ライフサイエンスコーパス: macule

1 typically appearing as a solitary pigmented macule.

2 ired in the melanocytes of the hypomelanotic macule.

3 zed by hyperpigmented and hypopigmented skin macules.

4 aracteristics of human NF1: [1] cafe-au-lait macules, [2] axillary/inguinal freckling, [3] shortened

5 On pathologic examination, 96% of miners had macules, 70% micronodules, 45% macronodules, 15% silicos

6 patients with 63 ambiguous pigmented facial macules and 12 control photodamaged facial areas were in

7 microscopy (RCM), ambiguous pigmented facial macules and establish a correlation between RCM, histopa

8 erall, q-type opacities were associated with macules and micronodules, whereas the large r-type opaci

9 with an 18-month history of scattered, brown macules and nodules up to 2 cm in size on his trunk and

10 20s presented with asymptomatic red to brown macules and papules.

11 ic autoimmune disease characterized by white macules and patches of the skin, having a negative impac

12 brows and cilia, along with depigmented skin macules and patches.

13 dermal melanocytes, resulting in depigmented macules and patches.

14 The patient presented with dark brown macules and sporadic erythema topped by bullous lesions.

15 cused on a single skin feature- cafe-au-lait macules-and partitioned the disease space into hierarchi

16 "Cafe-au-lait" macules (CALMs) and overall skin hyperpigmentation are e

17 Response to laser treatment for cafe au lait macules (CALMs) is inconsistent and difficult to predict

18 ividuals presenting mainly with cafe au lait macules (CALMs), axillary freckling, and macrocephaly.

19 ly characterised by the development of white macules due to the loss of functioning melanocytes in th

20 en presented with eruptions of hypopigmented macules following coryzal symptoms.

21 mary melanocytes isolated from hypomelanotic macules from 6 patients with TSC all exhibited reduced T

22 development of white, genetically revertant macules in red, diseased skin.

23 Benign melanotic macules (MAC) are the most frequent cause of lip pigment

24 sions of coal workers' pneumoconiosis (CWP): macules, micro- and macronodules (small and large fibrot

25 tegory 0/0 was often reported for cases with macules of mild to moderate grade and mild levels of mic

26 Pigmented macules of the glans penis, delayed motor development an

27 the gastrointestinal tract and by pigmented macules of the lips, buccal mucosa, and digits.

28 A skin biopsy of the macule on the abdomen revealed accumulation of mast cell

29 A skin biopsy of a brown macule on the left thigh revealed a dense accumulation o

30 gressively sclerotic and presented pigmented macules on a background of hypopigmentation and teleangi

31 ure glaucoma following the appearance of new macules on her upper extremities.

32 Pigmented facial macules on photodamaged skin are a clinical, dermoscopic

33 spective study of ambiguous pigmented facial macules on photodamaged skin was conducted in a tertiary

34 Six patients had pigmented macules on sun-exposed skin, but none developed a skin n

35 ents presented with hypo- and hyperpigmented macules over the body, resembling dyschromatosis univers

36 presentations were characterized by purpuric macules, papules, and confluent plaques predominantly on

37 ions progress through 4 well-defined stages (macules, papules, vesicles, and pustules) over 2 to 4 we

38 Body lice bites cause pruritic excoriated macules/papules and hyperpigmentation in areas where clo

39 Although the pruritus resolved, the brown macules remained for one year after the initial treatmen

40 r characterized by circumscribed depigmented macules resulting from the loss of cutaneous melanocytes

41 l lesions such as ulcerations, enanthema and macules-the involvement of the oral cavity in coronaviru

42 These macules were accompanied by pruritus and were positive f

43 Patients with TSC develop hypomelanotic macules (white spots), but the molecular mechanisms unde

44 y enhanced the diagnosis of pigmented facial macules with 91.7% sensitivity and 86.8% specificity.

45 Pink macules with a 3-cm diameter on the anterior left superi

46 immunodeficiency mice led to black-pigmented macules within 3 weeks of treatment.