ライフサイエンスコーパス: mammillary body

1 erebral cortex, hippocampus, cerebellum, and mammillary body.

2 of the mGluR2/3 agonist 2R,4R-APDC into the mammillary bodies.

3 brain regions including the hippocampus and mammillary bodies.

4 as well as subcortical regions including the mammillary bodies.

5 or temporal cortex, hippocampus, fornix, and mammillary bodies.

6 creased hippocampal activation and decreased mammillary bodies activity, while unsolvable anagrams we

7 ent study sought to identify the role of the mammillary bodies and their projections to the anterior

8 Although both mammillary body and mammillothalamic tract lesions resul

9 Normalized hippocampal, mammillary body and thalamic volumes were derived by man

10 orsal thalamic nucleus (AD) from the lateral mammillary body and the cortical afferents arriving thro

11 pus, subiculum, entorhinal cortex, amygdala, mammillary bodies, and septum were reported in a postmor

12 e limbic HAP-wave may travel through fornix, mammillary bodies, and the anterior nucleus of the thala

13 ventromedial, and premammillary nuclei, the mammillary body, and finally the substantia nigra and ve

14 y in areas of damage including the thalamus, mammillary body, and inferior colliculus.

15 e data support new functional models whereby mammillary bodies are important for coordinating hippoca

16 Although the thalamus and/or mammillary bodies are the primary sites of neuropatholog

17 regression analysis identified fornix FA and mammillary bodies as predictor of visual recall (R(2) =

18 The convergent evidence that mammillary body atrophy impairs recall but spares famili

19 Mammillary body atrophy is present in a number of neurol

20 emory loss when accompanied by fornix and/or mammillary body atrophy.

21 differed only with respect to the extent of mammillary body atrophy.

22 ral septum, ventral forebrain, hypothalamus, mammillary bodies, central and medial nuclei of the amyg

23 r injection was more laterally placed in the mammillary bodies, consistent with a projection to the l

24 Together, these results suggest that mammillary body damage causes an encoding deficit when l

25 the volumes of the left fornix and the left mammillary bodies decreased, the difference between reca

26 tylcholine after the lesion of the fornix or mammillary bodies did not increase the severity of the i

27 ngs are inconsistent with previous models of mammillary body function (those dominated by hippocampal

28 ulated to compare three possible theories of mammillary body function by increasing proactive interfe

29 demonstrating the crucial involvement of the mammillary bodies in post-encoding processing of spatial

30 sses, highlighting an important role for the mammillary bodies in the coordination of hippocampocorti

31 es, lesions were centred at the level of the mammillary bodies in the posterior hypothalamus.

32 nd demonstrate, for the first time, that the mammillary bodies, independently of the supramammillary

33 lthough the VTNg, and its projections to the mammillary bodies, is present across species, the size a

34 uch models downplay other projections to the mammillary bodies, leaving them largely ignored.

35 Rats with either selective, neurotoxic mammillary body lesions or discrete mammillothalamic tra

36 c memory impairment caused by lesions of the mammillary bodies, like fornix transection, was exacerba

37 Focusing on the mammillary bodies (MB), we discovered transcriptionally-

38 The mammillary body (MB), a subcortical node of the medial l

39 horseradish peroxidase was injected into the mammillary bodies of five cynomolgus monkeys (Macaca fas

40 The principal mammillary body output pathway, the mammillothalamic tra

41 study not only demonstrates that the primate mammillary bodies receive parallel inputs from the dorsa

42 Further projections to the mammillary body region arose from cells in the anterior

43 ficantly larger percent decreases in BOLD in mammillary bodies, secondary motor cortex, gustatory cor

44 ricted regions of cerebral cortex, thalamus, mammillary bodies, substantia nigra, and pineal glands t

45 phe nucleus, interpeduncular nucleus, medial mammillary body, supramammillary nucleus, posterior nucl

46 nuclei, interpeduncular nuclei, hippocampus, mammillary bodies, thalamus, and caudate nucleus.

47 those brain structures (e.g. hippocampus and mammillary bodies) that are now assumed to cause anterog

48 station, the highest level of mRNA is in the mammillary bodies, the posterior-most part of the hypoth

49 rtance of the hippocampal projections to the mammillary bodies, the present study tested the importan

50 e importance of the other major input to the mammillary bodies, the projections from the ventral tegm

51 rats, the two principal inputs reaching the mammillary bodies: the postcommissural fornix from the h

52 ly highlighted the hippocampal inputs to the mammillary bodies via the postcommissural fornix.

53 directly by overall volume and indirectly by mammillary body volume (which atrophies after fornix dam

54 Mammillary body volume significantly correlated with 13

55 sted for age and fornix volume, P<.0005) and mammillary body volumes (age-adjusted means 0.114 ml vs.

56 e volume of lesions and the proximity to the mammillary bodies were not different between the two gro

57 al nuclei provide major inputs to the rodent mammillary bodies, where they are thought to be importan