ライフサイエンスコーパス: lymphoid tissue

1 tic cell (DC) functions in mucosa-associated lymphoid tissue.

2 np40 is ubiquitous but strongly expressed in lymphoid tissue.

3 id not develop persistent CHIKV infection in lymphoid tissue.

4 p to B cells in the germinal centers (GC) of lymphoid tissue.

5 e peripherally, as well as reside within the lymphoid tissue.

6 oid dendritic cells (pDCs) in both blood and lymphoid tissue.

7 s, B cells and T cells in the gut-associated lymphoid tissue.

8 and having a unique conjunctival associated lymphoid tissue.

9 , specifically, in locations associated with lymphoid tissue.

10 ) expression in trout nasopharynx-associated lymphoid tissue.

11 of human memory B cells of mucosa-associated lymphoid tissue.

12 t virus in blood, CSF, intestinal tissue, or lymphoid tissue.

13 SAMHD1 antibodies were prevalent in tertiary lymphoid tissue.

14 marginal zone lymphoma of mucosa-associated lymphoid tissue.

15 o identify Ag-specific GC Tfh cells in human lymphoid tissue.

16 xis of inflammatory monocytes into blood and lymphoid tissue.

17 esponses in the periphery and gut-associated lymphoid tissue.

18 ecting the biology of Tfh in blood to Tfh in lymphoid tissue.

19 ion in peripheral blood compared to those in lymphoid tissue.

20 s and finally degraded inside DCs within the lymphoid tissue.

21 promote virus spread locally and to distant lymphoid tissues.

22 C3s and influenced ILC3 abundance in colonic lymphoid tissues.

23 B cell response to RSV in mucosa-associated lymphoid tissues.

24 form fundamental structural units supporting lymphoid tissues.

25 ates that GGT5 establishes a GGG gradient in lymphoid tissues.

26 cell numbers and functions in the liver and lymphoid tissues.

27 ation of immunomodulatory genes in secondary lymphoid tissues.

28 eflecting a redistribution of these cells to lymphoid tissues.

29 followed by rapid dissemination to systemic lymphoid tissues.

30 WD) replication had expanded to all systemic lymphoid tissues.

31 lusively within B cells of mucosa-associated lymphoid tissues.

32 immunity despite persistence of infection in lymphoid tissues.

33 on IL-7 amounts in the primary and secondary lymphoid tissues.

34 n of B cells in the hematopoietic system and lymphoid tissues.

35 phocytes, including CD4(+) T cells, into gut lymphoid tissues.

36 s exposure to monitor the immune response in lymphoid tissues.

37 distribution of multiple cell populations in lymphoid tissues.

38 roduction within pathologically inflamed non-lymphoid tissues.

39 om donor DCs that migrated from the graft to lymphoid tissues.

40 metastasized ovarian cancer cells within the lymphoid tissues.

41 tion by B cells, derived from gut-associated lymphoid tissues.

42 ction through continuous surveillance of non-lymphoid tissues.

43 mic immune response in primary and secondary lymphoid tissues.

44 re-activation cycles and clonal expansion in lymphoid tissues.

45 rrelating with the magnitude of Tfh cells in lymphoid tissues.

46 eostasis both in lymphoid tissues and in non-lymphoid tissues.

47 ng blood flow and immune cell recruitment in lymphoid tissues.

48 ation and regulates the growth of B cells in lymphoid tissues(1,2).

49 ZIKV infection caused a significant lymphoid tissue activation but limited induction of ZIKV

50 d5) bio-distributes at very low level to non-lymphoid tissues after skin immunisation.

51 Lymphoid tissues also express the immunoproteasome subun

52 n T cell-dependent clearance of CHIKV RNA in lymphoid tissue, although this effect was not observed i

53 eering work in the fields of T cell biology, lymphoid tissue anatomy, lymphocyte trafficking and muco

54 which deliver antigen to mucosal-associated lymphoid tissue and aim to elicit protective CTL-mediate

55 ncluding vaccine-induced bronchus-associated lymphoid tissue and CD8(+) effector memory T cells.

56 ion trajectory of regulatory T cells between lymphoid tissue and colon.

57 , induction of inducible bronchus-associated lymphoid tissue and correlates of bacterial killing, red

58 humanized rodent models developed with human lymphoid tissue and hematopoietic stem cell transplants.

59 in vivo characteristics, with high uptake in lymphoid tissue and hPBMC xenografts.

60 o resistance as an arm of the eye-associated lymphoid tissue and may also be susceptible to infection

61 al intervention targeting the gut-associated lymphoid tissue and microbiota.

62 A small population of B cells exists in lymphoid tissues and body cavities of mice that is disti

63 ) B cells in the peripheral blood, secondary lymphoid tissues and bone marrow.

64 cART reduced the size of HIV-1 reservoirs in lymphoid tissues and delayed HIV-1 rebound after cART ce

65 cient Trm cells progressively decayed in non-lymphoid tissues and expressed dysregulated Trm-specific

66 CXCR5+ SIV-specific CD8+ T cells in various lymphoid tissues and higher proportions of unique Gag-sp

67 he maintenance of immune homeostasis both in lymphoid tissues and in non-lymphoid tissues.

68 that only a small number of donor DCs reach lymphoid tissues and investigated how this limited popul

69 ever, it remains unclear whether GC Tfh exit lymphoid tissues and populate this cTfh pool.

70 s CD8(+) T-cell population was found only in lymphoid tissues and resided predominantly in the T-cell

71 ls (including Th follicular functionality in lymphoid tissues and Th2 responses in bronchoalveolar la

72 CD4 T cells can be readily detected in mouse lymphoid tissues and that latent HIV-1 was enriched in p

73 ontrols the normal organization of secondary lymphoid tissues and the neogenesis of ectopic lymphoid

74 of activated Tregs that readily migrate into lymphoid tissues and the pancreas while inhibiting autoa

75 increase their activation and migration into lymphoid tissues and the pancreas.

76 IL-1beta and its actions during EAE, in both lymphoid tissues and within the CNS.

77 irculating autoantibodies, lung perivascular lymphoid tissue, and elevated cytokines have been relate

78 r efficacy in controlling HIV replication in lymphoid tissues, and (ii) cytotoxic CD8(+) T cells with

79 full-thickness fetal skin, autologous fetal lymphoid tissues, and autologous fetal liver-derived hem

80 ful co-engraftment of human skin, autologous lymphoid tissues, and autologous immune cells in a roden

81 n full-thickness skin, along with autologous lymphoid tissues, and autologous immune cells.

82 romote cell-mediated transport of antigen to lymphoid tissues, and promote antigen retention in LNs.

83 f vaccine components to key immune cells and lymphoid tissues, and they can be highly multivalent, im

84 es that bind to CD8(+) T cells in the blood, lymphoid tissues, and tumors of mice.

85 esiding in B-cell follicles within secondary lymphoid tissues, are readily infected by AIDS viruses a

86 The role of lymphoid tissue as a potential source of HIV-1 rebound f

87 CWD pathogenesis have implicated pharyngeal lymphoid tissue as the earliest sites of prion accumulat

88 nd increase with age in systemic and mucosal lymphoid tissues as a heterogeneous population of polyfu

89 rostate, breast, colon, kidney, thyroid, and lymphoid tissues as well as NETs as reference.

90 follicular immunoreactivity in oropharyngeal lymphoid tissues at 1 and 2 months postexposure (MPE).

91 ut mice, SVNI replicated more efficiently in lymphoid tissues at early times postinfection and induce

92 a potent P2RY8 ligand that is detectable in lymphoid tissues at the nanomolar level.

93 cells and antigen-presenting cells (APCs) in lymphoid tissue, away from an immunosuppressive microenv

94 ganized and distinctive Bronchial Associated Lymphoid Tissue (BALT) not induced by any wild type Bord

95 cumulated within induced bronchus-associated lymphoid tissue (BALT) of tolerant mouse lungs.

96 cent work has shown that bronchus-associated lymphoid tissue (BALT), characterized by the development

97 emonstrate reduced accumulation in secondary lymphoid tissue because of low levels of proliferation 4

98 s decline drastically in the circulation and lymphoid tissues before death.

99 roviral DNA; virus is not detected in blood, lymphoid tissue, bone marrow and brain by nested and dig

100 a unique feature of peripheral MCs to affect lymphoid tissue-borne adaptive immunity over distance by

101 in healthy lung and lung lesions and in the lymphoid tissues bronchial lymph node, retropharyngeal l

102 dendritic cell (DC) homeostasis in secondary lymphoid tissues but necessary to regulate cellular meta

103 ily infects CD11b(+) DCs in non-lymphoid and lymphoid tissues, but spares the main cross-presenting C

104 The clearance of CHIKV RNA from lymphoid tissue by anti-CD137 MAb was associated with re

105 of dysregulated infected T cell motility in lymphoid tissue by preventing stable cellular polarizati

106 esolution of chronic alphavirus infection in lymphoid tissues by reducing the numbers of target cells

107 infused CD8 T cells, especially in secondary lymphoid tissues, by minimizing ex vivo culture/manipula

108 and the generation of Th1 cells in draining lymphoid tissues; (c) decreases graft infiltration of al

109 n together, these results suggest that human lymphoid tissues can be sites of silent IAV infections w

110 hed in intestinal macrophages, and a generic lymphoid tissue cDC cluster associated with Ccr7.

111 ssion of KLRG1 on infection-protective human lymphoid tissue central memory T cells and autoimmune-pr

112 Starting with cell encapsulation in digested lymphoid tissues, clusters of proliferating B cells with

113 ession at the injection site and in draining lymphoid tissue compared to a nonamphiphilic control and

114 ture antigen-presenting cells trafficking to lymphoid tissue compared with control (6.96 +/- 0.9 vs 1

115 ole of Nkx2.3 in colonic solitary intestinal lymphoid tissue composition and in colitis pathogenesis.

116 D20(+) B lymphocytes in peripheral blood and lymphoid tissues confirming that SGN-CD19B is pharmacody

117 ponses within the liver, adrenal glands, and lymphoid tissues contribute to EBOV pathogenesis.

118 ) T cell responses in the graft and regional lymphoid tissue [Correction added on January 9, 2019, af

119 We sought to investigate whether these lymphoid tissues could be sites of viral replication and

120 In lymphoid tissues, CSF1R-FRed highlighted diverse MPS pop

121 Unlike their counterparts in lymphoid tissues, cutaneous IgM-secreting cells were com

122 ynamics with pathological immune activation, lymphoid tissue damage progressing to significant immuno

123 sing a loss of CD4(+) T cell homeostasis and lymphoid tissue damage that lead to AIDS in HIV-1 and SI

124 (89)Zr-muS110 uptake in the spleen and other lymphoid tissues decreased and was comparable to uptake

125 al fluorescence microscopy of thin-sectioned lymphoid tissues demonstrated strong preferential locali

126 cluding TRM cells, in nasopharynx-associated lymphoid tissue, demonstrating its strong capacity to ex

127 these mice nor was the formation of enteric lymphoid tissue, demonstrating that the onset of RA sign

128 Rorc(fm+)) ILCs show a clear ILC3 phenotype, lymphoid tissue-derived Rorc(fm+) ILCs acquire an natura

129 the interface between circulating blood and lymphoid tissue, detect and respond to blood-borne antig

130 etion of NLK does not affect mouse health or lymphoid tissue development.

131 nd viral infections are well-known causes of lymphoid tissue disorganization, although the factors, b

132 lymphoma (FL) is a systemic neoplasm of the lymphoid tissue displaying germinal centre (GC) B cell d

133 ls out of lymphoid organs and subsequent non-lymphoid tissue distribution but also their phenotypic d

134 Ls), including DLBCLs with mucosa-associated lymphoid tissue (DLBCL[MALT]) and without ("pure" DLBCL)

135 dysbiotic hyperimmune response at secondary lymphoid tissues draining local gut and systemic circula

136 (+) T cells in B cell follicles of secondary lymphoid tissues during asymptomatic disease.

137 imately foster the establishment of tertiary lymphoid tissues during chronic neuroinflammatory condit

138 Despite their absence in secondary lymphoid tissues during homeostasis, gammadeltaT17 cells

139 and identification of leptomeningeal ectopic lymphoid tissue (ELT) in patients with multiple sclerosi

140 V infection both in the peripheral blood and lymphoid tissues, especially in the setting of persistin

141 contributes little to HIV infection of human lymphoid tissue ex vivo.

142 ry gp120, decreases viral infection of human lymphoid tissue ex vivo.

143 d the increased APC recruitment to secondary lymphoid tissues expand the scope of known adjuvant effe

144 s present evidence that stromal cells within lymphoid tissue express the Notch ligands Delta-like 1/4

145 es, and (ii) cytotoxic CD8(+) T cells within lymphoid tissues express low levels of chemokine recepto

146 on amplification occurs in the oropharyngeal lymphoid tissues followed by rapid dissemination to syst

147 ds that partially mimic the B-cell zone of a lymphoid tissue, for efficient and rapid generation of B

148 on, interactions with skin T cells, tertiary lymphoid tissue formation, and production of proinflamma

149 phocyte accumulation and bronchus-associated lymphoid tissue formation.

150 cluding TRM cells, in nasopharynx-associated lymphoid tissue from children and adults.

151 er investigation of ASFVsRNA2 detected it in lymphoid tissue from pigs with ASF.

152 Here we studied peripheral blood and lymphoid tissues from antiretroviral (ARV)-treated and A

153 protein (PrP(Sc)) was detected in brain and lymphoid tissues from intracranially and orally inoculat

154 Intestinal lymphoid tissues from SNF1 mice, females particularly, s

155 a new perspective on DCs as facilitators of lymphoid tissue function.

156 indication of short-lived ASCs in the local lymphoid tissue, further evidence of a TI-2 response to

157 The gut-associated lymphoid tissue (GALT) faces a considerable challenge.

158 a method for isolating human gut-associated lymphoid tissues (GALTs) that allows unprecedented profi

159 ation in intestine and in the gut-associated lymphoid tissues (GALTs).

160 and a site of viral replication, similar to lymphoid tissue, gut-associated lymphoid tissue or semen

161 The studies also revealed that the local lymphoid tissue had detectable FMDV-specific ASCs in the

162 By 4 MPE, the PrP(CWD) burden in all lymphoid tissues had increased and approached levels obs

163 reas, the diabetogenic insulitis lesion, and lymphoid tissues have revealed a broad repertoire of tar

164 8 RNA levels in various organs revealed that lymphoid tissues have the highest levels of expression o

165 ha) and caused inducible bronchus-associated lymphoid tissue (iBALT) formation in the lung.

166 iscovered that inducible bronchus-associated lymphoid tissue (iBALT) forms in response to infection w

167 Inducible bronchus-associated lymphoid tissue (iBALT) is a tertiary lymphoid structure

168 d cells in the inducible bronchus-associated lymphoid tissue (iBALT) likely contributed to dysregulat

169 ount for delayed gastrointestinal-associated lymphoid tissue immune recovery.

170 evaluation of humoral immunity and secondary lymphoid tissues in an established KS (Kmt2d(+/betaGeo))

171 longitudinally assess early HIV-1 spread in lymphoid tissues in humanized mice.

172 g latent viruses circulate between blood and lymphoid tissues in individuals on ART and support the i

173 ction in CD4(+) T cells that can be found in lymphoid tissues in infected individuals during ART.

174 hocyte migration and compartmentalization of lymphoid tissues in mammals, diversified in salmonids le

175 ll T lymphocytes in the peripheral blood and lymphoid tissues in mice and humans.

176 lustrated by impaired development of enteric lymphoid tissues in vitamin A-deficient mice.

177 ssification of Tumours of Haematopoietic and Lymphoid Tissues includes new criteria for the diagnosis

178 moking-related inducible bronchus-associated lymphoid tissue, induced regeneration of lung tissue, an

179 oxyvitamin D concentration in the peripheral lymphoid tissues, induced Treg cells.

180 al for the development of RORgammat(+) fetal lymphoid tissue inducer (LTi) cells and lymphoid organog

181 Lymphoid tissue inducer (LTi) cells are regarded as a su

182 nown to be expressed by natural killer (NK), lymphoid tissue inducer (LTi), and innate lymphoid cell

183 he pool of alpha4beta7(-) and alpha4beta7(+) lymphoid tissue inducer cell progenitors in the fetal li

184 nally, the percentage of cells thought to be lymphoid tissue inducer cells among donor ILCs was far h

185 ed the number of fetal liver progenitors and lymphoid tissue inducer cells in the neonatal intestine,

186 plantation and support the notion that human lymphoid tissue inducer cells may form in the fetus and

187 The dependence of lymphoid tissue inducer cells on vitamin A was furthermo

188 However, lymphoid tissue inducer cells were not affected in these

189 lated orphan receptor gammat is expressed on lymphoid tissue inducer cells.

190 utively high IL-22 expression was limited to lymphoid-tissue inducer (LTi) cells residing in lymph no

191 t, it negatively regulated genes specific to lymphoid tissue-inducer (LTi) or LTi-like ILC3 cells.

192 ant development of ILCs, including ILC2s and lymphoid-tissue-inducer-like cells.

193 uding the conventional natural killer cells, lymphoid tissue inducers, type 1, 2, and 3 with signific

194 ghlights differences in HIV-1 stimulation of lymphoid tissue inflammation and peripheral blood.

195 hin the major conduit of efferent lymph from lymphoid tissues into blood, the human thoracic duct.

196 cation scheme to segregate B cells from four lymphoid tissues into twelve unique subsets, including a

197 Lymphoid tissue is a key reservoir established by HIV-1

198 inflammatory stimulus, IFN-gamma signal from lymphoid tissue is detectable in vivo.

199 However, lymphoid tissue is rarely available from immunized human

200 The cellular homeostasis of lymphoid tissues is determined by the continuous interac

201 s to bone marrow, whereas migration to other lymphoid tissues is independent of B cell GRs.

202 Dissemination of HIV-1 throughout lymphoid tissues leads to systemic virus spread followin

203 in tissues of infected nonhuman primates and lymphoid tissue (LT) biopsies from infected humans.

204 t T cell-intrinsic role of mucosa-associated lymphoid tissue lymphoma translocation protein 1 (MALT1)

205 adenocarcinoma, or gastric mucosa-associated lymphoid tissue lymphoma.

206 follicles had features of mucosa-associated lymphoid tissue lymphoma.

207 rated superior efficacy in mucosa-associated lymphoid tissue lymphoma; however, improvements in EFS a

208 ere the most frequent, and mucosa-associated lymphoid tissue lymphomas (5.8%).

209 products that are secreted and accumulate in lymphoid tissues, mainly within lymph node germinal cent

210 Z) B-cell lymphomas of the mucosa-associated lymphoid tissue (MALT) arise from lymphoid populations t

211 ciated epithelial defects, mucosa-associated lymphoid tissue (MALT) hyperplasia, and dysplasia.

212 ile primary ocular adnexal mucosa-associated lymphoid tissue (MALT) lymphoma (POAML) is the most comm

213 sly received rituximab for mucosa-associated lymphoid tissue (MALT) lymphoma and steroids for prolong

214 mmunohistochemistry showed mucosa-associated lymphoid tissue (MALT) lymphoma with immunoglobulin kapp

215 e best OS in patients with mucosa-associated lymphoid tissue (MALT) lymphomas (HR = 0.26, 95%CI: 0.11

216 igens to interact with the mucosa-associated lymphoid tissue (MALT) to induce both mucosal and system

217 marginal zone lymphoma of mucosa-associated lymphoid tissue (MALT).

218 marginal zone lymphoma of mucosa-associated lymphoid tissue (MALT).

219 lymphocytes also establish residency in non-lymphoid tissues, most prominently at barrier sites, inc

220 corhynchus mykiss), a nasopharynx-associated lymphoid tissue (NALT) was recently described as a diffu

221 In the nasal-associated lymphoid tissue (NALT), a muted inflammatory response to

222 s a robust Th17 response in nasal-associated lymphoid tissue (NALT).

223 ncluding TRM cells in nasopharynx-associated lymphoid tissue(NALT) from children and adults.

224 nvestigated the role of the nasal-associated lymphoid tissues (NALTs), which are mucosal-associated l

225 Non-lymphoid tissues (NLTs) harbor a pool of adaptive immune

226 The data indicate that lymphoid tissues not only harbor expression of IAV prote

227 We studied the colonic solitary intestinal lymphoid tissue of Nkx2.3-deficient mice with immunofluo

228 gD CSR also was detected in nasal-associated lymphoid tissue of WT mice.

229 ed by PrP(CWD) accumulation in the brain and lymphoid tissues of affected animals.

230 Notably, SVA RNA has been detected in lymphoid tissues of infected animals several weeks follo

231 By analysing iNKT cells from lymphoid tissues of mice and humans we demonstrate that

232 The amount of PrP(Sc) in the brains and lymphoid tissues of positive pigs was small, especially

233 The lymphoid tissues of the gut are specialized for the indu

234 , similar to lymphoid tissue, gut-associated lymphoid tissue or semen.

235 kine and chemokine production that can alter lymphoid tissue organization, GC B cell development, and

236 13 dysregulation also contributes to loss of lymphoid tissue organization.

237 ICAM), suggesting they are early mesenchymal lymphoid tissue organizer (mLTo) cells.

238 receptor (LTbetaR) signaling is crucial for lymphoid tissue organogenesis and immune homeostasis.

239 ducing cells in circulation and in secondary lymphoid tissues, particularly the spleen (80%).

240 tes such as the lung into the gut-associated lymphoid tissues, Peyer's patches, and thus reduced the

241 that, in addition to antibody production in lymphoid tissues, plasma cells reside in healthy mouse a

242 The nose, paranasal sinuses, and associated lymphoid tissues play important roles in homeostasis and

243 hat form the reticular networks in organized lymphoid tissues, potentially linking two areas of fibro

244 on: a robust prion amplification in systemic lymphoid tissues prior to neuroinvasion and establishmen

245 VHD, whereas PD-L1 interactions with CD80 in lymphoid tissue promoted CD8+ T cell survival and expans

246 c lymphocytic leukemia (B-CLL) occurs within lymphoid tissue pseudofollicles.

247 on replication in the draining oropharyngeal lymphoid tissues, rapidly followed by dissemination to s

248 lassification of Tumors of Hematopoietic and Lymphoid Tissue; refractory disease or relapsed after au

249 rstood, the host responses within organs and lymphoid tissues remain poorly characterized.

250 sue injury in the liver, adrenal glands, and lymphoid tissues remains limited.

251 ion spread are generally observed: (i) early lymphoid tissue replication or (ii) direct neuroinvasion

252 atment fully suppresses viral replication in lymphoid tissue reservoirs.

253 ir functional properties, we found that only lymphoid-tissue resident Rorc(fm+) ILCs can suppress tum

254 Besides lymphoid tissue-resident Tfh cells, CXCR5(+) circulating

255 ating HIV reservoirs in peripheral blood and lymphoid tissues, residual sources of virus may remain i

256 w immune cells in the gut and gut-associated lymphoid tissues respond to IL-2C is not well characteri

257 reactive CD8(+) T cells isolated from murine lymphoid tissue retained developmentally plastic phenoty

258 In cultures with T cell-depleted lymphoid tissues, retinol enhanced Treg induction from D

259 Tem biology, including enhanced presence in lymphoid tissues, robust IL-2 production, and recall pot

260 Secondary lymphoid tissues share the important function of bringin

261 Analysis of lymphoid tissues showed early upregulation of genes that

262 ctures in the gut called solitary intestinal lymphoid tissues (SILT).

263 ith rheumatoid arthritis (RA) infiltrate non-lymphoid tissue sites, maneuver through extracellular ma

264 populations tend to have a greater amount of lymphoid tissue, smaller airways, and smaller lower faci

265 Solitary intestinal lymphoid tissues such as cryptopatches (CPs) and isolate

266 ptor and are preferentially localized in non-lymphoid tissues, such as mucosal barriers.

267 In the intestine, DC are found in organized lymphoid tissues, such as the mesenteric lymph nodes and

268 tion and production of IL-10 and IL-1beta in lymphoid tissue, suggesting a novel approach to HIV-1 th

269 Memory B cells are found in lymphoid and non-lymphoid tissues, suggesting that some may be tissue-res

270 ch are more radio-resistant than circulating/lymphoid tissue T cells.

271 opulations of viruses in the inoculum and in lymphoid tissue taken at 35 dpi.

272 g to, and poor persistence in, the secondary lymphoid tissues targeted by AIDS viruses.

273 The lymphoid tissue that drains the upper respiratory tract

274 odeficiency virus 1 infection are located in lymphoid tissues that are difficult to access.

275 ransport of solutes across it and associated lymphoid tissues that play a sentinel role.

276 -4)-positive T cells (compared with reactive lymphoid tissues) that outnumber PD-1-positive and lymph

277 In lymphoid tissues, the interactions of PD-L1 with CD80 au

278 Because they occur in the immunocyte-rich lymphoid tissues, they are easily accessible to antibodi

279 During chronic inflammation, tertiary lymphoid tissue (TLT) can form within an inflamed organ,

280 Therefore, modulating gut-associated lymphoid tissue to boost Tr1 cells may be important in t

281 F]-FDG-PET/CT imaging of immune processes in lymphoid tissues to identify patterns of inflammation in

282 ing fetal ILC1-like NKPs travel to secondary lymphoid tissues to initiate the formation of diversifie

283 corneal stroma quickly mature and migrate to lymphoid tissues to sensitize host T cells.

284 Sustained exposure of lymphoid tissues to vaccine antigens promotes humoral im

285 cells were selected in peripheral blood and lymphoid tissues upon HIV-1 infection.

286 erences in DC subsets localized in blood and lymphoid tissues versus skin, and a striking absence of

287 CAR T cell expansion in lymphoid tissue was demonstrated, and inhibition of lymp

288 ferential crosstalk among genes expressed in lymphoid tissues was predicted to be orchestrated by spe

289 d motifs and their tendency to accumulate in lymphoid tissue, we designed amphiphilic lipid-polymer c

290 ownregulation of humoral immune responses in lymphoid tissues were confirmed with flow cytometry.

291 ubs for HIV dissemination but that blood and lymphoid tissues were the main source; (c) that viral ex

292 ecific antibody-secreting cells in secondary lymphoid tissues were unaffected by depletion of CD4(+)

293 Strikingly, NALTs, unlike other lymphoid tissues, were not routinely surveyed during the

294 s the formation of germinal centers (GCs) in lymphoid tissues where self-reactive B cells expand and

295 lic processes in malignant lesions and local lymphoid tissues, which constitute the immunological tum

296 irs by reducing the CD4+ T-cell reservoir in lymphoid tissues, while increasing the microglia/reservo

297 T cells are significantly reduced in mucosal lymphoid tissues with disease progression.

298 In contrast to the lymph nodes and mucosal lymphoid tissues with well-defined entry and exit routes

299 fector Th1 CD4 T cells in ocular lesions and lymphoid tissues, with Treg becoming predominant over th

300 apidly followed by dissemination to systemic lymphoid tissues without evidence of neuroinvasion.