ライフサイエンスコーパス: malondialdehyde

1 oxidative stress (3-fold reduction in tissue malondialdehyde).

2 rescence parameters and their high levels of malondialdehyde.

3 d peroxidation biomarkers 8-isoprostanes and malondialdehyde.

4 nals, and lipid peroxidation products, e.g., malondialdehyde.

5 ication of dG residues with base propenal or malondialdehyde.

6 rom exposure of DNA to base propenals and to malondialdehyde.

7 odel for the exocylic M 1dG adduct formed by malondialdehyde.

8 ic replacement of the trimethylene tether by malondialdehyde.

9 uld be found in incubations of collagen with malondialdehyde.

10 wing a 4-day incubation of albumin with 8 mm malondialdehyde.

11 cies (RCS) acrolein, hydroxyl-2-nonenal, and malondialdehyde.

12 Peroxide value, malondialdehyde, 2,4-heptadienal and 2,4-decadienal were

13 cant suppression of oxidative stress (plasma malondialdehyde, 22%; 8-isoprostane-F(2alpha), 12%; P <

14 ction of IL-12 (-25% versus -4% versus -2%), malondialdehyde (-27% versus +5% versus +26%), and NT-pr

15 tive cells); and oxidative stress (increased malondialdehyde, 3-nitrotyrosine, and nicotinamide adeni

16 cally relevant reactive aldehydes (acrolein, malondialdehyde, 4-hydroxy-2-nonenal, and 4-oxo-2-nonena

17 We analyzed malondialdehyde, 4-hydroxynonenal (HNE), and 4-hydroxy-2

18 There was a significant increase of malondialdehyde, 4-hydroxynonenal adducts, 3-nitrotyrosi

19 Increased protein carbonyls, malondialdehyde, 4-hydroxynonenal-protein adducts, eleva

20 Malondialdehyde/4-hydroxyalkenals concentrations (mean +

21 region) showed the highest protection toward malondialdehyde (83.3% reduction).

22 ed to a decreased intestinal accumulation of malondialdehyde, a biomarker of lipid peroxidation.

23 Malondialdehyde, a genotoxic byproduct of lipid peroxida

24 nt brain regions removed for assay of VC and malondialdehyde, a marker of lipid peroxidation.

25 GPX was also positively associated with malondialdehyde, a marker of oxidative damage.

26 Levels of malondialdehyde, a marker of ROS generation and oxidant

27 Malondialdehyde, a marker of ROS-formation, was elevated

28 ell as accumulation of hydrogen peroxide and malondialdehyde, a product of lipid peroxidation.

29 Malondialdehyde, acetaldehyde, acrolein, and 4-hydroxyno

30 nol-fed ALDH2(-/-) mice had higher levels of malondialdehyde-acetaldehyde (MAA) adduct and greater he

31 bound immunodominant OSE adducts termed MAA (malondialdehyde-acetaldehyde-adducts), which are found o

32 ldehyde-bovine serum albumin (f-Alb) or 125I-malondialdehyde-acetaldehyde-bovine serum albumin (MAA-A

33 with the SR-binding ligands oxidized LDL and malondialdehyde-acetaldehyde-modified LDL.

34 as well as the higher hydrogen peroxide and malondialdehyde additionally contribute to premature sen

35 The conclusion from this work is that malondialdehyde adducts in the transcribed strand of exp

36 serum albumin also revealed two acid-labile malondialdehyde adducts of histidine in significant quan

37 and ROS generation, reduced the formation of malondialdehyde adducts, maintained a normal distributio

38 , glutathione), markers of oxidative stress (malondialdehyde, ADP-ribose) and nicotinic coenzymes (NA

39 Malondialdehyde, an indicator of oxidative stress indica

40 ith NAC reduced myocardial concentrations of malondialdehyde and 4-hydroxy-2(E)-nonenal, markers of o

41 tive stress, as indicated by the decrease of malondialdehyde and 4-hydroxynonenal content in BAL of R

42 Malondialdehyde and 4-hydroxynonenal were measured in br

43 rance of lipid peroxidation products such as malondialdehyde and 4-hydroxynonenal-2-nonenal.

44 c inflammation and oxidative stress (urinary malondialdehyde and 8-hydroxy-2'-deoxyguanosine, plasma

45 ression and serum levels of Klotho, improved malondialdehyde and 8-hydroxy-deoxy guanosine levels, an

46 Malondialdehyde and 8-oxodG were significantly associate

47 cant rise in the content of ATP-catabolites, malondialdehyde and ADP-ribose.

48 The biological aldehydes, malondialdehyde and base propenal, react with DNA to for

49 PB and Aroclor 1254 significantly enhanced malondialdehyde and H2O2 generation and NADPH oxidation

50 HDL-bound malondialdehyde and HDL-induced NO production by EC were

51 1 (VCAM-1), secretory phospholipase A2, and malondialdehyde and hydroxyalkenals (MDA+HAE) in elderly

52 munohistochemistry to detect the presence of malondialdehyde and hydroxynonenal adducts.

53 lected and analyzed for histology, levels of malondialdehyde and liver enzymes, gene expression, and

54 ry occluded rats demonstrated high levels of malondialdehyde and low levels of reduced glutathione an

55 paired biopsies, lower lobes contained more malondialdehyde and mtDNA deletions than upper lobes and

56 dation by-products as reflected by increased malondialdehyde and oxidized albumin.

57 AD decreased malondialdehyde and oxidized LDL at 7 d (35% and 11%, re

58 barbituric acid reactive substances (TBARS), malondialdehyde and phytosterol oxidation products (POPs

59 concentration of oxidative stress byproduct malondialdehyde and pro-inflammatory cytokine tumor necr

60 d nitrotyrosine and quantitative analysis of malondialdehyde and protein carbonyl in the liver.

61 After 12 weeks, RBBO significantly decreased malondialdehyde and restored superoxide dismutase, catal

62 P < 0.05) with the changes in CIELAB colour, malondialdehyde and sensory scoring than with the change

63 n of DNA with the lipid peroxidation product malondialdehyde and the DNA peroxidation product base pr

64 d glycoprotein were determined in serum, and malondialdehyde and total glutathione content were deter

65 Malondialdehyde and total glutathione content were measu

66 ions of protein (3-nitrotyrosine) and lipid (malondialdehyde) and increase GSH content both in bleomy

67 peroxidase was incubated with glycine, H2O2, malondialdehyde, and a lysine analog in PBS at a physiol

68 eta, protein carbonyl, higher nitrotyrosine, malondialdehyde, and Fas/Fas ligand than non-CAD (P<0.05

69 tic peptide), TNF-alpha, IL-6, IL-12, IL-17, malondialdehyde, and fetuin-a.

70 was evaluated by assessing the iron content, malondialdehyde, and glutathione levels, ferroptosis-rel

71 ts of oxidative stress: 4-hydroxy-2-nonenal, malondialdehyde, and glycolaldehyde.

72 electrophile signals such as phytoprostanes, malondialdehyde, and hexenal in plastids.

73 associated with higher airway 8-isoprostane, malondialdehyde, and IL-13 concentrations.

74 locked basal and maximal formation of CD and malondialdehyde, and lengthened the lag times of LDL, sd

75 tric oxide and exhaled breath condensate pH, malondialdehyde, and nitrite), and systemic inflammation

76 peroxidation products: hydroxy fatty acids, malondialdehyde, and phytoprostanes.

77 e synthase, 3-nitrotyrosine protein adducts, malondialdehyde, and protein carbonyls were also higher

78 ) as evidenced by the increased formation of malondialdehyde, and reduced antioxidant enzymes includi

79 phosphorylcholine (anti-PC) and Abs against malondialdehyde (anti-MDA) may be protective in chronic

80 tive stress markers, including antibodies to malondialdehyde (anti-MDA) protein adducts and to 4-hydr

81 om each species of milk was determined using malondialdehyde as an oxidation product marker.

82 otoxic aldehydes including methylglyoxal and malondialdehyde as substrates and the reduced form of ni

83 smin activity, ceruloplasmin protein, plasma malondialdehyde, benzylamine oxidase activity, erythrocy

84 II by HDL(NYHA-IIIb), and a higher amount of malondialdehyde bound to HDL(NYHA-IIIb) compared with HD

85 inflammatory cell infiltration, lung tissue malondialdehyde, bronchoalveolar lavage fluid protein co

86 d production of the oxidative stress marker, malondialdehyde by ~80%.

87 th a corresponding increase in the levels of malondialdehyde by-product (fourfold).

88 F(2)-isoprostanes, malondialdehyde, C-reactive protein, serum vitamin C, ca

89 through positive regulation on superoxidase, malondialdehyde, caspase-3 and Bax.

90 f SBP linearly (P < 0.01) decreased egg yolk malondialdehyde, cholesterol, and triglyceride, while in

91 oduced non-significantly different levels of malondialdehyde compared to the blank containing no ferr

92 sed amount of the lipid peroxidation product malondialdehyde compared to the wild type, suggesting th

93 as evidenced by higher circulating levels of malondialdehyde compared with WT controls.

94 g) in the meat of the SBs compared with the malondialdehyde concentration (1.79 +/- 0.17 micromol/25

95 There was a 71% reduction in the malondialdehyde concentration (mean +/- SD: 0.52 +/- 0.0

96 The plasma malondialdehyde concentration increased significantly in

97 groups, although, paradoxically, the plasma malondialdehyde concentration was significantly higher a

98 itro antioxidant activity was able to reduce malondialdehyde concentration.

99 83.2 +/- 2470.1 ng/mL, P = 0.03), and plasma malondialdehyde concentrations (-0.5 +/- 1.6 vs. +0.3 +/

100 fidence interval: -53.5, -15.7), and urinary malondialdehyde concentrations (-25.3%; 95% confidence i

101 Urinary malondialdehyde concentrations (micromol/g creatinine) d

102 production of malondialdehyde in burgers and malondialdehyde concentrations in plasma and urine after

103 urger meat before cooking was a reduction in malondialdehyde concentrations in the meat, plasma, and

104 erol concentrations (all P < 0.01) and lower malondialdehyde concentrations, which persisted after ad

105 relation to airway IL-13, 8-isoprostane, and malondialdehyde concentrations.

106 r meat and its effects on plasma and urinary malondialdehyde concentrations.

107 ) significantly declined (p < 0.05) with low malondialdehyde concentrations.

108 zed monoclonal autoantibody that reacts with malondialdehyde-conjugated LDL, was labeled with a NIRF

109 tformin also attenuated oxidative stress and malondialdehyde-containing protein levels, with correspo

110 Significant increase of malondialdehyde content and decrease of neutral red rete

111 Also, they showed lesser malondialdehyde content and electrolyte leakage compared

112 Malondialdehyde content as well as protein oxidation pro

113 , hydrogen peroxide, electrolyte leakage and malondialdehyde content than control.

114 uced glutathione levels were higher, whereas malondialdehyde content was lower, in the renal cortex o

115 Oxidative stress, indicated by malondialdehyde content, was greater in gr3 than the WT

116 blood flows, ventilatory pressures, and lung malondialdehyde content.

117 t peroxidation in the cakes, measured as the malondialdehyde content.

118 oss, pericarp browning, membrane leakage and malondialdehyde contents.

119 escribed for the quantification of the major malondialdehyde deoxyguanosine adduct, pyrimido[1,2-alph

120 Optimization was performed on both the malondialdehyde-deoxyguanosine (M(1)dG) adduct and the O

121 The malondialdehyde-deoxyguanosine adduct, 3-(2'-deoxy-beta-

122 The major malondialdehyde-derived adduct in DNA is 3-(2'-deoxy-bet

123 e provide indirect but strong evidence for a malondialdehyde-derived cross-link requiring just one ma

124 ligands C-reactive protein, pentraxin 3, and malondialdehyde epitopes.

125 E, such as oxidized phospholipids (OxPL) and malondialdehyde epitopes.

126 munohistochemistry confirmed the presence of malondialdehyde-epitopes and iron particles.

127 ), oligonucleotide 3'-phosphoglycolates (7), malondialdehyde equivalents (8 or 9), and furfural (10).

128 Here, we show that fluorescent malondialdehyde-formaldehyde (M2FA)-lysine adducts are i

129 ing periods considered in the present study, malondialdehyde formation was affected by the NaCl level

130 he effect of an antioxidant spice mixture on malondialdehyde formation while cooking hamburger meat a

131 y-5(Z),8(E),10(E)-heptadecatrienoic acid and malondialdehyde from PGH2, but not formation of PGE2.

132 rat pups had significantly higher levels of malondialdehyde, glutathione reductase enzyme activity,

133 oxidative damage was corroborated by higher malondialdehyde immunoreactivity in lesions from middle-

134 ation of advanced glycation end products and malondialdehyde in a dose-dependent manner.

135 The production of malondialdehyde in burgers and malondialdehyde concentra

136 gs at the end of reperfusion and assayed for malondialdehyde in combination with 4-hydroxyalkenals to

137 Reactive oxygen species and malondialdehyde in H2O2 treated CCD 841 CoN (CCD) and Ca

138 rotein), and markedly less nitrotyrosine and malondialdehyde in porphyrin-treated spinal cords relati

139 al-modified proteins, protein carbonyls, and malondialdehyde) in IFM with age.

140 Malondialdehyde increased in the cortex as VC supplement

141 Malondialdehyde increased with higher roasting temperatu

142 Lipid peroxidation, as measured by malondialdehyde, increased and antioxidants (superoxide

143 Malondialdehyde interstrand cross-links in DNA show stro

144 Malondialdehyde is an endogenous product of oxidative st

145 Stabilin-1 deficiency abrogated malondialdehyde-LDL (MDA-LDL) uptake by hepatic macropha

146 ts induced severe tissue damage and enhanced malondialdehyde levels and senescence symptoms, but not

147 idenced by a significant increase in hepatic malondialdehyde levels and upregulation of Nrf2-regulate

148 8-Hydroxy-deoxy guanosine and malondialdehyde levels as markers of oxidative stress we

149 had no apparent effect on fatty liver tissue malondialdehyde levels augmented by Jo2.

150 The increase in renal malondialdehyde levels caused by renal I/R was also sign

151 d mucosal graft morphology, diminished graft malondialdehyde levels demonstrating substantial reducti

152 Additionally, it lowered the malondialdehyde levels in plasma and abolished the incre

153 sser membrane damage as indicated by reduced malondialdehyde levels in transgenic leaves subjected to

154 oral neck fracture was assessed by measuring malondialdehyde levels using the thiobarbituric acid rea

155 ere also measured: Myeloperoxidase activity, malondialdehyde levels, and plasma nitrite/nitrate.

156 ted in increased reactive oxygen species and malondialdehyde levels, disruption of mitochondrial memb

157 ) levels and higher hepatic triglyceride and malondialdehyde levels.

158 (OSEs) reflecting oxidized phospholipids and malondialdehyde-like epitopes.

159 9 +/- 5.8 vs. 38.8 +/- 3.8; p < 0.0001), and malondialdehyde-like OSEs (93.9 +/- 7.9 vs. 54.7 +/- 3.9

160 percentage of circulating B-1 cells and anti-malondialdehyde-low-density lipoprotein IgM suggesting c

161 Synthetic ALE (malondialdehyde-lysine [MDA-Lys]) (50 micromol/l) could

162 onjugated with antibodies targeted to either malondialdehyde-lysine or oxidized phospholipid epitopes

163 olume, measurement of brain concentration of malondialdehyde (MDA) (an end-product of lipid peroxidat

164 xyguanosine (8-OHdG), protein carbonyls, and malondialdehyde (MDA) adducts of proteins, markers of ox

165 ably reduced levels of lipid hydroperoxides, malondialdehyde (MDA) and 4-hydroxy-trans2-hexenal (HHE)

166 Two lipid peroxidation end products, malondialdehyde (MDA) and 4-hydroxynonenal (4-HNE), show

167 Serum concentration of lipofuscine (LPS), malondialdehyde (MDA) and activity of total superoxide d

168 (SAM), and glutathione, and elevated plasma malondialdehyde (MDA) and alanine transaminase.

169 as measured by 2 lipid peroxidation markers, malondialdehyde (MDA) and F2-isoprostanes (IsoPs).

170 carbonyls resulting from lipid peroxidation (malondialdehyde (MDA) and hydroxynonenal) or carbohydrat

171 d glutathione (GSH) content, higher level of malondialdehyde (MDA) and lower levels of protein carbon

172 ns between secondary lipid oxidation product malondialdehyde (MDA) and selected beta-lactoglobulin (b

173 sical (MRI data) and biochemical parameters (Malondialdehyde (MDA) and starch content) from the peric

174 Serum malondialdehyde (MDA) and thioredoxin-1 (Trx1) levels we

175 s on metal ion-catalyzed oxidation of LDL to malondialdehyde (MDA) and to protein carbonyl and MetO d

176 ve oxygen species (ROS) scavenging including malondialdehyde (MDA) as a measure of lipid peroxidation

177 ing down the formation of hydroperoxides and malondialdehyde (MDA) compounds.

178 ameliorated the oxidative damage by reducing malondialdehyde (MDA) concentration and increasing antio

179 in hepatic pericytes, glutathione (GSH), and malondialdehyde (MDA) concentrations in liver; and serum

180 creased soluble protein content, proline and malondialdehyde (MDA) content as well as O2*(-) producti

181 s tolerance characteristics, including lower malondialdehyde (MDA) content, lower water loss rates, l

182 The soluble proteins and malondialdehyde (MDA) contents were stimulated by all te

183 We further show that malondialdehyde (MDA) epitopes, products of lipid peroxi

184 cid reactive substances (TBARS, 0.30-0.38 mg malondialdehyde (MDA) equivalents/kg mince) under low ox

185 re-slaughter stress i) increased post-mortem malondialdehyde (MDA) formation except in vacuum-stored

186 Oxidation events such as malondialdehyde (MDA) formation may produce specific, im

187 well as levels of free radical damage marker malondialdehyde (MDA) in blood and saliva of individuals

188 contents of free amino acid, carotenoid, and malondialdehyde (MDA) in egg yolk.

189 The levels of malondialdehyde (MDA) in gamma-ECS lines were also 27.3-

190 lpha (TNF-alpha) in plasma and TNF-alpha and malondialdehyde (MDA) in lung tissues were detected.

191 ioxidant contents and increased the level of malondialdehyde (MDA) in the hippocampus and striatum of

192 utathione peroxidase (GPx) and the levels of malondialdehyde (MDA) in the oedematous paw.

193 Malondialdehyde (MDA) is a natural and widespread genoto

194 Malondialdehyde (MDA) is an endogenous genotoxic product

195 Hepatic malondialdehyde (MDA) levels (a marker of lipid peroxida

196 at test, and brain homogenates, by measuring malondialdehyde (MDA) levels as a lipoperoxidation bioma

197 lop an accurate and fast method to determine malondialdehyde (MDA) levels in raw and processed meat.

198 Malondialdehyde (MDA) levels in the brain were measured

199 (a marker of oxidative stress) and increased malondialdehyde (MDA) levels in the hippocampus and amyg

200 Malondialdehyde (MDA) levels increased dramatically afte

201 ant effect on the fatty acid composition and malondialdehyde (MDA) levels of fresh eggs but reduced t

202 Colonic interleukin (IL)-1beta, IL-6, and malondialdehyde (MDA) levels were measured.

203 n liver and cerebellum in gulo(-/-) mice and malondialdehyde (MDA) levels were significantly increase

204 elated lipid peroxidation, measured as serum malondialdehyde (MDA) levels, correlates with delayed gr

205 panied by a concomitant decrease in cellular malondialdehyde (MDA) levels.

206 of oxidative stress and can be evaluated via malondialdehyde (MDA) levels.

207 the evaluation of methemoglobin (MetHb) and malondialdehyde (MDA) levels.

208 Moreover, a significant increase in malondialdehyde (MDA) production and a decrease in biofi

209 Measurement of malondialdehyde (MDA) showed that NACA reduced glutamate

210 The increased production of malondialdehyde (MDA) suggests that Ga stress could caus

211 onyls including isolevuglandins (IsoLGs) and malondialdehyde (MDA) that covalently modify proteins.

212 Malondialdehyde (MDA) was measured as a marker of oxidat

213 Circulating inflammatory markers and malondialdehyde (MDA) were measured before and after tre

214 The formation of malondialdehyde (MDA), 4-hydroxy-2-hexenal (HHE), 4-hydr

215 d the levels of oxidative damage biomarkers, malondialdehyde (MDA), 4-hydroxynonenal (4-HNE), aconita

216 Urinary malondialdehyde (MDA), a marker of lipid peroxidation, w

217 wn to arise in vitro in reactions of dG with malondialdehyde (MDA), a product of both lipid peroxidat

218 soluble antioxidants, the cellular amount of malondialdehyde (MDA), a product of lipid peroxidation,

219 es exposed during programmed cell death, and malondialdehyde (MDA), a reactive aldehyde degradation p

220 tes renal oxidative stress markers including malondialdehyde (MDA), advanced protein oxidation produc

221 rates lipid fragmentation products including malondialdehyde (MDA), an archetypal marker of PUFA oxid

222 At both low and high AA intakes, hepatic malondialdehyde (MDA), an indicator of oxidative stress,

223 Total antioxidant capacity, plasma levels of malondialdehyde (MDA), and activities of glutathione per

224 e base nitrogen (TVBN), peroxide value (PV), malondialdehyde (MDA), and bacteria levels.

225 ), blood levels of C-reactive protein (CRP), malondialdehyde (MDA), and CD11b/c positive cell ratio w

226 is study is to determine the serum levels of malondialdehyde (MDA), as a lipid peroxidation marker, a

227 quantification of conjugated dienes (CD) and malondialdehyde (MDA), but also by assessment of tocophe

228 DEN administration increased the levels of malondialdehyde (MDA), DNA fragmentation, caspase-3 and

229 length between functional groups were used: malondialdehyde (MDA), glutaraldehyde and hexamethylene

230 Cardiac tissue levels of malondialdehyde (MDA), glutathione (GSH), superoxide dis

231 In the hepatic tissue, the levels of malondialdehyde (MDA), glutathione (GSH), total choleste

232 derived aldehydes, 4-hydroxynonenal (4-HNE), malondialdehyde (MDA), glyoxal (GLY), atheronal-A (KA),

233 water stress, while significantly enhancing malondialdehyde (MDA), H(2)O(2), electrolyte leakage, ox

234 Biomarkers of oxidation, such as malondialdehyde (MDA), may represent independent indicat

235 Renal function, histopathology, malondialdehyde (MDA), myeloperoxidase levels, and antio

236 Malondialdehyde (MDA), nitric oxide (NO), superoxide dis

237 urobehavioral scores and infarction volumes, malondialdehyde (MDA), reactive oxygen species (ROS), an

238 num and ileum were analyzed by histology and malondialdehyde (MDA), respectively.

239 centrations of 4-hydroxyalkenals (4-HNE) and malondialdehyde (MDA), was reduced significantly by V-PY

240 In this study, levels of malondialdehyde (MDA), which is a significant product of

241 6, lipoprotein(a) [Lp(a)], autoantibodies to malondialdehyde (MDA)-LDL and copper-oxidized LDL (Cu-Ox

242 , immunoglobulin (Ig)G/IgM autoantibodies to malondialdehyde (MDA)-LDL, and apolipoprotein B (apoB)-i

243 munoglobulin (Ig)G and IgM autoantibodies to malondialdehyde (MDA)-low-density lipoprotein (LDL) and

244 ine monoclonal IgG antibody MDA2 targeted to malondialdehyde (MDA)-lysine epitopes or the human singl

245 rphology and contractility, Ca(2+) handling, malondialdehyde (MDA)-modified proteins, and ROS levels

246 w-density lipoprotein (LDL) cholesterol, and malondialdehyde (MDA).

247 oduct of the reaction of deoxyguanosine with malondialdehyde (MDA).

248 cysteine 328 (C328) by the oxidative adduct malondialdehyde (MDA).

249 to proteins where lysines have reacted with malondialdehyde (MDA).

250 to lipid peroxidation of liver tissue with a malondialdehyde (MDA)/free fatty acids (FFA) ratio of 0.

251 used for the analysis of lipid peroxidation (malondialdehyde [MDA]) and antioxidant enzymes (catalase

252 e aminotransferase (ALAT); oxidative stress (malondialdehyde [MDA], reduced glutathione/oxidative glu

253 self-ligands or altered self-ligands (e.g., malondialdehyde [MDA]-modified molecules) involved in ho

254 in B) and more oxidized (5.7 versus 1.5 nmol malondialdehyde/mg lipoprotein).

255 ious oxidation-specific neoepitopes, such as malondialdehyde-modified (MDA-modified) LDL (MDA-LDL) or

256 pecific epitopes, including IgM specific for malondialdehyde-modified LDL (low-density lipoprotein).

257 ion end products-modified LDL (AGE-LDL), and malondialdehyde-modified LDL (MDA-LDL) in IC and determi

258 noglobulin (Ig) G1, IgG2b, and IgG2c against malondialdehyde-modified LDL (MDA-LDL), presumably as a

259 plasma levels of IgM antibodies specific for malondialdehyde-modified LDL and inversely associates wi

260 The results demonstrate that injection of malondialdehyde-modified LDL promotes a Th2 response tha

261 ed lipoproteins, including autoantibodies to malondialdehyde-modified low-density lipoprotein (MDA-LD

262 m of recombinant mouse CD16 (sCD16) bound to malondialdehyde-modified low-density lipoprotein (MDALDL

263 hose on oxidized low-density lipoprotein and malondialdehyde-modified low-density lipoprotein.

264 bulin (Ig)-G (IgG) and IgM autoantibodies to malondialdehyde-modified, low-density lipoprotein (MDA-L

265 dehyde-derived cross-link requiring just one malondialdehyde molecule to link arginine and lysine, gi

266 injury, as evidenced by decreased levels of malondialdehyde, myeloperoxidase activity, and tumor nec

267 Moreover, SRT1720 decreased the levels of malondialdehyde, nitrotyrosine, and inducible nitric oxi

268 Although collagen is readily cross-linked by malondialdehyde, none of these particular products could

269 milk produced significantly lower amounts of malondialdehyde of 0.46+/-0.04mugMDA/ml after 3days at 3

270 ajor reaction product of deoxyguanosine with malondialdehyde or base propenals.

271 DL oxidizability, urinary F(2)-isoprostanes, malondialdehyde, or protein carbonyls in native plasma).

272 anosine with the lipid peroxidation product, malondialdehyde, or the DNA peroxidation product, base p

273 tments affected fillet lipid oxidation (free malondialdehyde), pigmentation and flavour volatile comp

274 se in lipid peroxidation, as assessed by LDL-malondialdehyde plasma concentration, was found in HC bu

275 AA repletion, led to oxidative stress (using malondialdehyde production as an index) and to major inc

276 on of ApoB, conjugated diene production, and malondialdehyde production through Cu(2+)-mediated oxida

277 significantly increase superoxide anion and malondialdehyde production to two-folds as compared to h

278 athione-s-transferase and catalase activity, malondialdehyde production, and induction of apoptosis.

279 avengers, Fe(2+) chelators and inhibitors of malondialdehyde production, while the essential oil was

280 g; and (4) interleukin-1beta, nitrotyrosine, malondialdehyde, protein carbonyl, and Fas/Fas ligand le

281 ock-out mice also showed increased levels of malondialdehyde, protein carbonyls, protein methionine s

282 increased levels of 4-hydroxy-2-nonenal- and malondialdehyde-protein adducts.

283 Base propenals and malondialdehyde react with DNA to form adducts, includin

284 tance, and liver weight, fat, triglycerides, malondialdehyde, reactive oxygen species and nitrotyrosi

285 ota rod tests), oxidative stress parameters (malondialdehyde, reduced glutathione, and superoxide dis

286 and oxidative stress: C-reactive protein and malondialdehyde, respectively.

287 ts improved the redox status by reducing the malondialdehyde serum levels and protein oxidative damag

288 hat the trimethylene tether and probably the malondialdehyde tether, as well, could be accommodated w

289 hamburger meat can promote the formation of malondialdehyde that can be absorbed after ingestion.

290 Plasma antioxidants, protein carbonyls, malondialdehyde, total antioxidant performance, LDL oxid

291 ively, and urinary lipid peroxidation marker malondialdehyde was decreased by 32 +/- 21% compared to

292 ventricular expression of the cytokines and malondialdehyde was induced in non-WM, whereas it was no

293 Lung malondialdehyde was significantly less in LH (LH, 0.33 +

294 F(2)-isoprostanes and malondialdehyde were lower in the GSTM1-0 and GSTT1-0 gr

295 -7,8-dihydro-2'-deoxyguanosine (8-oxodG) and malondialdehyde were measured in urine samples collected

296 lglucosinolate, 5-hydroxymethylfurfural, and malondialdehyde) were not detected in any tissue sample

297 ate dehydrogenase by 4-hydroxy-2-nonenal and malondialdehyde when co-incubated with NADP(+), reinforc

298 significantly decrease the concentration of malondialdehyde, which suggests potential health benefit

299 kely produced by endogenous formaldehyde and malondialdehyde with lysine.

300 n of peroxidation products, base propenal or malondialdehyde, with deoxyguanosine residues in DNA.