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1 CML hematopoietic stem cells and granulocyte-macrophage progenitors.
2 differentiation of murine 32Dcl3 granulocyte/macrophage progenitors.
3 her megakaryocyte/erythrocyte or granulocyte/macrophage progenitors.
4 aneous appearance of primitive erythroid and macrophage progenitors.
5  CSF, demonstrating that MIP-3 beta attracts macrophage progenitors.
6 teristics of colony-forming unit-granulocyte-macrophage progenitors.
7 austion and differentiation into granulocyte-macrophage progenitors.
8 ctly on the bone marrow to increase monocyte-macrophage progenitors.
9 rimed multipotent progenitors or granulocyte/macrophage progenitors.
10 itors but significantly lower in granulocyte-macrophage progenitors.
11 sistent with the immunophenotypic profile of macrophage progenitors.
12 genitors but relative sparing of granulocyte-macrophage progenitors.
13 renewing hematopoietic stem cells (HSCs) and macrophage progenitors.
14 oid progenitors and depletion of granulocyte-macrophage progenitors.
15 e very similar to that of normal granulocyte macrophage progenitors.
16 m common myeloid progenitors and granulocyte/macrophage progenitors.
17 d, common myeloid progenitors or granulocyte/macrophage progenitors.
18 264.7 cells and bone marrow-derived monocyte/macrophage progenitors.
19 s a severe reduction in myeloid (granulocyte/macrophage) progenitors.
20          The discovery of the fetal alveolar macrophage progenitor advances our understanding of huma
21 ese findings show that beyond its content of macrophage progenitors, adventitial Sca-1(+)CD45(+) cell
22  production prevented cancer-induced HSC and macrophage progenitor amplification and thus restrained
23 iption reduces both the leukemic granulocyte-macrophage progenitor and leukemia-initiating cell (LIC)
24 ith a hematopoietic shift toward granulocyte macrophage progenitor and myeloid cells.
25  progenitors committed to become granulocyte-macrophage progenitors and as megakaryocyte-erythroid pr
26 zed system of rapid migration of granulocyte-macrophage progenitors and committed macrophage-dendriti
27 e common myeloid progenitors and granulocyte-macrophage progenitors and down-regulation of proerythro
28 TPIP2 deficiency causes both an expansion of macrophage progenitors and increased responsiveness of m
29 ed with their normal counterpart granulocyte-macrophage progenitors and myeloblast precursors.
30  tumor-bearing mice induces the expansion of macrophage progenitors and the supply of macrophages.
31                      Splenic granulocyte and macrophage progenitors and their descendants were likewi
32 o differentiate into bipotential granulocyte/macrophage progenitors and their progeny.
33 rting increased proliferation by granulocyte/macrophage progenitors and, surprisingly, multipotent pr
34 g common myeloid progenitors and granulocyte-macrophage progenitors, and 4-1BB was inducible on activ
35 lls, common myeloid progenitors, granulocyte-macrophage progenitors, and megakaryocyte-erythroid prog
36 oattracts T cells, B cells, dendritic cells, macrophage progenitors, and NK cells and facilitates den
37 ctivation of beta-catenin in CML granulocyte-macrophage progenitors appears to enhance the self-renew
38 ge lineage outputs from a common granulocyte-macrophage progenitor are still not completely understoo
39                  Runx1-deficient granulocyte-macrophage progenitors are characterized by increased cl
40  are markedly increased, whereas granulocyte-macrophage progenitors are reduced.
41 on of unmethylated C/EBPalpha in granulocyte/macrophage progenitors by inhibiting Carm1 biases the ce
42 tion with cytomegalovirus, human granulocyte-macrophage progenitors carry the viral genome but fail t
43 -regulation in MDS is related to granulocyte-macrophage progenitor cell sensitivity to TRAIL-induced
44  in bone marrow-derived bipotent granulocyte macrophage progenitor cells (GM-colony forming cell [CFC
45 n myeloid progenitors (CMPs) and granulocyte-macrophage progenitor cells (GMPs) differentiate into PA
46 gs provide a framework for understanding how macrophage progenitor cells acquire tissue-specific phen
47 a vascular adventitial population containing macrophage progenitor cells and investigated their origi
48     ATI-2341-mediated release of granulocyte/macrophage progenitor cells from the bone marrow was con
49  dramatic increase in numbers of granulocyte-macrophage progenitor cells in the marrow and spleen.
50 cterization of resident vascular adventitial macrophage progenitor cells provides new insight into ad
51 es revealed that Sca-1(+)CD45(+) adventitial macrophage progenitor cells were not replenished via the
52                           Rather adventitial macrophage progenitor cells were upregulated in hyperlip
53 by C/EBPalpha-p42, and in normal granulocyte/macrophage progenitor cells, we detect C/EBPalpha bound
54 iferation of colony-forming unit-granulocyte-macrophage progenitor cells.
55 oid nodes, bone marrow cells and granulocyte-macrophage progenitor cells.
56 of common myeloid progenitor and granulocyte macrophage progenitor cells.
57  immunomodulatory cells (dendritic cells and macrophages), progenitor cells, vasa vasorum endothelial
58                                  Granulocyte-macrophage progenitors (CFU-GM) were present only during
59                    Unlike normal granulocyte-macrophage progenitors, CML granulocyte-macrophage proge
60 atopoietic stem cells (HSCs) and granulocyte-macrophage progenitors compared with wild-type controls.
61                                  Granulocyte/macrophage progenitors displayed a relatively normal pro
62  gadd45b-/- colony forming units granulocyte/macrophage progenitors displayed prolonged proliferation
63                                              Macrophage progenitors expressing the Shp-2 mutants disp
64 cyte-macrophage progenitors, CML granulocyte-macrophage progenitors formed self-renewing, replatable
65 deficient mice display increased bone marrow macrophage progenitor frequency and decreased tissue mac
66 ary leukemic colony-forming unit granulocyte/macrophage progenitors from patients with CML.
67 nt megakaryocyte-erythrocyte and granulocyte-macrophage progenitors give rise to unipotent progenitor
68     Here we use imaging to track granulocyte/macrophage progenitor (GMP) behaviour in mice during eme
69 -erythroid progenitor (MEP), and granulocyte-macrophage progenitor (GMP) cells, accompanied by increa
70 , common myeloid progenitor, and granulocyte/macrophage progenitor (GMP) cells.
71  primitive (CD34+CD38-) and late granulocyte-macrophage progenitor (GMP) cells.
72 istinct trajectories through the granulocyte-macrophage progenitor (GMP) compartment showing that AXL
73 f the lin-/Sca-1/c-kit (LSK) and granulocyte macrophage progenitor (GMP) compartments at the expense
74 mon myeloid progenitor (CMP) and granulocyte/macrophage progenitor (GMP) populations, and decreased t
75 ased numbers of DCs, even in the granulocyte-macrophage progenitor (GMP), which does not normally pos
76 nitors (EoPs), downstream of the granulocyte/macrophage progenitor (GMP).
77 ion protein selectively expanded granulocyte/macrophage progenitors (GMP) and enhanced their self-ren
78 factor critical for formation of granulocyte-macrophage progenitors (GMP) and leukemic GMP.
79 f splenic cells that derive from granulocyte/macrophage progenitors (GMP) compared with wild-type mic
80 xpands the numbers of LT-HSC and granulocyte/macrophage progenitors (GMP) resulting in chronic MPD.
81 ogenous leukemia (CML), abnormal granulocyte macrophage progenitors (GMP) with nuclear beta-catenin a
82 lls (HSC) or more differentiated granulocyte-macrophage progenitors (GMP).
83 ctivation of beta-catenin within granulocyte-macrophage progenitors (GMP).
84 ansion of phenotypically defined granulocyte macrophage progenitors (GMPs) and acquisition of self-re
85 i-potential progenitors (LMPPs), granulocyte-macrophage progenitors (GMPs) and multi-lymphoid progeni
86 n myeloid progenitors (CMPs) and granulocyte-macrophage progenitors (GMPs) but retained megakaryocyte
87 LPs]) and CMPs and their progeny granulocyte-macrophage progenitors (GMPs) can give rise to functiona
88 l cycle rate heterogeneity among granulocyte-macrophage progenitors (GMPs) determines their probabili
89 ique spatiotemporal mechanism of granulocyte-macrophage progenitors (GMPs) employed in emergency hema
90  We identify a recurrent loss of granulocyte-macrophage progenitors (GMPs) in the bone marrow of low
91  that myeloid differentiation to granulocyte macrophage progenitors (GMPs) is critical for LSC genera
92 ag1(-/-) mice, lineage-committed granulocyte-macrophage progenitors (GMPs) or bone marrow-derived mac
93 n myeloid progenitors (CMPs) and granulocyte-macrophage progenitors (GMPs) preferentially differentia
94 m wild-type and Dicer1-deficient granulocyte-macrophage progenitors (GMPs) revealed that 20 miRNA fam
95  have identified a population of granulocyte-macrophage progenitors (GMPs) that were highly enriched
96 xpression in MLL-AF9-transformed granulocyte macrophage progenitors (GMPs) that were initially Evi1(n
97  generation of granulocytes from granulocyte-macrophage progenitors (GMPs) were markedly reduced in S
98 mmon myeloid progenitors (CMPs), granulocyte/macrophage progenitors (GMPs), and thymocyte progenitors
99 e differentiation in bipotential granulocyte-macrophage progenitors (GMPs), its role in regulating ce
100 mmon myeloid progenitors (CMPs), granulocyte-macrophage progenitors (GMPs), megakaryocyte-erythrocyte
101 mon lymphoid progenitors (CLPs), granulocyte-macrophage progenitors (GMPs), megakaryocyte-erythrocyte
102 mon lymphoid progenitors (CLPs), granulocyte-macrophage progenitors (GMPs), or early thymic progenito
103 rogenitor differentiation toward granulocyte-macrophage progenitors (GMPs), resulting in a myeloproli
104 evated beta-catenin signaling in granulocyte macrophage progenitors (GMPs), which enables this popula
105 genitors partially overlaps with granulocyte-macrophage progenitors (GMPs).
106 HSCs/multipotent progenitors and granulocyte/macrophage progenitors have self-renewal capability, res
107 d LPS, and may play a role in trafficking of macrophage progenitors in and out of the bone marrow in
108                       Osteoclasts arise from macrophage progenitors in bone marrow (BMMs) as a conseq
109 ed numbers of peripheral-blood monocytes and macrophage progenitors in bone marrow.
110 phage depletion is associated with increased macrophage progenitors in bone marrow.
111 numbers of colonies derived from granulocyte-macrophage progenitors in cultures supplemented with low
112                       We identified alveolar macrophage progenitors in human fetal liver that express
113 sideration of the broader existence of local macrophage progenitors in other tissues.
114 osure to erythemal UV radiation can modulate macrophage progenitors in the BM such that their differe
115 bited some degree of toxicity to granulocyte/macrophage progenitors in the bone marrow of mice.
116 f myeloid-biased multipotent and granulocyte-macrophage progenitors in the bone marrow, resulting in
117  common myeloid progenitors, and granulocyte/macrophage progenitors in the spleen.
118 FU-E), mast cell and bipotential granulocyte/macrophage progenitors in the yolk sac.
119                    C/EBPalpha(-/-) FL lacked macrophage progenitors in vitro and had impaired ability
120 rogenitor lineage skewing toward granulocyte-macrophage progenitors, increased colony-forming unit gr
121 odel by transplanting hPSC-derived primitive macrophage progenitors into neonatal mouse brains.
122 logy reported in TTP (-/-) animals, and that macrophage progenitors may be involved in the transplant
123                                              Macrophage progenitors migrate to tissues perinatally, w
124          Steady-state numbers of granulocyte-macrophage progenitors, myeloid-lineage cells and mature
125                                   Clonogenic macrophage progenitors of fetal origin were present in t
126                              The granulocyte-macrophage progenitor pool from patients with CML in bla
127  the same myeloid-restricted pre-granulocyte-macrophage progenitor (pre-GM) (Lin(-)Sca-1(-)c-Kit(+)CD
128 HSC, multipotent progenitor, and granulocyte/macrophage progenitor proliferation and reactive neutrop
129 rophage colony-forming unit (CFU-GM) growth, macrophage progenitor proliferation, and activation of t
130                During latency in granulocyte-macrophage progenitors, RC2710 DNA was detected at level
131 begins to be up-regulated at the granulocyte-macrophage progenitor stage and continues throughout mye
132 leukemia evolving from committed granulocyte-macrophage progenitors that have acquired the self-renew
133 tal latent infection of cultured granulocyte-macrophage progenitors, the viral genome was detected in
134 eukaemias initiated in committed granulocyte macrophage progenitors through introduction of the MLL-A
135                                Consistently, macrophage progenitors transduced with the Shp-2 mutants
136 nonuclear cells (BMNCs) are a rich source of macrophage progenitors used for treating chronic inflamm
137  niche postirradiation for human granulocyte-macrophage progenitors via reduced murine CD47 and physi
138  but differentiation from CMP to granulocyte/macrophage progenitor was decreased, and the mature gran
139 nd progenitor cells, notably granulocyte and macrophage progenitors, which produced CD11b(+) Ly-6C(hi
140 ique in that it is initiated by pericyte and macrophage progenitors, with endothelial cell recruitmen

 
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