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1 pigment epithelium), and H35.50 (unspecified macular degeneration).
2 used on diabetic retinopathy and age-related macular degeneration.
3 etinitis pigmentosa, but also in age-related macular degeneration.
4 ise likely causal biomarkers for age-related macular degeneration.
5 of vision loss in patients with age-related macular degeneration.
6 get for diabetic retinopathy and age-related macular degeneration.
7 on for patients with neovascular age-related macular degeneration.
8 m many retinal diseases, such as age-related macular degeneration.
9 lions of patients suffering from age-related macular degeneration.
10 taract surgery, corneal transplantation, and macular degeneration.
11 ible blindness in the setting of age-related macular degeneration.
12 liferative vitreoretinopathy and age-related macular degeneration.
13 nd choroidal vascular disease in age-related macular degeneration.
14 isual outcomes for patients with age-related macular degeneration.
15 n eye diseases like glaucoma and age-related macular degeneration.
16 macular degeneration (AMD) and 2 with myopic macular degeneration.
17 al ocular diseases including keratoconus and macular degeneration.
18 ambiguity about cone survival in age-related macular degeneration.
19 her macular disease including AMD and myopic macular degeneration.
20 mes and drives the pathogenesis of Stargardt macular degeneration.
21 tients with advanced neovascular age-related macular degeneration.
22 recommended for patients who are at risk for macular degeneration.
23 te unexpected cell types in diseases such as macular degeneration.
24 tics and diagnostics directed at age-related macular degeneration.
25 s less than that for other diseases, such as macular degeneration.
26 atrophy, and 2 eyes (2.8%) developed cystoid macular degeneration.
27 atients affected by dry atrophic age-related macular degeneration.
28 nd monitoring human eye disorders, including macular degeneration.
29 membrane probably also has implications for macular degeneration.
30 get in patients with neovascular age-related macular degeneration.
31 y (GA) secondary to nonexudative age-related macular degeneration.
32 ells to treat conditions such as age-related macular degeneration.
33 lytic uremic syndrome (aHUS) and age-related macular degeneration.
34 treatment schemes in neovascular age-related macular degeneration.
35 es like retinitis pigmentosa and age-related macular degeneration.
38 commonly marketed ocular conditions included macular degeneration (35), optic neuritis (18), retiniti
39 abnormalities (26.6% versus 7.3%), exudative macular degeneration (5.2% versus 0.1%), and geographic
40 bservational study and found that history of macular degeneration (a proxy for complement-activation
42 ing hypothesis of the Alabama Study on Early Macular Degeneration (ALSTAR2) is that early AMD is a di
43 bnormal deposits associated with age-related macular degeneration, Alzheimer's disease, and many othe
48 s were diagnosed with coincident age-related macular degeneration (AMD) and 2 with myopic macular deg
49 to 90 years with GA secondary to age-related macular degeneration (AMD) and best-corrected visual acu
50 ssociated with increased risk of age-related macular degeneration (AMD) and disease progression, but
52 and anxiety among subjects with age-related macular degeneration (AMD) and its association with AMD
53 sociated with the development of age-related macular degeneration (AMD) and other complementopathies.
54 susceptible genetic variants to age-related macular degeneration (AMD) and response to as-needed int
55 ges of patients with neovascular age-related macular degeneration (AMD) and to demonstrate its use to
56 non-neovascular and neovascular age-related macular degeneration (AMD) and to provide recommendation
57 icity on the association between age-related macular degeneration (AMD) and vision-specific functioni
58 t prediction models for advanced age-related macular degeneration (AMD) are based on a restrictive se
59 drusen and without nGA nor late age-related macular degeneration (AMD) at baseline were included.
60 he progression of nonneovascular age-related macular degeneration (AMD) attributed to anteriorly migr
61 d in eyes with large drusen from age-related macular degeneration (AMD) before and after the drusen s
62 tify metabolites associated with age-related macular degeneration (AMD) by performing the largest met
63 ecline in the risk of developing age-related macular degeneration (AMD) continued for people born dur
64 nning may be secondary to active age-related macular degeneration (AMD) disease progression in both e
65 SIs to assess visual function in age-related macular degeneration (AMD) found CSI guided analysis det
66 o slow or prevent progression of age-related macular degeneration (AMD) from its early stages to visi
69 iction models for progression of age-related macular degeneration (AMD) have been developed, but the
70 y population, worldwide cases of age-related macular degeneration (AMD) have seen a dramatic increase
71 ve eyes diagnosed with exudative age-related macular degeneration (AMD) in comparison with eyes with
77 (VEGF) treatment of neovascular age-related macular degeneration (AMD) is a highly effective advance
84 rongest susceptibility genes for age-related macular degeneration (AMD) is complement factor H (CFH);
95 thological neovascularization in age-related macular degeneration (AMD) or diabetic macular edema (DM
96 assess whether older adults with age-related macular degeneration (AMD) or glaucoma performed worse o
97 n but are strongly implicated in age-related macular degeneration (AMD) pathogenesis, a second single
99 LLQ) in patients with a range of age-related macular degeneration (AMD) severity are associated with
101 t often in eyes with neovascular age-related macular degeneration (AMD) that had type 1 macular neova
102 ophy (GA) is an advanced form of age-related macular degeneration (AMD) that leads to progressive and
103 bolomic profile of patients with age-related macular degeneration (AMD) using mass spectrometry (MS).
105 ages from eyes with nonexudative age-related macular degeneration (AMD) were graded for the presence
106 standard care for patients with age-related macular degeneration (AMD) who are being considered for
107 y SD OCT study participants with age-related macular degeneration (AMD) with bilateral large drusen o
109 iking phenotypic similarities to age-related macular degeneration (AMD), a common and genetically com
110 t common genetic contributors to age-related macular degeneration (AMD), a leading cause of irreversi
112 re implicated in the etiology of age-related macular degeneration (AMD), a major cause of blindness i
113 tor contributing to the risk for age-related macular degeneration (AMD), a retinal disease involving
114 as retinitis pigmentosa (RP) and age-related macular degeneration (AMD), are among the leading causes
115 idal neovascularization (CNV) in age-related macular degeneration (AMD), but central questions regard
116 th literacy of 225 patients with age-related macular degeneration (AMD), diabetic macular edema (DME)
117 ng ocular comorbidity other than age-related macular degeneration (AMD), diabetic retinopathy, glauco
118 PPS and to drusen, nonexudative age-related macular degeneration (AMD), exudative AMD, hereditary ma
119 rse range of diseases, including age-related macular degeneration (AMD), glaucoma and refractive erro
120 ic atrophy (GA), a late stage of age-related macular degeneration (AMD), is a major cause of blindnes
121 vent irreversible vision loss in age-related macular degeneration (AMD), it is critical to detect ret
122 genetic variants associated with age-related macular degeneration (AMD), one of the leading causes of
123 t in patients suffering from wet age-related macular degeneration (AMD), particularly when refractory
124 genous retinal detachment (RRD), age-related macular degeneration (AMD), proliferative diabetic retin
126 In assessing the severity of age-related macular degeneration (AMD), the Age-Related Eye Disease
127 E) underlies the pathogenesis of age-related macular degeneration (AMD), the leading cause of irrever
128 n implicated in the pathology of age-related macular degeneration (AMD), the most common cause of adu
129 ic loci that are associated with age-related macular degeneration (AMD), the most common cause of inc
130 ntributes to the pathogenesis of age-related macular degeneration (AMD), the role of retinal perfusio
132 uited patients with intermediate age-related macular degeneration (AMD), without other vitreoretinal
169 years or older with neovascular age-related macular degeneration and a baseline best-corrected visua
170 n C, a recessive risk factor for age-related macular degeneration and Alzheimer's disease, in both in
171 during treatment for neovascular age-related macular degeneration and are associated with poor visual
172 l name, CFH) are associated with age-related macular degeneration and atypical hemolytic uremic syndr
177 es for retinal diseases, such as age-related macular degeneration and inherited retinal dystrophies,
180 atform for understanding the pathogenesis of macular degeneration and other related degenerative diso
181 nant I62-CFH (protective against age-related macular degeneration) and V62-CFH functioned equivalentl
182 dema, 32 (25.8%) had neovascular age-related macular degeneration, and 32 (25.8%) had other causes of
183 oid-associated diseases, including dementia, macular degeneration, and diabetes mellitus, in epidemio
184 ic features, including glaucoma, age-related macular degeneration, and epiretinal membrane, require s
186 including diabetic retinopathy, age-related macular degeneration, and past cataract surgery, were re
187 body donor eyes with neovascular age-related macular degeneration are limited by the time span from d
188 inal pigment epithelium (RPE) of age-related macular degeneration (ARMD) patients and therefore could
189 d with neovascularization in wet age-related macular degeneration (ARMD), choriocapillaris degenerati
192 als to diverse disorders such as age-related macular degeneration, atypical hemolytic uremic syndrome
194 ive loss of vision in young males, a form of macular degeneration called X-linked retinoschisis (XLRS
195 f ophthalmic diseases, including age-related macular degeneration, cataracts, diabetic retinopathy, g
196 al diseases such as retinitis pigmentosa and macular degeneration cause irreversible vision loss in m
197 uch as diabetic retinopathies, glaucoma, and macular degeneration, cause the death of retinal neurons
198 ic atrophy is a blinding form of age-related macular degeneration characterized by retinal pigmented
200 rd of diabetic macular edema and age-related macular degeneration clinical trials registered on Clini
201 tformin were less likely to have age-related macular degeneration compared with those not taking the
203 iabetic retinopathy, neovascular age-related macular degeneration, diabetic macular edema, and retina
204 dry eye syndrome) and posterior (age-related macular degeneration, diabetic retinopathy and glaucoma)
205 bute to vascular inflammation in age-related macular degeneration, diabetic retinopathy, and glaucoma
206 sting for age, gender, glaucoma, age-related macular degeneration, diabetic retinopathy, cataract, gl
208 Progression to exudative 'wet' age-related macular degeneration (exAMD) is a major cause of visual
211 ould be of clinical and research interest in macular degeneration, for example in estimating visual p
212 tinal dystrophies and late-stage age-related macular degeneration, for which treatments remain limite
215 ) published by the International Age-related Macular Degeneration Genomics consortium of 33,526 (16,1
216 d for information on the topics of cataract, macular degeneration, glaucoma, diabetic retinopathy, an
217 without concomitant ocular pathology such as macular degeneration, glaucoma, Sicca syndrome, epiretin
218 telangiectasia type 2 (MacTel), a late-onset macular degeneration, has been linked to a loss in the r
219 roidal neovascularization due to age-related macular degeneration in the study eye were randomized an
220 tinoschisis (XLRS) is one of the most common macular degenerations in young males, with a worldwide p
223 tages of geographic atrophy (GA) age-related macular degeneration is characterised by the demise of p
224 raphic atrophy (GA) secondary to age-related macular degeneration is considered a single entity.
225 (GA), a progressive dry form of age-related macular degeneration is elusive and there is currently n
230 uro-ophthalmology.(1-5) Humans with juvenile macular degeneration (JMD) show significant blood-oxygen
231 Intervention in Early Stages of Age-Related Macular Degeneration (LEAD) study is a 36-month, multice
232 erited retinal degenerations and age-related macular degeneration.Literature discussed here focuses o
234 presence of coarse stereopsis in humans with macular degeneration (MD), which affects the central ret
236 mes in patients with neovascular age-related macular degeneration (nAMD) during anti-vascular endothe
237 ens in patients with neovascular age-related macular degeneration (nAMD) from the TReat and extEND (T
238 he natural course of neovascular age-related macular degeneration (nAMD) is essential in discussing p
240 ata of patients with neovascular age-related macular degeneration (nAMD) treated with intravitreal af
241 ion in patients with neovascular age-related macular degeneration (nAMD) treated with intravitreal in
242 hy (GA) in eyes with neovascular age-related macular degeneration (nAMD) treated with ranibizumab.
244 mes in patients with neovascular age-related macular degeneration (nAMD) who received anti-vascular e
245 in the management of neovascular age-related macular degeneration (nAMD), diabetic retinal disease (D
258 tion for Atrophy Associated with Age-Related Macular Degeneration on OCT, it is unclear whether decre
259 pigment epithelium in eyes with age-related macular degeneration or central serous chorioretinopathy
260 f age or older with a diagnosis of exudative macular degeneration or diabetic macular edema requiring
262 eases such as Stargardt disease, age-related macular degeneration or retinitis pigmentosa urgently re
263 antly with sets found by GWAS of age-related macular degeneration (P=1.4 x 10(-12)), ulcerative colit
264 several human diseases including age-related macular degeneration, paroxysmal nocturnal hemoglobinuri
266 emplary search for patients with age-related macular degeneration, performed cataract surgery, and at
267 rcept injections for neovascular age-related macular degeneration presented 4 weeks after his most re
268 ,188 Medicare beneficiaries with neovascular macular degeneration receiving >=1 anti-vascular endothe
270 near-sightedness, diabetic retinopathy, and macular degeneration, respectively, with all pairwise co
271 ss of photoreceptors in atrophic age-related macular degeneration results in severe visual impairment
272 from the early drusen stage to the advanced macular degeneration stage that leads to blindness, rema
274 ssible lack of success in clinical trials in macular degeneration that have targeted inflammatory age
276 r age, gender, and a documented diagnosis of macular degeneration, the use of a BLF IOL was not predi
277 urity, diabetic retinopathy, and age-related macular degeneration, threaten the visual health of chil
278 rusen, retinal pigmentary abnormalities, and macular degeneration to age and sex were studied in 4926
283 chedule, and CATT (Comparison of Age-Related Macular Degeneration Treatments) study and VIEW (VEGF Tr
284 ncluding an epiretinal membrane, age-related macular degeneration, vitreomacular traction, and cystoi
285 r inhibitors (anti-VEGF) for wet age-related macular degeneration (wAMD), and to acquire a snapshot o
286 national registry of neovascular age-related macular degeneration was analyzed with an intention-to-t
289 lume scans of 1094 patients with age-related macular degeneration, we generated a vocabulary of 20 lo
291 hy of prematurity (ROP), and wet age-related macular degeneration (wet AMD) have been found to have e
292 r glaucoma, Brolucizumab for wet age-related macular degeneration (wet AMD), Luxturna for retinitis p
293 Similarities to OCT features in age-related macular degeneration, where mitochondrial dysfunction ha
295 ophy outcome(s) in patients with age-related macular degeneration who received anti-VEGF treatment we
296 g 1185 patients with neovascular age-related macular degeneration who were enrolled in the clinical t
299 PCV) is a variant of neovascular age-related macular degeneration with distinct phenotypes, treatment
300 ents may proceed to an early-onset end-stage macular degeneration with frank atrophy of the RPE or ne