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1 .8 g/dL; median, 7.5 g/dL), which was mostly macrocytic (10/11 [91%]) with concomitant thrombocytopen
2 erythroblasts, explain why these anemias are macrocytic, and show why children with GATA1 mutations h
3                    STM infection resulted in macrocytic anemia and enhanced splenomegaly with destruc
4  Tc1 mice do not develop leukemia, they have macrocytic anemia and increased extramedullary hematopoi
5 ications of vitamin B-12 deficiency, such as macrocytic anemia and neurological complications affecti
6  mechanism is underscored in 5q- syndrome, a macrocytic anemia caused by a large monoallelic deletion
7                    Sf3b1(K700E) mice develop macrocytic anemia due to a terminal erythroid maturation
8 mitochondrial respiration and development of macrocytic anemia in mtDNA-mutator mice.
9              A 47-year-old woman with severe macrocytic anemia markedly improved during the second an
10                                              Macrocytic anemia occurring in patients with fatigue sug
11        Classical 5q- syndrome is an acquired macrocytic anemia of the elderly.
12 s, was causally associated with a pernicious/macrocytic anemia pattern, i.e. decreased erythrocyte co
13  of the mtDNA polymerase, PolgA, and develop macrocytic anemia similar to that of patients with MDS.
14            The disease is characterized by a macrocytic anemia that remains poorly understood.
15                   In this group, the risk of macrocytic anemia was 63% lower than in the placebo grou
16 hat mutant animals develop an age-dependent, macrocytic anemia with abnormal erythroid maturation and
17                                              Macrocytic anemia with abnormal erythropoiesis is a comm
18  defect observed in both individuals and the macrocytic anemia with megaloblastic features of the mor
19 pc (del/+)) accelerated the development of a macrocytic anemia with monocytosis, early features of t-
20  that is deleted neonatally develop a severe macrocytic anemia with proerythroblast maturation arrest
21 se (the more severe case also presented with macrocytic anemia).
22 duced serum vitamin B12 concentrations, mild macrocytic anemia, and fecal loss of Na+ and K+, the lat
23 ion in deep brain nuclei, mild normocytic to macrocytic anemia, and gastrointestinal symptoms.
24 ad to many clinical abnormalities, including macrocytic anemia, cardiovascular diseases, birth defect
25  is characterized by red blood cell aplasia, macrocytic anemia, clinical heterogeneity, and increased
26 cartilage, and pulmonary lesions, as well as macrocytic anemia, during the treatment process for feve
27 inal features of the 5q- syndrome, including macrocytic anemia, erythroid hypoplasia, and megakaryocy
28 , encoding the ribosomal protein S14) caused macrocytic anemia, prominent erythroid dysplasia and mon
29 in SFXN4 as a cause of mitochondriopathy and macrocytic anemia.
30  of the MDS progenitors, which corrected the macrocytic anemia.
31 neutrophils and monocytes, associated with a macrocytic anemia.
32 ia, a disorder characterized by hypoplastic, macrocytic anemia.
33 are responsible for Pearsons syndrome with a macrocytic anemia.
34 characterized by vitamin B-12 deficiency and macrocytic anemia.
35 iculocytes, hence recapitulating features of macrocytic anemia.
36 ythroid differentiation because they develop macrocytic anemia.
37 ly with multiple female individuals who have macrocytic anemia.
38 cking the heme exporter FLVCR1 have a severe macrocytic anemia; however, the mechanisms that underlie
39 unction could contribute to the emergence of macrocytic anemias, for instance, due to nutritional def
40 inant effects; mild pigmentation defects and macrocytic, hypoplastic anemia occur in heterozygous mic
41 -deficient (PCFT(-/-)) mice developed severe macrocytic normochromic anemia and pancytopenia.
42 ctivation or inhibition of mTORC1 results in macrocytic or microcytic anemia, respectively.
43      Diamond Blackfan Anemia (DBA) is a rare macrocytic red blood cell aplasia that usually presents
44 ice develop massive splenomegaly, basophilic macrocytic red blood cells, and anemia as they age.
45 ions of myopathic exercise intolerance and a macrocytic sideroblastic anemia.