ライフサイエンスコーパス: marginal zone

1 ocalization of virus and pDCs to the splenic marginal zone.

2 vesicle to their destination in the ciliary marginal zone.

3 anotubes in the spleen were found within the marginal zone.

4 o the perimeter of follicles adjacent to the marginal zone.

5 +) Mphis and other cells in the red pulp and marginal zone.

6 ophils that localize in the red pulp and the marginal zone.

7 pulp of SCD mice without distinct B, T, and marginal zones.

8 [1.63-6.62]), follicular (3.01 [1.95-4.63]), marginal zone (1.90 [1.13-3.18]), and T-cell (2.11 [1.17

9 aly, autoantibody production, frequencies of marginal zone and B1 B cells, and renal pathology compar

10 cell development, which results in increased marginal zone and decreased follicular B cell numbers.

11 nal zone B cells migrate continually between marginal zone and follicles and establishes the marginal

12 Marginal zone B cells shuttle between the marginal zone and follicles with at least one-fifth of t

13 differentiation of transitional B cells into marginal zone and follicular B cells.

14 k secreted and membrane-bound Ab, yet harbor marginal zone and follicular B cells.

15 p a two-photon microscopy procedure to study marginal zone and follicular B-cell movement in the live

16 lp follicles, markedly increased size of the marginal zone and germinal centers, and increased expres

17 finity self-reactive specificities in murine marginal zone and human naive B cells.

18 ells and substantially rescues maturation of marginal zone and Iglambda(+) B cells, but not Igkappa(+

19 B cell immunophenotype, with a deficiency in marginal zone and memory B cells and an increased freque

20 Zeb1 is also required for marginal zone and peritoneal B-1 B-cell development as w

21 ited abnormal elevated IDO expression in the marginal zone and red pulp and inhibition of IDO markedl

22 KLRG1(-) and KLRG1(+) CD8(+) T cells in the marginal zone and red pulp, which ceases prior to the fi

23 ed spleen with marked disorganization of the marginal zone and red pulp.

24 We find that iNKT cells consolidate in the marginal zone and require dendritic cells lining the spl

25 ls were located in the region of the splenic marginal zone and were not detected in blood or other se

26 ecursor cell subsets that become infected in marginal zones and then migrate into GCs as fully mature

27 nguishes them from follicular, transitional, marginal zone, and B1 cells.

28 on impaired generation of mature follicular, marginal zone, and B1a B lymphocytes.

29 so possessed a reduced proliferative ciliary marginal zone, and decreased and disorganized Muller gli

30 lusters or heterotopias were detected in the marginal zone, and disorganization of cortical cells ind

31 m V(H)B1-8 knock-in mice, we evaluated B-1b, marginal zone, and follicular B cell responses to the TI

32 lacked lymph nodes, Peyer's patches, splenic marginal zones, and follicular dendritic cells and faile

33 o the apical membrane, that is enriched at a marginal zone apical to tight junctions, and that drives

34 ginal zone and follicles and establishes the marginal zone as a site of S1PR1-dependent B-cell exit f

35 Marginal zone B (MZ B) cells can rapidly produce antibod

36 al zone precursors (MZPs) differentiate into marginal zone B (MZB) cells under a signaling pathway in

37 Splenic marginal zone B (MZB) cells, positioned at the interface

38 populations: B1, B2 (follicular B, FOB), and marginal zone B (MZB) cells.

39 etermine whether transitional B cells become marginal zone B (MZB) or follicular B (FoB) cells in the

40 A recent study showed that marginal zone B cell and B1 B cell populations are drama

41 iously known as INK1117) did not disrupt the marginal zone B cell compartment and did not block T cel

42 of B cells into the follicular mature versus marginal zone B cell compartment.

43 ise bone marrow B lymphopoiesis, but splenic marginal zone B cell development failed, and B cells und

44 TCH2, a gene encoding a protein required for marginal zone B cell development, in 25 of 99 ( approxim

45 tients with chronic lymphocytic leukemia and marginal zone B cell lymphoma also underwent integrin-me

46 , 76% of p53(rev/rev) mice developed splenic marginal zone B cell lymphomas, indicating sensitivity o

47 ressed germinal center formation and reduced marginal zone B cell numbers, similar to a pan-class I i

48 ) mice, IgM and IgG3 Ab responses as well as marginal zone B cell plasma cell numbers and peritoneal

49 raf2 deletion, Traf2DN-tg mice show expanded marginal zone B cell population and have constitutive p1

50 n of IgA plasma cells and also the enigmatic marginal zone B cell subset that is poorly understood in

51 artments and a strong orientation toward the marginal zone B cell subset.

52 etes insulin binders from the follicular and marginal zone B cell subsets.

53 omparable to that seen in old follicular and marginal zone B cell subsets.

54 Marginal zone B cells (MZB) are a mature B cell subset t

55 Marginal zone B cells (MZB) participate in the early imm

56 CD22(-/-) background have restored levels of marginal zone B cells and Ab responses compared with def

57 O also leads to reduction of Ag transport by marginal zone B cells and affects the subsequent immune

58 -cell development, but led to a reduction of marginal zone B cells and an increase in splenic B1 B ce

59 B cell maturation, showed reduced numbers of marginal zone B cells and class-switched cells, and were

60 KLF2 knockout mice have increased numbers of marginal zone B cells and decreased numbers of B1 phenoy

61 , allowing Notch 2-driven differentiation of marginal zone B cells and of Esam(+) dendritic cells.

62 rmore, Lbw2 congenics had greater numbers of marginal zone B cells and reduced expansion of peritonea

63 he number of spontaneous germinal center and marginal zone B cells and the level of autoantibody are

64 at phenotypes in the heart, endothelium, and marginal zone B cells are attributed to haploinsufficien

65 We show that marginal zone B cells are highly motile and exhibit long

66 ollicular B cells are decreased and those of marginal zone B cells are increased in spleens of CD28(-

67 or example, peritoneal B-1 cells and splenic marginal zone B cells exhibited significantly increased

68 Marginal zone B cells expand in the knockout spleen, whe

69 wever, it increased T1 B cells and decreased marginal zone B cells in the spleen.

70 e marrow and profound loss of follicular and marginal zone B cells in the spleen.

71 hereas splenic B-1 cells were unaffected and marginal zone B cells increased.

72 This study shows that marginal zone B cells migrate continually between margin

73 differs dramatically from the follicular and marginal zone B cells repertoires and is defined by dist

74 w that the Ab repertoire of CD21(hi)/CD23(-) marginal zone B cells shows persistent increase in level

75 Marginal zone B cells shuttle between the marginal zone

76 , a population that resembles murine splenic marginal zone B cells that mount T-independent antibody

77 ular repertoire, indicating that the loss of marginal zone B cells was not due to diversion to the fo

78 up-regulated Nod1, including follicular and marginal zone B cells with natural autoreactivity.

79 follicular regulatory cells, an expansion of marginal zone B cells, and early increases in regulatory

80 subsets of B cells, in mature follicular and marginal zone B cells, and in activated B cells, includi

81 yed defects in multiple cell types including marginal zone B cells, B1 B cells, IL-10-producing B cel

82 l(-/-) mice, finding a relative expansion of marginal zone B cells, B1 cells, and plasma cells associ

83 IgM when the cells mature into follicular or marginal zone B cells, but the transacting factors respo

84 d Zfp318 exon 10 abolished IgD expression on marginal zone B cells, decreased IgD on follicular B cel

85 As with mouse marginal zone B cells, human IgM(+)CD27(+) B cells activ

86 e latency mice, i.e., increased frequency of marginal zone B cells, hyperplasia, and hyperglobulinemi

87 served more mature naive B cells, especially marginal zone B cells, in BLyS-treated mice.

88 We found that in addition to marginal zone B cells, mature follicular B cells signifi

89 immune response at various levels, including marginal zone B cells, plasmacytoid dendritic cells and

90 m follicles to the marginal zone, but unlike marginal zone B cells, they fail to undergo integrin-med

91 Transcriptomic analyses of resting marginal zone B cells, which generate plasma cells with

92 racellular ATP and loss of CD21 and CD62L on marginal zone B cells.

93 he spleen and enabled efficient shuttling of marginal zone B cells.

94 llular, albeit with a relative deficiency of marginal zone B cells.

95 9-activated peripheral B cells, and splenic marginal zone B cells.

96 B cells mature into follicular and then into marginal zone B cells.

97 n use the 44.1-idiotype and are dependent on marginal zone B cells.

98 e development of mature B1a, follicular, and marginal zone B cells.

99 1b peritoneal cells, but not in B1a cells or marginal zone B cells.

100 and suggest that the 44.1-Id is derived from marginal zone B cells.

101 eas heterozygous deficiency caused a loss of marginal zone B cells.

102 ost dramatically affected the maintenance of marginal zone B cells.

103 the physiological differentiation of spleen marginal zone B cells.

104 llectively, these findings demonstrate B-1b, marginal zone B, and follicular B subsets significantly

105 is, including increased development of B-1a, marginal zone B, gamma/delta (gammadelta) T cells, and n

106 fibrosis identified a common upregulation of marginal zone B- and B1-cell-specific protein (MZB1), th

107 t frequent lymphoma subtypes were extranodal marginal zone B-cell lymphoma (EMZL) (n = 177, 68%), fol

108 Extranodal marginal zone B-cell lymphoma (EMZL) was the most freque

109 s lacking Ig translocations, such as splenic marginal zone B-cell lymphoma or Waldenstrom macroglobul

110 patient, who had liver granuloma, extranodal marginal zone B-cell lymphoma, and autoimmune neutropeni

111 king phenotypes-gender imbalance and splenic marginal zone B-cell lymphoma-emerged in combination wit

112 , diffuse large B-cell lymphoma, and splenic marginal zone B-cell lymphoma.

113 orbidities associated with primary cutaneous marginal zone B-cell lymphomas (PCMZLs).

114 GCs, showing a modest increase in naive and marginal-zone B cells and a significant decrease in GC B

115 haperone that is preferentially expressed in marginal-zone B cells and is highly upregulated during p

116 iptional hub that determined the identity of marginal-zone B cells by promoting their proper localiza

117 Nkx2-3-deficient mice exhibit the absence of marginal-zone B cells, transgenic mice with expression o

118 or studying the biology and therapy of human marginal-zone B-cell lymphomas.

119 y and the remaining cells fail to situate in marginal zone bridging channels.

120 r B cells also transit from follicles to the marginal zone, but unlike marginal zone B cells, they fa

121 beta, and included genes expressed by normal marginal zone cells and memory B cells.

122 B cell development validate the identity of marginal zone cells and the maturation status of human C

123 However, in contrast to the follicular and marginal zone cells, ABCs displayed significant somatic

124 ressed by progenitors in the circumferential marginal zone (CMZ) and is upregulated by Muller glia in

125 Retinal progenitors in the circumferential marginal zone (CMZ) and Muller glia-derived progenitors

126 rogenitor population residing in the ciliary marginal zone (CMZ) at the retinal peripheral margin.

127 erentiation, and stem cells from the ciliary marginal zone (CMZ) being responsible for late neurogene

128 our and retinal morphology including ciliary marginal zone (CMZ) cell death and decreased photorecept

129 cts stem and progenitor cells in the ciliary marginal zone (CMZ) of the amphibian retina, a well-char

130 The ciliary marginal zone (CMZ) of the zebrafish retina contains a p

131 at the rim of the retina, called the ciliary marginal zone (CMZ).

132 elopment as shown by a dramatically expanded marginal zone compartment and extensive receptor editing

133 enance of cells in the murine follicular and marginal zone compartments is thought to involve differi

134 tion stages and allows the classification of marginal zone-derived (JAM-C-positive) and germinal cent

135 Interestingly the dorso-lateral marginal zone (DLMZ) is only able to promote the express

136 ce with expression of NKX2-3 in B cells show marginal-zone expansion that leads to the development of

137 d require dendritic cells lining the splenic marginal zone for activation following administration of

138 -borne antigens, lymphocyte migration in the marginal zone has not been intravitally visualized due t

139 The splenic marginal zone is a unique microenvironment where residen

140 Follicular B-cell egress via the marginal zone is sphingosine-1-phosphate receptor-1 (S1P

141 al numbers but with abnormal distribution of marginal zone-like and memory B cells.

142 ils; macrophages; natural killer cells (NK); Marginal Zone-like B cells (MZB); gammadelta T cells; an

143 xposed women had more atypical MBC and fewer marginal zone-like MBC, and their levels correlated with

144 argin) and the retreated ice sheet (with the marginal zone located far inland).

145 sion, as were mantle cell lymphoma (0 of 5), marginal zone lymphoma (0 of 6), follicular lymphoma (0

146 or rarely expressed in samples from splenic marginal zone lymphoma (2/20; 10%), CLL (1/26; 4%), mult

147 The most frequent subtype was extranodal marginal zone lymphoma (EMZL) (68.4% [180 of 263]), foll

148 The histologic subtype was extranodal marginal zone lymphoma (EMZL) in 51 patients (81.0%).

149 re most consistent with pulmonary extranodal marginal zone lymphoma (ENMZL; Fig 2 ).

150 Marginal zone lymphoma (MZL) is a heterogeneous B-cell m

151 Marginal zone lymphoma (MZL) is the third most common su

152 uminari et al demonstrate that patients with marginal zone lymphoma (MZL) who experience early progre

153 s macroglobulinemia (n = 2, 11%), extranodal marginal zone lymphoma (n = 2, 11%), plasmablastic lymph

154 , Waldenstrom macroglobulinemia (n = 4), and marginal zone lymphoma (n = 3).

155 10 patients with follicular lymphoma (n=50), marginal zone lymphoma (n=30), and small lymphocytic lym

156 Nodal marginal zone lymphoma (NMZL) is a rare form of indolent

157 Nodal marginal zone lymphoma (NMZL) is a rare, indolent B-cell

158 Nodal marginal zone lymphoma (NMZL) is a small B-cell neoplasm

159 lamydophila psittaci (Cp) and ocular adnexal marginal zone lymphoma (OAMZL) and the efficacy of doxyc

160 5% confidence interval (CI): 2.25, 4.30) and marginal zone lymphoma (OR = 5.80, 95% CI: 3.82, 8.80);

161 across major B-cell NHL subtypes, including marginal zone lymphoma (P-interaction = 0.02) and follic

162 the catalogue of somatic variants in splenic marginal zone lymphoma (SMZL) and to provide a well-anno

163 ell tumor that is distinguished from splenic marginal zone lymphoma (SMZL) by the different pattern o

164 Splenic marginal zone lymphoma (SMZL) is a B cell malignancy of

165 Splenic marginal zone lymphoma (SMZL) is a mature B-cell neoplas

166 Splenic marginal zone lymphoma (SMZL) is a rare B-cell malignanc

167 ned significance (IgM-MGUS), 84 with splenic marginal zone lymphoma (SMZL), and 52 with B-cell chroni

168 Splenic marginal zone lymphoma (SMZL), the most common primary l

169 tiate from HCL-like disorders (e.g., splenic marginal zone lymphoma and HCL variant).

170 ortunity to better understand the biology of marginal zone lymphoma and optimize therapy by using dem

171 Exposure of splenic marginal zone lymphoma cell lines to a demethylating age

172 Splenic marginal zone lymphoma is a rare lymphoma.

173 ercent of the cases reported were extranodal marginal zone lymphoma of mucosa-associated lymphoid tis

174 y accepted prognostic indices for extranodal marginal zone lymphoma of mucosa-associated lymphoid tis

175 stemic treatment of patients with extranodal marginal zone lymphoma of mucosa-associated lymphoid tis

176 bserved across strata of sex for CLL/SLL and marginal zone lymphoma subtypes as well as age for the f

177 Extranodal marginal zone lymphoma was the predominant (76.2%) histo

178 ution, patients with follicular lymphoma and marginal zone lymphoma were given lenalidomide, orally,

179 n profiling identifies 2 subtypes of splenic marginal zone lymphoma with different clinical and genet

180 ALL, B-chronic lymphocytic leukemia, splenic marginal zone lymphoma) is well characterized, there is

181 atified by histology (follicular lymphoma vs marginal zone lymphoma), treatment intent (palliative or

182 leukemias/lymphomas (HCL-variant and splenic marginal zone lymphoma).

183 w-grade non-Hodgkin lymphoma, 27% extranodal marginal zone lymphoma).

184 rent treatments (eg, HCL-variant and splenic marginal zone lymphoma).

185 Of 27 evaluable patients with marginal zone lymphoma, 18 (67%) had a complete response

186 ith HCL, 1 with HCL variant, 91 with splenic marginal zone lymphoma, 29 with Waldenstrom macroglobuli

187 rapy were 71% in follicular lymphoma, 78% in marginal zone lymphoma, 67% in mantle cell lymphoma, and

188 7 with non-mucosa-associated lymphoid tissue marginal zone lymphoma, and 38 with lymphoplasmacytic ly

189 e local control, with follicular lymphoma or marginal zone lymphoma, who had received no previous tre

190 atients with IgM-MGUS progressed to WM or to marginal zone lymphoma.

191 le cell, or other indolent lymphomas such as marginal zone lymphoma.

192 ll lymphoma, 53 follicular lymphoma, and one marginal zone lymphoma.

193 patients), small lymphocytic lymphoma (28), marginal-zone lymphoma (15), and lymphoplasmacytic lymph

194 The most common subtypes were extranodal marginal-zone lymphoma (EMZL) (37% [n = 32]), follicular

195 samples from patients with multiple myeloma, marginal-zone lymphoma, or IgM monoclonal gammopathy of

196 Extranodal marginal zone lymphomas (EMZL) are the most common lymph

197 Marginal zone lymphomas (MZLs) are indolent nonfollicula

198 The histological distribution was 37 marginal zone lymphomas (MZLs), 2 lymphoplasmacytic lymp

199 lobulin (Ig)G4-related disease (IgG4-RD) and marginal zone lymphomas (MZLs).

200 d IGH translocations, have been described in marginal zone lymphomas (MZLs); however, these known gen

201 B) signaling, have been described in splenic marginal zone lymphomas and multiple myeloma.

202 gic and genetic types, and primary cutaneous marginal zone lymphomas recognizing 2 different subtypes

203 16 follicular lymphomas (FLs), 9 extranodal marginal zone lymphomas, and 8 reactive lymph nodes and

204 tumour cells from a subset of patients with marginal-zone lymphomas, but not with other B-cell malig

205 al clinical and biological features of human marginal-zone lymphomas.

206 Marginal zone macrophages (MZM) are strategically locate

207 Marginal zone macrophages (MZMs) act as a barrier to ent

208 The splenic marginal zone macrophages (MZMs) are important for estab

209 s in the spleens of BXD2 lupus mice disrupts marginal zone macrophages (MZMs), which normally clear A

210 Accordingly, marginal zone macrophages and pDC precursors were elimin

211 ) BMDC-dependent protection required CD169(+)marginal zone macrophages and the macrophage-derived che

212 cial effects require microparticle uptake by marginal zone macrophages expressing the scavenger recep

213 1 expression and IL-10 production by splenic marginal zone macrophages leading to Ag-specific T cell

214 ystem, namely Kupffer cells of the liver and marginal zone macrophages of the spleen.

215 Marginal zone macrophages, some DCs, and myeloid cells i

216 irrors aged Bim(-/-) mice, including loss of marginal zone macrophages, splenomegaly, lymphadenopathy

217 it the clearance of apoptotic cells (ACs) by marginal zone macrophages.

218 -1-mediated capture by subcapsular sinus and marginal zone metallophilic macrophages for trans-infect

219 Furthermore, the splenic marginal zone microarchitecture was substantially distur

220 Marginal zone (MZ) and B1 B cells have the capacity to r

221 -3 in B cells resulted in significantly more marginal zone (MZ) and fewer follicular (FO) B cells.

222 ion of apoptotic cell (AC) debris within the marginal zone (MZ) and increased loading of AC Ags on MZ

223 the time of leading process contact with the marginal zone (MZ) and occurs primarily by neurite exten

224 Innate-like splenic marginal zone (MZ) and peritoneal cavity B1 B lymphocyte

225 the majority of NKT cells patrol around the marginal zone (MZ) and red pulp (RP) of the spleen.

226 es from splenic stromal cells located in the marginal zone (MZ) and requires B cells that express lym

227 on of irf4-deleted follicular B cells in the marginal zone (MZ) area.

228 Marginal zone (MZ) B and B1 cells were not rescued, indi

229 elopment, and LFNG and MFNG are required for marginal zone (MZ) B cell development.

230 Kruppel-like factor 3 (KLF3, BKLF) increases marginal zone (MZ) B cell numbers, a phenotype shared wi

231 BCs lacked CD11b or CD11c expression but had marginal zone (MZ) B cell phenotypes and colonized the s

232 Marginal zone (MZ) B cells are an innate-like population

233 Although splenic marginal zone (MZ) B cells are considered to be importan

234 Splenic marginal zone (MZ) B cells are innate-like lymphocytes t

235 portant roles in promoting the generation of marginal zone (MZ) B cells at the expense of follicular

236 Expansion of autoimmune-prone marginal zone (MZ) B cells has been implicated in type 1

237 g peripheral antigen-inexperienced naive and marginal zone (MZ) B cells in acute and chronic HIV-1 in

238 Therefore, the contribution of marginal zone (MZ) B cells in experimental atheroscleros

239 chanism in which follicular translocation of marginal zone (MZ) B cells in the spleens of BXD2 lupus

240 Marginal zone (MZ) B cells reside in the splenic MZ and

241 ase in splenic macrophages, neutrophils, and marginal zone (MZ) B cells that was inhibited by IL-10 s

242 y, type I IFNs increase the translocation of marginal zone (MZ) B cells to the follicular region of t

243 ordinates immunometabolic reconfiguration of marginal zone (MZ) B cells, a pre-activated lymphocyte s

244 s study, we found binding of PTX3 to splenic marginal zone (MZ) B cells, an innate-like subset of ant

245 After 2 h, OVA-TNP was detected on marginal zone (MZ) B cells, and a substantial amount of

246 -independent (TI) antibody production by the marginal zone (MZ) B cells, leaving the contribution of

247 ell-specific Notch2-deficient mice that lack marginal zone (MZ) B cells.

248 decrease in memory B cells, particularly of marginal zone (MZ) B cells.

249 ection, with significant colocalization with marginal zone (MZ) B cells.

250 of anti-gp120 B cells in follicular (FO) and marginal zone (MZ) B-cell compartments of naive WT mice

251 gered by increased plasma cell frequency and marginal zone (MZ) B-cell hyperplasia.

252 Extranodal marginal zone (MZ) B-cell lymphomas of the mucosa-associ

253 splenic B-cell numbers, mostly of the B1 and marginal zone (MZ) B-cell subtypes; 2) enlarged germinal

254 egg chamber development, being lost from the marginal zone (MZ) in stage 9 before abruptly returning

255 The marginal zone (MZ) in the spleen contains specialized su

256 and genetic approaches to delete SIGN-R1(+) marginal zone (MZ) macrophages and reveal their specific

257 us-4 (MuHV-4) enters the spleen by infecting marginal zone (MZ) macrophages, which provided a conduit

258 The marginal zone (MZ) of the spleen contains multiple cell

259 (IL-10)-dependent cellular crosstalk in the marginal zone (MZ) that promoted bacterial infection.

260 ere we identified RORgammat(+) ILCs near the marginal zone (MZ), a splenic compartment that contains

261 llular matrix (ECM) niche in the spleen, the marginal zone (MZ), characterized by the basement membra

262 haracteristic perifollicular rim marking the marginal zone (MZ), which is the interface between the n

263 to DC-inhibitory receptor 2 (DCIR2) found on marginal zone (MZ)-associated CD8alpha(-) DCs in mice le

264 ese tissues revealed respective expansion of marginal zone (MZ)-like B cells (IgD(+)IgM(+)CD43(neg)CD

265 Mutations in NOTCH2, a gene required for marginal-zone (MZ) B cell development, represent the mos

266 d low blood memory B-cells counts (including marginal zone [MZ] B-cell counts).

267 by IgD and CD27 expression: IgD(+)CD27(+) ("marginal zone [MZ]"), IgD(-)CD27(+) ("memory," including

268 ding to the 'modern-scale' ice sheet (with a marginal zone near the present ice-sheet margin) and the

269 ngly, the arrangement of the retinal ciliary marginal zone niche results in a spatially biased random

270 rtant signaling centers including the dorsal marginal zone, notochord and floorplate.

271 IM, localized largely to the splenic marginal zone of naive CD11b-diphtheria toxin (DT) recep

272 -55 degrees C for 10 minutes, simulating the marginal zone of RFA treatment.

273 On OCT, in the marginal zone of the lesions, a disappearance of the int

274 me-capturing macrophages were present in the marginal zone of the spleen and in the subcapsular sinus

275 ghout the spleen at rest, consolidate in the marginal zone of the spleen early after activation, and

276 ells of lymphoid tissues and in cells in the marginal zone of the spleen, while administration of an

277 ternating currents applied to the middle and marginal zones of isolated TM segments evoke motions at

278 mesoderm-inducing signals to the vegetal and marginal zones of the pre-gastrula Xenopus laevis embryo

279 ng being restricted to basal VSNs and at the marginal zones of the VNO: the site of neurogenesis.

280 n ICECAP aerogeophysical data, demarcate the marginal zones of two distinct quasi-stable EAIS configu

281 primarily in GCs and not in the T cell zone, marginal zone, or red pulp areas of the spleen.

282 xpressed B surface markers compatible with a marginal zone origin.

283 osis with an immunophenotype consistent with marginal-zone origin (CBL-MZ) is poorly understood.

284 Costimulatory blockade increased IL-10 in marginal zone precursor (MZP) B cells, but not other sub

285 ly elevated CD69 and CD86 observed in RBP(+) marginal zone precursor B cells in the spleens of BXD2 m

286 ent for NCSTN during the type 2 transitional-marginal zone precursor stage and peritoneal B-1 B cell

287 requency and greater numbers of RBP-reactive marginal zone precursor, transitional T3, and PDL-2(+)CD

288 Mouse splenic marginal zone precursors (MZPs) differentiate into margi

289 entry points for peripheral Ags: the splenic marginal zone, red pulp, and lymph node sinuses.

290 persistence maps anatomically to the splenic marginal zone/red pulp and is defined by prolonged motil

291 ce, with exosomes freely accessing the outer marginal zone rim of SIGN-R1(+) macrophages and F4/80(+)

292 architecture and the function of the splenic marginal zone significantly influence the pathogenesis o

293 0 was significantly increased on FO, but not marginal zone, splenic B cells after SAT development.

294 transitional 2 (T2), follicular mature, and marginal zone subsets identified in mice.

295 ssed in multiple cell types in the preplate, marginal zone, subventricular zone (SVZ), and ventricula

296 Males had a smaller marginal zone than females with a repertoire that was di

297 se that during gastrulation Pnhd acts in the marginal zone to contribute to mesoderm heterogeneity vi

298 ntified 9395 patients with iNHL (follicular, marginal zone, Waldenstrom macroglobulinemia) treated wi

299 plate cells extend a primary dendrite to the marginal zone, whereas all dendrites of P7 subplate cell

300 cortical neurons overmigrate and invade the marginal zone, which are characteristics similar to a ph