ライフサイエンスコーパス: lymphocytic choriomeningitis

1 NA viruses that cause human diseases such as lymphocytic choriomeningitis, Bolivian hemorrhagic fever

2 clearance of murine gamma-herpesvirus 68 and lymphocytic choriomeningitis clone 13 and reversed T cel

3 IgG-antigen complexes formed during chronic lymphocytic choriomeningitis infection can interfere wit

4 f HIV infection in humans and during chronic lymphocytic choriomeningitis infection in mice.

5 lls were demonstrated in vivo during chronic lymphocytic choriomeningitis infection.

6 and therapy, we focused on two RNA viruses: lymphocytic choriomeningitis (LCMV) and influenza (Flu).

7 s of coinfection, including the well-studied lymphocytic choriomeningitis (LCMV) and Pichinde (PICV)

8 for diabetes antigen tetramers and to LCMV (lymphocytic choriomeningitis)-reactive CD8+ T cells.

9 his concept, we have developed a recombinant lymphocytic choriomeningitis virus (LCMV) (rLCMVDeltaGP/

10 oteins of all known pathogenic arenaviruses, lymphocytic choriomeningitis virus (LCMV) and Lassa, Jun

11 essing of GPC from the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) and LASV, whic

12 infection with two natural mouse pathogens, lymphocytic choriomeningitis virus (LCMV) and murine cyt

13 -deficient (Prf1(--)) mice are infected with lymphocytic choriomeningitis virus (LCMV) and secondary

14 T cells were generated after infection with lymphocytic choriomeningitis virus (LCMV) and that these

15 Here we show that the arenaviruses lymphocytic choriomeningitis virus (LCMV) and the clinic

16 eoproteins (NPs) of the Old World arenavirus lymphocytic choriomeningitis virus (LCMV) and the New Wo

17 ce with Leishmania major and 2 wk later with lymphocytic choriomeningitis virus (LCMV) and then monit

18 Mice were infected with lymphocytic choriomeningitis virus (LCMV) and treated wi

19 topes from unrelated and pathogenic viruses, lymphocytic choriomeningitis virus (LCMV) and vaccinia v

20 te viral infections, such as infections with lymphocytic choriomeningitis virus (LCMV) and vaccinia v

21 In this study, using lymphocytic choriomeningitis virus (LCMV) and Zika virus

22 yelin oligodendrocyte glycoprotein (MOG) and lymphocytic choriomeningitis virus (LCMV) antigens, resp

23 Using lymphocytic choriomeningitis virus (LCMV) as a model of

24 owing infection with a persistent variant of lymphocytic choriomeningitis virus (LCMV) but have no im

25 We show that lymphocytic choriomeningitis virus (LCMV) can also inhib

26 Lymphocytic choriomeningitis virus (LCMV) can cause acut

27 Lymphocytic choriomeningitis virus (LCMV) can establish

28 ablishment of life-long chronic infection by lymphocytic choriomeningitis virus (LCMV) Cl 13 but does

29 and hepatitis C virus or those of mice with lymphocytic choriomeningitis virus (LCMV) clone 13 (CL13

30 virus (ECTV) when chronically infected with lymphocytic choriomeningitis virus (LCMV) clone 13 (CL13

31 ly shown that mice chronically infected with lymphocytic choriomeningitis virus (LCMV) clone 13 (CL13

32 nic phase of infection with HIV in humans or lymphocytic choriomeningitis virus (LCMV) clone 13 in mi

33 ) is maximally induced on APCs at day 2 post-lymphocytic choriomeningitis virus (LCMV) clone 13 infec

34 effector CD8 T cells from mice infected with lymphocytic choriomeningitis virus (LCMV) clone 13 into

35 During chronic infection with lymphocytic choriomeningitis virus (LCMV) clone 13, miR-

36 aride (LPS) with that of a persistent virus, lymphocytic choriomeningitis virus (LCMV) clone 13, on e

37 Lymphocytic choriomeningitis virus (LCMV) clone 13, whic

38 rst 12-24 hours after mice are infected with lymphocytic choriomeningitis virus (LCMV) clone 13, whic

39 from a septic event are more susceptible to lymphocytic choriomeningitis virus (LCMV) clone-13 infec

40 toire response against a massively exhausted lymphocytic choriomeningitis virus (LCMV) epitope.

41 report that mice persistently infected with lymphocytic choriomeningitis virus (LCMV) exhibit a seve

42 this study we found that mice infected with lymphocytic choriomeningitis virus (LCMV) exhibit global

43 rmore, pDC-deficient animals failed to clear lymphocytic choriomeningitis virus (LCMV) from hematopoi

44 Mice immunized with H(2)O(2)-inactivated lymphocytic choriomeningitis virus (LCMV) generated cyto

45 -defective adenovirus vectors expressing the lymphocytic choriomeningitis virus (LCMV) glycoprotein (

46 Ad5, Ad26, Ad35, and Ad48 vectors expressing lymphocytic choriomeningitis virus (LCMV) glycoprotein (

47 we immunized mice with Ad5 vectors encoding lymphocytic choriomeningitis virus (LCMV) glycoprotein (

48 enic CD4(+) and CD8(+) T cells responding to lymphocytic choriomeningitis virus (LCMV) identified mul

49 onse during acute primary infection with the lymphocytic choriomeningitis virus (LCMV) in mice is sig

50 n with hepatitis B and C virus in humans and lymphocytic choriomeningitis virus (LCMV) in mice.

51 viruses pseudotyped with the glycoprotein of lymphocytic choriomeningitis virus (LCMV) in vitro.

52 ulated on virus-specific CTLs during chronic lymphocytic choriomeningitis virus (LCMV) infection and

53 in mice changes the outcome to a subsequent lymphocytic choriomeningitis virus (LCMV) infection and

54 Cs) is amplified during chronic versus acute lymphocytic choriomeningitis virus (LCMV) infection and

55 d the role of IAPs in T-cell immunity during lymphocytic choriomeningitis virus (LCMV) infection by p

56 Although cellular immunity to acute lymphocytic choriomeningitis virus (LCMV) infection has

57 Recent studies in chronic lymphocytic choriomeningitis virus (LCMV) infection have

58 IL-10 production over the course of chronic lymphocytic choriomeningitis virus (LCMV) infection in a

59 ced in both CD4(+) and CD8(+) T cells during lymphocytic choriomeningitis virus (LCMV) infection in a

60 ls contribute to establishment of persistent lymphocytic choriomeningitis virus (LCMV) infection in m

61 ector function of CD8 T cells during chronic lymphocytic choriomeningitis virus (LCMV) infection in m

62 t CD70 blockade during an acute or a chronic lymphocytic choriomeningitis virus (LCMV) infection in m

63 In contrast to MCMV, lymphocytic choriomeningitis virus (LCMV) infection in m

64 Lymphocytic choriomeningitis virus (LCMV) infection indu

65 Using acute and chronic lymphocytic choriomeningitis virus (LCMV) infection mode

66 Using the lymphocytic choriomeningitis virus (LCMV) infection mode

67 ll activation kinetics and viral loads after lymphocytic choriomeningitis virus (LCMV) infection of C

68 this study, we used a mouse model of chronic lymphocytic choriomeningitis virus (LCMV) infection to a

69 of virus-specific CD8 T cells during chronic lymphocytic choriomeningitis virus (LCMV) infection to e

70 se ranging from aseptic meningitis following lymphocytic choriomeningitis virus (LCMV) infection to h

71 The primary CD8(+) T-cell response to acute lymphocytic choriomeningitis virus (LCMV) infection was

72 found that during the first week of chronic lymphocytic choriomeningitis virus (LCMV) infection, bef

73 (+) T cells are essential for clearance of a lymphocytic choriomeningitis virus (LCMV) infection, but

74 ty, Hegazy et al. report that in response to lymphocytic choriomeningitis virus (LCMV) infection, ful

75 During persistent lymphocytic choriomeningitis virus (LCMV) infection, IFN

76 In a mouse model of persistent lymphocytic choriomeningitis virus (LCMV) infection, it

77 (+) T-cell formation following immunization, lymphocytic choriomeningitis virus (LCMV) infection, or

78 evelop fatal pathology during early systemic lymphocytic choriomeningitis virus (LCMV) infection, sug

79 nt mice exhibit reduced IFN-I responses upon lymphocytic choriomeningitis virus (LCMV) infection, whi

80 differentiation in vitro and in vivo during lymphocytic choriomeningitis virus (LCMV) infection.

81 e subsets to HLH-like disease severity after lymphocytic choriomeningitis virus (LCMV) infection.

82 induction of exhaustion in mice with chronic lymphocytic choriomeningitis virus (LCMV) infection.

83 viral replication in the context of chronic lymphocytic choriomeningitis virus (LCMV) infection.

84 d IL-21 production at late stages of chronic lymphocytic choriomeningitis virus (LCMV) infection.

85 D-L1) blockade in the mouse model of chronic lymphocytic choriomeningitis virus (LCMV) infection.

86 y reproduced the manifestations of HLH after lymphocytic choriomeningitis virus (LCMV) infection.

87 of CD8 T cells are virus specific following lymphocytic choriomeningitis virus (LCMV) infection.

88 during the early stages of acute and chronic lymphocytic choriomeningitis virus (LCMV) infection.

89 worldwide-distributed prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) is a neglected

90 worldwide-distributed prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) is a neglected

91 e globally distributed prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) is a neglected

92 worldwide-distributed prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) is an importan

93 dult mice with the clone 13 (CL13) strain of lymphocytic choriomeningitis virus (LCMV) is extensively

94 The prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) is widely used

95 g (ARM) or chronic Clone 13 (C13) strains of lymphocytic choriomeningitis virus (LCMV) led to two dis

96 Ins2-GP(Tg) mice injected with lymphocytic choriomeningitis virus (LCMV) lost islet sym

97 lowing infection with the clone 13 strain of lymphocytic choriomeningitis virus (LCMV) or influenza v

98 In mice infected with lymphocytic choriomeningitis virus (LCMV) or Listeria mo

99 evidence that infection of mice with either lymphocytic choriomeningitis virus (LCMV) or pneumonia v

100 anasal infection with the systemic pathogens lymphocytic choriomeningitis virus (LCMV) or vaccinia vi

101 t isografts using RIP-LCMV mice expressing a lymphocytic choriomeningitis virus (LCMV) protein in the

102 Systemic low-dose infection of mice with lymphocytic choriomeningitis virus (LCMV) results in mas

103 l differentiation after acute infection with lymphocytic choriomeningitis virus (LCMV) strain Armstro

104 combinant antigen variant-expressing chronic lymphocytic choriomeningitis virus (LCMV) strains, we un

105 generated in mice persistently infected with lymphocytic choriomeningitis virus (LCMV) suppressed mul

106 The lymphocytic choriomeningitis virus (LCMV) system constit

107 d mice persistently infected from birth with lymphocytic choriomeningitis virus (LCMV) to demonstrate

108 We used the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) to develop a g

109 The recombinant engineering of trisegmented lymphocytic choriomeningitis virus (LCMV) to express two

110 ted the ability of the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) to interfere w

111 ility of the prototypic Old World arenavirus lymphocytic choriomeningitis virus (LCMV) to interfere w

112 cine (HB-101) consisting of 2 nonreplicating lymphocytic choriomeningitis virus (LCMV) vectors expres

113 ion-experienced T(regs) generated upon acute Lymphocytic Choriomeningitis Virus (LCMV) WE and Vaccini

114 Lymphocytic choriomeningitis virus (LCMV) WE variant 2.2

115 -cell responses to chimeric vaccines against lymphocytic choriomeningitis virus (LCMV) were assessed

116 leoprotein (NP) of the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) with the least

117 uced by chronic infections such as HIV, HPV, lymphocytic choriomeningitis virus (LCMV), and schistoso

118 tion) of the NP of the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV), as well as th

119 costimulatory molecule expression induced by lymphocytic choriomeningitis virus (LCMV), CD28/B7-media

120 per that the prototype member in the family, lymphocytic choriomeningitis virus (LCMV), disables the

121 deficient (Prf1(-/-)) mice are infected with lymphocytic choriomeningitis virus (LCMV), disease is dr

122 The prototypic arenavirus, lymphocytic choriomeningitis virus (LCMV), is a model fo

123 worldwide-distributed prototypic arenavirus, lymphocytic choriomeningitis virus (LCMV), is a neglecte

124 d by coinfecting mice with L. guyanensis and lymphocytic choriomeningitis virus (LCMV), or the sand f

125 We infected mice with chronic lymphocytic choriomeningitis virus (LCMV), performed RNA

126 The prototypic arenavirus, lymphocytic choriomeningitis virus (LCMV), provides inve

127 ruses, including Ebola virus, Marburg virus, lymphocytic choriomeningitis virus (LCMV), rabies virus,

128 Lymphocytic choriomeningitis virus (LCMV), the prototype

129 us (IAV) and in memory phase challenged with lymphocytic choriomeningitis virus (LCMV), which we show

130 T(M) from lymphocytic choriomeningitis virus (LCMV)-immune and H60

131 ate protein (AP)-fed controls, LP feeding in lymphocytic choriomeningitis virus (LCMV)-immune mice re

132 In this environment, lymphocytic choriomeningitis virus (LCMV)-infected iFRCs

133 Here, we show that lymphocytic choriomeningitis virus (LCMV)-specific CD8(+

134 Here, we show that lymphocytic choriomeningitis virus (LCMV)-specific CD8+

135 duced expression of CCL21 in murine spleens, lymphocytic choriomeningitis virus (LCMV)-specific T cel

136 protect mice against infection with chronic lymphocytic choriomeningitis virus (LCMV).

137 n the S segment of the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV).

138 ntiviral effector T cells postinfection with lymphocytic choriomeningitis virus (LCMV).

139 itro and in vivo during acute infection with lymphocytic choriomeningitis virus (LCMV).

140 oma cells that replicate the negative-strand lymphocytic choriomeningitis virus (LCMV).

141 were challenged with the Armstrong strain of lymphocytic choriomeningitis virus (LCMV).

142 protection against viral infection, such as lymphocytic choriomeningitis virus (LCMV).

143 eted mice infected with the mouse arenavirus lymphocytic choriomeningitis virus (LCMV).

144 T) donor mice and infected the chimeras with lymphocytic choriomeningitis virus (LCMV).

145 mphohistiocytosis (HLH) after infection with lymphocytic choriomeningitis virus (LCMV).

146 fection of WASP-deficient (WAS KO) mice with lymphocytic choriomeningitis virus (LCMV).

147 pe I IFN response to many viruses, including lymphocytic choriomeningitis virus (LCMV).

148 eficient (Prf1 (-/-)) mice are infected with lymphocytic choriomeningitis virus (LCMV).

149 iral loads in mice chronically infected with lymphocytic choriomeningitis virus (LCMV).

150 -Z interaction for the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV).

151 Ia and class II molecules were infected with lymphocytic choriomeningitis virus (LCMV).

152 ells generated after systemic infection with lymphocytic choriomeningitis virus (LCMV).

153 ctivating TCR signals after clearance of the lymphocytic choriomeningitis virus (LCMV).

154 f cells) during acute infection of mice with lymphocytic choriomeningitis virus (LCMV).

155 uring acute, but not chronic, infection with lymphocytic choriomeningitis virus (LCMV).

156 e in perforin-deficient mice is triggered by lymphocytic choriomeningitis virus (LCMV).

157 ry pathway in mice chronically infected with lymphocytic choriomeningitis virus (LCMV).

158 D8(+) T cells following acute infection with lymphocytic choriomeningitis virus (LCMV).

159 on with vesicular stomatitis virus (VSV) and lymphocytic choriomeningitis virus (LCMV).

160 tivity against three distinct ssRNA viruses: lymphocytic choriomeningitis virus (LCMV); influenza A v

161 replaced with a variant glycoprotein of the lymphocytic choriomeningitis virus (LCMV-GP), creating a

162 t viruses, murine cytomegalovirus (MCMV) and lymphocytic choriomeningitis virus (LCMV; Cl13), in thei

163 infected Il18-transgenic (Il18tg) mice with lymphocytic choriomeningitis virus (LCMV; strain Armstro

164 Ad5) prime followed by replication-defective lymphocytic choriomeningitis virus (rLCMV) boost elicite

165 We constructed recombinant lymphocytic choriomeningitis virus (rLCMV) featuring eit

166 at recombinants of the prototypic arenavirus lymphocytic choriomeningitis virus (rLCMVs), whose S-IGR

167 maintained on CD8(+) T cells during chronic lymphocytic choriomeningitis virus and hepatitis C virus

168 complex I was also required for infection of lymphocytic choriomeningitis virus and human parainfluen

169 4 failed to expand and to protect from acute lymphocytic choriomeningitis virus and Leishmania major

170 s and we describe its use in the contexts of lymphocytic choriomeningitis virus and Mycobacterium tub

171 d CD8(+) T cell responses to infections with lymphocytic choriomeningitis virus and the intracellular

172 only two mammarenaviruses, the widely spread lymphocytic choriomeningitis virus and the recently desc

173 luenza A virus and EBV in humans and between lymphocytic choriomeningitis virus and vaccinia virus in

174 infection of a naive C57BL/6 recipient with lymphocytic choriomeningitis virus and vaccinia virus, f

175 t in Atf3 showed enhanced viral clearance in lymphocytic choriomeningitis virus and vesicular stomati

176 When administered at the beginning of lymphocytic choriomeningitis virus Armstrong infection a

177 yzed viperin expression in vivo during acute lymphocytic choriomeningitis virus Armstrong infection,

178 cell-dependent antiviral immunity using the lymphocytic choriomeningitis virus Armstrong strain acut

179 Using lymphocytic choriomeningitis virus Armstrong to probe th

180 e was followed by acute viral infection with lymphocytic choriomeningitis virus Armstrong.

181 e infection with the natural murine pathogen lymphocytic choriomeningitis virus become more resistant

182 of type I IFNs, and in persistently infected lymphocytic choriomeningitis virus carrier mice, which c

183 pecific CD8 T cells during acute and chronic lymphocytic choriomeningitis virus challenges, but did n

184 Pathogenic HIV or lymphocytic choriomeningitis virus chronic infections di

185 We find that the Old World arenaviruses lymphocytic choriomeningitis virus clone 13 (LCMV Cl13)

186 n PTPN22 resist chronic viral infection with lymphocytic choriomeningitis virus clone 13 (LCMV cl13).

187 ls without altering Slamf6(+) numbers during lymphocytic choriomeningitis virus clone 13 infection.

188 ssential for viral control during persistent lymphocytic choriomeningitis virus clone 13 infection.

189 s control and prevented chronic infection in lymphocytic choriomeningitis virus clone 13- and reduced

190 The G protein from lymphocytic choriomeningitis virus endowed VSV with the

191 isella strains expressing well-characterized lymphocytic choriomeningitis virus epitopes, we found th

192 recombinant adenovirus vector expressing the lymphocytic choriomeningitis virus glycoprotein (LCMVgp)

193 ted phenotype during chronic infections with lymphocytic choriomeningitis virus in mice and hepatitis

194 (but not a slow-spreading acute) isolate of lymphocytic choriomeningitis virus induced large-scale m

195 Infection with lymphocytic choriomeningitis virus induces monopoiesis i

196 ay) on early T cell attrition in response to lymphocytic choriomeningitis virus infection and during

197 exhibited a normal acute response (day 8) to lymphocytic choriomeningitis virus infection but generat

198 Consistent with this finding, we show that lymphocytic choriomeningitis virus infection can directl

199 ined T cell responses to acute or persistent lymphocytic choriomeningitis virus infection in IFN-lamb

200 erived T cells are comparable in controlling lymphocytic choriomeningitis virus infection in mice and

201 regulates T cell dysfunction during chronic lymphocytic choriomeningitis virus infection in mice, an

202 ncoding the TGF-beta receptor during chronic lymphocytic choriomeningitis virus infection in mice, an

203 In a model of chronic lymphocytic choriomeningitis virus infection in mice, we

204 each stable levels of memory following acute lymphocytic choriomeningitis virus infection in mice.

205 notherapeutic effects of IL-7 during chronic lymphocytic choriomeningitis virus infection in mice.

206 s in virus-specific CD8 T cells during acute lymphocytic choriomeningitis virus infection in mice.

207 sential for the control of acute and chronic lymphocytic choriomeningitis virus infection in the join

208 c memory CD4(+) T cell subpopulations in the lymphocytic choriomeningitis virus infection model, we f

209 We used a murine model of HLH, involving lymphocytic choriomeningitis virus infection of perforin

210 Here we demonstrate that chronic lymphocytic choriomeningitis virus infection rapidly tri

211 cell-extrinsic manner early following acute lymphocytic choriomeningitis virus infection to suppress

212 rvation of antiviral immune responses, acute lymphocytic choriomeningitis virus infection was used.

213 pDC development and serum IFN-I responses to lymphocytic choriomeningitis virus infection were augmen

214 Here, we show that during chronic murine lymphocytic choriomeningitis virus infection, activation

215 ults in reduced viral clearance in models of lymphocytic choriomeningitis virus infection, and also p

216 y respond during the early stages of chronic lymphocytic choriomeningitis virus infection, and that t

217 after protein immunization; however, during lymphocytic choriomeningitis virus infection, B cells in

218 In chronic lymphocytic choriomeningitis virus infection, blockade o

219 Upon lymphocytic choriomeningitis virus infection, Cmah(-/-)

220 , as dual TCR cells predominated response to lymphocytic choriomeningitis virus infection, comprising

221 rrantly upregulated during memory to chronic lymphocytic choriomeningitis virus infection, limiting f

222 Following acute lymphocytic choriomeningitis virus infection, memory CD8

223 Here we show that, following lymphocytic choriomeningitis virus infection, resident m

224 Using the experimental mouse model of lymphocytic choriomeningitis virus infection, we demonst

225 Following lymphocytic choriomeningitis virus infection, we found m

226 nd naive CD8(+) T cells to acute and chronic lymphocytic choriomeningitis virus infection, we show th

227 4hi CD8 and CD4 T cells at days 2 to 3 after lymphocytic choriomeningitis virus infection, when type

228 CD8(+) T cells to mount a robust response to lymphocytic choriomeningitis virus infection, with both

229 sion and increased cytokine production after lymphocytic choriomeningitis virus infection, yet DGK-de

230 virus infection to those in acute or chronic lymphocytic choriomeningitis virus infection.

231 cked gp33-specific CD8(+) T cells during RRV-lymphocytic choriomeningitis virus infection.

232 ere also found in mouse liver within 24 h of lymphocytic choriomeningitis virus infection.

233 i-CD20 before or different times after acute lymphocytic choriomeningitis virus infection.

234 d day-8 effector CD8 T cells following acute lymphocytic choriomeningitis virus infection.

235 e a crucial function in viral clearance upon lymphocytic choriomeningitis virus infection.

236 ptional profiles and chromatin states during lymphocytic choriomeningitis virus infection.

237 ry CD8 T-cell generation and responses after lymphocytic choriomeningitis virus infection.

238 exhaustion by Tim-3 and PD-1 during chronic lymphocytic choriomeningitis virus infection.

239 ed the early T cell attrition resulting from lymphocytic choriomeningitis virus infection.

240 ephalitis, and defective immune responses to lymphocytic choriomeningitis virus infection.

241 risk of developing HLH immunopathology after lymphocytic choriomeningitis virus infection.

242 TE/TEM and TRM subsets was overcome by acute lymphocytic choriomeningitis virus infection; neverthele

243 isingly, they also show that chronic HIV and lymphocytic choriomeningitis virus infections have a ver

244 G (IgG), and were unable to resolve chronic lymphocytic choriomeningitis virus infections.

245 CD8 T cells responding to acute and chronic lymphocytic choriomeningitis virus infections.

246 ated stimulation during primary responses to lymphocytic choriomeningitis virus lowered the magnitude

247 To improve upon this, we used the murine lymphocytic choriomeningitis virus model and adenoviral

248 for CTL activation was also verified in the lymphocytic choriomeningitis virus model, in which IFN-b

249 r infection by two other NS RNA viruses, the lymphocytic choriomeningitis virus of the Arenaviridae f

250 splantation, the impact of an infection with lymphocytic choriomeningitis virus on prenatal allospeci

251 to acute or protracted viral infection with lymphocytic choriomeningitis virus or during autoimmune

252 und to be in cell cycle after infection with lymphocytic choriomeningitis virus or vesicular stomatit

253 ronic, but not acute, infection of mice with lymphocytic choriomeningitis virus results in a marked e

254 Infection of Spi6 knockout mice with lymphocytic choriomeningitis virus revealed impaired sur

255 namics of this cell turnover, we transferred lymphocytic choriomeningitis virus specific memory CD8 T

256 n T cell responses in mice given variants of lymphocytic choriomeningitis virus that cause acute or p

257 tly reduced in mice lacking B cells, whereas lymphocytic choriomeningitis virus titers were dramatica

258 el of perforin (Prf1)(KO) mice infected with lymphocytic choriomeningitis virus to genetically elimin

259 vity of A3 against representative Old World (lymphocytic choriomeningitis virus) and New World (Junin

260 RNA was also detected in cells infected with lymphocytic choriomeningitis virus, an ambisense RNA vir

261 for JUNV and for vesicular stomatitis virus, lymphocytic choriomeningitis virus, and dengue virus but

262 uthia mandrillaris, Cryptococcus neoformans, lymphocytic choriomeningitis virus, and West Nile virus.

263 We found that in mice infected with lymphocytic choriomeningitis virus, colocalization of vi

264 After infection of mice with lymphocytic choriomeningitis virus, IL-2Ralpha-deficient

265 ections with murine CMV (MCMV), but not with lymphocytic choriomeningitis virus, induce CD25 on NK ce

266 did not efficiently promote CD8 immunity to lymphocytic choriomeningitis virus, nor did cav-1(-/-) O

267 After infection with lymphocytic choriomeningitis virus, pearl mice developed

268 Using murine infection with lymphocytic choriomeningitis virus, we demonstrate that,

269 contraction phase of an acute infection with lymphocytic choriomeningitis virus, we found that virus-

270 aring the influenza hemagglutinin or GP from lymphocytic choriomeningitis virus, which enter through

271 The clone 13 (Cl13) variant of lymphocytic choriomeningitis virus--a prototype of Old W

272 ely transferring splenocytes from individual lymphocytic choriomeningitis virus-immune donors into pa

273 mmune diabetes development in the CD8-driven lymphocytic choriomeningitis virus-induced model of type

274 at Bim has a dual role in the development of lymphocytic choriomeningitis virus-induced, T cell-media

275 Using the highly dynamic model system of lymphocytic choriomeningitis virus-mediated hepatitis an

276 theless, neither germinal center B cells nor lymphocytic choriomeningitis virus-specific Ab levels we

277 rs, compromised Ig switch and low avidity of lymphocytic choriomeningitis virus-specific Abs despite

278 of HIV-specific human CD8 T cells or chronic lymphocytic choriomeningitis virus-specific CD8 T cells

279 on fundamental T-cell functions, "exhausted" lymphocytic choriomeningitis virus-specific cells losing

280 ing and expansion of both primary and memory lymphocytic choriomeningitis virus-specific CTL, which c

281 +) memory B cells and the systemic levels of lymphocytic choriomeningitis virus-specific IgG2 Abs wer

282 ed T cells from peripheral T cells using the lymphocytic choriomeningitis virus-specific P14 TCR.

283 is virus infection, Cmah(-/-) mice make more lymphocytic choriomeningitis virus-specific T cells than

284 and IFN-I signaling blockade on the residual lymphocytic choriomeningitis virus-triggered CTL respons

285 ell generation in response to infection with lymphocytic choriomeningitis virus.

286 8(+) T cell fate during acute infection with lymphocytic choriomeningitis virus.

287 determinant derived from the glycoprotein of lymphocytic choriomeningitis virus.

288 T cells during acute infection of mice with lymphocytic choriomeningitis virus.

289 g an immune response to acute infection with lymphocytic choriomeningitis virus.

290 ibody production during acute infection with lymphocytic choriomeningitis virus.

291 onted with a highly disseminating variant of lymphocytic choriomeningitis virus.

292 loped encephalitis from organ donor-acquired lymphocytic choriomeningitis virus.

293 ls) to antiviral immunity after infection by lymphocytic choriomeningitis virus.

294 or the ability to mount a recall response to lymphocytic choriomeningitis virus.

295 hitecture in mice persistently infected with lymphocytic choriomeningitis virus.

296 nfluenza, H1N1; measles; dengue; rabies; and lymphocytic choriomeningitis virus.

297 pment of T-cell immunity by using the murine lymphocytic choriomeningitis virus.

298 t RNA viruses, including West Nile virus and lymphocytic choriomeningitis virus.

299 cell populations after acute infection with lymphocytic choriomeningitis virus.

300 T cell responses during acute infection with lymphocytic choriomeningitis virus.

301 conditionally ablated in T cells, with acute lymphocytic choriomeningitis virus.