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1              Sixty-two percent of cases were malnourished.
2 fourth of outpatients with Crohn disease are malnourished.
3 therapeutic feeding centres for the severely malnourished.
4 eve an adequate coverage of all the severely malnourished.
5 or tuberculosis episodes, and 238 (59%) were malnourished.
6                    Over 75% of patients were malnourished.
7 s disproportionately causing death among the malnourished.
8  1 in 5 patients undergoing HNC surgery were malnourished.
9 tic criteria used, 3%-17% were classified as malnourished.
10 lnourished (or suspected to be), or severely malnourished.
11 skin parasite burdens were equivalent in the malnourished (3% protein) and control mice, but in the m
12        One death occurred in a significantly malnourished 62-year-old female.
13 unodeficiency virus (HIV), and 87 (30%) were malnourished; 90 (31%) had confirmed pulmonary tuberculo
14 outcome, defined as being alive, not acutely malnourished according to the inclusion criteria (MUAC >
15                                              Malnourished adolescents and young adults with anorexia
16 malized emotional and cognitive behaviors in malnourished adult mice.
17 e HIV treatment, with particular urgency for malnourished adults initiating ART.
18 data obtained suggest strongly that severely malnourished adults, particularly those with edema, reco
19 n the nutritional rehabilitation of severely malnourished adults.
20                                              Malnourished African children had a normal FEV1/FVC rati
21              Compared to nourished patients, malnourished AMI patients have a more unfavorable progno
22  Almost one-third (29%) of the subjects were malnourished and almost two-thirds (63%) were at risk of
23 patients with low creatinine production were malnourished and had low serum creatinine levels and cre
24          Patients with analbuminemia are not malnourished and individuals with simple malnutrition ar
25  These findings suggest that, in the acutely malnourished and infected state, children with edematous
26 reased substantially among children who were malnourished and infected with human immunodeficiency vi
27 ospital admission (period 1), when they were malnourished and infected; approximately 8 d after admis
28              As all children were moderately malnourished and many had inflammation, this probably ex
29 en with edematous or nonedematous SCU in the malnourished and recovered states.
30 supplementary feeding centres for moderately malnourished and therapeutic feeding centres for the sev
31 al extracellular 5-HT did not differ between malnourished and well-nourished controls in either the m
32 are children who earlier were more seriously malnourished and who are survivors of a composite proces
33                                              Malnourished and young children are particularly suscept
34 mpste24(-/-) mice gain weight slowly, appear malnourished, and exhibit progressive hair loss.
35                      Children who are young, malnourished, and infected with HIV have significant ris
36 s of acute and chronic infections in normal, malnourished, and transgenic mice.
37 olved but they were still anthropometrically malnourished; and at recovery (study 3), when weight-for
38                     However, for the younger malnourished animals (PND15 and PND30), PSA(1) values we
39 ntrols, although the bacillary burden in the malnourished animals continued to rise.
40 actericidal nitrogen oxides was depressed in malnourished animals in the lungs specifically at early
41           Stimulation of the MRN in behaving malnourished animals may markedly affect the recurrent n
42  animals, with the change occurring later in malnourished animals when compared to control ones.
43 provide part of the bases for alterations in malnourished animals' response to stress.
44 dendritic and terminal 5-HT autoreceptors in malnourished animals, in order to understand possible me
45 it from REM sleep to SWS in both control and malnourished animals, with the change occurring later in
46 in vitro were markedly suppressed in protein-malnourished animals.
47 decreased in the medial prefrontal cortex of malnourished animals.
48 urther risk factors for death included being malnourished (aOR = 4.2; 95% CI 2.1-8.7); having oral th
49  (study 1), when they were both infected and malnourished; approximately 11 d after admission (study
50 on fails, many patients become progressively malnourished, as evidenced by reduced levels of albumin,
51                                     Children malnourished at birth had increased Entamoeba histolytic
52  were obese and 50% of Turkish children were malnourished at last observation (P < 0.005).
53 oversampled from mothers who were anaemic or malnourished at SUMMIT enrolment.
54       Many patients with cystic fibrosis are malnourished at the time of diagnosis.
55 ell-recognized lung disease of premature and malnourished babies.
56 formulation, MDCF-2, improved weight gain in malnourished Bangladeshi children compared to a more cal
57 who require antiretroviral therapy (ART) are malnourished because of a combination of HIV-associated
58 d after admission (period 2), when they were malnourished but free of infection; and approximately 54
59 ical phase 1), when they were still severely malnourished but no longer infected (clinical phase 2),
60 ical phase 1), when they were still severely malnourished but no longer infected (clinical phase 2),
61 ical phase 1), when they were still severely malnourished but no longer infected (clinical phase 2),
62                          Children who became malnourished by 12 months of age were more likely to hav
63 ed, respectively; 71.8% were at least mildly malnourished by at least 1 score.
64 inoma (HCT-8) cells and weaned nourished and malnourished C57BL/6 mice, following outcomes of growth
65 nosa aerosols were lower in the lungs of the malnourished C57BL/6J mice relative than in lungs of mic
66 73; 95% CI: 1.20, 2.49) were associated with malnourished cases, and the total burden of these pathog
67 long-term clinical trials evaluating IDPN in malnourished CHD patients.
68 posed as a potential therapeutic approach in malnourished CHD patients.
69 higher in children living with HIV (60%) and malnourished children (62%).
70  = .07); the tendency was more pronounced in malnourished children (7 [88%] vs 2 [28%]).
71                                     Severely malnourished children (weight for age z score, <-3.0) yo
72 s associated with malnutrition in Bangladesh.Malnourished children [weight-for-age z score (WAZ) <-2]
73 neumocystis pneumonia was first diagnosed in malnourished children and has more recently been found i
74 tosporidium infection places on the lives of malnourished children and immunocompromised individuals.
75 ides no benefit for those in gravest danger: malnourished children and immunocompromised patients.
76 he only approved treatment is ineffective in malnourished children and immunocompromised people.
77 ly currently approved drug is ineffective in malnourished children and immunocompromised people.
78    Cryptosporidiosis is a serious disease in malnourished children and in people with malignancies or
79 ice can mimic some metabolic changes seen in malnourished children and may help elucidate our underst
80 pots successfully detected these episodes in malnourished children and were acceptable to mothers.
81                                              Malnourished children are at increased risk for death du
82                                  Acutely ill malnourished children are at risk of being overfed when
83 ampling of peripheral blood from 81 severely malnourished children at the University Teaching Hospita
84 by comparing the severity of malnutrition in malnourished children before and during the war.
85          These results suggest that severely malnourished children can mount only a partial APP respo
86                             The mortality of malnourished children did not increase during the war.
87 penditure (REE) trajectories in hospitalized malnourished children during and after hospitalization.
88   Empirically treating hospitalized severely malnourished children during the stabilization phase wit
89 uring the acute phase of illness in severely malnourished children exceeded REE.
90 ause of morbidity and mortality in young and malnourished children from low- and middle-income countr
91                 Promoting catch-up growth in malnourished children has health benefits, but recent ev
92      We investigated the effect of an SFP on malnourished children in Guinea-Bissau who were returnin
93  diarrhea (95% CI, -69% to -16%) vs severely malnourished children in the control group.
94 istributions to vulnerable subgroups such as malnourished children is one approach to reaching those
95 nting" pathogen and suggest that, similarly, malnourished children may be at increased risk of G. lam
96  exposed to but not infected with HIV and in malnourished children not infected with HIV.
97  to HIV but uninfected and 90% (53 to 98) in malnourished children not infected with HIV.
98 therapeutic exposures were more common among malnourished children than among age-matched healthy chi
99                  Because the hypolactasia of malnourished children was associated with much lower lac
100       Four hundred eighteen and 425 severely malnourished children were randomized to F75 and mF75, r
101 er double-blind trial, hospitalized severely malnourished children were randomized to receive standar
102 s treatment outcomes in children, especially malnourished children who are at high risk of mortality.
103   This triple cohort study demonstrates that malnourished children with acute cryptosporidiosis mount
104 nosuppressive phenotype of DCs from severely malnourished children with endotoxemia provide a rationa
105                                           In malnourished children with HIV infection, a paradoxical
106                                 For the most malnourished children with MUAC <115 mm or edema, the st
107  MDCF-2 and members of the gut microbiota of malnourished children with potential implications for fu
108 parasites and is a leading cause of death in malnourished children worldwide.
109 ne development, respiratory infections among malnourished children, and limited work specifically on
110  longer-term consequences of the disease for malnourished children, involving growth stunting and cog
111 h higher anemia prevalence and among acutely malnourished children, respectively.
112 l was conducted in southern Malawi with 1178 malnourished children.
113 l status and the increase in mortality among malnourished children.
114 cultural and socioeconomic background of the malnourished children.
115  organ transplant recipients, travelers, and malnourished children.
116 ontribute to increased mortality in severely malnourished children.
117 nd how this can be used for interventions in malnourished children.
118 cterial pharmacokinetics, particularly among malnourished children.
119 ower socioeconomic status, and among acutely malnourished children.
120 s has shown poor outcomes among supplemented malnourished children.
121 sing improves malaria protection in young or malnourished children.
122 rolonged parasite shedding, especially among malnourished children.
123 en designed to repair the gut communities of malnourished children.
124 shed mice that is consistent with results in malnourished children.
125 c alterations that we previously observed in malnourished children.
126           There is also a high proportion of malnourished children.
127 ting the efficacy of supplementary foods for malnourished children.
128 and that diarrhea was more severe in already malnourished children.
129 nces the immune system, and is suppressed in malnourished children.
130 ribe normal DC maturation in the majority of malnourished children.
131 posed versus non-exposed (ie, previously non-malnourished) children.
132 ous (n = 11) SCU when they were infected and malnourished (clinical phase 1), when they were still se
133 d undernutrition when they were infected and malnourished (clinical phase 1), when they were still se
134 ous (n = 11) SCU when they were infected and malnourished (clinical phase 1), when they were still se
135  commonly consumed staple, will benefit many malnourished communities in rural Africa burdened with h
136         The children with Crohn disease were malnourished compared with the ulcerative colitis and co
137  of >=25 kg/m(2) were moderately or severely malnourished, depending on the nutritional index used.
138 tly shown clinical promise by protecting the malnourished embryonic kidney from adverse developmental
139  72.0 +/- 14.2 years and 42.6% patients were malnourished (GNRI < 92).
140  Length of stay differed by 11 d between the malnourished group and the nutritionally at-risk group (
141                       At 15 days of age, the malnourished group exhibits greater early inhibition of
142 igilance state, while at 30 days of age, the malnourished group exhibits greater facilitation at IPIs
143 ed (3% protein) and control mice, but in the malnourished group, a greater percentage (39.8 and 4.0%,
144 ontrast, peritoneal macrophages from protein-malnourished guinea pigs produced a higher level of TGF-
145 quently became inadvertently and transiently malnourished had compromised cell-mediated immunity comp
146   It does not address pregnant women who are malnourished, have symptoms of iron deficiency anemia, o
147                      The repeatedly infected malnourished host did not produce interleukin-10, a majo
148  a rationale for the theory that elderly and malnourished hosts may also represent norovirus reservoi
149  for compromised antimicrobial resistance in malnourished hosts remain obscure.
150 help explain the increased susceptibility of malnourished human populations to arsenic.
151 es are to address the plight of the severely malnourished in an efficient and effective manner.
152 ldren with acute cryptosporidiosis were more malnourished (including measures of stunting [P=.03] and
153                                              Malnourished individuals are especially vulnerable to in
154 ents when applied enterally, particularly in malnourished individuals.
155 ca, targeting susceptible HIV+, malarial, or malnourished individuals.
156 otein intake may improve vaccine efficacy in malnourished individuals.
157 secretor mothers, and 11 (57.89%) of the non-malnourished infants had secretor mothers.
158                                         Many malnourished infants have reduced lactase specific activ
159 test the hypothesis that the hypolactasia of malnourished infants results from transcriptional suppre
160 er Bg_2D9 is superior to EVC001 for treating malnourished infants who consume a diet with limited bre
161 , human immunodeficiency virus-infected, and malnourished infants).
162 enzyme proteins and activities were lower in malnourished infants, and partial villus atrophy was pre
163                                           In malnourished infants, lactase messenger RNA (mRNA) was r
164 the low lactase activities commonly found in malnourished infants.
165 bial community development in healthy versus malnourished infants/children, we performed a time-serie
166                    Lymph node cells from the malnourished, infected mice produced increased levels of
167 hat only men had lower insulin if previously malnourished: insulin (pmol/L) at 120 min was 311 (95% C
168 w many children in a particular area will be malnourished is fundamental to planning an effective ope
169  on language among children who were acutely malnourished (mean difference: 0.31) at baseline; on lan
170 railty, Functional Outcomes, and Recovery of malnourished medical inpatients Trial (EFFORT) is a prag
171 railty, Functional Outcomes, and Recovery of Malnourished Medical Inpatients Trial (EFFORT), a random
172 railty, Functional Outcomes, and Recovery of malnourished medical inpatients Trial) comparing individ
173 Frailty, Functional Outcome, and Recovery of Malnourished Medical Inpatients Trial, which compared th
174 Frailty, Functional Outcomes and Recovery of malnourished medical inpatients) trial.
175 fatal course of tuberculosis observed in the malnourished mice could be reversed by restoring a full
176                                              Malnourished mice exhibited a tissue-specific diminution
177                                    Faltering malnourished mice exhibited impaired compensatory respon
178                                              Malnourished mice show an inability to control sublethal
179 a striking deficiency in bile acid levels in malnourished mice that is consistent with results in mal
180 f the granulomatous reaction was observed in malnourished mice throughout the entire course of infect
181  the higher visceral parasite burdens in the malnourished mice were not due to a deficit in local par
182  were reduced in the lymph nodes of infected malnourished mice, as were CCR2-bearing monocytes/macrop
183 icantly lower in the spleen and liver of the malnourished mice.
184 s present both before and after infection in malnourished mice.
185 draining lymph nodes of L. donovani-infected malnourished mice.
186 n in vivo, were not detectably diminished in malnourished, mycobacteria-infected mice.
187 old infants who had SAM (n = 26) or were non-malnourished (n = 19) and were analyzed for constituent
188                                Majority were malnourished nonobese (57.7%), followed by malnourished
189 ves demonstrated least favorable survival in malnourished nonobese group, followed by malnourished ob
190 h nourished nonobese group as the reference, malnourished nonobese had higher all-cause mortality (ha
191                                              Malnourished nonobese had highest all-cause mortality (3
192 o the following: (1) nourished nonobese, (2) malnourished nonobese, (3) nourished obese, and (4) maln
193 e malnourished nonobese (57.7%), followed by malnourished obese (18.8%), nourished nonobese (16.9%),
194 all-cause mortality (38.6%), followed by the malnourished obese (35.8%), nourished nonobese (21.4%),
195 nt increase in mortality was observed in the malnourished obese (hazard ratio, 1.31 [95% CI, 0.94-1.8
196  in malnourished nonobese group, followed by malnourished obese, nourished nonobese, and nourished ob
197 ished nonobese, (3) nourished obese, and (4) malnourished obese.
198 bjects admitted to subacute care were either malnourished or at risk of malnutrition.
199 t infected with HIV, including those who are malnourished or who have been exposed to HIV.
200 assified as adequately nourished, moderately malnourished (or suspected to be), or severely malnouris
201  via modifications in the immunoepigenome of malnourished parents, and these may contribute to interg
202 , and potentially focus on a more profoundly malnourished patient population.
203                                              Malnourished patients (BMI of <20) were excluded.
204 fely with minimal risk of adverse outcome in malnourished patients and in the critically ill and crit
205 perative drainage only in selected cases (in malnourished patients and in those with hypoalbuminemia,
206 ral nutrition may be an effective therapy in malnourished patients before undergoing elective surgery
207          Survival was significantly lower in malnourished patients compared to patients without myost
208                                              Malnourished patients had lower rates of combination med
209                                              Malnourished patients have increased levels of C reactiv
210 han parenteral hyperalimentation in severely malnourished patients may improve survival.
211                           Enteral feeding in malnourished patients may result in rapid growth of gut
212                                     Severely malnourished patients require more blood products during
213 , it is of particular importance to identify malnourished patients so that nutritional therapy can be
214 idely preferred to enteral nutrition (EN) in malnourished patients undergoing allogeneic stem-cell tr
215                            Female, obese and malnourished patients were at high risk of extracellular
216                    Nutritional management in malnourished patients with cirrhosis should be undertake
217 thropometry, and immune function in severely malnourished patients with cirrhosis.
218                                        Among malnourished patients with PU, 8 weeks of supplementatio
219 ibilities including: nutrition treatment for malnourished patients, optimization of growth and develo
220 individualised nutritional interventions for malnourished patients, should become part of routine cli
221 k of overt HE in patients with cirrhosis; in malnourished patients, the amelioration of nutritional s
222 ects of immunonutrition are most apparent in malnourished patients.
223 from severe acute malnutrition and their non-malnourished peers in parallel for 6 months in Mali (nin
224 entres as the sole mode of treating severely malnourished people during famine is inappropriate and o
225 d intervention for the treatment of severely malnourished people.
226 iate treatment for large numbers of severely malnourished people.
227 m this cohort study highlights a chronically malnourished population with much of the burden associat
228 riculture, which could feed about 30% of the malnourished population.
229                     Measles among displaced, malnourished populations can result in a high case fatal
230                Lethargy is characteristic of malnourished populations, but little is known about the
231 ndard care than with parenteral nutrition in malnourished populations.
232 tous (n = 7) SCU when they were infected and malnourished (postadmission day approximately 3; clinica
233 l and offspring anthropometry for moderately malnourished pregnant women receiving ready-to-use suppl
234 t neocortex in normal and prenatally protein malnourished rats ages 15, 30, 90, and 220 postnatal day
235 r to stimulation was significantly higher in malnourished rats as compared to well-nourished controls
236 5-HT) fibers in the hippocampal formation of malnourished rats as well as increased levels of 5-HT in
237 pal mossy fiber zone) in 90- and 220-day-old malnourished rats compared with age- and sex-matched con
238  adult but not in early postnatal developing malnourished rats may help ensure continued breeding and
239      Stimulation of this nucleus at 20 Hz in malnourished rats resulted in a significantly diminished
240  the ileum and cecum was less in the protein-malnourished rats than in the normally nourished animals
241  and AMPA receptors quantified in prenatally malnourished rats was comparable to the density quantifi
242                                           In malnourished rats, stress produced an increase in 5-HT i
243 vitro was decreased significantly in protein-malnourished rats, whereas E. coli binding to insoluble
244 atched controls but not in 15- or 30-day-old malnourished rats.
245  hippocampal formation of prenatally protein malnourished rats.
246 e nucleus (MRN) in unanesthetized prenatally malnourished rats.
247 -nourished rats but did not alter release in malnourished rats.
248 nd 8.9% patients were moderately or severely malnourished, respectively; 71.8% were at least mildly m
249  group than in the nonedematous group in the malnourished state but not in the recovered state.
250                       When compared with the malnourished state value, leucine flux at recovery doubl
251  group than in the nonedematous group in the malnourished state, but in the recovered state, it was f
252  postnatal development, a way of classifying malnourished states, and a parameter for judging therape
253  with the duration of dialysis (vintage) and malnourished status and was negatively associated with s
254 The Geriatric Depression Score was higher in malnourished subjects than in nutritionally at-risk subj
255         In the MNA-assessed group of largely malnourished subjects, 25% of subjects required readmiss
256 renteral nutrition is beneficial in severely malnourished surgical patients.
257                                              Malnourished, tuberculous guinea pigs harbored only 20 a
258  pancreata within 2 months of age and became malnourished, underweight, hypoglycemic, and hypothermic
259 cipation was restricted to patients who were malnourished, were able to drink oral supplements, and w
260 te lymphoblastic leukemia rarely are overtly malnourished, which makes this population ideal for an i
261 proximately a billion people are chronically malnourished while our agricultural systems are concurre
262 +/-SD) BMI was 21.6 (+/-3.78) kg/m(2) (18.3% malnourished with BMI<18.5); and among women for whom th

 
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