ライフサイエンスコーパス: mansoni

1 ity (IC(5)(0): 0.3 muM) on adult Schistosoma mansoni .

2 on two-day old schistosomula of Schistosoma mansoni.

3 in Batf3(-/-) mice infected with Schistosoma mansoni.

4 pression or after infection with Schistosoma mansoni.

5 by the cercarial larvae stage of Schistosoma mansoni.

6 TC, TT (P = 0.03; OR = 11]) infected with S. mansoni.

7 ere infected percutaneously with Schistosoma mansoni.

8 th muscles of the human parasite Schistosoma mansoni.

9 the digenetic trematode parasite Schistosoma mansoni.

10 last-like cells in the trematode Schistosoma mansoni.

11 tions, as revealed by genetic analyses of S. mansoni.

12 ng in response to challenge with Schistosoma mansoni.

13 ff, were naturally infected with Schistosoma mansoni.

14 fibrosis in mice following infection with S mansoni.

15 ely by the eggs of the trematode Schistosoma mansoni.

16 ction with the helminth parasite Schistosoma mansoni.

17 om liver fibrosis following infection with S mansoni.

18 pounds against T. cruzi, L. donovani, and S. mansoni.

19 fter infection with the helminth parasite S. mansoni.

20 tions with the helminth parasite Schistosoma mansoni.

21 e against the parasitic helminth Schistosoma mansoni.

22 iate Th1 response to the parasite Shistosoma mansoni.

23 ed eIF4E from the human parasite Schistosoma mansoni.

24 te host of the human blood fluke Schistosoma mansoni.

25 Ixodes ricinus, and the flatworm Schistosoma mansoni.

26 (SULT) in the parasitic flatworm Schistosoma mansoni.

27 granulomas in mice infected with Schistosoma mansoni.

28 various developmental stages of Schistosoma mansoni.

29 this species as a suitable snail host for S. mansoni.

30 blood-feeding trematode parasite Schistosoma mansoni.

31 mission of the human blood fluke Schistosoma mansoni.

32 45%), Mansonella perstans (21%), Schistosoma mansoni (18%), and Plasmodium falciparum (11%).

33 r LDL-cholesterol levels were observed in S. mansoni (2.37 vs 2.80 mmol/L, -0.25 [-0.49,-0.02] p=0.04

34 DL-c levels were associated with Schistosoma mansoni (2.37 vs 2.80 mmol/L; -0.25 [95% CI, -.49 to -.0

35 f information obtained through RNA-Seq in S. mansoni (88 libraries).

36 dontis, a filarial nematode, and Schistosoma mansoni, a blood fluke.

37 bits male-specific expression in Schistosoma mansoni, a derived, dioecious flatworm.

38 The micro-exon genes (MEG) of Schistosoma mansoni, a parasite responsible for the second most wide

39 g novel stem cell populations of Schistosoma mansoni, a prevalent parasite that infects hundreds of m

40 ction with the helminth parasite Schistosoma mansoni, Ab regulates hepatic inflammation, and local pr

41 aused by the parasitic trematode Schistosoma mansoni after deposition of eggs in the liver and intest

42 Th2-mediated immune response to Schistosoma mansoni Ags.

43 n (VSVG) for the transduction of Schistosoma mansoni and delivery of reporter transgenes into schisto

44 results show that the genomes of Schistosoma mansoni and Drosophila melanogaster lack detectable DNA

45 arasitological examination for diagnosing S. mansoni and flow cytometry for lymphocyte (CD3, CD4, CD8

46 major liver-residing pathogens, Schistosoma mansoni and hepatitis B virus (HBV), is barely understoo

47 n schoolchildren coinfected with Schistosoma mansoni and hookworm.

48 f schoolchildren coinfected with Schistosoma mansoni and hookworm.

49 ected with the helminth parasite Schistosoma mansoni and observed an upregulation of CD14 expression

50 domain from Sm-TSP-2, a TSP from Schistosoma mansoni and one of the better prospects for the developm

51 hey could be applied for the detection of S. mansoni and other parasites in settings without reliable

52 After adjusting for Schistosoma mansoni and Plasmodium infection, we estimated a signifi

53 se-causing agents, Schistosoma japonicum, S. mansoni and S. haematobium, are blood flukes that have c

54 of oxamniquine derivatives that kill both S. mansoni and S. haematobium, the two species responsible

55 scence modeling reveals the speciation of S. mansoni and S. rodhaini as 107.5-147.6KYA, a period whic

56 fluence both the distribution of Schistosoma mansoni and Schistosoma haematobium and the incidence of

57 Collectively, S. mansoni and several other schistosomes are responsible f

58 Schistosoma mansoni (and rarely Schistosoma haematobium) intestinal

59 luble egg Ag (SEA) obtained from Schistosoma mansoni, and by RA.

60 vatives was tested against adult Schistosoma mansoni, and values in the micromolar range (26-68 muM)

61 The differences between Schistosoma mansoni- and Schistosoma japonicum-induced hepatic granu

62 anded RNA interference (RNAi) in Schistosoma mansoni, appraises delivery systems for transgenesis and

63 trate that somatic stem cells in Schistosoma mansoni are biased towards generating a population of ce

64 Hepatitis C virus (HCV) and Schistosoma mansoni are major causes of chronic liver disease (CLD)

65 flukes Schistosoma japonicum and Schistosoma mansoni are the first major human platyhelminth pathogen

66 arasitic helminth worms, such as Schistosoma mansoni, are endemic in regions with a high prevalence o

67 with helminth parasites, such as Schistosoma mansoni, are often chronic and characterized by the deve

68 by targeting omega-1 (omega1) of Schistosoma mansoni as proof of principle.

69 These results indicate that chronic S. mansoni attenuates the severity of P. knowlesi coinfecti

70 rst application of population genomics to S. mansoni based on high-coverage resequencing data from 10

71 ta subunit of the human parasite Schistosoma mansoni (beta(Sm)), a motif that does not occur in other

72 va during natural infection with Schistosoma mansoni, but the role of TGF-beta is less clear.

73 s can be made resistant to infection with S. mansoni by first inducing hemocyte proliferation with Bg

74 infected with juvenile and adult Schistosoma mansoni by incorporating a weak base functional group in

75 Sm-p80, the large subunit of Schistosoma mansoni calpain, is a leading antigen candidate for a sc

76 We sought to determine whether Schistosoma mansoni causes experimental PH associated with pulmonary

77 tion with the parasitic helminth Schistosoma mansoni causes significant liver fibrosis and extracellu

78 Schistosoma mansoni cercariae display specific behavioral responses

79 y/secretory products released by Schistosoma mansoni cercariae rapidly produce IL-10 as a result of M

80 f mice to repeated doses (4x) of Schistosoma mansoni cercariae, compared to a single dose (1x), resul

81 6J mice were percutaneously infected with S. mansoni cercariae, followed by i.v. injection of eggs.

82 p (n = 3) were infected with 500 Schistosoma mansoni cercariae.

83 ce IL-10 in vivo early postinfection with S. mansoni cercariae.

84 Eight rhesus macaques were exposed to S. mansoni cercariae.

85 , TSLPR(-/-) mice were also infected with S. mansoni cercariae.

86 el of Mtb infection, we demonstrated that S. mansoni coinfection or immunization with S. mansoni egg

87 e whether children infected with Schistosoma mansoni develop protection-related immune responses afte

88 mice infected with the helminth Schistosoma mansoni develop severe CD4 T cell-mediated hepatic granu

89 ough a recent treatment trial in Schistosoma mansoni did not detect this association.

90 In contrast to other helminth infections, S. mansoni did not elicit a Foxp3(+) Treg cell response dur

91 etect DNA traces of the parasite Schistosoma mansoni directly in the aquatic environment, where the n

92 y product of eggs from the parasitic worm S. mansoni, efficiently triggers basophils to release the i

93 n-derived Ag (soluble extract of Schistosoma mansoni egg [SEA]) that induces type 2 immunity.

94 response to the helminth soluble Schistosoma mansoni egg Ag, which conditions DCs to induce Th2 respo

95 at rabbit IgG antibodies against Schistosoma mansoni egg antigens (SmSEA) cross-react with allergens

96 three invertebrate allergens reacted with S. mansoni egg antigens and variably with schistosome cerca

97 mansoni coinfection or immunization with S. mansoni egg antigens can reversibly impair Mtb-specific

98 We collected up to 12 S. mansoni egg counts from 414 children aged 6-12 years bef

99 significantly associated with increasing S. mansoni egg IgG1 titers and RF titers of >or=80 (adjuste

100 irmed by in vivo studies using a Schistosoma mansoni egg-challenged mouse model, a well-studied syste

101 ith recombinant helminth-derived Schistosoma mansoni egg-derived omega1 (omega1), a potent inducer of

102 rticipates in the development of Schistosoma mansoni egg-induced CD4(+) Th2 responses, it plays only

103 basophils for protective immunity against S. mansoni egg-induced pathology during the patent stage of

104 The IL-4-inducing principle from Schistosoma mansoni eggs (IPSE/alpha-1), the major secretory product

105 CFA, aluminum salts (Alum), and Schistosoma mansoni eggs (Sm Egg).

106 ribonuclease (RNase) secreted by Schistosoma mansoni eggs and abundantly present in soluble egg antig

107 t of this model, we find that only mature S. mansoni eggs are shed into the feces of mice and humans.

108 Schistosoma mansoni eggs contain factors that trigger potent Th2 res

109 and subsequently challenged with Schistosoma mansoni eggs developed pulmonary hypertension associated

110 gainst Trichuris muris worms and Schistosoma mansoni eggs do not develop in mice with IRF-4-deficient

111 The immune response to S. mansoni eggs is characterized by increased Th2 cells, eo

112 S. mansoni eggs lodge in the hepatic sinusoids of infected

113 f the variation in the number of Schistosoma mansoni eggs per gram of fecal matter.

114 sitized and intravenously challenged with S. mansoni eggs to induce experimental PH.

115 tes, in the presence of S. haematobium or S. mansoni eggs were investigated.

116 ens (SEA; a soluble extract from Schistosoma mansoni eggs) inhibit the activation of DCs in response

117 Following exposure to Schistosoma mansoni eggs, a model of Th2 cytokine-mediated disease t

118 re M-IPSE), a major protein secreted from S. mansoni eggs, can infiltrate host cells.

119 uced in the liver and lung in response to S. mansoni eggs, confirmed by both DNA microarray and real-

120 sure to bleomycin (BLM), but not Schistosoma mansoni eggs, IL-17A produced by CD4(+) and gammadelta(+

121 People in regions of Schistosoma mansoni endemicity slowly acquire immunity, but why this

122 To this end, the targeting of Schistosoma mansoni epigenetic enzymes, which regulate the parasitic

123 Children (8-10 years old) were tested for S. mansoni every 4 months and treated with praziquantel whe

124 ident tissue macrophages, which encounter S. mansoni excretory/secretory products during infection, a

125 ning differential gene expression between S. mansoni-exposed schistosome-resistant and susceptible sn

126 Schistosoma mansoni exposure results in prototypical type-2 inflamma

127 examining children infected with Schistosoma mansoni from 6 schools in Uganda that had previously rec

128 Up to 20 different S. mansoni fucosyltransferase (SmFucT) genes can be found i

129 evidence for cytosine methylation in the S. mansoni genome.

130 d structural annotation of lncRNAs in the S. mansoni genome.

131 improve the environmental surveillance of S. mansoni Given the proper method and guideline developmen

132 l host for the human blood fluke Schistosoma mansoni Granulins are growth factors that drive prolifer

133 oup-I: patients with chronic schistosomiasis mansoni, group-II: HCV patients without cirrhosis, group

134 Vitamin A-deficient mice infected with S. mansoni had disrupted liver granuloma architecture and i

135 o systematically explore this hypothesis, S. mansoni hemozoin was purified and added to in vitro bone

136 VSVG-pseudotyped MLV for transgenesis of S. mansoni, herald a tractable pathway forward toward germl

137 nity to the intestinal trematode Schistosoma mansoni Here, we report that abrogation of IL-4 receptor

138 prepared as potent inhibitors of Schistosoma mansoni histone deacetylase 8 (smHDAC8).

139 lecules that cross-react with rabbit anti-S. mansoni IgG antibodies in extracts of the house dust mit

140 xplore selected protein-encoding genes of S. mansoni implicated in the disease process.

141 ome IgE are associated with resistance to S. mansoni in children, and these immunological parameters

142 s a report of the successful detection of S. mansoni in freshwater samples by using aquatic eDNA.

143 ansmission of the human parasite Schistosoma mansoni in Kenya.

144 ina, are the major intermediate hosts for S. mansoni in sub-Saharan Africa, where more than 90% of gl

145 which have antagonistic interactions with S. mansoni in their shared Biomphalaria vector snails.

146 dysmic ratios of 1 and 2 against Schistosoma mansoni in vitro.

147 chool-age children in an area endemic for S. mansoni in western Kenya.

148 lyze the reproductive biology of Schistosoma mansoni in-depth we isolated complete ovaries and testes

149 volved in the human blood fluke (Schistosoma mansoni) in Brazil in the 1970s.

150 tes activation markers in chronic HCV and S. mansoni induced CLD that may have a role in disease prog

151 -4Ralpha signaling on B cells exacerbated S. mansoni-induced mortality and pathology in BALB/c mice,

152 Experimental S mansoni-induced pulmonary vascular disease relies on can

153 ammals by the parasitic helminth Schistosoma mansoni induces antibodies to glycan antigens in worms a

154 S. mansoni-infected Arg I-deficient bone marrow chimeras de

155 S. mansoni-infected Cd14(-/-) mice also presented with smal

156 say, and immunohistochemistry in liver of S. mansoni-infected hamsters, Huh7 cells, primary hepatocyt

157 blood, spleen, and hepatic granulomas of S. mansoni-infected high-pathology CBA mice and low-patholo

158 d eosinophils from the livers of Schistosoma mansoni-infected mice.

159 n was analyzed in offspring from Schistosoma mansoni-infected mothers mated during the TH1, TH2, or r

160 ing to the development of liver cancer in S. mansoni-infected patients.

161 that removal of C. sukari would increase S. mansoni-infected snails by two-fold.

162 Previously, in a group of S. mansoni-infected Ugandan males, we showed that IgE respo

163 e elimination target was reached only for S. mansoni infection (in Burkina Faso, Burundi, and Rwanda

164 uracy of 3 different diagnostic tests for S. mansoni infection (stool microscopy [samples prepared by

165 ed by Schistosoma haematobium or Schistosoma mansoni infection by quantifying gene expression in the

166 t inflammation is controlled during acute S. mansoni infection by two distinct, organ-specific mechan

167 nfection and women with and those without S. mansoni infection from separate villages in rural Tanzan

168 Participants with moderate to heavy S. mansoni infection had lower triglycerides, LDL-cholester

169 S. mansoni infection had no consistent association with chi

170 Children with > or =2 repeat diagnoses of S. mansoni infection had significantly increased levels of

171 There was no evidence for S. mansoni infection having a similar suppressive effect on

172 ponses during the first 3 weeks of murine S. mansoni infection in C57BL/6 mice, a time when larval pa

173 nsitive screening option for asymptomatic S. mansoni infection in Eritrean refugees, compared with st

174 gical characteristics, caused by Schistosoma mansoni infection in IL-4 receptor alpha-deficient mice

175 by either acute or chronic HBV infection, S mansoni infection influenced HBV infection outcomes in a

176 Intensive MDA reduced Schistosoma mansoni infection intensity: the prevalence from Kato Ka

177 s at least 400 eggs per gram of feces for S. mansoni infection or as more than 50 eggs per 10 ml of u

178 pot, not associated with study arm, where S. mansoni infection prevalence and intensity did not decre

179 Instead, S. mansoni infection resulted in accumulation of high argin

180 over three consecutive days for Schistosoma mansoni infection simultaneously by age group at baselin

181 We used a murine model of Schistosoma mansoni infection to further investigate whether the per

182 S. mansoni infection was associated with lower total choles

183 ubstantial heritability for the burden of S. mansoni infection was confirmed in these Brazilian famil

184 to -.07]; P = .01) and moderate to heavy S. mansoni infection with lower triglycerides, LDL-c, and d

185 otal of 81% of baboons exposed to chronic S. mansoni infection with or without praziquantel treatment

186 rch as well as CCA for mapping surveys of S. mansoni infection, although additional diagnostic tools

187 During the patent phase of Schistosoma mansoni infection, Foxp3(+) Treg cells are activated and

188 paired Th2 cell responses during Schistosoma mansoni infection, Schistosoma egg antigen (SEA) immuniz

189 obium infection as compared to those with S. mansoni infection, which may influence HIV acquisition a

190 r73), and AP-1/DNA-binding in response to S. mansoni infection.

191 activation of c-Jun and STAT3 by Schistosoma mansoni infection.

192 then studied in mice harboring a chronic S. mansoni infection.

193 single-day double KK for the diagnosis of S. mansoni infection.

194 ty and mortality of the TKO mice following S mansoni infection.

195 children with > or =2 repeat diagnoses of S. mansoni infection.

196 ages between 11 women with and 29 without S. mansoni infection.

197 igrants from endemic regions for Schistosoma mansoni infection.

198 Traditionally, human Schistosoma mansoni infections have been detected using stool micros

199 able tools to detect ongoing and previous S. mansoni infections, including in endemic regions where t

200 As with Schistosoma mansoni infections, the pathology of urogenital schistos

201 itive new serological markers of Schistosoma mansoni infections, we have compiled a recombinant prote

202 concomitant chronic HCV and schistosomiasis mansoni infections.

203 Despite reductions in S. mansoni intensity and hookworm prevalence, intensive MDA

204 The larvae of Schistosoma mansoni invade their mammalian host by utilizing a serin

205 The intravascular trematode Schistosoma mansoni is a causative agent of schistosomiasis, a disea

206 Schistosoma mansoni is a parasitic fluke that infects millions of pe

207 We show S. mansoni is impacted by cattle and wild vertebrates becau

208 The trematode Schistosoma mansoni is one of the etiological agents of schistosomia

209 Schistosoma mansoni is responsible for the neglected tropical diseas

210 hree main human schistosome species, only S. mansoni is sensitive to oxamniquine therapy despite the

211 Further development of S. mansoni is subsequently prevented by C. sukari's presenc

212 Schistosoma mansoni is the causative agent of intestinal schistosomi

213 The blood fluke Schistosoma mansoni is the causative agent of the intestinal form of

214 prominent role in regulating immunity to S. mansoni larvae and that the character of the initial imm

215 develop in Biomphalaria pfeifferi unless S. mansoni larvae are present in the same snail.

216 Induction of inflammation by S mansoni, liver fibrosis, and mortality increased greatly

217 In addition, we find strong evidence that S. mansoni migrated to the New World with the 16-19th Centu

218 al regions that are commonly the sites of S. mansoni miracidium penetration.

219 that the transition from the free-living S. mansoni miracidium to parasitic mother sporocyst depends

220 ctivity in the juvenile (WBR: 18.9-43.1%) S. mansoni mouse model.

221 ine (DA) are reduced during the course of S. mansoni multiplication and transformation within the sna

222 S. mansoni, N. americanus, S. stercoralis, T. trichiura, M.

223 eening approach to identify inhibitors of S. mansoni NAD(+) catabolizing enzyme (SmNACE), a receptor

224 re very similar to those of its congener, S. mansoni, offering the prospect of designing chemicals th

225 sed on this evidence, we hypothesize that S. mansoni omega-1 acts by limiting the interaction of DCs

226 elpful tool for understanding the role of S. mansoni on malaria parasitemia and antimalarial immune r

227 s an intermediate snail host for Schistosoma mansoni, one of the important schistosomes infecting man

228 of B. glabrata to infection with Schistosoma mansoni or Echinostoma paraensei, and functions as an op

229 ther hepatosplenomegaly or infection with S. mansoni or P. falciparum.

230 ccording to schistosome species (Schistosoma mansoni or S. haematobium), number of treatment rounds,

231 th P. falciparum that are coinfected with S. mansoni or S. haematobium.

232 omiasis due to the chronic infection with S. mansoni or S. japonicum associated with liver periportal

233 ts from a chronic infection with Schistosoma mansoni or Schistosoma japonicum.

234 n DCs responding to the helminth Schistosoma mansoni or the allergen house dust mite (HDM).

235 Our results indicate that S. mansoni originated in East Africa and experienced a decl

236 determined the responsiveness to Schistosoma mansoni over a 2-year period, when reinfection was restr

237 ium-Plasmodium falciparum versus Schistosoma mansoni-P. falciparum) has produced conflicting results.

238 of the initial immune response invoked by S. mansoni parasites contrasts with the responses to other

239 Schistosoma mansoni parasites of both sexes recovered from infected

240 was significantly decreased in HCV and/or S. mansoni patients.

241 ress SMDR2, a Pgp homologue from Schistosoma mansoni (Platyhelminthes), in Chinese hamster ovary (CHO

242 Differences were not observed in the S. mansoni-prevalent villages between 11 women with and 29

243 n example, the human blood fluke Schistosoma mansoni produces highly fucosylated glycan structures on

244 B. glabrata/S. mansoni provides a useful model system for investigating

245 igens were associated with lower Schistosoma mansoni reinfection intensity, while no associations bet

246 ombinant antigen vaccine against Schistosoma mansoni remains elusive, in part because the parasite de

247 th high prevalences of S. haematobium and S. mansoni, respectively.

248 he case of the human blood fluke Schistosoma mansoni, responsible for intestinal bilharzia, the pheno

249 tion with the trematode parasite Schistosoma mansoni results in a distinct heterogeneity of disease s

250 tion with the trematode helminth Schistosoma mansoni results in a parasite egg-induced, CD4 T-cell-me

251 In the mouse, infection with Schistosoma mansoni results in an egg-producing infection and associ

252 he eggs of the helminth parasite Schistosoma mansoni (schistosome egg Ag (SEA)) leads to the inductio

253 ctures of the GTPase domain of a Schistosoma mansoni septin (SmSEPT10), one bound to GDP and the othe

254 SmSrpQ, one of two S. mansoni serpins found in larval secretions, is only expr

255 vitro testing of synthetic substrates of S. mansoni sirtuin 2 (SmSirt2) and kinetic experiments on a

256 After challenge with Schistosoma mansoni (Sm) eggs, Retnla(-/-) mice developed exacerbate

257 Schistosoma mansoni (Sm) infection has been linked with an increased

258 specificity among Ugandan rural Schistosoma mansoni (Sm)-endemic communities, proximate urban commun

259 nology among Ugandans from rural Schistosoma mansoni (Sm)-endemic islands (n = 209), and from proxima

260 ctional surveys in Ugandan rural Schistosoma mansoni (Sm)-endemic islands, and in nearby mainland urb

261 (legumains) from the bloodfluke, Schistosoma mansoni (SmAE), and the hard tick, Ixodes ricinus (IrAE)

262 mine kinase from the blood fluke Schistosoma mansoni (SmTK) belongs to the phosphagen kinase (PK) fam

263 regation mediated by HIVgp120 or Schistosoma mansoni soluble egg Ag accelerated maximal CXCR4 express

264 ransmission based on antibody response to S. mansoni soluble egg antigen (SEA) with stool-based measu

265 challenged intratracheally with Schistosoma mansoni soluble egg antigens (SEAs) to induce robust Th2

266 tive chemotherapy strategies for Schistosoma mansoni, spatial scan statistics were used to find infec

267 RF-4-deficient DCs also effectively prime S. mansoni-specific Th2 responses.

268 rom the intestine, is sufficient to prime S. mansoni-specific Th2 responses.

269 ly binds to hemocytes and the tegument of S. mansoni sporocysts in a sugar-inhibitable fashion sugges

270 fied Schistosoma haematobium and Schistosoma mansoni surveys done in, respectively, 9318 and 9140 uni

271 as been hampered by the lack of validated S. mansoni targets.

272 nd SmCalp2) are expressed in the Schistosoma mansoni tegument.

273 uding the allergen-like proteins Schistosoma mansoni tegumental-allergen-like 1 protein (SmTAL1), SmT

274 The digenetic trematode Schistosoma mansoni that causes the form of schistosomiasis found in

275 erference (RNAi) screen in adult Schistosoma mansoni that examined the function of 2216 genes.

276 er directional and balancing selection in S. mansoni that may facilitate adaptation to the human host

277 We have targeted a protein of Schistosoma mansoni that plays an important role in the surface memb

278 sessed the lncRNAs complement of Schistosoma mansoni, the blood fluke that causes schistosomiasis, ra

279 tion with the trematode helminth Schistosoma mansoni, the severity of CD4 T cell-mediated hepatic gra

280 their cattle enable C. sukari to exploit S. mansoni, thereby limiting transmission of this human pat

281 -based virtual screening (VS) of Schistosoma mansoni thioredoxin glutathione reductase (SmTGR) inhibi

282 infected with the human parasite Schistosoma mansoni through mechanisms that are currently unclear.

283 The site of S. mansoni transmission was molluscicided throughout.

284 illages with different levels of Schistosoma mansoni transmission.

285 Using data from a study of Schistosoma mansoni (trematode) infections in Biomphalaria glabrata

286 observed between infection with Schistosoma mansoni, Trichuris, or Strongyloides species and P. falc

287 hammerhead ribozyme derived from Schistosoma mansoni under conditions that permit detailed observatio

288 lar to other metazoan pathogens, Schistosoma mansoni undergoes transcriptional and developmental regu

289 In vitro studies with adult Schistosoma mansoni using several substrates suggest that the excret

290 nails, the intermediate host for Schistosoma mansoni, using Illumina MiSeq sequencing of the bacteria

291 Schistosoma mansoni was associated with lower total cholesterol (4.2

292 of the hammerhead ribozyme from Schistosoma mansoni was monitored with double electron-electron reso

293 S. mansoni was positively associated with Dermatophagoides-

294 thesis of highly fucosylated N-glycans by S. mansoni, we examined the ability of ten selected SmFucTs

295 vely recent empirical studies on Schistosoma mansoni, we use a mathematical model to investigate the

296 died extensively for the related organism S. mansoni, which is more amenable to laboratory studies.

297 in sera from mice infected with Schistosoma mansoni, which revealed the presence of both IgM and IgG

298 Emergence of Schistosoma mansoni with reduced sensitivity to praziquantel, the dr

299 ections were prepared from ~400 mum thick S. mansoni worm couples, comparing several microembedding a

300 assay false positivity and the levels of S. mansoni worm IgG1 and IgG2 and Plasmodium falciparum IgG