ライフサイエンスコーパス: maternal exposure

1 significant infant plasma exposures (3-8% of maternal exposures).

2 (SIRT1) pathways in the liver, regardless of maternal exposure.

3 sure signifying the importance of preventing maternal exposure.

4 TB and LBW are dependent upon the periods of maternal exposure.

5 opportunity for conception that may minimize maternal exposure.

6 nce offspring had with high fat food was via maternal exposure.

7 arly in gestation, where they may respond to maternal exposures.

8 dysregulation was more pronounced than with maternal exposure alone, an average increase of 4.06-fol

9 o evaluate the potential association between maternal exposure and GDM development.

10 Positive relationship between maternal exposure and newborn bilirubin level is also qu

11 whether outcomes differ between progenitor (maternal) exposure and direct embryonic exposure.

12 fants by both passive antibodies and reduced maternal exposure, and will provide valuable information

13 ubfoot, but studies of risks associated with maternal exposures are lacking.

14 r childhood germ-cell tumors associated with maternal exposures before pregnancy, during pregnancy, a

15 Three latent maternal exposure clusters were determined: a high-risk,

16 Both male and female offspring from all maternal exposure conditions were compared to offspring

17 Prospective questionnaires provided maternal exposure data; case status was established from

18 Information on maternal exposure during pregnancy or direct exposure to

19 Here, we provide evidence that maternal exposure during pregnancy to environmentally re

20 We tested hypotheses that maternal exposure during the periconceptional period to

21 gs highlight the importance of understanding maternal exposures during pregnancy and factors influenc

22 Several maternal exposures during pregnancy are considered predi

23 isk among young children have suggested that maternal exposures during pregnancy, such as low-dose ra

24 hyroid system and neurodevelopment following maternal exposure from early gestation through lactation

25 cerns about their pregnancy outcomes through maternal exposure have emerged, and clinical comparative

26 aly has genetic causes, some associated with maternal exposures including radiation, tobacco smoke, a

27 Among those with available maternal exposure information, at age 20, 421 offspring

28 F(4) generations and that paternal, but not maternal, exposure is most important for the reproductiv

29 Following arsenite maternal exposure of the F0 generation, subsequent gener

30 (95% confidence interval [CI], 1.3-2.6) for maternal exposure only and 2.4 (CI, 1.6-3.6) for neonata

31 ong women of reproductive age as a proxy for maternal exposures, or estimating the impact of risk fac

32 is review focuses on the mechanisms by which maternal exposures, particularly to pathogens, diet, and

33 Maternal exposure prior to conception did not impact off

34 ether the decreased risks observed for these maternal exposures reflected a true association or were

35 revented by CB(1) receptor antagonism during maternal exposure, showing that the CB(1) receptor under

36 Maternal exposure to 0.2% lead acetate in drinking water

37 In the female offspring, the effects of maternal exposure to a high-fat diet during pregnancy on

38 Therefore, only 9 days of maternal exposure to a sodium-restricted diet is require

39 ubstantial differences between the effect of maternal exposure to a TLR7/8 activator as compared to a

40 Maternal exposure to adversity during pregnancy has been

41 amine the epidemiological studies addressing maternal exposure to air pollutants and fetal growth dur

42 l study investigated the association between maternal exposure to air pollutants, carbon monoxide, ni

43 Although maternal exposure to air pollution can potentially have

44 Evidence supports an association between maternal exposure to air pollution during pregnancy and

45 Maternal exposure to ambient air pollution has been asso

46 s suggest some positive associations between maternal exposure to ambient PM10 during the first two m

47 Intrauterine inflammation and maternal exposure to ambient PM2.5 during preconception

48 Our goal was to determine whether maternal exposure to an AhR ligand directly alters CD4+

49 e earthquake shows substantial evidence that maternal exposure to an unambiguously stressful event re

50 re similar to those of the main analysis for maternal exposure to anorexia nervosa and bulimia nervos

51 oral clefts, and urinary tract defects after maternal exposure to antiepileptic drugs were 2.2 (95 pe

52 kely than females to develop psychosis after maternal exposure to any bacterial infection during preg

53 these latter data revealed an association of maternal exposure to any pesticides during the first tri

54 Only maternal exposure to both iron excess and inflammation,

55 Maternal exposure to Cd is associated with fetal growth

56 ify an intergenerational association between maternal exposure to childhood abuse and risk for autism

57 Regardless of maternal exposure to childhood abuse, in response to a s

58 Maternal exposure to cocaine increased gene expression o

59 However, it is unclear whether maternal exposure to common human allergens such as hous

60 Thus, maternal exposure to DEA reduces the number of neural pr

61 In conclusion, maternal exposure to Delta9-THC effectively compromised

62 AD that emerged in early life as a result of maternal exposure to DEP.

63 We created a mouse model that linked maternal exposure to DEPs with asthma susceptibility in

64 Using a mouse model, here we showed that maternal exposure to diesel exhaust particles (DEP) pred

65 ort an association between neuroblastoma and maternal exposure to diuretic agents, antiinfective agen

66 Maternal exposure to dLAN dampened delayed type hypersen

67 We evaluated maternal exposure to drinking water brominated trihalome

68 quartile (7.36 to 12.67 mug/L) of estimated maternal exposure to drinking water with lithium and the

69 Maternal exposure to each by-product was evaluated for e

70 programming and transgenerational effects of maternal exposure to early-life stress on several phenot

71 We found that maternal exposure to Ecig in the second and third trimes

72 mortality risks are particularly related to maternal exposure to El Nino-like conditions in the 0th-

73 Maternal exposure to environmental contaminants during p

74 Atopic status of the mother and maternal exposure to environmental factors are associate

75 There is a gap in knowledge how maternal exposure to environmental tobacco smoke (ETS) i

76 nationwide cohort study of Danish children, maternal exposure to epidural analgesia during labor was

77 In the current study, we show that maternal exposure to estrogens, including 17alpha- and b

78 Maternal exposure to ETS in first trimester was associat

79 ng gestation in a tissue specific manner and maternal exposure to exogenous retinoids during pregnanc

80 Maternal exposure to fine PM increased stillbirths; redu

81 ion against GBS disease in infancy following maternal exposure to GBS colonization.

82 Our data demonstrate that maternal exposure to HDM during pregnancy increases asth

83 alyses showed a positive association between maternal exposure to herbicides during the postnatal per

84 The authors examined whether maternal exposure to herpes virus type 2 is associated w

85 Maternal exposure to high MF during pregnancy may be a n

86 We investigated the role of maternal exposure to human influenza virus [HI] in C57BL

87 Maternal exposure to increased steroid hormones, includi

88 as observed between serologically documented maternal exposure to influenza and bipolar disorder in o

89 lutamate antipsychotics are both affected by maternal exposure to influenza virus, with increased hea

90 ors examined whether serologically confirmed maternal exposure to influenza was associated with an in

91 Serological documentation of maternal exposure to influenza was determined using the

92 ations between parental stressors, including maternal exposure to intimate partner violence (IPV), ma

93 Maternal exposure to intranasal triamcinolone during pre

94 ns in juvenile nonhuman primate tissue after maternal exposure to intravenous gadoteridol during preg

95 We found a strong link between maternal exposure to IPV and health outcomes for childre

96 We examined the association of maternal exposure to IPV with early initiation of breast

97 lf-life of approximately 62 years), and even maternal exposure to lead decades before pregnancy can s

98 We assessed the effect of maternal exposure to low concentrations of ambient air p

99 ine model of bronchopulmonary aspergillosis, maternal exposure to mainstream CS increases AHR after a

100 Maternal exposure to mild daily stress during pregnancy

101 k of offspring ASD in relation to a combined maternal exposure to multiple (up to 42) common and rare

102 The synthetic effects of maternal exposure to multiple risk factors could increas

103 Every IQR increase in estimated geocoded maternal exposure to natural source of lithium in drinki

104 CIs for ASD according to estimated geocoded maternal exposure to natural source of lithium in drinki

105 ) were used to estimate associations between maternal exposure to nitrogen dioxide (NO2), dietary int

106 We investigated the association between maternal exposure to nitrogen dioxide and fine particula

107 2-2005), we investigated the associations of maternal exposure to nitrogen dioxide, nitrogen oxides,

108 study was to evaluate whether high levels of maternal exposure to O3, SO2, NO2, CO are related to inc

109 There is evidence in animal models that maternal exposure to PAHs during pregnancy is associated

110 sistent with the results for the cell lines, maternal exposure to PAHs was associated with hypermethy

111 Maternal exposure to particulate matter <= 2.5 mum in di

112 We explored the association between maternal exposure to particulate matter (PM) air polluti

113 Steps should be taken to decrease maternal exposure to phthalates during pregnancy.

114 dy aimed to explore the associations between maternal exposure to physical, sexual, or emotional viol

115 could reduce the risk of PTD associated with maternal exposure to PM in ambient air during pregnancy.

116 Our findings suggest that maternal exposure to PM(2.5) and specific constituents m

117 aimed to investigate the association between maternal exposure to PM2.5 and IUI in the Boston Birth C

118 Higher maternal exposure to PM2.5 during pregnancy, particularl

119 emiological and animal studies indicate that maternal exposure to pollutants that bind the aryl hydro

120 owever, it has yet to be established whether maternal exposure to polycyclic aromatic hydrocarbons (P

121 While maternal exposure to polyethylene did not impact fetal g

122 In this study, we investigated the effect of maternal exposure to polyethylene micro- and nanoplastic

123 Maternal exposure to PolyI:C induced a potent antiviral

124 und between acute lymphoblastic leukemia and maternal exposure to professional pest exterminations (o

125 serorevert at a younger age (P = .0052), and maternal exposure to protease inhibitors was associated

126 Accordingly, our findings suggest that maternal exposure to psychosocial stressors during pregn

127 We found that maternal exposure to seasonal energy restriction in uter

128 It is well documented that maternal exposure to second-hand smoke (SHS) during preg

129 Maternal exposure to second-trimester fever was associat

130 Our goal was to explore whether maternal exposure to select phthalates and phenols is as

131 In conclusion, maternal exposure to SHS during pregnancy may lead to LB

132 g identified two plausible pathways by which maternal exposure to SHS during pregnancy might cause LB

133 significantly and positively associated with maternal exposure to SHS during pregnancy, lower placent

134 determine the effects in adult offspring of maternal exposure to stress and alcohol during pregnancy

135 However, the association between maternal exposure to stressful events during pregnancy a

136 We tested the hypothesis that maternal exposure to stressful life events would be asso

137 Maternal exposure to systemic antibiotics during pregnan

138 Maternal exposure to TCDD resulted in durable changes in

139 No significant effects were observed after maternal exposure to the endocrine disruptor bisphenol A

140 intrauterine environment as conveyed by the maternal exposure to the general life environment, the s

141 Gallagher et al. (2013) show that maternal exposure to the infection-induced inflammatory

142 This study shows that maternal exposure to this endocrine-active compound shif

143 Adjustment for maternal exposure to tobacco smoke during pregnancy and

144 Low maternal exposure to ultraviolet B radiation may play a

145 rmations to estimate the association between maternal exposure to VOCs adjusting for sex, mother's ag

146 We investigated maternal exposures to air pollutants during weeks 2-8 of

147 Maternal exposures to environmental factors during pregn

148 ase-control study of the association between maternal exposures to environmental sulfur dioxide and t

149 ariates, there were associations of TGA with maternal exposures to herbicides (odds ratio (OR) = 2.8;

150 tes suggests an association between VLBW and maternal exposures to high levels of air pollution.

151 Maternal exposures to higher levels of PFOA, PFHpS, and

152 Maternal exposures to most chemicals were not associated

153 lations and is usually considered to mediate maternal exposures to the fetus through a direct supply

154 rhabditis elegans strains, paternal, but not maternal, exposure to ionizing radiation leads to transg