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1                             CD11b+ monocytic/macrophagic cells (Mo/Mph), which infiltrate into skin a
2 int, small numbers of infiltrating monocytic/macrophagic cells are already apparent.
3 n of class II MHC+CD11b+Gr-1+ and -monocytic/macrophagic cells in the dermis and epidermis.
4                             CD11b+ monocytic/macrophagic cells that infiltrate human skin after in vi
5  while the activities in P388D1 and RAW264.7 macrophagic cells were considerably higher than the basa
6                    Taken together, monocytic/macrophagic cells with high IL-10 and low IL-12 expressi
7 ocytes or class II MHC+CD11b+Gr-1+ monocytic/macrophagic cells.
8  the molecular determinants of monocytic and macrophagic commitment by profiling the EGR1 transcripti
9 ng the bacteria to gain direct access to the macrophagic cytoplasm.
10 vigate the 48-h "viability gate" and undergo macrophagic differentiation has remained elusive.
11 w levels in progenitor cells of granulocytic-macrophagic, erythroid, or megakaryocytic lineage detect
12 oach, we showed that muscles did not require macrophagic hif1a or hif2a to regenerate.
13                  These results indicate that macrophagic HIFs do not play a crucial role during skele
14 an IRF1 is essential for IFN-gamma-dependent macrophagic immunity to mycobacteria, but largely redund
15   Inborn errors of human IFN-gamma-dependent macrophagic immunity underlie mycobacterial diseases, wh
16 ron viral diseases, type II interferon intra-macrophagic infections, interleukin-6 pyogenic bacterial
17 surface receptors of IL-18-responsive NK and macrophagic KG-1 cells.
18 1, the causative gene of DHS, at hepatic and macrophagic levels.
19 or tuberculous mycobacteria, and other intra-macrophagic microbes.
20 ain metabolic abnormalities in patients with macrophagic myofascitis (MMF) and the relationship with
21 tently downregulated in the CD11b+ monocytic/macrophagic population (p < 0.0002).
22 increased to 70%, the infiltrating monocytic/macrophagic population had risen to 1.2% of the total de
23 lative to 0 h by the CD11b+ dermal monocytic/macrophagic population peaked at 24-48 h and tapered the
24 dermal CD11b+ (CD1-, 3-, 24-, 56-) monocytic/macrophagic population.
25 studies, HIV production was increased in the macrophagic U1 cell line expressing the HIV genome after