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1 tally induced Staphylococcus aureus clinical mastitis.
2 tools to identify S. aureus CC during bovine mastitis.
3 ctiae, one of the causative agents of bovine mastitis.
4 ntributing to higher milk viral loads during mastitis.
5 site detection and field diagnosis of bovine mastitis.
6 ient previously diagnosed with granulomatous mastitis.
7 ads in breast milk were not increased during mastitis.
8 ical processes that occur during LPS-induced mastitis.
9 ontribute to milk composition changes during mastitis.
10 hanisms affected in cows more susceptible to mastitis.
11 3.3, 95% CI: 1.92, 5.62) strongly predicted mastitis.
12 uarters suffering recurrent cases of E. coli mastitis.
13 (CoNS) from bovine clinical and subclinical mastitis.
14 subspecies zooepidemicus, a cause of bovine mastitis.
15 f early inflammatory responses during bovine mastitis.
16 amammary therapy for the treatment of bovine mastitis.
17 as up-regulated on leukocytes from cows with mastitis.
18 were studied during Escherichia coli-induced mastitis.
19 7, a serum-resistant isolate from a cow with mastitis.
20 ssociations with clinical characteristics of mastitis.
21 tory pathways involved in the development of mastitis.
22 reducing ROS production in S aureus-induced mastitis.
23 ate immune cell responders during infectious mastitis.
24 ing for on-site pathogen detection of bovine mastitis.
25 ing of the molecular interactome involved in mastitis.
26 ductal mastitis and idiopathic granulomatous mastitis.
27 d modules associated with clinical traits of mastitis.
28 NAs as early diagnostic biomarkers of bovine mastitis.
29 were found in Egyptian cattle suffering from mastitis.
30 (based on HMO profile) was not associated to mastitis.
31 cular diagnosis and biological therapies for mastitis.
32 oblotting, with a stronger reactivity in SAU mastitis.
33 ntly influence the pathophysiology of bovine mastitis.
34 m dairy cattle with transient and persistent mastitis.
35 cells and in a mouse model of staphylococcal mastitis.
36 ent approach to treat lactational infectious mastitis.
37 the development of chronic S. aureus-related mastitis.
38 the dairy industry are used to treat bovine mastitis.
39 ays relevant to bovine S. aureus subclinical mastitis.
40 hanistic studies on susceptibility of bovine mastitis.
41 is an alga increasingly isolated from bovine mastitis.
42 lead to the development of vaccines against mastitis.
43 ysbiosis of the milk microbiome that permits mastitis.
44 rine macrophages and using a murine model of mastitis.
45 could make dairy animals more vulnerable to mastitis.
46 he azithromycin group had lower incidence of mastitis (0.3% vs 0.5%; RD, -0.24 [95% CI, -0.47 to -0.0
50 Although the etiology and diagnosis of acute mastitis (AM) is well established, little is known about
51 -32 (IL-32) in Staphylococcus aureus-induced mastitis, an inflammation of the mammary gland, is uncle
52 inflammatory breast diseases (granulomatous mastitis and acute suppurative mastitis) and mean ADC va
57 lococcus aureus are a common cause of bovine mastitis and can result in both clinical (CM) or subclin
58 ith the progression of S. aureus subclinical mastitis and could be used as powerful biomarkers for th
59 nd miRNAs modulate immune response in bovine mastitis and could potentially serve as disease biomarke
60 GT-II is the genotype associated with acute mastitis and decreased milk production, although its pat
61 ection of the mammary gland with necrotizing mastitis and high fever was observed for both H5N1 isola
62 ce genes complicates the treatment of bovine mastitis and highlights the serious threat to public hea
63 th peripheral and local sites of cattle with mastitis and identified important granulocyte-specific f
65 series of 9 severe gynecological infections (mastitis and pelvic cellulitis) occurring in the French
66 ein, we develop a murine model of autoimmune mastitis and provide a detailed characterization of its
67 ive understanding of the metabolic status of mastitis and provide new insights into its impact on the
70 understanding of the epidemiology of E. coli mastitis and suggest that pathogen adaptation and host s
71 ghest homology with a GBS strain causing cow mastitis and that the 1992 ST-1 strain differed from ser
73 associated with symptomatic and asymptomatic mastitis and with the quantity of HIV-1 RNA and DNA in m
74 rial and viral infections, which can lead to mastitis and, in some cases, vertical transmission to of
75 granulomatous mastitis and acute suppurative mastitis) and mean ADC value of metastatic lymph nodes.
76 have been identified as causative agents in mastitis, and are traditionally diagnosed by bacterial c
77 ens but have so far not been associated with mastitis, and DNA of bacteria that are currently not kno
78 te immune responses, reduces the severity of mastitis, and facilitates clearance and neutralization o
79 Escherichia coli strains that caused bovine mastitis, and have since been implicated in many physiol
80 ing genital ulcer disease, chorioamnionitis, mastitis, and malnutrition in HIV-infected women, and of
83 s pneumonia, pharyngitis, otitis, arthritis, mastitis, and reproductive disorders in cattle and bison
86 y endemic clones of S. aureus causing bovine mastitis around the world and traced them back to 4 inde
87 cus aureus (Newbould) that developed chronic mastitis as assessed by bacteria and somatic cell counts
89 notable differences in the genomes of bovine mastitis-associated and human clones of S. aureus and pr
90 omparative genomic analysis between a bovine mastitis-associated clone, RF122, and the recently seque
92 f 9.5% reported provider-diagnosed lactation mastitis at least once during the 12-week period, with 6
93 ss-sectional study, laboratory indicators of mastitis (breast milk sodium [Na(+)] concentration, sodi
94 Staphylococcus aureus commonly causes bovine mastitis, but bovine strains, unlike human isolates of S
97 e findings indicate that 38% of all clinical mastitis cases and 63% of the PTEs attributed to S. uber
99 s sequences were the third most prevalent in mastitis cases diagnosed as Staphylococcus aureus by cul
100 were the second most prevalent sequences in mastitis cases diagnosed as Streptococcus dysgalactiae b
101 were the second most prevalent sequences in mastitis cases diagnosed as Trueperella pyogenes by cult
103 olates of Streptococcus uberis from clinical mastitis cases in a study of 52 commercial dairy herds o
104 Forty-one percent of all clinical E. coli mastitis cases occurred in just 2.2% of the population.
105 arison between subacute (SAM) and acute (AM) mastitis cases revealed differences related to the etiol
107 y farm from recently calved cows or clinical mastitis cases were cultured, and 181 isolates were iden
108 of previous lactation and count of positive mastitis cases) to encoded MALDI-TOF spectra, which incr
109 acter, and Staphylococcus, often involved in mastitis cases, were the most abundant genera across tre
115 The phylogenetic analysis classified all mastitis-causing K. pneumoniae into two major phylogroup
116 r a better understanding of the virulence of mastitis-causing K. pneumoniae strains and may lead to t
118 Thus, we concluded P. zopfii GT-II is a mastitis-causing pathogen that triggers severe inflammat
121 itis, we compared the microbiome of clinical mastitis (CM, n = 14) and healthy (H, n = 7) milk sample
125 choice of the antibiotic to treat cows with mastitis could be determined based on the naked eye.
126 samples from women with laboratory-diagnosed mastitis (defined as elevated BM Na(+) levels) were 5.4-
130 fied DNA of bacteria that are known to cause mastitis, DNA of bacteria that are known pathogens but h
131 ssociated with diseases of cattle, including mastitis, endocarditis, orchitis, and endometritis.
134 er; postpartum infections (puerperal sepsis, mastitis), fever, and malaria; and use of antibiotics du
135 inancial and animal welfare impact of bovine mastitis globally, improved tools are urgently needed th
138 I: 1.37, 8.54), and (for women with no prior mastitis history) using a manual breast pump (OR = 3.3,
139 Globally, 44 of 108 women (41%) developed mastitis; however, the percentage of women with mastitis
140 olecular regulatory mechanisms during bovine mastitis immune response and could lay the foundation fo
151 ) H5N1 virus has been associated with severe mastitis in dairy cows, leading to decreased milk produc
155 with susceptibility to ketosis and clinical mastitis in Jersey and Holstein dairy cattle, respective
159 titis; however, the percentage of women with mastitis in the probiotic group (25% [n = 14]) was signi
160 y important in the epidemiology of S. uberis mastitis in the United Kingdom, with cow-to-cow transmis
163 until they stopped breastfeeding to describe mastitis incidence, mastitis treatment, and any associat
164 ved in the host defense of the udder against mastitis infection and that selective recruitment of the
181 cell line (MAC-T) by a Staphylococcus aureus mastitis isolate to study the potential role of intracel
188 pressed the LPS-induced immune response in a mastitis mouse model and that SAMC inhibited LPS-induced
189 ed breast milk sodium levels consistent with mastitis occurred in 16.4% of HIV-1-infected women and w
191 itis treatment, and any associations between mastitis occurrence and hypothesized host characteristic
193 ike pandemics and livestock diseases such as mastitis, often lead to unethical practices that comprom
196 ipple thrush) in the same 3-week interval as mastitis (OR = 3.4, 95% CI: 1.37, 8.54), and (for women
197 cracks and nipple sores in the same week as mastitis (OR = 3.4, 95% CI: 2.04, 5.51), using an antifu
199 clinical presentations in cattle, including mastitis, otitis, arthritis, and reproductive disorders.
202 e involved in the response of the udder to a mastitis pathogen and if the type of mastitis pathogen i
203 er to a mastitis pathogen and if the type of mastitis pathogen influenced the subset composition of t
205 taphylococcus aureus is the major contagious mastitis pathogen, accounting for approximately 15-30% o
207 cussion centers around descriptions of novel mastitis pathogens in Streptococcaceae, Staphylococcacea
208 evealed alterations in the gut microbiota of mastitis rats characterized by an increased abundance of
209 evels of IFIH1, Tnfaip8l2, IRGM, and IRF5 in mastitis rats, which suggests that mastitis triggers an
210 reast-feeding (OR, 1.7; 95% CI, 1.0-2.9) and mastitis (relative risk [RR], 3.9; 95% CI, 1.2-12.7) wer
216 ted with increased transmission overall, and mastitis (RR, 21.8; 95% CI, 2.3-211.0) and breast absces
217 mphocytes in cows with S. aureus subclinical mastitis (SA group) and healthy controls (CK) were gener
219 cterial load was significantly higher in the mastitis samples and decreased after clinical symptoms d
221 itive benign lesions, such as: granulomatous mastitis, sclerosing adenosis, chronic inflammation, fat
224 th significantly higher prevalence in bovine mastitis strains, compared to human-sourced or dairy env
226 ide isolated from the cell envelop of bovine mastitis Streptococcus dysgalactiae 2023 is reported for
227 The chronic nature of bovine staphylococcal mastitis suggests that some products or components of S.
229 minate strains of S. uberis causing clinical mastitis that are likely to be responsive or unresponsiv
233 85.7% of cases of recurrent quarter E. coli mastitis, the same genotype was implicated as the cause
234 ics are effective in the treatment of bovine mastitis, they do not address the regeneration of mammar
236 althy cows and cows with naturally occurring mastitis to determine if distinct alphabeta and gammadel
237 reastfeeding to describe mastitis incidence, mastitis treatment, and any associations between mastiti
238 d IRF5 in mastitis rats, which suggests that mastitis triggers an inflammatory response and immune st
240 numbers observed in cows with streptococcal mastitis was due to a parallel increase in both CD4(+) a
242 ollected from cows in August, 1998, although mastitis was evident among cows on the suspected farm.
243 requency in the same week or the week before mastitis was included in the model (for the same week: 7
247 served in milk from cows with staphylococcal mastitis was primarily due to increased numbers of CD4(+
249 the acute host response to Escherichia coli mastitis, we analyzed gene expression patterns of approx
250 tion between microbiome diversity and bovine mastitis, we compared the microbiome of clinical mastiti
251 experiment modeling phage therapy for bovine mastitis, we observed pathogenicity island transfer betw
252 h confirmed staphylococcal and streptococcal mastitis were characterized by increased numbers of gamm
253 olved in recurrent cases of clinical E. coli mastitis were compared by DNA fingerprinting with entero
255 m cows with staphylococcal and streptococcal mastitis were due to a selective recruitment of a distin
256 y virus (HIV) type 1 load in breast milk and mastitis were examined as risk factors for vertical tran
257 very, HIV-1 load and sodium (an indicator of mastitis) were measured in breast milk from 334 HIV-1-in
259 t in a logistic regression model, history of mastitis with a previous child (odds ratio (OR) = 4.0, 9
260 f the nipple, previous treatment for Candida mastitis with oral or topical antifungals was ineffectiv
261 defined the changes occurring in SAU and NAS mastitis, with potential for improving detection (Proteo