ライフサイエンスコーパス: m-cresol

1 samples were higher in gamma-nonalactone and m-cresol.

2 reaction specificity in HDO of furfural and m-cresol.

3 ne at the meta position, producing primarily m-cresol.

4 in comparison, produces 97% p-cresol and 3% m-cresol.

5 250 microM and produced 90% p-cresol and 10% m-cresol.

6 fferent with resorcinol than with phenol and m-cresol.

7 eniently prepared in five steps from 2-amino-m-cresol.

8 usters Ti(16) O(16) (OEt)(32) , linkers, and m-cresol.

9 catalyzed the oxidation of catechol, 6-amino-m-cresol, 2-amino-m-cresol, and 2-amino-4-chlorophenol.

10 4-Chloro-m-cresol (4-CmC) is a clinically relevant activator of t

11 4-Chloro-m-cresol (4-CmC) is a potent and specific activator of t

12 ponse to high [K(+)], caffeine, and 4-chloro-m-cresol (4-CMC), the maximal tensions generated in Stac

13 sometric specific force followed by 4-chloro-m-cresol (4-CmC)-evoked maximal contracture force in sin

14 RyR3), are efficiently activated by 4-chloro-m-cresol (4-CmC).

15 onsistent with this interpretation, 4-chloro-m-cresol (4-CMC; 100 microm) increases the rate of Ca(2+

16 owed that the RYR-stimulating agent 4-chloro-m-cresol (4CmC) induced Ca(2+) release and thereby confi

17 ,4,6-Trichlorophenol (2) and 2,4,6-trichloro-m-cresol (5) react with calcium hypochlorite (Ca(OCl)(2)

18 +) stores, since the application of 4-chloro-m-cresol, a direct type 1 ryanodine receptor activator,

19 -loaded CD19(+) B and DAKIKI cells, 4-chloro-m-cresol, a potent activator of Ca2+ release mediated by

20 could be restored by application of 4-chloro-m-cresol, a ryanodine receptor agonist, indicating that

21 and ryanodine but not for Ca(2+) or 4-chloro-m-cresol, although they all induced Ca(2+) release.

22 4-Chloro-m-cresol, an activator of the skeletal muscle ryanodine

23 i.e., alcohol dehydration and alkylation of m-cresol and 2-propanol in the liquid phase, at high tem

24 ring precursors 60 and 68 were prepared from m-cresol and 3-ethylphenol, respectively.

25 Both 4-chloro-m-cresol and halothane caused adenosine accumulation in

26 91%) and methylhydroquinone (9%), to oxidize m-cresol and p-cresol to 4-methylcatechol (100%), and to

27 cells was significantly altered by 4-chloro-m-cresol and ryanodine.

28 ation of catechol, 6-amino-m-cresol, 2-amino-m-cresol, and 2-amino-4-chlorophenol.

29 ffusion coefficients of benzene, anthracene, m-cresol, and p-nitrophenol in enhanced-fluidity liquid

30 The oxidative half-reaction of PHHY using m-cresol as a substrate is similarly affected by the mut

31 gands studied in resorcinol >> phenol > or = m-cresol as determined from their overall free energies

32 Phenolic ligands, e.g., phenol and m-cresol, bind to 2Zn(II)-insulin hexamers and induce a

33 1, they were much less sensitive to 4-chloro-m-cresol (CMC).

34 rization or RyR agonists (caffeine, 4-chloro-m-cresol) compared with wtRyR1.

35 imately 5-fold increase in the percentage of m-cresol formation relative to that of the natural isofo

36 e para-monooxygenase variant that formed 75% m-cresol from toluene and 100% m-nitrophenol from nitrob

37 was applied and validated for PCMC (4-chloro-m-cresol), household derived antimicrobial agent with no

38 roximately 0.0001), suggesting that 4-chloro-m-cresol-induced adenosine could readily distinguish bet

39 n hexamer containing two zinc ions, with two m-cresol molecules bound at each dimer-dimer interface s

40 on, near-quantitative deuterium retention in m-cresol obtained from 4-(2)H(1)-toluene, and partial lo

41 lymphocytes incubated with 0-10 mM 4-chloro-m-cresol or 0-10.7 mM halothane.

42 Similarly, at 1 mM 4-chloro-m-cresol or 0.96 mM halothane, adenosine levels were sig

43 ,5-benzenetricarbaldehyde building blocks in m-cresol or acetic acid, named RT-COF-1 or RT-COF-1Ac/RT

44 after RyR activation (caffeine and 4-chloro-m-cresol) or beta-adrenergic stimulation (isoproterenol)

45 eceptor (10 mM caffeine, 200 microM 4-chloro-m-cresol, or 10 mM KCl).

46 rescein at pH 6.5, phenol red at pH 7.5, and m-cresol purple at pH 8.5) which permitted separation of

47 tic analyses showed areas under the 4-chloro-m-cresol receiver-operating characteristic curves near m

48 Although 4-chloro-m-cresol receiver-operating characteristic curves reveal

49 ptible B cells treated with 0.75 mM 4-chloro-m-cresol relative to controls.

50 nodine receptor Ca channels agonist 4-chloro-m-cresol was compared in blood lymphocytes from malignan

51 -3-hexenol, acetic acid, benzyl alcohol, and m-cresol, while the addition of oxygen significantly inf

52 to probe the mechanism of the alkylation of m-cresol with isopropyl alcohol in scCO(2) using Nafion