ライフサイエンスコーパス: marker gene

1 ich could be fully characterised by a single marker gene.

2 expressed or under which conditions it is a marker gene.

3 ognition receptors and neutrophil activation marker genes.

4 e revealed subpopulations of cells and their marker genes.

5 , there is a need for alternative selectable marker genes.

6 regulating the expression of major decidual marker genes.

7 gen fixation - and could be visualised using marker genes.

8 tant can alter the expression pattern of EAS marker genes.

9 s of the 16S rRNA gene or other phylogenetic marker genes.

10 peration that merely requires a set of known marker genes.

11 riant reconstruction within species-specific marker genes.

12 s and analysed expression of cardiac lineage marker genes.

13 rentiation and mRNA expression of osteoclast marker genes.

14 munity (MTI), inducing the expression of MTI marker genes.

15 eta) signaling pathway and enhances fibrosis marker genes.

16 nd expression of hormone related or meristem marker genes.

17 ltilocus sequence typing and strain-specific marker genes.

18 ogenic HPV18 genome was replaced by CpG free marker genes.

19 e senescence-specific and pathogen-resistant marker genes.

20 damage and elevated expression of DNA damage marker genes.

21 chronous occurrences of preplacodal and otic marker genes.

22 fq transcriptomes, mainly on shade-avoidance marker genes.

23 troscopy and the induction of singlet oxygen marker genes.

24 nes that are co-regulated with characterized marker genes.

25 inar identity using canonical cortical layer marker genes.

26 PPP1R14a and other neuronal differentiation marker genes.

27 esses and have elevated expression of stress marker genes.

28 ncluding previously characterized NF-induced marker genes.

29 howed enhanced expression of leaf senescence marker genes.

30 ribosome binding sites, and yeast selectable marker genes.

31 on in the regulatory regions of pluripotency marker genes.

32 oxidative burst and expression of PTI early marker genes.

33 pe frequencies in tissue using low-abundance marker genes.

34 ith graded, regional expression of 1 or more marker genes.

35 arker genes or by specifying a custom set of marker genes.

36 ered by their lack of universal phylogenetic marker genes.

37 3 (MPK3) and MPK6, and induction of immunity marker genes.

38 to quantify gene expression with a panel of marker genes.

39 llectively define thousands of corresponding marker genes.

40 eq that can identify previously undetectable marker genes.

41 ost organisms by analyzing metatranscriptome marker genes.

42 , by using spatial patterns of expression of marker genes(5,6)-which often do not exist.

43 g frame downstream of the melanoma stem cell marker gene ABCB5.

44 mics and denitrification-specific functional marker gene abundance and expression.

45 RKNs and investigated the expression of PTI marker genes after RKN infection using both quantitative

46 An analysis of cell activity marker genes after stimuli exposure revealed that only t

47 y, mineralization, and up-regulation of bone marker genes (alkaline phosphatase/ALPL, osteopontin/SPP

48 s, which cannot be predicted by phylogenetic marker genes alone.

49 l tools, including highly specific stem cell marker genes along with more rigorous experimental metho

50 The expression level of early osteogenic marker genes, ALP, Runx2, and type I collagen, which pla

51 more, TSH increased expression of osteoblast marker genes ALPL (8.2+/-4.6-fold), RANKL (21+/-5.9-fold

52 Marker gene analysis indicated that both sets of cells d

53 of cyclooxygenase 2 (COX-2) expression as a marker gene and by using gene expression profiling to id

54 earning approach leveraging a combination of marker gene and genome features, we identified 10,295 in

55 upregulated and mediate the VSMC contractile marker gene and PDGFRbeta expression in differentiating

56 eir bacterial and eukaryotic communities via marker gene and shotgun metagenomic sequencing.

57 1-related tissue-specific changes of various marker genes and a significant transcriptomic overlap wi

58 lf18, as measured by the induction of immune marker genes and callose deposition.

59 ancer datasets are correlated with the known marker genes and enriched in cancer-relevant Kyoto Encyc

60 Novel marker genes and gene signatures of mesangial cells, vas

61 y expressed genes, including important tuber marker genes and genes involved in cell growth, transcri

62 l-type annotation using predefined cell type marker genes and identifies driver regulators from diffe

63 xpressed Gtpbp2 can induce ventral-posterior marker genes and localize to cell nuclei in Xenopus anim

64 says of enzyme activities, lipid biomarkers, marker genes and microscopy indicate heterogeneously dis

65 nactive UC, however, was the mobilization of marker genes and proteins for the Epithelial Mesenchymal

66 sed expression of osteoblast differentiation marker genes and reduced expression of genes associated

67 ecreased expression of a subset of adipocyte marker genes and reduced plasma TNFalpha levels.

68 Moreover, induction of stress marker genes and ROS-scavenging enzyme genes under vario

69 the expression of early and late odontoblast marker genes and stage-specific proteases involved in de

70 between the expression levels of these known marker genes and the expression of the coregulated genes

71 umber of genes expressed per cell, number of marker genes and their fold change, and number of single

72 d plants was assessed upon the expression of marker genes and transcriptomic analysis.

73 Marker-gene and metagenomic sequencing have profoundly e

74 anti-Plasmodium falciparum effector genes, a marker gene, and the autonomous gene-drive components ar

75 thods have been developed to infer clusters, marker genes, and cell lineage, none yet integrate these

76 sing oxygen consumption rate, brown-specific marker genes, and miR-30b and 378, which were abrogated

77 Lithotrophic marker genes appear more often in highly acidophilic lin

78 These marker genes are associated with a broad range of photot

79 However, when marker genes are expected to be only up to fold change o

80 Based on our study, we found that when marker genes are expressed at fold change of 4 or more,

81 we find that while many of the early muscle marker genes are reprogrammed, global gene expression an

82 lely responsible for transcript induction of marker genes around 3 to 6 h after chemical inhibition o

83 sphate (Pi) offers an alternative selectable marker gene as demonstrated for tobacco and maize.

84 bstitutes for antibiotic/herbicide-dependent marker genes as well as surprisingly sensitive reporters

85 tified Cox7a1, well-known as brown adipocyte marker gene, as a cold-responsive protein of brown adipo

86 Surprising stochastic expression of marker genes associated with differentiated cell types w

87 Phylogenetic marker gene-based studies have identified many bacteria

88 t of genes, and recapitulating expression of marker genes by BAC transgenesis or knock-in has generat

89 NP delivery, a plasmid carrying a selectable marker gene can be co-delivered with the RNP to enrich f

90 cell clusters by examining the expression of marker genes can be subjective and labor-intensive.

91 We demonstrate that this small panel of marker genes can recover pseudotimes that are consistent

92 dditions to the genome such as insertions of marker gene cassettes or functional elements, without th

93 1-linked regulatory elements proximal to the marker gene CD69.

94 odel of meningioma and validate the high ADC marker genes CDH2 and PTPRZ1 as potential targets for me

95 to induction of the important pro-apoptotic marker gene chop.

96 ession, rescued decreased intestinal barrier marker genes (claudin 3 and claudin 15) expression, and

97 ia abundances, assessed by quantification of marker genes, consistently displayed a four order of mag

98 s and down-regulation of the G2/M cell-cycle marker gene, CYCB1;1 TCP20 and NLP6&7 also support root

99 ents based on quantitative PCR of functional marker genes decreased but were still detectable after 4

100 article, we design the feature selection and marker gene detection as a multi-view multi-objective cl

101 silico method, for identifying subpopulation marker genes directly from the original mixed gene expre

102 t assigned cell-type identity based on known marker genes, distinguishing five major groups: neural p

103 the expression of osteoclastogenesis-related marker genes during osteoclastogenesis, and prevented os

104 Marker genes enable measurement of transduction and allo

105 on of the PATHOGENESIS-RELATED GENE1 defense marker gene, enhanced reactive oxygen species (ROS) burs

106 pic defense response, including pathogenesis marker gene expression and cell death, increase in older

107 naling is required for the maintenance of DP marker gene expression and feather regeneration, excessi

108 Th17 differentiation by digoxin lowered Th17 marker gene expression and IL-17 production and strongly

109 ated with significant reduction in adipocyte marker gene expression and subcutaneous lipoatrophy.

110 Positive correlation of marker gene expression between both cell lines and resem

111 activation and PHI1, WRKY33, and FRK1 immune marker gene expression but have reduced reactive oxygen

112 genously applied brassinolide and altered BR marker gene expression demonstrate that zmbri1-RNAi tran

113 d BEC and hepatocyte proliferation and liver marker gene expression during liver regeneration.

114 ryonic head morphogenesis, and developmental marker gene expression in combinatorial ey and toy knock

115 vitro confirmed that miRNAs are required for marker gene expression in mature brown adipocytes.

116 the observed enrichment of lineage-specific marker gene expression in Nanog-negative cells is associ

117 Selected marker gene expression indicated stage one (0-3 d.p.i) a

118 We found that all marker gene expression levels were significantly elevate

119 Quantification of JA and SA, marker gene expression, and cell death confirmed that in

120 sistance to virulent pathogens, high defence marker gene expression, and salicylic acid accumulation.

121 Current methods enable targeting based on marker gene expression, development, anatomical projecti

122 pora lutea, as evident by the lack of luteal marker gene expression, marked reduction of vascularizat

123 s with a deficit in dermomyotome and myotome marker gene expression, suggesting that Ripply1 promotes

124 inducible expression of groEL activated PTI marker gene expression.

125 h poor clinical outcome and the loss of LSEC marker gene expression.

126 , without altering adipocyte differentiation marker gene expression.

127 itro bone formation and decreased osteogenic marker gene expression; promoter methylation analysis by

128 f which is characterized by specific surface markers, gene-expression patterns, and distinct function

129 ses, notably delayed upregulation of the PTI marker gene FLG22-INDUCED RECEPTOR-LIKE KINASE1, reduced

130 CAM-marker genes followed the opposite pattern.

131 ings greatly expand the diversity of the key marker gene for anaerobic alkane cycling and outline the

132 Recent discoveries have shown that the marker gene for anaerobic methane cycling (mcrA) is more

133 transcriptional control and a low-expression marker gene for selection.

134 KEY MESSAGE: A new selectable marker gene for stable transformation of the plastid gen

135 A new selectable marker gene for stable transformation of the plastid gen

136 nary relationships between amoA, a conserved marker gene for Thaumarchaeota, and soil characteristics

137 lt and larval zebrafish, thereby identifying marker genes for >30 mature types and several developmen

138 encing analyses revealed the deregulation of marker genes for basal and secretory cells upon CS expos

139 ll types and define novel cell-type-specific marker genes for both cultivars.

140 P-overlying cells displayed the induction of marker genes for cell types undergoing developmental cel

141 Furthermore, only some of the proposed marker genes for cortical Sst neurons were expressed in

142 ll major retinal cell types are observed and marker genes for each cell type are identified.

143 rence cell type-specific gene signatures, or marker genes for each cell type, which may not be availa

144 critical problem of the absence of validated marker genes for many (including novel) subpopulations,

145 The development of new marker genes for plastid transformation is of crucial im

146 xpression of posterior Hox genes and that of marker genes for presomitic mesoderm and the chordoneura

147 In agreement, marker genes for SnRK1 activity are upregulated in sr45-

148 induction of DA1 and BB expression, several marker genes for the transition from proliferation to ex

149 curately captured differential expression of marker genes for type II (e.g. the Tas1r genes, Plcb2, T

150 iral genomes and study viral diversity using marker genes from free viruses.

151 n and identifying relevant and non-redundant marker genes from high dimensional gene expression data

152 eria, bacteria, and antimicrobial resistance marker genes from lower respiratory tract specimens (spu

153 ylinositol 3-phosphate] and up-regulation of marker genes from the synthesis phase of the cell cycle

154 discoverable from the perspective of genetic markers, genes, genome regions or phenotypes, via graphi

155 The expression of the trichome development marker genes GLABRA2 (GL2) and Ethylene Receptor2 (ETR2)

156 Reporters for trichome developmental marker genes GLABRA2 (GL2) and Ethylene Receptor2 (ETR2)

157 uilt with eight of these quiescent stem cell marker genes (GSE70696_QNPbyTAP) was sufficient to disti

158 ant enrichment of rodent quiescent stem cell marker genes (GSE70696_QNPbyTAP).

159 Combinations of cell-type marker genes had predictive power for neuronal responses

160 Expression patterns of specific marker genes have been used to characterize some limited

161 n of downstream target genes for the hypoxia marker gene, hypoxia-inducible factor 1alpha (HIF1A).

162 udotemporal ordering, lineage inference, and marker gene identification are inferred via a structured

163 ither relied on the presence of a selectable marker gene in the insertion cassette or occurred at low

164 ngo system exhibit increased expression of a marker gene in the presence of dopamine and blue-light e

165 and reduced the expression level of the HCC marker gene in the Tak1-deficient liver.

166 Using 23 promoter fragments of six UPR marker genes in a high-throughput enhanced yeast one-hyb

167 xpression of previously identified NP and NC marker genes in adult human NP cells from a range of age

168 A qPCR time course of (1)O2 induced systemic marker genes in directly and indirectly connected leaves

169 gene expression of Alpl and other osteogenic marker genes in mouse osteoblasts and mesenchymal stem c

170 e dataset and removes aberrant expression of marker genes in PBMC datasets.

171 he cassette stably and efficiently expressed marker genes in progeny under either an artificial or an

172 expressed with multiple stem/progenitor cell marker genes in prostate epithelial cells and acts as a

173 accumulation and the expression of adipocyte marker genes in the cultured cells, and shRNA knockdown

174 tochrome B (eYHB) as plant-derived selection marker genes in the model plant Arabidopsis (Arabidopsis

175 ed expression of key MSN and neural activity marker genes in the NAc.

176 Bone-related genes in tibia and barrier marker genes in the small intestine were analyzed by qua

177 evidenced by lower mRNA levels of macrophage marker genes including F4/80, Cd68, Cd11b, Cd11c, and ma

178 Expressions of osteogenic marker genes including Osterix, but not Runx2, were upre

179 the expression of osteoclastogenesis-related marker genes, including CtsK, Nfatc1, Trap, Ctr.

180 ed expression of key Merkel cell lineage/MCC marker genes, including HES6, SOX2, ATOH1, and KRT20 Of

181 ion inhibits the induction of major decidual marker genes, including IGFBP1, WNT4 and PRL.

182 The expression of the M1 marker genes interleukin 1beta (IL1beta) and interleukin

183 ification for 16S rRNA or other phylogenetic marker genes is critical for microbiome research.

184 nstructing 16S ribosomal RNA, a phylogenetic marker gene, is usually required to analyze the composit

185 ical signaling, expression of the JA pathway marker gene JAZ10 in leaves distal to wounds was enhance

186 th reduced steady-state expression of immune marker genes, leading to increased susceptibility to the

187 S and higher expression levels of senescence marker genes, leading to premature senescence of KO sili

188 A multi-marker gene-level analysis of rare (<1% frequency) singl

189 sponses, notably delayed upregulation of PTI marker genes, lower callose deposition, and mitogen-acti

190 curate novel method enabling reference-based marker gene matching (picking Operational Taxonomic Unit

191 (i.e., reference-independent), phylogenetic marker gene (MG)-based operational taxonomic units (mOTU

192 e used to automatically detect cell-specific marker genes (MGs) located on the scatter radii of mixed

193 bundance analysis on sparse high-dimensional marker gene microbiome data.

194 k for PERMANOVA power estimation tailored to marker-gene microbiome studies that will be analyzed by

195 to decode premature stop codons in metabolic marker gene mRNAs, that can be used as in vivo reporters

196 Specific marker genes newly identified for the early hormone resp

197 playing decreased negative selection-related marker genes (Nur77 and CD5high) in CD4 single positive

198 analysis demonstrated reduced expression of marker genes of alternative activation in monocytes sort

199 most upregulated genes were Ms4a8a and Ym1, marker genes of alternatively activated myeloid cells.

200 Using hierarchical clustering of the marker genes of each sub-group, ClusterMap matches the s

201 Transcriptional regulation of marker genes of medium spiny neurons (MSNs) allowed best

202 ons that develop chronological expression of marker genes of non-neural ectoderm, preplacodal ectoder

203 Marker genes of six autotrophic pathways are detected in

204 regulation of Hippo pathway target genes and marker genes of TGF-beta signaling, including biomarkers

205 (ROS) production and the expression of redox marker genes of the cytosol and mitochondrion.

206 ly correlated with the viral burden and with marker genes of the IFN antiviral cascade.

207 n the production of ROS and the induction of marker genes of the JA, ET and SA signalling pathways in

208 levels, weight, histology, or expression of marker genes of the thyroid gland.

209 tically increased expression of osteogenesis marker genes only in the BMP group.

210 dentified from a comprehensive collection of marker genes or by specifying a custom set of marker gen

211 proaches require a priori information on the marker genes or composition of known subpopulations.

212 egrees C and air concentrations of bacterial-marker-genes (p < 0.0001, R(2) = 0.999, n = 6), pointing

213 it upregulated expression of beige adipocyte marker genes, particularly during an HFD, in association

214 full induction of the JA/ET defense pathway marker gene PLANT DEFENSIN1.2 (PDF1.2) and for resistanc

215 nsition of these cells to a unique stem cell marker gene-positive, extracellular matrix-remodeling, "

216 ly, an elevated expression of the adipogenic marker genes PPARgamma and Cebpalpha with a concomitant

217 gulated the expression of an oligodendrocyte marker gene PPP1R14a and other neuronal differentiation

218 d genes, including ICS1 and the canonical SA marker gene, PR1.

219 nd (v) pairwise identity of an additional 73 marker genes present in >90% prokaryotes.

220 NA-seq reveals that MMPs express a number of marker genes previously assigned to mesenchymal stem/pro

221 4, LGR5, TP63 (p63), as well as neural crest marker genes PSIP1 (p75(NTR)), PAX3, SOX9, AP2B1 (AP-2be

222 ements and deletion of the intervening color marker gene, recapitulating the reversion of the UBE2T d

223 level of RAB18- and RD19A-drought-responsive marker genes relative to WT plants.

224 ge-subunit ribosomal RNA and psbA protein D1 marker genes, revealed them to be identical to Ramicrust

225 driven by identification of novel stem cell marker genes, revealing the existence of quiescent, slow

226 -based methods such as database alignment or marker gene search, which limits the set of detectable c

227 Split marker gene segments fused to protein splicing elements

228 rops include the dearth of proven selectable marker gene-selection combinations and tissue culture me

229 ence (MIMS), and Minimum Information about a Marker Gene Sequence (MIMARKS).

230 VAMPS allows researchers using marker gene sequence data to analyze the diversity of mi

231 Users can upload marker gene sequences and associated metadata; reads are

232 taxonomic classification of any phylogenetic marker gene sequences.

233 ed microbiome assembly outcomes using fungal marker gene sequencing and measured susceptibility of th

234 Marker gene sequencing of 16S ribosomal genes revealed t

235 Here, using high-throughput marker gene sequencing of soils collected from 18 sites

236 niques have seldom been applied to microbial marker gene sequencing studies, which have mostly relied

237 al windows, we used high-resolution 16S rRNA marker gene sequencing to examine outcomes in our mouse

238 We characterized bacterial communities using marker gene sequencing, determined N(2) -fixation rates

239 ene assemblies are a powerful alternative to marker gene set and 16S short reads.

240 e automated dynamic selection of single copy marker gene sets.

241 AHOMEOBOXGENE1 (ATH1) maintains the meristem marker gene SHOOT MERISTEMLESS (STM) expression in the l

242 Marker gene silencing assays and genome-wide RNA sequenc

243 d protein extracts induce the Mi-1.2 defense marker gene SlWRKY72b, indicating that both saliva and e

244 allowed excision and recycling of selectable marker genes (SMGs).

245 , and decreases in expression of mesenchymal marker genes SNAI1, SNAI2, and TWIST1.

246 We redefine a common core of marker genes specific to methanogenic, anaerobic methano

247 g the importance of replication in microbial marker gene studies.

248 he expressions of the atrioventricular canal marker genes, such as tbx2b, hyaluronan synthase 2 (has2

249 it sporadic expression of many of these same marker genes, suggesting the existence of a general prog

250 a common data structure for working with 16S marker-gene survey data in R.

251 cilitate working with 16S rRNA databases and marker-gene survey feature data.

252 rRNA sequence databases, and mgFeatures for marker-gene survey feature data.

253 Amplicon-based marker gene surveys form the basis of most microbiome an

254 ly higher expression of chondrogenic-related marker genes than static culture at day 21.

255 I gene becomes a highly efficient selectable marker gene that facilitates the selection of transgenic

256 ies' gene catalog is measured using a set of marker genes that are expected to be present.

257 Finally, we identified novel marker genes that characterize rare neuronal populations

258 that learns pseudotimes from a small set of marker genes that might ordinarily be used to retrospect

259 adata about the specimen source analyzed and marker genes that serve as the most useful features in m

260 The prognostic marker genes that were identified when the shape of the

261 tissue, and yeast cell cycle, revealed novel marker genes that were otherwise undetectable using exis

262 to inhibit the expression of a Pi starvation marker gene (the high-affinity Pi transporter PHT1;4).

263 thus far have relied on a single selectable marker gene, the spectinomycin resistance gene aadA.

264 d increased intestinal inflammatory cytokine marker genes (Tnfalpha, IL17, Ifngamma) expression, resc

265 that leverages prior knowledge of cell-type marker genes to annotate single-cell RNA sequencing data

266 We used high-throughput sequencing on three marker genes to characterize effects on bacterial, funga

267 Here we used high-throughput sequencing of marker genes to elucidate the responses of soil fungal,

268 f community fingerprinting and sequencing of marker genes to identify community changes associated wi

269 we adopt a selection system that allows for marker genes to integrate into the genome through homolo

270 ession measurements of a small subset of 100 marker genes to predict transcriptome-wide gene abundanc

271 S and 18S rRNA gene have been widely used as marker genes to profile diversity of microorganisms in e

272 on, coverage and the presence of single-copy marker genes to separate microbial genomes from non-mode

273 he highest percentage match (45% of proximal marker genes [TPM threshold =15]), with pig kidney cells

274 Investigation of the effect on marker gene transcript levels suggests that the Xoc dite

275 e promoters, resulting in suppression of SMC marker gene transcription and, consequently, in inhibiti

276 ecreased expression levels of the osteoclast marker genes TRAP, Cathepsin K, dendritic cell-specific

277 ted activation of ADH1 and activates hypoxia marker genes under both anoxic and normoxic conditions.

278 olymerase chain reaction (qPCR) of the nifH (marker gene used to identify biological nitrogen fixatio

279 e generated MSTN-mutant Meishan pigs with no marker gene via zinc finger nucleases (ZFN) technology.

280 decreased transactivation of EMT-associated marker genes, vimentin and fibronectin.

281 The abundance of bacterial marker genes was identified in sediments contaminated by

282 nrichment of RNA polymerase II at primed PTI marker genes was observed in environmentally challenged

283 Escherichia coli (using blaNDM and mcr-1 as marker genes), we sampled poultry, dogs, sewage, wild bi

284 Using a panel of spermatogonial cell marker genes, we demonstrate that Sox3 is expressed with

285 Using a large collection of marker genes, we found that vascular identity was specif

286 ations and analyses of core and phylogenetic marker genes, we show that patterns of diversity for the

287 capturing strain variation in clade-specific marker genes were able to distinguish among 100s of indi

288 , H2O2- and photooxidative stress-responsive marker genes were found to be dramatically up-regulated

289 eoclast numbers and expression of osteoclast marker genes were increased in parallel with reduced Erk

290 UCROSE NONFERMENTING-RELATED KINASE1 (SnRK1) marker genes were most affected in reproductive tissue.

291 pe and Ppard null cells, while macrophage/DC marker genes were up-regulated.

292 pipelines validate their results using known marker genes (which are not widely available for all typ

293 racterized by diagnostic quiescent stem cell marker genes, which may potentially be used clinically a

294 ed strain were simultaneously substituted by marker genes, which were subsequently excised using Cre/

295 a dark period induced stress and cell death marker genes while reducing photosynthetic efficiency.

296 tracked by differential expression of three marker genes: Wilms' tumor 1, transgelin, and myxovirus

297 ss C. cellulans, which revealed 49 potential marker genes with known association to human infections,

298 epair activated expression of the selectable marker gene within the donor locus.

299 ation needed to select suitable phylogenetic marker genes within particular lineages.

300 ssion of hypertrophy-related and profibrotic marker genes, without affecting intracellular detoxifyin