ライフサイエンスコーパス: lymphoid cell

1 mechanism of p53-induced cell death in pre-B lymphoid cells.

2 ranulocytes; and reductions in type 3 innate lymphoid cells.

3 function and potentiating responses of other lymphoid cells.

4 issue homeostasis and repair, such as innate lymphoid cells.

5 ly through IL-13 production by goup 2 innate lymphoid cells.

6 etic system, was restricted predominantly to lymphoid cells.

7 per development of adaptive, but not innate, lymphoid cells.

8 hemokine axis and by IL-13 expressing innate lymphoid cells.

9 eosinophils, mast cells, and group 3 innate lymphoid cells.

10 meostasis by shaping the fate of myeloid and lymphoid cells.

11 s, IL1B+ monocytes, and fewer group 1 innate lymphoid cells.

12 pon daunorubicin-induced DNA damage of human lymphoid cells.

13 ppressor cells at the cost of tumor-reactive lymphoid cells.

14 rection of functional defects in myeloid and lymphoid cells.

15 d antigens is a cardinal feature of adaptive lymphoid cells.

16 ecretion from mature miR-146a-/- myeloid and lymphoid cells.

17 ), lymphoid tissue inducer (LTi), and innate lymphoid cell 1 (ILC1) cells, but not ILC2 or ILC3 cells

18 oes not alter the balance of NK cell/ innate lymphoid cell 1 generation and slightly decreases the nu

19 in beta1, distinguishes NK cells from innate lymphoid cells 1 and other leukocytes.

20 and straightforward identification of innate lymphoid cell 2 progenitor populations.

21 Finally, the discovery of a prominent innate lymphoid cell-2 cluster links the single-cell RNA sequen

22 tudies that predated the discovery of innate lymphoid cells-2 cells.

23 endritic cells, natural killer cells, innate lymphoid cells-2, and CD (cluster of differentiation)-8

24 ing a strong atheroprotective role of innate lymphoid cells-2.

25 eport that MAIT cells repress group 2 innate lymphoid cell activation and restrict allergen-induced a

26 ed permeability, IL-33 levels, type 2 innate lymphoid cell activation, and T(h)2 cell differentiation

27 T cells, classic NK cells, and type 1 innate lymphoid cells), all of which can produce large amounts

28 Type 2 innate lymphoid cells and basophils were scarce in BAL fluid.

29 tion of chemokine responses in primary human lymphoid cells and cell lines that express CXCR4.

30 colon tissues, which activated type 2 innate lymphoid cells and dendritic cells to promote differenti

31 en induces IL-13 secretion by group 2 innate lymphoid cells and enteroid gene expression consistent w

32 nate immune effector cells at the expense of lymphoid cells and erythrocytes.

33 mbers of eosinophils, IL-13(+) type 2 innate lymphoid cells and IL-13(+)CD4(+) T cells and IL-5 and I

34 ically marked as an active enhancer in human lymphoid cells and not monocytes.

35 ily on mast cells, Th2 cells, group 2 innate lymphoid cells and regulatory T cells, and to a lesser e

36 cells and antigen-presenting cells to innate lymphoid cells and regulatory T cells.

37 During development innate lymphoid cells and specialized lymphocyte subsets coloni

38 ulted in loss of AHR-dependent type 3 innate lymphoid cells and T helper 17 cells and increased susce

39 observed increased numbers of group 3 innate lymphoid cells and Th17 cells and enhanced transcription

40 bsence of other inflammatory stimuli, innate lymphoid cells, and adaptive immunity.

41 ils, mast cells, T(H)2 cells, group 2 innate lymphoid cells, and antigen-presenting cells; and T3 CRS

42 latory interactions between lymphoid and non-lymphoid cells, and correlated pyridoxate with p53-regul

43 ress, required extrinsic signals from innate lymphoid cells, and limited bacterial dissemination.

44 ether with CD14(+) CD16(+) monocytes, innate lymphoid cells, and natural killer cells.

45 ely, eosinophils, Th2 T cells, type 2 innate lymphoid cells, and possibly Foxp3+ Tregs protect agains

46 epithelial cells, macrophages, type 2 innate lymphoid cells, and TH2 cells along with increased Il33

47 l developmental intermediates, non-NK innate lymphoid cells, and the capacity for NK cells to adapt a

48 phatic endothelial cells, tumor-infiltrating lymphoid cells, and tissue-resident macrophages.

49 lly, we provide evidence that group 2 innate lymphoid cells are a source of IL-13, which promotes lun

50 Adipose type 1 innate lymphoid cells (AT1-ILCs) promote pro-inflammatory macro

51 Natural killer (NK) cells are innate lymphoid cells being explored as they engage tumor cells

52 ich previously has only been associated with lymphoid cell biology.

53 eletion led to an overall increase in innate lymphoid cells (CD45(+)lin(-)CD25(+) cells) and IL-13(+)

54 nd lymphoid (natural killer cells and innate lymphoid cells) cell populations of the innate immune sy

55 n natural killer (NK) cells and other innate lymphoid cells, competent enhancers are primed during li

56 hether engrafted miR-210-positive myeloid or lymphoid cells contribute to paracrine miR-210 delivery,

57 ression but differed in terms of myeloid and lymphoid cell counts.

58 Myeloid and lymphoid cell cytokine secretion was measured after in v

59 pacity of MYC-driven normal and neoplastic B lymphoid cells depends on MNT, a MYC-related transcripti

60 After a high fat diet, mice lacking CD69 on lymphoid cells developed large atheroma plaque along wit

61 ucial at several stages of T cell and innate lymphoid cell development and differentiation.

62 Here, we review roles for Ikaros factors in lymphoid cell development, differentiation, and function

63 D94(+)NKp80(-)) NKDIs promoted non-NK innate lymphoid cell differentiation at the expense of NK cell

64 whether AHR antagonism could promote innate lymphoid cell differentiation from hESCs.

65 During early pregnancy, decidual innate lymphoid cells (dILCs) interact with surrounding materna

66 cy increased IL-5 production, group 2 innate lymphoid cell expansion, and host resistance to the hook

67 In adaptive lymphoid cells EZH2 prevented the premature expression o

68 d protein axis specifies innate and adaptive lymphoid cell fate.

69 At pre-tumorous stages, lymphoid cells from the animals exhibit deregulated phos

70 , we detect ERbeta protein in testis, ovary, lymphoid cells, granulosa cell tumours, and a subset of

71 holic acid induced intestinal group 3 innate lymphoid cell IL-22 secretion through GATA binding prote

72 se (Th2 cells, M2 macrophages, type 2 innate lymphoid cells, IL-33, IL-4, IL-13, and mucus) that dire

73 e mutations, Langerhans cells, type 2 innate lymphoid cells, IL-33, TSLP) that have important roles i

74 s to suppress excessive activation of innate lymphoid cell (ILC) 2 during allergic lung inflammation.

75 They are also required for innate lymphoid cell (ILC) development and maintenance, and con

76 ate early T cell progenitor (ETP) and innate lymphoid cell (ILC) development remains unclear.

77 We investigated NK cell and innate lymphoid cell (ILC) dynamics and function in rhesus maca

78 commitment to both the T cell and the innate lymphoid cell (ILC) lineages in mammals.

79 eported the selective loss of group 3 innate lymphoid cell (ILC) number and function in a Dock8-defic

80 Little is known about innate lymphoid cell (ILC) populations in the human gut, and th

81 g multiparameter flow cytometry human innate lymphoid cell (ILC) subsets can be detected in the circu

82 ed the development of tissue-resident innate lymphoid cell (ILC) subsets.

83 Although innate lymphoid cells (ILC) and natural killer (NK) cells are f

84 Innate lymphoid cells (ILC) are a heterogeneous family of immun

85 Innate lymphoid cells (ILC) are lymphocytes that lack an antige

86 ral killer (NK) cells are a subset of innate lymphoid cells (ILC) capable of recognizing stressed and

87 ssed eosinophils, T cells, Tregs, and innate lymphoid cells (ILC) from peripheral blood using flow cy

88 Innate lymphoid cells (ILC) play critical roles in regulating i

89 Recently, several groups of innate lymphoid cells (ILC), distinct from NK cells in developm

90 ritical negative regulator of group 2 innate lymphoid cells (ILC-2s).

91 ells are known to convert to a type 1 innate lymphoid cell (ILC1)-like phenotype in response to TGF-b

92 , we show that tissue-resident type 1 innate lymphoid cells (ILC1) serve an essential early role in h

93 activation of CD3(-) NK1.1(+) group 1 innate lymphoid cells (ILC1) within the FRT, essential for recr

94 Group 1 innate lymphoid cells (ILC1s) are effectors of innate immunity

95 the features that distinguish type 1 innate lymphoid cells (ILC1s) from natural killer (NK) cells is

96 Although type 1 innate lymphoid cells (ILC1s) have been originally found as liv

97 ntly "time stamp" NK cells and type 1 innate lymphoid cells (ILC1s) to characterize the dynamics of t

98 IL-33 drives colonic group 2 innate lymphoid cell (ILC2) activation during infection and IL-

99 L-33 synergistically promoted group 2 innate lymphoid cell (ILC2) activation to induce innate allergi

100 binant HpARI abrogated IL-33, group 2 innate lymphoid cell (ILC2) and eosinophilic responses to Alter

101 ds: Cytokines, viral load, and type 2 innate lymphoid cell (ILC2) levels in nasal aspirates, collecte

102 Alternaria-induced pulmonary group 2 innate lymphoid cell (ILC2) responses and IL-33 release.

103 nstrate that helminth-induced group 2 innate lymphoid cell (ILC2) responses are exaggerated in the ab

104 y gene expression, and TH and group 2 innate lymphoid cell (ILC2) responses.

105 Here we identify a group 2 innate lymphoid cell (ILC2) subpopulation that can convert into

106 nt repairing AAMs, as well as group-2 innate lymphoid cell (ILC2)-mediated AAM activation.

107 e report that tissue-resident group 2 innate lymphoid cells (ILC2) accumulate in the choroid plexus o

108 Group 2 innate lymphoid cells (ILC2) are stimulated by IL-33 to increas

109 Group 2 innate lymphoid cells (ILC2) are tissue-resident innate effecto

110 Group 2 innate lymphoid cells (ILC2) are tissue-resident, long-lived in

111 creased the proportion of ST2-bearing innate lymphoid cells (ILC2) in blood and kidneys, and adoptive

112 Type 2 innate lymphoid cells (ILC2) mediate inflammatory immune respon

113 Type 2 innate lymphoid cells (ILC2) share cytokine and transcription f

114 ecule), potentiated Tregs and group 2 innate lymphoid cells (ILC2) to prevent renal injury.

115 )-Rorasg/sg mice deficient in group 2 innate lymphoid cells (ILC2), and C57BL/6 wild-type mice treate

116 Group-2 innate lymphoid cells (ILC2), type-2 cytokines, and eosinophils

117 ls expressing CRTh2), eosinophils and innate lymphoid cells (ILC2).

118 It also caused a reduction in innate lymphoid cell, ILC2, and IL-9(+) and IL-13(+) ILC2 numbe

119 ct on the differentiation of Group 2 Innate -Lymphoid -Cells (ILC2s) in the pathogenesis of asthma.

120 e expansion of IL-13-producing type 2 innate lymphoid cells (ILC2s) and dependent on IL-25 and IL-33.

121 ocation and release, increased type 2 innate lymphoid cells (ILC2s) and monocyte-derived dendritic ce

122 Group 2 innate lymphoid cells (ILC2s) and type 2 helper T cells (Th2 ce

123 Type-2 innate lymphoid cells (ILC2s) are a potent source of T-helper 2

124 Group 2 innate lymphoid cells (ILC2s) are a potential innate source of

125 Type-2 innate lymphoid cells (ILC2s) are a recently discovered immune

126 Group 2 innate lymphoid cells (ILC2s) are a recently identified group o

127 of allergic inflammation, and type 2 innate lymphoid cells (ILC2s) are an important cell type for me

128 The recently discovered group 2 innate lymphoid cells (ILC2s) are capable of secreting copious

129 Group 2 innate lymphoid cells (ILC2s) are effective producers of IL-5 a

130 Group 2 innate lymphoid cells (ILC2s) are effector cells of type 2 immu

131 Current models propose that group 2 innate lymphoid cells (ILC2s) are generated in the bone marrow.

132 Human type 2 innate lymphoid cells (ILC2s) are identified by coupled detecti

133 Type 2 Innate lymphoid cells (ILC2s) are implicated in helminth infect

134 Group 2 innate lymphoid cells (ILC2s) are implicated in host defense an

135 Group 2 innate lymphoid cells (ILC2s) are important mediators of allerg

136 Group 2 innate lymphoid cells (ILC2s) are key regulators of type 2 infl

137 Group 2 innate lymphoid cells (ILC2s) are rare innate immune cells that

138 Group 2 innate lymphoid cells (ILC2s) are the dominant innate lymphoid

139 Group 2 innate lymphoid cells (ILC2s) are tissue-resident cells promine

140 d interleukin (IL)-9-producing type 2 innate lymphoid cells (ILC2s) as the mediators of a molecular a

141 ablished, the newly identified type 2 innate lymphoid cells (ILC2s) can also contribute to orchestrat

142 controlled lineage tracing of group 2 innate lymphoid cells (ILC2s) during this period remains unstud

143 Group 2 innate lymphoid cells (ILC2s) expand in the lungs of mice durin

144 Group 2 innate lymphoid cells (ILC2s) have recently been shown to play

145 Group 2 innate lymphoid cells (ILC2s) have tissue-resident competence a

146 ced eosinophilia and expanded group 2 innate lymphoid cells (ILC2s) in aspirin-exacerbated respirator

147 limited regarding the role of group 2 innate lymphoid cells (ILC2s) in regulating humoral immunity.

148 models of allogeneic BMT, that type 2 innate lymphoid cells (ILC2s) in the lower GI tract are sensiti

149 IL-33-induced accumulation of type 2 innate lymphoid cells (ILC2s) in the lungs, which perpetuate ty

150 Group 2 innate lymphoid cells (ILC2s) mediate allergic immunity but hav

151 servations in prostate cancer.Group 2 innate lymphoid cells (ILC2s) modulate inflammatory and allergi

152 t activation of lung-resident group 2 innate lymphoid cells (ILC2s) orchestrated suppression of natur

153 Group 2 innate lymphoid cells (ILC2s) play a key role in the initiation

154 The recently identified type 2 innate lymphoid cells (ILC2s) play significant roles in the pat

155 Group 2 innate lymphoid cells (ILC2s) possess indispensable roles durin

156 Group 2 innate lymphoid cells (ILC2s) produce type 2 cytokines, and alt

157 sis at mucosal barriers, with group 2 innate lymphoid cells (ILC2s) producing type 2 cytokines and co

158 In innate immunity, IL-33 and group 2 innate lymphoid cells (ILC2s) provide an essential axis for rap

159 Group 2 innate lymphoid cells (ILC2s) regulate immunity, inflammation,

160 Group 2 innate lymphoid cells (ILC2s) regulate inflammation and immunit

161 Group 2 innate lymphoid cells (ILC2s) represent a subset of newly disco

162 Type 2 innate lymphoid cells (ILC2s) represent an important type 2 imm

163 Type 2 innate lymphoid cells (ILC2s) resemble TH2 cells and produce th

164 Group 2 innate lymphoid cells (ILC2s) reside in multiple organs in the

165 Group 2 innate lymphoid cells (ILC2s) secrete high amounts of T(H)2 cyt

166 nt in eliciting and activating type 2 innate lymphoid cells (ILC2s) that may act throughout allergic

167 o define the tissue niches of group 2 innate lymphoid cells (ILC2s), which are critical instigators o

168 Group 2 innate lymphoid cells (ILC2s), which promote tissue eosinophili

169 results in the activation of group 2 innate lymphoid cells (ILC2s), which subsequently drive increas

170 phenotype that is dependent on type 2 innate lymphoid cells (ILC2s).

171 n and activation of intestinal type-2 innate lymphoid cells (ILC2s).

172 e inflammation, especially in group 2 innate lymphoid cells (ILC2s).

173 ne (IL-5, IL-13) production by type 2 innate lymphoid cells (ILC2s).

174 (Treg) cells and the emerging type 2 innate lymphoid cells (ILC2s).

175 activity of islet-associated group 2 innate lymphoid cells (ILC2s).

176 inflammation, eosinophils and group 2 innate lymphoid cells (ILC2s).

177 cts of IL-35 were assessed on group 2 innate lymphoid cells (ILC2s); dendritic cells primed with thym

178 ing IL-22 production from the group 3 innate lymphoid cell (ILC3) in an aryl hydrocarbon receptor dep

179 eukin-22 (IL-22), produced by group 3 innate lymphoid cells (ILC3) and gammadelta T cells, is an impo

180 ant and persistent decrease in type 3 innate lymphoid cells (ILC3) in the lamina propria.

181 Here we demonstrate that group 3 innate lymphoid cells (ILC3) regulate steady state interactions

182 te activation hubs containing group 3 innate lymphoid cells (ILC3) that continuously produce interleu

183 sponses that are controlled by type-3 innate lymphoid cells (ILC3)(1-3).

184 Type 3 innate lymphoid cells (ILC3s) are critical for lung defense aga

185 Group 3 innate lymphoid cells (ILC3s) are important regulators of the i

186 Group 3 innate lymphoid cells (ILC3s) are major regulators of inflammat

187 CCR6- group 3 innate lymphoid cells (ILC3s) are mediators of intestinal immun

188 that, in the small intestine, group-3 innate lymphoid cells (ILC3s) are the dominant cellular source

189 Group 3 innate lymphoid cells (ILC3s) are the innate counterparts of Th

190 delta (gammadelta) T cells and Type 3 innate lymphoid cells (ILC3s) as important producers of IL-22.

191 Group 3 innate lymphoid cells (ILC3s) have emerged as master regulators

192 T helper 17 (Th17) cells and group 3 innate lymphoid cells (ILC3s) in response to the gut microbiota

193 RORgammat(+) group 3 innate lymphoid cells (ILC3s) maintain intestinal homeostasis t

194 ukin-22 (IL-22) production by group 3 innate lymphoid cells (ILC3s) protects against pathobionts tran

195 Group 3 innate lymphoid cells (ILC3s) regulate intestinal immunity and

196 Group 3 innate lymphoid cells (ILC3s) sense environmental signals that

197 As such, group 3 innate lymphoid cells (ILC3s) that reside in the intestinal muc

198 Group 3 innate lymphoid cells (ILC3s), but not ILC1s or ILC2s, were sim

199 but not T cells, B cells nor group 3 innate lymphoid cells (ILC3s), is critical specifically for the

200 Innate lymphoid cells (ILCs) and CD4(+) T cells produce IL-22,

201 compartmentalization of skin-resident innate lymphoid cells (ILCs) and modulation of sebaceous glands

202 Innate lymphoid cells (ILCs) are critical for host defense and

203 Innate lymphoid cells (ILCs) are enriched at barrier surfaces o

204 Innate lymphoid cells (ILCs) are generated early during ontogen

205 athways underlying the development of innate lymphoid cells (ILCs) are mostly unknown.

206 Innate lymphoid cells (ILCs) are rapidly-responding cells that

207 Innate lymphoid cells (ILCs) are strategically positioned at mu

208 Innate lymphoid cells (ILCs) are the most recently identified l

209 Innate lymphoid cells (ILCs) are tissue-resident "first respond

210 Innate lymphoid cells (ILCs) are tissue-resident lymphocytes ca

211 Innate lymphoid cells (ILCs) are tissue-resident lymphocytes th

212 Innate lymphoid cells (ILCs) are tissue-resident sentinels that

213 Innate lymphoid cells (ILCs) communicate with other haematopoie

214 Innate lymphoid cells (ILCs) consist of a heterogeneous family

215 Intestinal innate lymphoid cells (ILCs) contribute to the protective immun

216 Although innate lymphoid cells (ILCs) functionally analogous to T helper

217 Innate lymphoid cells (ILCs) guard epithelial tissue integrity

218 In particular, the discovery of innate lymphoid cells (ILCs) has opened entirely new avenues fo

219 Innate lymphoid cells (ILCs) have an important role in the immu

220 Innate lymphoid cells (ILCs) have been classified into "functio

221 al importance of an emerging group of innate lymphoid cells (ILCs) in health and disease.

222 However, the involvement of innate lymphoid cells (ILCs) in immune responses to infection w

223 s and they control the development of innate lymphoid cells (ILCs) in the bone marrow.

224 ne production by CRTH2(-)IL7Ralpha(-) innate lymphoid cells (ILCs) is unknown.

225 factor alpha [TNF-alpha]) produced by innate lymphoid cells (ILCs) located in the colon secondary to

226 Depleting group I innate lymphoid cells (ILCs) or infecting Trail(-/-) mice compl

227 Innate lymphoid cells (ILCs) patrol environmental interfaces to

228 Innate lymphoid cells (ILCs) play critical roles in immune home

229 Although innate lymphoid cells (ILCs) play fundamental roles in mucosal

230 Innate lymphoid cells (ILCs) play important functions in immuni

231 Innate lymphoid cells (ILCs) play important roles in host defen

232 or the first time what roles, if any, innate lymphoid cells (ILCs) play in HSV-1 infection.

233 Innate lymphoid cells (ILCs) play strategic roles in tissue hom

234 Innate lymphoid cells (ILCs) promote tissue homeostasis and imm

235 Recent findings that innate lymphoid cells (ILCs) regulate adaptive T cell responses

236 iller (NK) cells are cytotoxic type 1 innate lymphoid cells (ILCs) that defend against viruses and me

237 wo independent cohorts, we found that innate lymphoid cells (ILCs) were depleted in the blood and gut

238 Innate lymphoid cells (ILCs) were originally classified based o

239 y macrophages, dendritic cells (DCs), innate lymphoid cells (ILCs), and natural killer (NK) cells.

240 pid depletion of all three subsets of innate lymphoid cells (ILCs), ILC1s, ILC2s and ILC3s, which is

241 GR is selectively deleted in NKp46(+) innate lymphoid cells (ILCs), we demonstrated a major role for

242 rescued IL-22 production from group 3 innate lymphoid cells (ILCs), whereas IL-6 administration durin

243 ique characteristics of skin-resident innate lymphoid cells (ILCs).

244 Ti) cells are regarded as a subset of innate lymphoid cells (ILCs).

245 , demonstrating an important role for innate lymphoid cells (ILCs).

246 ular bacterial mutant, is produced by innate lymphoid cells (ILCs).

247 ged with the discovery of a family of innate lymphoid cells (ILCs): ILC1s, ILC2s, and ILC3s.

248 Further, myeloid and lymphoid cells in ascites were predominantly linked to t

249 sh biopsy specimens while sparing the normal lymphoid cells in the tumor microenvironment.

250 rived cytokines that activate group 2 innate lymphoid cells, induce migration and activation of dendr

251 Characteristic of memory responses, more lymphoid cells infiltrated the prostate in a second infe

252 Maturation of lymphoid cells is controlled by the action of stage and

253 in multiple chronically infected myeloid and lymphoid cell lines using an anti-ASP monoclonal antibod

254 proliferation in the P493-6 and RAMOS human lymphoid cell lines.

255 escribed as a diffuse network of myeloid and lymphoid cells located in the olfactory organ of fish.

256 dent leukocyte populations, including innate lymphoid cells, macrophages, natural killer and natural

257 prisingly, besides CD4(+) T cells and innate lymphoid cells, mast cells are a source of GM-CSF in thi

258 with pathways associated with type 2 innate lymphoid cells, monocytes, neutrophil trafficking, and T

259 phages, neutrophils, dendritic cells, innate lymphoid cells, myeloid-derived suppressor cells, and na

260 el mechanistic roles for macrophages, innate lymphoid cells, natural killer cells, innate gammadelta

261 They either differentiate into innate lymphoid cells or migrate to the thymus to give rise to

262 t may interact with immune cells to regulate lymphoid cell organization and type 2 inflammation.

263 mphoid cells (ILC2s) are the dominant innate lymphoid cell population in the lungs at steady state, a

264 roteins were detected in both epithelial and lymphoid cell populations expressing CD155 in the tonsil

265 tion and function of mature T, B, and innate lymphoid cell populations including natural killer (NK)

266 sturbing the development of GATA-3-dependent lymphoid cell populations.

267 tosis and markedly decreasing premalignant B lymphoid cell populations.

268 nce of mast cells, natural killer and innate lymphoid cell precursors in the yolk sac.

269 uced IL-22 expression, which required innate lymphoid cells, prevented microbiota encroachment and pr

270 hat chronic SIV-infected gut contains innate lymphoid cells producing inflammatory cytokines.

271 ment of protocols for robust granulocyte and lymphoid cell production from hPSCs, for adoptive immuno

272 system is characterized by a high myeloid-to-lymphoid cell ratio that includes a novel, lipopolysacch

273 ssed on natural killer (NK) cells and innate lymphoid cells, recognizes PDGF-DD.

274 suppressor, as lack of Ikaros in developing lymphoid cells results in leukemia.

275 eased STAT1 and STAT3 signaling responses in lymphoid cell subsets after surgery, consistent with enh

276 PRKCQ gene expression was assessed in innate lymphoid cell subsets purified from human PBMCs and mous

277 ct is eventually rescued by RPL10-R98S mouse lymphoid cells that acquire 5-fold more secondary mutati

278 atural killer cells constitute potent innate lymphoid cells that play a major role in both tumor immu

279 Lymphoid cells that produce interleukin (IL)-17 cytokine

280 (IELs) are a large and diverse population of lymphoid cells that reside between the intestinal epithe

281 While SBS85 is a denoted signature of AID in lymphoid cells, the etiologies of SBS37 and SBS39 are un

282 es can modulate the behaviour of myeloid and lymphoid cells, thereby empowering anticancer immunity a

283 T(reg) cells, which activated group 2 innate lymphoid cells to provide a feed-forward mechanism for a

284 pithelial cells, dendritic cells, and innate lymphoid cells translates to T-cell outcomes, with an em

285 ese mice, ozone increased lung IL-13+ innate lymphoid cells type 2 (ILC2) and IL-13+ gammadelta T cel

286 IL-13 from T helper 2 (Th2) cells and innate lymphoid cells type 2 (ILCs), and increased airway smoot

287 e cytokines, especially IL-33, target innate lymphoid cells type 2 to produce type 2 cytokines.

288 pend on IL-22, a cytokine produced by innate lymphoid cells type 3 (ILC3) during Enterobacteriaceae i

289 rked contrast to previous studies with non-B lymphoid cell types, for which K8.1A is known to be disp

290 (vagina and cervix), whereas APCs and innate lymphoid cells were mainly located in the upper tract (u

291 inflammation, mast cells, and group 3 innate lymphoid cells were more enriched in adult-onset severe

292 The lymphoid cells were positive for CD3, CD4, CD5, CD7 and

293 or not expressing CD69 on either myeloid or lymphoid cells were subjected to a high fat diet.

294 ns of specific subsets of T cells and innate lymphoid cells, which are key drivers of inflammatory di

295 ils and mast cells and the intriguing innate lymphoid cells, which are present in the circulation and

296 ges and interleukin (IL)-22-producing innate lymphoid cells, which in turn promote pSTAT3 signalling

297 ue injury in sepsis, activates type 2 innate lymphoid cells, which promote polarization of M2 macroph

298 of using an immunomodulatory mAb to regulate lymphoid cells, which then recruit and activate myeloid

299 RAS oncogene family (RAP1) in human Kit 225 lymphoid cells, which, in turn, is regulated by the epid

300 es of alloantigens, the cross talk of innate lymphoid cells with damaged epithelia and with the recip