ライフサイエンスコーパス: lymphocyte

1 ed clusters of macrophages scattered between lymphocytes.

2 eceptor temporarily upregulated by activated lymphocytes.

3 (+) cell differentiation into CD56(+) innate lymphocytes.

4 trophils, and decreased progenitor cells and lymphocytes.

5 pecification programs of innate and adaptive lymphocytes.

6 eans to redirect the natural properties of T lymphocytes.

7 age trafficking when co-cultured with immune lymphocytes.

8 or activation and persistence of cytotoxic T lymphocytes.

9 et of gamma/delta T-cell receptor-expressing lymphocytes.

10 l blood, and elevated bronchoalveolar lavage lymphocytes.

11 globin, alkaline phosphatase, platelets, and lymphocytes.

12 as partial-body exposures produce fewer such lymphocytes.

13 ear factor of activated T-cells (NF-AT) in T-lymphocytes.

14 subtypes, dendritic cells, granulocytes, and lymphocytes.

15 ation with Epstein-Barr virus (EBV)-positive lymphocytes.

16 ed mechanical differences compared to normal lymphocytes.

17 s while promoting the development of B and T lymphocytes.

18 s well as increased populations of CD73(+) B lymphocytes.

19 n how Hivep3 impacts the maturation of these lymphocytes.

20 achieved 8.2 % gene recombination in mouse T lymphocytes.

21 d activation of antigen-specific cytotoxic T lymphocytes.

22 tinocytes, fibroblasts, dendritic cells, and lymphocytes.

23 in tumor-infiltrating, Fas receptor-positive lymphocytes.

24 umbers of CD4(+) T lymphocytes and CD20(+) B lymphocytes.

25 fic phenotype did not extend to other innate lymphocytes.

26 with their cognate receptors on cytotoxic T lymphocytes.

27 -CD40L interactions between DC1 and CD4(+) T lymphocytes.

28 o and in vitro contraction of activated CD8+ lymphocytes.

29 d cells actively engulf invading living Th17 lymphocytes, a process mediated by expression of activat

30 associated with clinical characteristics and lymphocyte aberrations.

31 ethod, we characterized the presence of anti-lymphocyte Abs in the whole cohort of 72 patients, as we

32 ciated invariant T (MAIT) cells are innate T lymphocytes activated by bacteria that produce vitamin B

33 , paradigm for lncRNA-mediated modulation of lymphocyte activation and signaling, with implications f

34 e production, and reduced expression of many lymphocyte activation genes.

35 philic immunoreceptor of the SLAM (signaling lymphocyte activation molecule) family-on either platele

36 cts gene expression in a manner dependent on lymphocyte activation status, contributing to the interi

37 Infiltration of T lymphocytes, activation of microglia, and their interpla

38 sponse of patient-matched tumor infiltrating lymphocytes against antigens diferentially presented aft

39 We characterized lymphocyte and monocyte populations as well as antigen-s

40 of antigen processing and presentation; more lymphocytes and associated cytokines; decreased extracel

41 T lymphocytes but fewer numbers of CD4(+) T lymphocytes and CD20(+) B lymphocytes.

42 intratumoral S100 dendritic cells, and CD8 T lymphocytes and CD57 natural killer cells in the ALNs(-)

43 proach to quantify MiHAs presented by B-cell lymphocytes and determined their expression levels by LC

44 human transcription factor TFIIH, in both B lymphocytes and epithelial cells, we hypothesized that S

45 Electron microscopy of patient-derived lymphocytes and fibroblasts from both patients with VPS1

46 examines interactions between group 2 innate lymphocytes and gastric microbes that enhance IgA produc

47 had significantly increased percentages of T lymphocytes and higher levels of a wide array of inflamm

48 ins (LMPs), is expressed in newly infected B lymphocytes and in post-transplant lymphomas.

49 agy, leading to the survival of autoreactive lymphocytes and increased autoantibody.

50 We visualized the interactions of lymphocytes and LVS-infected macrophages using confocal

51 Furthermore, there were higher lymphocytes and M1 macrophage infiltration in high mutat

52 We show that apoptotic lymphocytes and macrophages release specific metabolites

53 he repression of multiple gene programs in B lymphocytes and maintains the adult B-2 cell fate.

54 between the T-cell receptor of CD4-positive lymphocytes and MHC II on antigen-presenting cells.

55 n conclusion, we identified that circulating lymphocytes and monocytes of individuals simultaneously

56 Significant increases were also apparent for lymphocytes and monocytes, accompanied by locally elevat

57 immune system and is secreted by cytotoxic T lymphocytes and natural killer cells to help eliminate v

58 th ligand 1 (PD-L1) microenvironment in live lymphocytes and selectively labeled within an immunosyna

59 Close proximity between cytotoxic T lymphocytes and tumour cells is required for effective i

60 dicated that CD20(+) B lymphocytes, CD8(+) T lymphocytes, and CD11c(+) cells are susceptible to IAV i

61 enic bacteria, remained nonimmunogenic for T lymphocytes, and increased bioavailability in tear fluid

62 r role in dictating the migratory pattern of lymphocytes, and thus vaccine efficacy in mucosal tissue

63 , respectively, within skin-homing/cutaneous lymphocyte antigen (CLA)(+) versus systemic/CLA(-) T cel

64 ell death 1 ligand 1 (PD-L1) and cytotoxic T lymphocyte antigen 4 (CTLA-4) immune checkpoints led to

65 Despite administration of cytotoxic T lymphocyte antigen 4 (CTLA-4) immunoglobulin (Ig), Qa-1

66 ge-specific effects of CTLA4-Ig (cytotoxic T-lymphocyte antigen 4-Ig), a fusion protein blocking cost

67 ndosome-localized thiolreductase (GILT), and lymphocyte antigen 6 family member E (LY6E), the three c

68 Lymphocyte antigen receptor repertoire deep sequencing d

69 rce of diverse immunoglobulin repertoires, B lymphocytes are an indispensable part of humoral immunit

70 TIPE-deficient T lymphocytes are completely pilot-less: they are unable t

71 B lymphocytes are important players of the adaptive immune

72 embryogenesis and continuing even as matured lymphocytes are primed and educated in adult tissue.

73 Lymphocytes are some of the most motile cells of vertebr

74 Th17 lymphocytes are therefore critical for selective CNS ent

75 eptides derive from human proteins, to which lymphocytes are tolerant.

76 rs in the lamina propria and intraepithelial lymphocytes are unaffected.

77 sults from this study demonstrate that tumor lymphocytes, assessed by TCR repertoire quantification b

78 express the skin homing receptors cutaneous lymphocyte associated antigen and CCR4 (Ccr4), associate

79 mmed death ligand 1 (PD-L1), and cytotoxic T lymphocyte-associated protein 4 (CTLA-4) for cancer immu

80 cell death protein 1 (PD-1) and cytotoxic T lymphocyte-associated protein 4 (CTLA-4), which limit T

81 death ligand 1 (PD-1:PD-L1) and cytotoxic T lymphocyte-associated protein 4 (CTLA-4):B7-1 are among

82 2 received a combination of anticytotoxic T-lymphocyte-associated protein 4 and anti-PD-1 antibodies

83 o follow-up suggests a potential effect on T-lymphocytes at HLA-B(*)55:01.

84 e line is an ideal tool to study cytotoxic T lymphocyte biology and to optimize personalized immunoth

85 e review advances in understanding of innate lymphocyte biology with a focus on skin disease and the

86 pment and/or expansion of immune-suppressive lymphocytes (Bregs and Tregs).

87 quencies of CD19+CD24hiCD38hi transitional B lymphocytes (Bregs) increased from a median of 6% before

88 l mucosal monocytes/macrophages and CD8(+) T lymphocytes but fewer numbers of CD4(+) T lymphocytes an

89 he metabolic shift induced in human CD4(+) T lymphocytes by stimulation is characterized by an upregu

90 Lymphocyte CB(2)depletion also exacerbated JWH133 self-a

91 y infected biopsies indicated that CD20(+) B lymphocytes, CD8(+) T lymphocytes, and CD11c(+) cells ar

92 ining bronchoalveolar lavage (BAL) fluid for lymphocyte cellular analysis, and suggestions for transb

93 ions were made in favor of obtaining BAL for lymphocyte cellular analysis, transbronchial lung cryobi

94 Latest median donor myeloid and lymphocyte chimerism were 100% (range, 0-100) and 100% (

95 isplayed a significant decrease in hepatic B lymphocytes compared to untreated mice as assessed by hi

96 Within FLAgs, the lymphocytes compartmentalize according to their chemokin

97 Lymphocyte conditioned media (CM) induced epithelial-mes

98 subset of TNBCs with high tumor-infiltrating lymphocyte content.

99 (N0 vs rest), number of tumour-infiltrating lymphocytes (continuous variable), subtype (HER2-enriche

100 Immune T lymphocytes control the replication of Francisella withi

101 Low absolute lymphocyte count (ALC) has potential to be a useful and

102 4] x 10(9) cells/L, p = 6.93 x 10(-57)), but lymphocyte count was less consistent (0.016 [0.007, 0.02

103 itive and potentially causal relationship of lymphocyte count with systolic BP and diastolic BP.

104 the curve >0.70), as were low albumin level, lymphocyte count, monocyte count, and ratio of periphera

105 2.1 ug/mL, P = .004) levels, and lower nadir lymphocyte counts (0.09 vs. 0.4 x 10(3) /uL, P = .006).

106 rbidities, more systemic symptoms and higher lymphocyte counts and C-reactive protein levels.

107 e 46 patients who died were older, had lower lymphocyte counts and estimated glomerular filtration ra

108 The lead SNP was also associated with lower lymphocyte counts and in silico follow-up suggests a pot

109 tics of temperature, C-reactive protein, and lymphocyte counts mirrored the remitting/relapsing SARS-

110 erum albumin and cholesterol concentrations, lymphocyte counts, age, body mass index (BMI), complicat

111 group significantly depended on infiltrating lymphocyte counts, with patients who showed both an earl

112 ients with an early hypoxia response but low lymphocyte counts.

113 These TMPs inhibit cytotoxic T lymphocyte (CTL) activity both in vitro and in vivo, and

114 modeling, and suppression of the cytotoxic T lymphocyte (CTL) response.

115 gnature, and the ratio of CD8(+) cytotoxic T lymphocytes (CTL) to CD68(+) macrophages both predict di

116 d infiltration and activation of cytotoxic T lymphocytes (CTLs) and CD103+ cross-presenting dendritic

117 lytic lymphocytes-including CD8+ cytotoxic T lymphocytes (CTLs) and natural killer cells-and regulate

118 ogical synapse formation between cytotoxic T lymphocytes (CTLs) and the target cells they aim to dest

119 n vivo molecular imaging of CD8+ cytotoxic T lymphocytes (CTLs) in response to anti-PD-L1 therapy.

120 teins also occurs from activated cytotoxic T lymphocytes (CTLs) where they have recently been reporte

121 lls to evade killing mediated by cytotoxic T lymphocytes (CTLs), we performed genome-wide CRISPR scre

122 ) expression to evade killing by cytotoxic T lymphocytes (CTLs).

123 resents a new model of peripherin-reactive B lymphocyte-dependent autoimmune neuritis.

124 ic HL (nodular sclerosis, mixed cellularity, lymphocyte-depleted or lymphocyte-rich HL), which accoun

125 ancreatitis, adrenocortical hyperplasia, and lymphocyte depletion of spleen and lymph nodes.

126 tious mononucleosis and cancers, including B lymphocyte-derived Burkitt lymphoma and immunocompromise

127 rized endocervical cells with HIV-1-infected lymphocyte-derived cells, we discovered endocervical+BVA

128 Besides, boldine reduced the Th17-lymphocyte detection and response and increased the Treg

129 etection and response and increased the Treg-lymphocyte detection and response in periodontitis-affec

130 Lymphocyte development is a complex and coordinated path

131 utations in the EOMES gene (12%) involved in lymphocyte development, PI3K-AKT/mTOR (6%), and loss-of-

132 ether antiviral response of peripheral blood lymphocytes differs between HG patients and healthy indi

133 ver, delivering large biologics to primary T lymphocytes directly in vivo is technically challenging

134 Therefore, immune lymphocytes directly limit intracellular bacterial repli

135 CD8(+) tumor-infiltrating lymphocytes displayed low expression of CD2 in the major

136 ntly, we found that peripheral monocytes and lymphocytes do not express substantial amounts of pro-in

137 lipoxin A(4) (LXA(4)) has emerging roles in lymphocyte-driven diseases.

138 that is linked to its latent infection of B lymphocytes, during which virus replication is not suppo

139 y mechanisms by which LXRalphabeta governs T lymphocyte education and illuminate LXRalphabeta's indis

140 The T lymphocyte, especially its capacity for antigen-directed

141 Additionally, SARS-CoV-2-specific memory lymphocytes exhibited characteristics associated with po

142 e of Wnt5A-depleted MDSC, tumor-infiltrating lymphocytes expressed decreased PD-1 and LAG3, suggestin

143 However, adult Ezh2-deficient B lymphocytes expressed Lin28b, which encodes an RNA-bindi

144 e main sources of Ca(2+) influx in mammalian lymphocytes following antigen receptor stimulation are C

145 Moreover, both CD4(+) and CD8(+) T lymphocytes from Mtb infected, metformin treated animals

146 factors, and outcomes of post-HCT AIC and B-lymphocyte function following rituximab.

147 act on the nuclear factor-kappaB pathway and lymphocyte function.

148 ding IL-17, and increased consumption of the lymphocyte growth factor IL-7 before motor onset of HD.

149 of LVS-infected macrophages with LVS-immune lymphocytes halted LVS replication and inhibited the spr

150 B lymphocytes have immunological functions beyond Ab produ

151 Moreover, mice lacking Th17 lymphocytes have reduced BBB leakage, microglial activat

152 ripheral immune cells, such as monocytes and lymphocytes, have also been found to play an important r

153 The molecular mechanisms regulating lymphocyte homing into lymph nodes are only partly under

154 in vivo immune abnormalities consistent with lymphocyte hyperactivity.

155 diated inhibition of RXR Function, PI3K in B lymphocytes, iCOS-iCOSL in T helper cells, and the role

156 repair activities across thousands of human lymphocytes, identifying known and novel cell-type-speci

157 genitors, but these cells maintained their B lymphocyte identity.

158 In a subset of patients, lymphocyte immunophenotyping, quantitative real-time PCR

159 ning was also observed in tumor-infiltrating lymphocytes in a subset of TNBCs with high tumor-infiltr

160 focused on understanding the role played by lymphocytes in autoimmune diseases of the central nervou

161 ents with COPD and monocytes/macrophages and lymphocytes in both patients with COPD and control subje

162 the phenotypes and HBV-specific response of lymphocytes in CHB patients stratified by serum HBsAg le

163 mpaired thymic development of PLZF(+) innate lymphocytes in germ-free (GF) neonatal mice is restored

164 eletion of the tumor suppressor TRAF3 from B lymphocytes in mice leads to the prolonged survival of m

165 esults suggest a substantial role for CD8(+) lymphocytes in suppressing the latency reversal effect o

166 ning demonstrated the presence of CD8+CD161+ lymphocytes in the arterial wall of two unruptured intra

167 , a large proportion of islet-infiltrating B lymphocytes in the NOD mouse model of T1D produce Abs di

168 vely correlated with the density of detected lymphocytes in tumor tissues, while expression of the ex

169 essential for antigen recognition by T and B lymphocytes in viral-host interaction.

170 ematopoietic cells was observed in cytolytic lymphocytes-including CD8+ cytotoxic T lymphocytes (CTLs

171 ociated with decreased IFNgamma response and lymphocyte infiltration in patient tumors.

172 owed both an early hypoxia response and high lymphocyte infiltration levels exhibiting significantly

173 ed with ISGs, CD4, CD8A, CD8B, and the tumor-lymphocyte infiltration phenotype.

174 Gene signature analysis confirmed the lymphocyte infiltration profile consistent with the hist

175 focal lymphocytes with moderate perivascular lymphocyte infiltration.

176 induces cell-cycle arrest in MF/SS malignant lymphocytes, inhibiting their proliferation but not thei

177 ment of innate (NK) and innate-like adaptive lymphocytes (iNKT, MAIT, and Vdelta2(+) gammadelta T cel

178 the exocrine glands become infiltrated with lymphocytes instigating severe damage to the salivary an

179 cal intracellular signaling molecule for T-B lymphocyte interactions, drives T follicular helper (Tfh

180 flammatory cells including neutrophils and T lymphocytes into the skin and hyperkeratosis/hyperplasia

181 oxygen and nutrients and recruit circulating lymphocytes into the SLO parenchyma, where they encounte

182 atments, and by adoptive transfer of treated lymphocytes into uninfected humanized mice.

183 , blood film microscopy to detect vacuolated lymphocytes is a rapid, readily accessible, and sensitiv

184 Engineering T lymphocytes is an emerging approach in a variety of biom

185 k alleles, and drug-induced proliferation of lymphocytes isolated from patients support a role for th

186 kines release (cytokine storm) and loss of T lymphocytes (leukopenia) characterize the most aggressiv

187 functional hypoxia imaging and infiltrating lymphocyte levels as a potential predictor for treatment

188 Lymphocyte lineage specification and commitment requires

189 entional T cells, a heterogeneous class of T lymphocytes (MAIT, gammadeltaT, and iNKT cells) with pot

190 In T lymphocytes, MAL is found at the immunological synapse a

191 hoid organ that plays an essential role in T lymphocyte maturation and selection during development o

192 CD8+CD161+ lymphocytes may have an important role in the inflammato

193 achieved with immunotherapy that relies on T lymphocyte-mediated recognition of tumor antigens.

194 Lymphocyte migration is essential for adaptive immune su

195 A subset of tumor-associated lymphocytes, most prominent in uveal melanoma, showed po

196 of chronic inflammatory cells, consisting of lymphocytes (n = 9), plasma cells (n = 6), and histiocyt

197 ings reveal novel functions of cullin 3 in B lymphocytes, namely regulating CD22 surface expression a

198 ecular pilot, akin to a ship's pilot, guides lymphocyte navigation, the nature of this pilot is unkno

199 a plasmacytoid dendritic cell-type I IFN-T/B lymphocyte network.

200 74% neutrophils [normal range, 40%-80%], 24% lymphocytes [normal range, 20%-40%], and 2% eosinophils

201 Developing lymphocytes of jawed vertebrates cleave and combine dist

202 report that mice deficient in conventional T lymphocytes or recombination-activating gene (Rag) faile

203 through hindering access to key epitopes by lymphocytes or through altering immune responses by bind

204 ological data on stromal tumour-infiltrating lymphocytes, PAM50 subtypes, and expression of 55 genes

205 ograms, and these cells acquired a fetal B-1 lymphocyte phenotype in vitro and in vivo.

206 8 HIV-negative MSM and 72 HIV-negative MSW T-lymphocyte phenotyping was performed 3 times biennially.

207 B lymphocytes play a central role in host immunity.

208 B lymphocytes play a critical role in adaptive immunity.

209 ies that highlight the important role that B lymphocytes play in cardiac and vascular disease.

210 Decidual NK cells (dNK) are the main lymphocyte population in early pregnancy decidual mucosa

211 killer T (iNKT) cells are a rare innate-like lymphocyte population that recognizes glycolipids presen

212 ng revealed reductions and rapid recovery in lymphocyte populations across lymphocyte subsets.

213 Mice developed expanded germinal center B lymphocyte populations as in other models of AID deficie

214 cy and provide preliminary insights into the lymphocyte populations that are key for the antiviral re

215 assess the clonal richness and diversity of lymphocyte populations; to track clone members over time

216 sical Hodgkin lymphoma, the disease-defining lymphocyte-predominant cells in NLPHL are consistently p

217 ccount for the majority of cases, or nodular lymphocyte-predominant HL.

218 nscription factors, specifically in murine B lymphocyte progenitors, but these cells maintained their

219 had significantly (p <= 0.05) increased CD4+ lymphocyte programmed death-1 and monocyte programmed de

220 creased neutrophils, accompanied by hampered lymphocyte proliferation but increased monocyte phagocyt

221 Lymphocyte proliferation was inhibited after 2 h of nora

222 Low total helper T lymphocyte proportions and low naive helper T cell propo

223 ortant role for a myelin protein, myelin and lymphocyte protein (MAL), in the process of cell-to-cell

224 il to lymphocyte ratio (NLR) and monocyte to lymphocyte ratio (MLR) are respectively associated with

225 High neutrophil to lymphocyte ratio (NLR) and monocyte to lymphocyte ratio

226 rognostic score (mGPS) and the neutrophil to lymphocyte ratio (NLR) and survival in patients with lun

227 In addition, the derived neutrophil-to-lymphocyte ratio and platelet count were significantly a

228 r level, neutrophil count, and neutrophil-to-lymphocyte ratio were all predictive of mortality (area

229 ory rate, white blood cell count, neutrophil/lymphocyte ratio, and lactate dehydrogenase level.

230 se (MELD) scores, AFP levels, and neutrophil-lymphocyte ratios (NLR); were more likely to have tumors

231 approaches, including peptide elution, mixed lymphocyte reaction, T-cell receptor (TCR) deep sequenci

232 Reltecimod, a CD 28 T-lymphocyte receptor mimetic, inhibits T-cell stimulation

233 oligomerization domain-containing receptors, lymphocyte receptors, and cytokine receptors.

234 ngs reveal a role for vascular structures in lymphocyte recirculation through the spleen, indicating

235 cells during EAU resulting in alteration of lymphocyte repertoire by increasing levels of autoreacti

236 critical for central tolerance and diverse T-lymphocyte repertoire development, to provide lifelong d

237 d a TMP-specific H-2K(b)-restricted CD8(+) T lymphocyte response upon immunotherapy with cyclophospha

238 nodes induce activation of tumor-specific T-lymphocyte responses that can result in cytolytic target

239 a, and phosphorylated STAT3 as well as swift lymphocyte restoration.

240 Conditional deletion of Tph1 in lymphocytes resulted in selective impairment of ILC2(INF

241 s, mixed cellularity, lymphocyte-depleted or lymphocyte-rich HL), which account for the majority of c

242 e of the mesenteric adipose streaks contains lymphocyte-rich organoids comprised of a highly compacte

243 Antihuman lymphocyte serum (L1S1) antibody completely prevented ac

244 d progressively declined, whereas CD8+CD161+ lymphocytes significantly increased at days 5-9.

245 lymph node structures and therefore specific lymphocyte subpopulations.

246 However, they are the most abundant T-lymphocyte subset in some epithelial barriers such as mo

247 However, function of STAT3 in the B lymphocyte subset is not well understood.

248 Absolute numbers of lymphocyte subsets and serum immunoglobulin levels were

249 id recovery in lymphocyte populations across lymphocyte subsets.

250 mbers over time, between tissues, and across lymphocyte subsets; to detect clonal expansion; and to d

251 thway are upregulated on tumour-infiltrating lymphocytes, suggesting that IL-18 therapy could enhance

252 ses breast cancer cells to evade cytotoxic T-lymphocyte surveillance, which leads to tumor growth.

253 n investigated in detail; CD4(+) cytotoxic T lymphocytes (suspected of promoting disease) and a speci

254 ears, with HIV viral load <50 copies/mL, and lymphocyte T-CD4 level >=200 cells/uL in the past 24 and

255 Cytotoxic T lymphocytes (T) and natural killer cells are the main cy

256 rates relies on the presence of B and T cell lymphocytes that aggregate in specific body sites to for

257 the context of cellular therapies may yield lymphocytes that are able to withstand harsh tumor metab

258 s several common pathways in MF/SS malignant lymphocytes that are associated with control of cell-cyc

259 s a cell surface receptor present on B and T lymphocytes that is universally and strongly expressed o

260 lta T cells (gammadeltaT17 cells) are innate lymphocytes that participate in type 3 immune responses

261 te lymphoid cells (ILCs) are tissue-resident lymphocytes that promote immunity to pathogens at mucosa

262 rease of dendritic cells and infiltration of lymphocytes throughout the pancreatic tumor compared to

263 bility to promote the continuous growth of B lymphocytes, thus providing significant new insight into

264 irect competition between tumor-infiltrating lymphocytes (TIL) and cancer cells for metabolic resourc

265 how that P2X7 activity in tumor-infiltrating lymphocytes (TIL) induces cellular senescence and limits

266 nvironment harboring more tumor infiltrating lymphocytes (TILs) and up-regulation of inflammatory sig

267 vivo-expanded autologous tumor-infiltrating lymphocytes (TILs) can mediate complete regression of ce

268 expansion and function of tumor-infiltrating lymphocytes (TILs) for treating cancer patients with ado

269 epitopes recognized by tumor-infiltrating T lymphocytes (TILs) from eight melanoma patients for all

270 The distribution of tumor-infiltrating lymphocytes (TILs) within the tumor microenvironment pro

271 (ACT) of ex vivo expanded tumor-infiltrating lymphocytes (TILs).

272 stochemical stainings for tumor-infiltrating lymphocytes, tissue-based hypoxia, and microvascular mar

273 t tumors induce de novo steroidogenesis in T lymphocytes to evade anti-tumor immunity.

274 ection and differentiation of the responding lymphocytes to generate a multitude of cell fates.

275 nly serves to amplify the number of specific lymphocytes to mount a robust protective response to the

276 For CD8 T lymphocytes to mount responses to cancer and virally-inf

277 Trafficking of alpha(4)beta(7)-expressing lymphocytes to the gut is mediated by MAdCAM, the natura

278 mph node addressin, and the extravasation of lymphocytes to the omental and mesenteric adipose tissue

279 hil interactions during migration on overall lymphocyte trafficking patterns in confined environments

280 erous genes, including genes encoding innate lymphocyte transcription factors, specifically in murine

281 lipidoids that are particularly potent in T lymphocytes transfection.

282 Regulatory T lymphocytes (Treg) play an important role in preventing

283 intervention induces functional regulatory T lymphocytes (Tregs) in mouse lung experimentally inflame

284 Dividing lymphocytes typically rely on aerobic glycolysis over ox

285 other NCRs that are expressed on activated T lymphocytes, V-type immunoglobulin domain-containing sup

286 ity of neutrophils to recirculate, much like lymphocytes via L-selectin and high endothelial venules

287 Co-localization of B- and T-lymphocytes was observed.

288 Cultured lymphocytes were analyzed for cytokine secretion profile

289 Subsequently, vaccine-primed lymphocytes were collected and reinfused after a standar

290 Blood lymphocytes were quantified by flow cytometry and antige

291 the evidence for clonal expansion in innate lymphocytes, which has primarily been observed in natura

292 Notably, transfection of the affected lymphocytes with MAGT1 mRNA restored both N-glycosylatio

293 Skin biopsy revealed acanthosis and focal lymphocytes with moderate perivascular lymphocyte infilt

294 tions, neutrons produce more heavily-damaged lymphocytes with multiple micronuclei/binucleated cell,

295 in vitro transduction of normal human CD4+ T lymphocytes with NPM-ALK results in their immortalizatio

296 atural killer T (iNKT) cells are innate-like lymphocytes with unique signaling requirements for their

297 ng the parallels between innate and adaptive lymphocytes, with a particular focus on NK cells and CD8

298 analysis of specific migratory behaviors of lymphocytes within the tissue microenvironment can provi

299 ural killer T (iNKT) cells are self-reactive lymphocytes, yet how this lineage attains self-tolerance

300 that sorting the most migratory cytotoxic T lymphocytes yields a pool of cells with enhanced cytotox