ライフサイエンスコーパス: lymphocytic choriomeningitis virus

1 ell generation in response to infection with lymphocytic choriomeningitis virus.

2 8(+) T cell fate during acute infection with lymphocytic choriomeningitis virus.

3 determinant derived from the glycoprotein of lymphocytic choriomeningitis virus.

4 T cells during acute infection of mice with lymphocytic choriomeningitis virus.

5 g an immune response to acute infection with lymphocytic choriomeningitis virus.

6 ibody production during acute infection with lymphocytic choriomeningitis virus.

7 onted with a highly disseminating variant of lymphocytic choriomeningitis virus.

8 loped encephalitis from organ donor-acquired lymphocytic choriomeningitis virus.

9 ls) to antiviral immunity after infection by lymphocytic choriomeningitis virus.

10 or the ability to mount a recall response to lymphocytic choriomeningitis virus.

11 hitecture in mice persistently infected with lymphocytic choriomeningitis virus.

12 nfluenza, H1N1; measles; dengue; rabies; and lymphocytic choriomeningitis virus.

13 pment of T-cell immunity by using the murine lymphocytic choriomeningitis virus.

14 t RNA viruses, including West Nile virus and lymphocytic choriomeningitis virus.

15 cell populations after acute infection with lymphocytic choriomeningitis virus.

16 infection with the prototypic mouse pathogen lymphocytic choriomeningitis virus.

17 ronic infection of mice with viruses such as lymphocytic choriomeningitis virus.

18 T cell responses during acute infection with lymphocytic choriomeningitis virus.

19 conditionally ablated in T cells, with acute lymphocytic choriomeningitis virus.

20 The clone 13 (Cl13) variant of lymphocytic choriomeningitis virus--a prototype of Old W

21 RNA was also detected in cells infected with lymphocytic choriomeningitis virus, an ambisense RNA vir

22 maintained on CD8(+) T cells during chronic lymphocytic choriomeningitis virus and hepatitis C virus

23 complex I was also required for infection of lymphocytic choriomeningitis virus and human parainfluen

24 4 failed to expand and to protect from acute lymphocytic choriomeningitis virus and Leishmania major

25 s and we describe its use in the contexts of lymphocytic choriomeningitis virus and Mycobacterium tub

26 d CD8(+) T cell responses to infections with lymphocytic choriomeningitis virus and the intracellular

27 only two mammarenaviruses, the widely spread lymphocytic choriomeningitis virus and the recently desc

28 luenza A virus and EBV in humans and between lymphocytic choriomeningitis virus and vaccinia virus in

29 infection of a naive C57BL/6 recipient with lymphocytic choriomeningitis virus and vaccinia virus, f

30 t in Atf3 showed enhanced viral clearance in lymphocytic choriomeningitis virus and vesicular stomati

31 vity of A3 against representative Old World (lymphocytic choriomeningitis virus) and New World (Junin

32 for JUNV and for vesicular stomatitis virus, lymphocytic choriomeningitis virus, and dengue virus but

33 uthia mandrillaris, Cryptococcus neoformans, lymphocytic choriomeningitis virus, and West Nile virus.

34 When administered at the beginning of lymphocytic choriomeningitis virus Armstrong infection a

35 yzed viperin expression in vivo during acute lymphocytic choriomeningitis virus Armstrong infection,

36 cell-dependent antiviral immunity using the lymphocytic choriomeningitis virus Armstrong strain acut

37 Using lymphocytic choriomeningitis virus Armstrong to probe th

38 e was followed by acute viral infection with lymphocytic choriomeningitis virus Armstrong.

39 e infection with the natural murine pathogen lymphocytic choriomeningitis virus become more resistant

40 of type I IFNs, and in persistently infected lymphocytic choriomeningitis virus carrier mice, which c

41 pecific CD8 T cells during acute and chronic lymphocytic choriomeningitis virus challenges, but did n

42 Pathogenic HIV or lymphocytic choriomeningitis virus chronic infections di

43 matopoietic or nonhematopoietic cells during lymphocytic choriomeningitis virus clone 13 (LCMV CL-13)

44 We find that the Old World arenaviruses lymphocytic choriomeningitis virus clone 13 (LCMV Cl13)

45 n PTPN22 resist chronic viral infection with lymphocytic choriomeningitis virus clone 13 (LCMV cl13).

46 ls without altering Slamf6(+) numbers during lymphocytic choriomeningitis virus clone 13 infection.

47 ssential for viral control during persistent lymphocytic choriomeningitis virus clone 13 infection.

48 s control and prevented chronic infection in lymphocytic choriomeningitis virus clone 13- and reduced

49 We found that in mice infected with lymphocytic choriomeningitis virus, colocalization of vi

50 The G protein from lymphocytic choriomeningitis virus endowed VSV with the

51 isella strains expressing well-characterized lymphocytic choriomeningitis virus epitopes, we found th

52 recombinant adenovirus vector expressing the lymphocytic choriomeningitis virus glycoprotein (LCMVgp)

53 After infection of mice with lymphocytic choriomeningitis virus, IL-2Ralpha-deficient

54 ely transferring splenocytes from individual lymphocytic choriomeningitis virus-immune donors into pa

55 ted phenotype during chronic infections with lymphocytic choriomeningitis virus in mice and hepatitis

56 ections with murine CMV (MCMV), but not with lymphocytic choriomeningitis virus, induce CD25 on NK ce

57 (but not a slow-spreading acute) isolate of lymphocytic choriomeningitis virus induced large-scale m

58 mmune diabetes development in the CD8-driven lymphocytic choriomeningitis virus-induced model of type

59 at Bim has a dual role in the development of lymphocytic choriomeningitis virus-induced, T cell-media

60 Infection with lymphocytic choriomeningitis virus induces monopoiesis i

61 ay) on early T cell attrition in response to lymphocytic choriomeningitis virus infection and during

62 exhibited a normal acute response (day 8) to lymphocytic choriomeningitis virus infection but generat

63 Consistent with this finding, we show that lymphocytic choriomeningitis virus infection can directl

64 ined T cell responses to acute or persistent lymphocytic choriomeningitis virus infection in IFN-lamb

65 erived T cells are comparable in controlling lymphocytic choriomeningitis virus infection in mice and

66 regulates T cell dysfunction during chronic lymphocytic choriomeningitis virus infection in mice, an

67 ncoding the TGF-beta receptor during chronic lymphocytic choriomeningitis virus infection in mice, an

68 In a model of chronic lymphocytic choriomeningitis virus infection in mice, we

69 each stable levels of memory following acute lymphocytic choriomeningitis virus infection in mice.

70 notherapeutic effects of IL-7 during chronic lymphocytic choriomeningitis virus infection in mice.

71 s in virus-specific CD8 T cells during acute lymphocytic choriomeningitis virus infection in mice.

72 sential for the control of acute and chronic lymphocytic choriomeningitis virus infection in the join

73 c memory CD4(+) T cell subpopulations in the lymphocytic choriomeningitis virus infection model, we f

74 We used a murine model of HLH, involving lymphocytic choriomeningitis virus infection of perforin

75 Here we demonstrate that chronic lymphocytic choriomeningitis virus infection rapidly tri

76 cell-extrinsic manner early following acute lymphocytic choriomeningitis virus infection to suppress

77 rvation of antiviral immune responses, acute lymphocytic choriomeningitis virus infection was used.

78 pDC development and serum IFN-I responses to lymphocytic choriomeningitis virus infection were augmen

79 Here, we show that during chronic murine lymphocytic choriomeningitis virus infection, activation

80 ults in reduced viral clearance in models of lymphocytic choriomeningitis virus infection, and also p

81 y respond during the early stages of chronic lymphocytic choriomeningitis virus infection, and that t

82 after protein immunization; however, during lymphocytic choriomeningitis virus infection, B cells in

83 In chronic lymphocytic choriomeningitis virus infection, blockade o

84 Upon lymphocytic choriomeningitis virus infection, Cmah(-/-)

85 , as dual TCR cells predominated response to lymphocytic choriomeningitis virus infection, comprising

86 rrantly upregulated during memory to chronic lymphocytic choriomeningitis virus infection, limiting f

87 Following acute lymphocytic choriomeningitis virus infection, memory CD8

88 Here we show that, following lymphocytic choriomeningitis virus infection, resident m

89 Using the experimental mouse model of lymphocytic choriomeningitis virus infection, we demonst

90 Following lymphocytic choriomeningitis virus infection, we found m

91 nd naive CD8(+) T cells to acute and chronic lymphocytic choriomeningitis virus infection, we show th

92 4hi CD8 and CD4 T cells at days 2 to 3 after lymphocytic choriomeningitis virus infection, when type

93 CD8(+) T cells to mount a robust response to lymphocytic choriomeningitis virus infection, with both

94 sion and increased cytokine production after lymphocytic choriomeningitis virus infection, yet DGK-de

95 cked gp33-specific CD8(+) T cells during RRV-lymphocytic choriomeningitis virus infection.

96 ere also found in mouse liver within 24 h of lymphocytic choriomeningitis virus infection.

97 i-CD20 before or different times after acute lymphocytic choriomeningitis virus infection.

98 d day-8 effector CD8 T cells following acute lymphocytic choriomeningitis virus infection.

99 e a crucial function in viral clearance upon lymphocytic choriomeningitis virus infection.

100 ptional profiles and chromatin states during lymphocytic choriomeningitis virus infection.

101 ry CD8 T-cell generation and responses after lymphocytic choriomeningitis virus infection.

102 exhaustion by Tim-3 and PD-1 during chronic lymphocytic choriomeningitis virus infection.

103 ed the early T cell attrition resulting from lymphocytic choriomeningitis virus infection.

104 ephalitis, and defective immune responses to lymphocytic choriomeningitis virus infection.

105 risk of developing HLH immunopathology after lymphocytic choriomeningitis virus infection.

106 virus infection to those in acute or chronic lymphocytic choriomeningitis virus infection.

107 TE/TEM and TRM subsets was overcome by acute lymphocytic choriomeningitis virus infection; neverthele

108 isingly, they also show that chronic HIV and lymphocytic choriomeningitis virus infections have a ver

109 G (IgG), and were unable to resolve chronic lymphocytic choriomeningitis virus infections.

110 CD8 T cells responding to acute and chronic lymphocytic choriomeningitis virus infections.

111 his concept, we have developed a recombinant lymphocytic choriomeningitis virus (LCMV) (rLCMVDeltaGP/

112 oteins of all known pathogenic arenaviruses, lymphocytic choriomeningitis virus (LCMV) and Lassa, Jun

113 essing of GPC from the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) and LASV, whic

114 infection with two natural mouse pathogens, lymphocytic choriomeningitis virus (LCMV) and murine cyt

115 -deficient (Prf1(--)) mice are infected with lymphocytic choriomeningitis virus (LCMV) and secondary

116 T cells were generated after infection with lymphocytic choriomeningitis virus (LCMV) and that these

117 Here we show that the arenaviruses lymphocytic choriomeningitis virus (LCMV) and the clinic

118 eoproteins (NPs) of the Old World arenavirus lymphocytic choriomeningitis virus (LCMV) and the New Wo

119 ce with Leishmania major and 2 wk later with lymphocytic choriomeningitis virus (LCMV) and then monit

120 Mice were infected with lymphocytic choriomeningitis virus (LCMV) and treated wi

121 topes from unrelated and pathogenic viruses, lymphocytic choriomeningitis virus (LCMV) and vaccinia v

122 te viral infections, such as infections with lymphocytic choriomeningitis virus (LCMV) and vaccinia v

123 In this study, using lymphocytic choriomeningitis virus (LCMV) and Zika virus

124 yelin oligodendrocyte glycoprotein (MOG) and lymphocytic choriomeningitis virus (LCMV) antigens, resp

125 Using lymphocytic choriomeningitis virus (LCMV) as a model of

126 owing infection with a persistent variant of lymphocytic choriomeningitis virus (LCMV) but have no im

127 We show that lymphocytic choriomeningitis virus (LCMV) can also inhib

128 Lymphocytic choriomeningitis virus (LCMV) can cause acut

129 Lymphocytic choriomeningitis virus (LCMV) can establish

130 ablishment of life-long chronic infection by lymphocytic choriomeningitis virus (LCMV) Cl 13 but does

131 and hepatitis C virus or those of mice with lymphocytic choriomeningitis virus (LCMV) clone 13 (CL13

132 virus (ECTV) when chronically infected with lymphocytic choriomeningitis virus (LCMV) clone 13 (CL13

133 ly shown that mice chronically infected with lymphocytic choriomeningitis virus (LCMV) clone 13 (CL13

134 nic phase of infection with HIV in humans or lymphocytic choriomeningitis virus (LCMV) clone 13 in mi

135 ) is maximally induced on APCs at day 2 post-lymphocytic choriomeningitis virus (LCMV) clone 13 infec

136 effector CD8 T cells from mice infected with lymphocytic choriomeningitis virus (LCMV) clone 13 into

137 During chronic infection with lymphocytic choriomeningitis virus (LCMV) clone 13, miR-

138 aride (LPS) with that of a persistent virus, lymphocytic choriomeningitis virus (LCMV) clone 13, on e

139 Lymphocytic choriomeningitis virus (LCMV) clone 13, whic

140 rst 12-24 hours after mice are infected with lymphocytic choriomeningitis virus (LCMV) clone 13, whic

141 from a septic event are more susceptible to lymphocytic choriomeningitis virus (LCMV) clone-13 infec

142 toire response against a massively exhausted lymphocytic choriomeningitis virus (LCMV) epitope.

143 report that mice persistently infected with lymphocytic choriomeningitis virus (LCMV) exhibit a seve

144 this study we found that mice infected with lymphocytic choriomeningitis virus (LCMV) exhibit global

145 rmore, pDC-deficient animals failed to clear lymphocytic choriomeningitis virus (LCMV) from hematopoi

146 Mice immunized with H(2)O(2)-inactivated lymphocytic choriomeningitis virus (LCMV) generated cyto

147 -defective adenovirus vectors expressing the lymphocytic choriomeningitis virus (LCMV) glycoprotein (

148 Ad5, Ad26, Ad35, and Ad48 vectors expressing lymphocytic choriomeningitis virus (LCMV) glycoprotein (

149 we immunized mice with Ad5 vectors encoding lymphocytic choriomeningitis virus (LCMV) glycoprotein (

150 enic CD4(+) and CD8(+) T cells responding to lymphocytic choriomeningitis virus (LCMV) identified mul

151 onse during acute primary infection with the lymphocytic choriomeningitis virus (LCMV) in mice is sig

152 n with hepatitis B and C virus in humans and lymphocytic choriomeningitis virus (LCMV) in mice.

153 viruses pseudotyped with the glycoprotein of lymphocytic choriomeningitis virus (LCMV) in vitro.

154 ulated on virus-specific CTLs during chronic lymphocytic choriomeningitis virus (LCMV) infection and

155 in mice changes the outcome to a subsequent lymphocytic choriomeningitis virus (LCMV) infection and

156 Cs) is amplified during chronic versus acute lymphocytic choriomeningitis virus (LCMV) infection and

157 d the role of IAPs in T-cell immunity during lymphocytic choriomeningitis virus (LCMV) infection by p

158 Although cellular immunity to acute lymphocytic choriomeningitis virus (LCMV) infection has

159 Recent studies in chronic lymphocytic choriomeningitis virus (LCMV) infection have

160 IL-10 production over the course of chronic lymphocytic choriomeningitis virus (LCMV) infection in a

161 ced in both CD4(+) and CD8(+) T cells during lymphocytic choriomeningitis virus (LCMV) infection in a

162 ls contribute to establishment of persistent lymphocytic choriomeningitis virus (LCMV) infection in m

163 ector function of CD8 T cells during chronic lymphocytic choriomeningitis virus (LCMV) infection in m

164 t CD70 blockade during an acute or a chronic lymphocytic choriomeningitis virus (LCMV) infection in m

165 In contrast to MCMV, lymphocytic choriomeningitis virus (LCMV) infection in m

166 Lymphocytic choriomeningitis virus (LCMV) infection indu

167 Using acute and chronic lymphocytic choriomeningitis virus (LCMV) infection mode

168 Using the lymphocytic choriomeningitis virus (LCMV) infection mode

169 ll activation kinetics and viral loads after lymphocytic choriomeningitis virus (LCMV) infection of C

170 It is dramatic at 2 to 4 days following lymphocytic choriomeningitis virus (LCMV) infection of m

171 We show here that persistent lymphocytic choriomeningitis virus (LCMV) infection of m

172 this study, we used a mouse model of chronic lymphocytic choriomeningitis virus (LCMV) infection to a

173 of virus-specific CD8 T cells during chronic lymphocytic choriomeningitis virus (LCMV) infection to e

174 se ranging from aseptic meningitis following lymphocytic choriomeningitis virus (LCMV) infection to h

175 The primary CD8(+) T-cell response to acute lymphocytic choriomeningitis virus (LCMV) infection was

176 found that during the first week of chronic lymphocytic choriomeningitis virus (LCMV) infection, bef

177 (+) T cells are essential for clearance of a lymphocytic choriomeningitis virus (LCMV) infection, but

178 ty, Hegazy et al. report that in response to lymphocytic choriomeningitis virus (LCMV) infection, ful

179 During persistent lymphocytic choriomeningitis virus (LCMV) infection, IFN

180 In a mouse model of persistent lymphocytic choriomeningitis virus (LCMV) infection, it

181 (+) T-cell formation following immunization, lymphocytic choriomeningitis virus (LCMV) infection, or

182 evelop fatal pathology during early systemic lymphocytic choriomeningitis virus (LCMV) infection, sug

183 nt mice exhibit reduced IFN-I responses upon lymphocytic choriomeningitis virus (LCMV) infection, whi

184 differentiation in vitro and in vivo during lymphocytic choriomeningitis virus (LCMV) infection.

185 e subsets to HLH-like disease severity after lymphocytic choriomeningitis virus (LCMV) infection.

186 induction of exhaustion in mice with chronic lymphocytic choriomeningitis virus (LCMV) infection.

187 viral replication in the context of chronic lymphocytic choriomeningitis virus (LCMV) infection.

188 d IL-21 production at late stages of chronic lymphocytic choriomeningitis virus (LCMV) infection.

189 D-L1) blockade in the mouse model of chronic lymphocytic choriomeningitis virus (LCMV) infection.

190 y reproduced the manifestations of HLH after lymphocytic choriomeningitis virus (LCMV) infection.

191 of CD8 T cells are virus specific following lymphocytic choriomeningitis virus (LCMV) infection.

192 during the early stages of acute and chronic lymphocytic choriomeningitis virus (LCMV) infection.

193 worldwide-distributed prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) is a neglected

194 worldwide-distributed prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) is a neglected

195 e globally distributed prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) is a neglected

196 worldwide-distributed prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) is an importan

197 dult mice with the clone 13 (CL13) strain of lymphocytic choriomeningitis virus (LCMV) is extensively

198 The prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) is widely used

199 g (ARM) or chronic Clone 13 (C13) strains of lymphocytic choriomeningitis virus (LCMV) led to two dis

200 Ins2-GP(Tg) mice injected with lymphocytic choriomeningitis virus (LCMV) lost islet sym

201 lowing infection with the clone 13 strain of lymphocytic choriomeningitis virus (LCMV) or influenza v

202 In mice infected with lymphocytic choriomeningitis virus (LCMV) or Listeria mo

203 evidence that infection of mice with either lymphocytic choriomeningitis virus (LCMV) or pneumonia v

204 anasal infection with the systemic pathogens lymphocytic choriomeningitis virus (LCMV) or vaccinia vi

205 t isografts using RIP-LCMV mice expressing a lymphocytic choriomeningitis virus (LCMV) protein in the

206 Systemic low-dose infection of mice with lymphocytic choriomeningitis virus (LCMV) results in mas

207 l differentiation after acute infection with lymphocytic choriomeningitis virus (LCMV) strain Armstro

208 combinant antigen variant-expressing chronic lymphocytic choriomeningitis virus (LCMV) strains, we un

209 generated in mice persistently infected with lymphocytic choriomeningitis virus (LCMV) suppressed mul

210 The lymphocytic choriomeningitis virus (LCMV) system constit

211 d mice persistently infected from birth with lymphocytic choriomeningitis virus (LCMV) to demonstrate

212 We used the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) to develop a g

213 The recombinant engineering of trisegmented lymphocytic choriomeningitis virus (LCMV) to express two

214 ted the ability of the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) to interfere w

215 ility of the prototypic Old World arenavirus lymphocytic choriomeningitis virus (LCMV) to interfere w

216 cine (HB-101) consisting of 2 nonreplicating lymphocytic choriomeningitis virus (LCMV) vectors expres

217 ion-experienced T(regs) generated upon acute Lymphocytic Choriomeningitis Virus (LCMV) WE and Vaccini

218 Lymphocytic choriomeningitis virus (LCMV) WE variant 2.2

219 -cell responses to chimeric vaccines against lymphocytic choriomeningitis virus (LCMV) were assessed

220 leoprotein (NP) of the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) with the least

221 uced by chronic infections such as HIV, HPV, lymphocytic choriomeningitis virus (LCMV), and schistoso

222 tion) of the NP of the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV), as well as th

223 costimulatory molecule expression induced by lymphocytic choriomeningitis virus (LCMV), CD28/B7-media

224 per that the prototype member in the family, lymphocytic choriomeningitis virus (LCMV), disables the

225 deficient (Prf1(-/-)) mice are infected with lymphocytic choriomeningitis virus (LCMV), disease is dr

226 The prototypic arenavirus, lymphocytic choriomeningitis virus (LCMV), is a model fo

227 worldwide-distributed prototypic arenavirus, lymphocytic choriomeningitis virus (LCMV), is a neglecte

228 d by coinfecting mice with L. guyanensis and lymphocytic choriomeningitis virus (LCMV), or the sand f

229 We infected mice with chronic lymphocytic choriomeningitis virus (LCMV), performed RNA

230 The prototypic arenavirus, lymphocytic choriomeningitis virus (LCMV), provides inve

231 ruses, including Ebola virus, Marburg virus, lymphocytic choriomeningitis virus (LCMV), rabies virus,

232 Lymphocytic choriomeningitis virus (LCMV), the prototype

233 us (IAV) and in memory phase challenged with lymphocytic choriomeningitis virus (LCMV), which we show

234 T(M) from lymphocytic choriomeningitis virus (LCMV)-immune and H60

235 ate protein (AP)-fed controls, LP feeding in lymphocytic choriomeningitis virus (LCMV)-immune mice re

236 In this environment, lymphocytic choriomeningitis virus (LCMV)-infected iFRCs

237 Here, we show that lymphocytic choriomeningitis virus (LCMV)-specific CD8(+

238 Here, we show that lymphocytic choriomeningitis virus (LCMV)-specific CD8+

239 duced expression of CCL21 in murine spleens, lymphocytic choriomeningitis virus (LCMV)-specific T cel

240 protect mice against infection with chronic lymphocytic choriomeningitis virus (LCMV).

241 n the S segment of the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV).

242 ntiviral effector T cells postinfection with lymphocytic choriomeningitis virus (LCMV).

243 itro and in vivo during acute infection with lymphocytic choriomeningitis virus (LCMV).

244 oma cells that replicate the negative-strand lymphocytic choriomeningitis virus (LCMV).

245 were challenged with the Armstrong strain of lymphocytic choriomeningitis virus (LCMV).

246 protection against viral infection, such as lymphocytic choriomeningitis virus (LCMV).

247 eted mice infected with the mouse arenavirus lymphocytic choriomeningitis virus (LCMV).

248 T) donor mice and infected the chimeras with lymphocytic choriomeningitis virus (LCMV).

249 mphohistiocytosis (HLH) after infection with lymphocytic choriomeningitis virus (LCMV).

250 fection of WASP-deficient (WAS KO) mice with lymphocytic choriomeningitis virus (LCMV).

251 pe I IFN response to many viruses, including lymphocytic choriomeningitis virus (LCMV).

252 iral loads in mice chronically infected with lymphocytic choriomeningitis virus (LCMV).

253 eficient (Prf1 (-/-)) mice are infected with lymphocytic choriomeningitis virus (LCMV).

254 -Z interaction for the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV).

255 Ia and class II molecules were infected with lymphocytic choriomeningitis virus (LCMV).

256 ells generated after systemic infection with lymphocytic choriomeningitis virus (LCMV).

257 ctivating TCR signals after clearance of the lymphocytic choriomeningitis virus (LCMV).

258 f cells) during acute infection of mice with lymphocytic choriomeningitis virus (LCMV).

259 uring acute, but not chronic, infection with lymphocytic choriomeningitis virus (LCMV).

260 e in perforin-deficient mice is triggered by lymphocytic choriomeningitis virus (LCMV).

261 ry pathway in mice chronically infected with lymphocytic choriomeningitis virus (LCMV).

262 D8(+) T cells following acute infection with lymphocytic choriomeningitis virus (LCMV).

263 on with vesicular stomatitis virus (VSV) and lymphocytic choriomeningitis virus (LCMV).

264 tivity against three distinct ssRNA viruses: lymphocytic choriomeningitis virus (LCMV); influenza A v

265 replaced with a variant glycoprotein of the lymphocytic choriomeningitis virus (LCMV-GP), creating a

266 t viruses, murine cytomegalovirus (MCMV) and lymphocytic choriomeningitis virus (LCMV; Cl13), in thei

267 infected Il18-transgenic (Il18tg) mice with lymphocytic choriomeningitis virus (LCMV; strain Armstro

268 ated stimulation during primary responses to lymphocytic choriomeningitis virus lowered the magnitude

269 Using the highly dynamic model system of lymphocytic choriomeningitis virus-mediated hepatitis an

270 To improve upon this, we used the murine lymphocytic choriomeningitis virus model and adenoviral

271 for CTL activation was also verified in the lymphocytic choriomeningitis virus model, in which IFN-b

272 did not efficiently promote CD8 immunity to lymphocytic choriomeningitis virus, nor did cav-1(-/-) O

273 r infection by two other NS RNA viruses, the lymphocytic choriomeningitis virus of the Arenaviridae f

274 splantation, the impact of an infection with lymphocytic choriomeningitis virus on prenatal allospeci

275 could be cross-reactive with three different lymphocytic choriomeningitis virus, one Pichinde virus,

276 to acute or protracted viral infection with lymphocytic choriomeningitis virus or during autoimmune

277 und to be in cell cycle after infection with lymphocytic choriomeningitis virus or vesicular stomatit

278 After infection with lymphocytic choriomeningitis virus, pearl mice developed

279 ronic, but not acute, infection of mice with lymphocytic choriomeningitis virus results in a marked e

280 Infection of Spi6 knockout mice with lymphocytic choriomeningitis virus revealed impaired sur

281 Ad5) prime followed by replication-defective lymphocytic choriomeningitis virus (rLCMV) boost elicite

282 We constructed recombinant lymphocytic choriomeningitis virus (rLCMV) featuring eit

283 at recombinants of the prototypic arenavirus lymphocytic choriomeningitis virus (rLCMVs), whose S-IGR

284 namics of this cell turnover, we transferred lymphocytic choriomeningitis virus specific memory CD8 T

285 theless, neither germinal center B cells nor lymphocytic choriomeningitis virus-specific Ab levels we

286 rs, compromised Ig switch and low avidity of lymphocytic choriomeningitis virus-specific Abs despite

287 of HIV-specific human CD8 T cells or chronic lymphocytic choriomeningitis virus-specific CD8 T cells

288 on fundamental T-cell functions, "exhausted" lymphocytic choriomeningitis virus-specific cells losing

289 ing and expansion of both primary and memory lymphocytic choriomeningitis virus-specific CTL, which c

290 +) memory B cells and the systemic levels of lymphocytic choriomeningitis virus-specific IgG2 Abs wer

291 ed T cells from peripheral T cells using the lymphocytic choriomeningitis virus-specific P14 TCR.

292 is virus infection, Cmah(-/-) mice make more lymphocytic choriomeningitis virus-specific T cells than

293 n T cell responses in mice given variants of lymphocytic choriomeningitis virus that cause acute or p

294 tly reduced in mice lacking B cells, whereas lymphocytic choriomeningitis virus titers were dramatica

295 We infected FtDKO mice with lymphocytic choriomeningitis virus to generate and track

296 el of perforin (Prf1)(KO) mice infected with lymphocytic choriomeningitis virus to genetically elimin

297 and IFN-I signaling blockade on the residual lymphocytic choriomeningitis virus-triggered CTL respons

298 Using murine infection with lymphocytic choriomeningitis virus, we demonstrate that,

299 contraction phase of an acute infection with lymphocytic choriomeningitis virus, we found that virus-

300 aring the influenza hemagglutinin or GP from lymphocytic choriomeningitis virus, which enter through