in vitro, can be used to
185                                              Lymphoblastoid cell line (LCL) is a common tool to study
186 n and subsequently almost completely ablated lymphoblastoid cell line (LCL) outgrowth.
187  (WES) are whole blood (WB) and immortalized lymphoblastoid cell line (LCL).
188 addition, human LRRK2 G2019S patient-derived lymphoblastoid cell lines (LCL) demonstrated increased m
189 l transcriptomics data from a panel of human lymphoblastoid cell lines (LCL) to infer drug response n
190 tore iNKT recognition in EBV-infected cells, lymphoblastoid cell lines (LCL) were treated with AM580,
191 inhibitor olaparib (AZD2281) of 5 ATM mutant lymphoblastoid cell lines (LCL), an ATM mutant MCL cell
192 allowed the establishment of MDV-transformed lymphoblastoid cell lines (LCL).
193 latency II NPC C666-1 cells, and latency III lymphoblastoid cell lines (LCL).
194 ntly reported gene expression changes in 480 lymphoblastoid cell lines (LCLs) after in vitro simvasta
195 lysis of RNA stability in seven human HapMap lymphoblastoid cell lines (LCLs) and analyzed the effect
196 lomere repeat elements (SREs) in transformed lymphoblastoid cell lines (LCLs) and human embryonic ste
197 otein isoforms across 68 Yoruba (YRI) HapMap lymphoblastoid cell lines (LCLs) and identified 12 cis a
198 on measured via RNA-seq analysis in adipose, lymphoblastoid cell lines (LCLs) and skin.
199                       Using EBV-immortalized lymphoblastoid cell lines (LCLs) as a model, we found th
200 umented the DNA methylation pattern in human lymphoblastoid cell lines (LCLs) as well as identified s
201                 B lymphocytes converted into lymphoblastoid cell lines (LCLs) by an Epstein-Barr viru
202           Patient-derived cell lines such as lymphoblastoid cell lines (LCLs) could represent the ide
203  lymphocytes into indefinitely proliferating lymphoblastoid cell lines (LCLs) depends on the concerte
204       Using a data set of gene expression in lymphoblastoid cell lines (LCLs) derived from 210 HapMap
205 tosine modifications at 283,540 CpG sites in lymphoblastoid cell lines (LCLs) derived from independen
206   We developed an in vitro model of PMD with lymphoblastoid cell lines (LCLs) derived from participan
207               Here we report that RP-mutated lymphoblastoid cell lines (LCLs) established from DBA pa
208                                        Using lymphoblastoid cell lines (LCLs) established with EBV re
209                                          How lymphoblastoid cell lines (LCLs) evolve from the infecte
210 ene expression profiles using microarrays on lymphoblastoid cell lines (LCLs) from 413 cases and 446
211 romatin profiling for three histone marks in lymphoblastoid cell lines (LCLs) from 75 sequenced indiv
212  Previously we hypothesized that a subset of lymphoblastoid cell lines (LCLs) from children with auti
213  Here we analyze m(6)A mRNA modifications in lymphoblastoid cell lines (LCLs) from human, chimpanzee
214 tified H3K4me3-associated genomic regions in lymphoblastoid cell lines (LCLs) from humans, chimpanzee
215 enome-wide association studies involving 523 lymphoblastoid cell lines (LCLs) from individuals of Eur
216 A21 to assess trisomic protein expression in lymphoblastoid cell lines (LCLs) from patients with DS a
217                                              Lymphoblastoid cell lines (LCLs) from some children with
218  B cell lymphoma infected with HHV-6B, (iii) lymphoblastoid cell lines (LCLs) from subjects with inhe
219                 Using RNA-sequence data from lymphoblastoid cell lines (LCLs) from the TwinsUK cohort
220 EBV to convert human B cells into long-lived lymphoblastoid cell lines (LCLs) in vitro requires the c
221  lymphocytes into continuously proliferating lymphoblastoid cell lines (LCLs) in vitro through manipu
222  for in vitro transformation of B cells into lymphoblastoid cell lines (LCLs) is activation of the NF
223 rther demonstrated that knockdown of H2AX in lymphoblastoid cell lines (LCLs) led to the upregulation
224 ion, elicited lytic EBV in latently infected lymphoblastoid cell lines (LCLs) partially via Toll-like
225         Epstein-Barr virus (EBV) transformed lymphoblastoid cell lines (LCLs) provide a conveniently
226 r Virus (EBV) conversion of B-lymphocytes to Lymphoblastoid Cell Lines (LCLs) requires four EBV nucle
227 hocytes (RBLs) leads to the establishment of lymphoblastoid cell lines (LCLs) that can grow indefinit
228  transformed into continuously proliferating lymphoblastoid cell lines (LCLs) that carry EBV DNA as e
229                          The ZIIRmt-infected lymphoblastoid cell lines (LCLs) that did grow out exhib
230                                              Lymphoblastoid cell lines (LCLs) were cocultured with au
231          Populations of human-derived HapMap lymphoblastoid cell lines (LCLs) were infected with RSV.
232  between vitamin D receptor (VDR) binding in lymphoblastoid cell lines (LCLs), chromatin states in LC
233                     In contrast to wild-type lymphoblastoid cell lines (LCLs), dividing LCLs establis
234                        ChIP-seq performed on lymphoblastoid cell lines (LCLs), expressing epitope-tag
235                                        Human lymphoblastoid cell lines (LCLs), generated through Epst
236                          In lytic-permissive lymphoblastoid cell lines (LCLs), pulse exposure to the
237                                In GS-derived lymphoblastoid cell lines (LCLs), the proportion of ITPR
238 amining Epstein-Barr virus (EBV)-transformed lymphoblastoid cell lines (LCLs), we identified four EBV
239 sforms B cells to continuously proliferating lymphoblastoid cell lines (LCLs), which represent an exp
240 ll proliferation leading to the outgrowth of lymphoblastoid cell lines (LCLs).
241 oth in newly infected primary B cells and in lymphoblastoid cell lines (LCLs).
242 A polymerase II (Pol II) occupancy in Yoruba lymphoblastoid cell lines (LCLs).
243 s drives their indefinite proliferation into lymphoblastoid cell lines (LCLs).
244 loci and facilitate KLF14 gene expression in lymphoblastoid cell lines (LCLs).
245 roliferation and through transformation into lymphoblastoid cell lines (LCLs).
246  critical for B-lymphocyte transformation to lymphoblastoid cell lines (LCLs).
247 ansformation into indefinitely proliferating lymphoblastoid cell lines (LCLs).
248  response using 277 ethnically defined human lymphoblastoid cell lines (LCLs).
249 ls in response to autologous EBV-transformed lymphoblastoid cell lines (LCLs).
250 and transformation of quiescent B cells into lymphoblastoid cell lines (LCLs).
251 ISPR/Cas9 loss-of-function screens in BL and lymphoblastoid cell lines (LCLs).
252 n vitro, EBV transforms primary B cells into lymphoblastoid cell lines (LCLs).
253  conducted a pharmacogenomic study using 266 lymphoblastoid cell lines (LCLs).
254 lation from chromatin to proteins, in Yoruba lymphoblastoid cell lines (LCLs).
255 s, nasopharyngeal carcinoma (NPC) cells, and lymphoblastoid cell lines (LCLs).
256 a panel of iPSCs from 58 well-studied Yoruba lymphoblastoid cell lines (LCLs); 14 of these lines were
257  vitro results in their immortalization into lymphoblastoid cell lines (LCLs); this latency program i
258 s (CTLs) reactivated using EBV-transformed B-lymphoblastoid cells lines (LCLs) contained minor popula
259 eral blood mononuclear cells, monocytes, and lymphoblastoid cell lines, leading to enhanced autophagi
260                          Infection of the JY lymphoblastoid cell line limited the accumulation of a m
261 gh-throughput sequencing data sets for human lymphoblastoid cell lines mapped to the EBV genome.
262 miR-M4 from the MDV-induced lymphoma-derived lymphoblastoid cell line MDCC-HP8.
263  loci in genome-wide expression data sets of lymphoblastoid cell lines (n = 1,830) and were related t
264                                              Lymphoblastoid cell lines obtained from a patient and fr
265 elationship between COMETs and haplotypes in lymphoblastoid cell lines of African and European origin
266  profiling of Epstein-Barr virus-transformed lymphoblastoid cell lines of all 270 individuals genotyp
267                                     In seven lymphoblastoid cell lines of Asian, European and African
268                                     The BJAB lymphoblastoid cell line often serves as a model for B c
269 -infected B lymphocytes and are critical for lymphoblastoid cell line outgrowth.
270 nscription factor MEF2A in 32 distinct human lymphoblastoid cell lines, providing insights into the m
271                                        Human lymphoblastoid cell line (Raji), a poor host for HCV pse
272 lls or NK-cell clones with HLA-C2(+) CCR7(+) lymphoblastoid cell lines resulted in increased CCR7 upt
273                   Using whole-genome DNA and lymphoblastoid cell line RNA sequencing data from 360 Eu
274 common CNVs in a cohort of 50 radiosensitive lymphoblastoid cell lines (RS-LCLs) derived from patient
275  perfectly-matched probes were identified in lymphoblastoid cell-line samples through comparison with
276 entified as eQTL in monocytes, liver tissue, lymphoblastoid cell lines, T cells, and fibroblasts are
277 uencing in ATAC-seq libraries generated from lymphoblastoid cell lines: targeted cleavage of mitochon
278  exhibits higher expression in BLM-sensitive lymphoblastoid cell lines than insensitive cell lines up
279  CAMKK2 in human brains (P=1.1 x 10(-6)) and lymphoblastoid cell lines (the lowest P=8.4 x 10(-6)).
280                      Here we report that, in lymphoblastoid cell lines, the translocation additionall
281                  Furthermore, in the GM12878 lymphoblastoid cell line, these three genes are in a con
282 s, which leads to continuously proliferating lymphoblastoid cell lines through examination of the exp
283 kata-EBV and Raji), and cells from an EBV(+) lymphoblastoid cell line, thus suggesting a specific ass
284 C-seq by sequencing the methylome of a human lymphoblastoid cell line to approximately 8.6x high-qual
285 red heteroplasmies in mtRNA from 446 human B-lymphoblastoid cell lines to their corresponding mtDNA u
286 for PBL-to-atrium; and from 0.81 to 0.98 for lymphoblastoid cell line-to-PBL based on cross-validatio
287 In an expression quantitative trait study in lymphoblastoid cell lines, transcript levels of the MTDH
288 logous dendritic cells and EBV-transformed B-lymphoblastoid cell lines transduced with an adenoviral
289 nd drug sensitivity measurements in 24 human lymphoblastoid cell lines, was applied to a panel of 12
290                             Using the HapMap lymphoblastoid cell lines, we combine 1000 Genomes genot
291                       Using RNA derived from lymphoblastoid cell lines, we find that of 46 informativ
292                    Applying riboHMM to human lymphoblastoid cell lines, we identified 7273 novel codi
293           Using metaphase spreads from human lymphoblastoid cell lines, we previously showed how immu
294 sed gene editing approach in MDV-transformed lymphoblastoid cell lines, we show that MDV-miR-M4, desp
295 y members were enrolled, blood was obtained, lymphoblastoid cell lines were immortalized, deoxyribonu
296  to the establishment of permanently growing lymphoblastoid cell lines, whereas CD40L/IL-4 blasts hav
297  transcription factor binding sites in human lymphoblastoid cell lines, which is comprised of sites c
298 cellular phenotypes in three patient-derived lymphoblastoid cell lines with three variants: p.Gly535A
299 iation in genome-wide gene expression in 107 lymphoblastoid cell lines, with alleles ranging from 15
300 ranscription initiation across several human lymphoblastoid cell lines (Yoruba population) and detect

 
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