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1 diversity driven by the presence of dominant matrilines.
2 read relatively recently across all examined matrilines.
3 of matrix remodeling associated 5b (mxra5b), matrilin 1 (matn1), and the transcription factor kruppel
4 h matrilin 1, and that the total ablation of matrilin 1 expression has no impact on disease severity
5  investigate the hypothesis that deletion of matrilin 1 would abolish the formation of matrilin 1/mat
6 not dependent on hetero-oligomerization with matrilin 1, and that the total ablation of matrilin 1 ex
7  that were homozygous for V194D and null for matrilin 1.
8 ed aggregates and caused the co-retention of matrilin 1.
9 nt on the formation of hetero-oligomers with matrilin 1.
10 of matrilin 1 would abolish the formation of matrilin 1/matrilin 3 hetero-oligomers, eliminate the se
11 us angiogenesis inhibitors, we have purified matrilin-1 (MATN-1) and have demonstrated, for the first
12                                              Matrilin-1 and epiphycan were specific for rib and trach
13 ss of this domain occurs as a consequence of matrilin-1 deficiency.
14                                              Matrilin-1 displays a multiphasic expression during zebr
15 region, matrilin-2 will form a homotrimer as matrilin-1 does.
16 on zone, in addition to its co-assembly with matrilin-1 during endochondral ossification.
17 n of early chondrocytes that do not activate matrilin-1 expression rather than by redifferentiation f
18 c molecule with a noncollagenous A domain of matrilin-1 fused to the N terminus of NC1.
19 nase gene has been targeted to exon 1 of the matrilin-1 gene (Matn1) to investigate the origins of ar
20 e chondrocytes did not turn on expression of matrilin-1 in contrast to the other chondrocytes of the
21                   However, gene targeting of matrilin-1 in mouse did not lead to pronounced phenotype
22  appears to function in the matrix linked to matrilin-1 in the form of disulfide-bonded heteromeric m
23           Later, when the skeleton develops, matrilin-1 is expressed mainly in cartilage.
24                        The results show that matrilin-1 is indispensible for zebrafish cartilage form
25 expression at about 15 h post-fertilization, matrilin-1 is present throughout the zebrafish embryo wi
26                                              Matrilin-1 is the prototypical member of the matrilin pr
27        During the early expression phase the matrilin-1 knockdown had no effects on cell morphology,
28 with that of the mature chondrocyte-abundant matrilin-1 mRNA.
29                      Morpholino knockdown of matrilin-1 results both in overall growth defects and in
30 derived from cells that have never expressed matrilin-1 whereas the remainder of the chondrocytes in
31  that, in primary chondrocyte cultures, CMP (matrilin-1) forms a filamentous network, which is made u
32                          Matrilin-3 and CMP (matrilin-1) were prominent in equimolar amounts in fetal
33 sults indicate a molecular stoichiometry of (matrilin-1)2(matrilin-3)2.
34                     One potential antigen is matrilin-1, a cartilage matrix protein found uniquely in
35 ain organization to cartilage matrix protein/matrilin-1, but information on the protein structure is
36                                              Matrilin-1, matrilin-4, epiphycan, and thrombospondin-4
37 de corresponding to the C-terminal domain of matrilin-1.
38 ndrocytes in the cartilage anlagen expresses matrilin-1.
39 directed toward the cartilage matrix protein matrilin-1.
40 rmined that the extracellular matrix protein matrilin-2 (MATN2) is an endogenous neuronal molecule th
41 astrocytes secrete Inhibin A, downregulating Matrilin-2 and blocking myelin repair and recovery.SIGNI
42  differentiation, and Inhibin A inhibits OPC Matrilin-2 expression and inhibits OPC differentiation.
43                        In functional assays, Matrilin-2 induces OPC differentiation, and Inhibin A in
44                                              Matrilin-2 is a member of von Willebrand factor A contai
45                         Within ECM proteins, Matrilin-2 is induced early after stroke and then rapidl
46 n of oligodendrocyte precursor production of Matrilin-2 limit OPC differentiation, tissue repair, and
47                                     In vivo, Matrilin-2 promotes motor recovery after white matter st
48      We also observed that in cultured AVICs matrilin-2 stimulation activated the NOD-, LRR-, and pyr
49 he low sequence identity within this region, matrilin-2 will form a homotrimer as matrilin-1 does.
50 f laminin gamma3 chain, nidogen-2, netrin-4, matrilin-2, and matrilin-4 is described in the cornea fo
51                     The addition of IL-38 to matrilin-2-treated AVICs suppressed caspase-1 activation
52 ICs exposed to the proinflammatory stimulant matrilin-2.
53  including Inhibin A-a negative regulator of Matrilin-2.
54 sponding to the C-terminal sequence of mouse matrilin-2.
55                                 Mutations in matrilin 3 can result in multiple epiphyseal dysplasia (
56                              Retained mutant matrilin 3 formed disulfide-bonded aggregates and caused
57  1 would abolish the formation of matrilin 1/matrilin 3 hetero-oligomers, eliminate the secretion of
58 us, it was proposed that secretion of mutant matrilin 3 may be dependent on the formation of hetero-o
59                                       Mutant matrilin 3 oligomers form non-native disulfide-bonded ag
60                  We showed that secretion of matrilin 3 V194D mutant protein is not dependent on hete
61 ar matrix, and the amount of retained mutant matrilin 3 was not noticeably increased.
62               A similar proportion of mutant matrilin 3 was present in the extracellular matrix, and
63 cellular retention of the majority of mutant matrilin 3 was previously observed in a murine model of
64 oligomers, eliminate the secretion of mutant matrilin 3, and influence disease severity.
65                         Type IX collagen and matrilin-3 (MATN3), also accumulate in the rER cisternae
66 n result from mutations in the gene encoding matrilin-3 (MATN3).
67                                              Matrilin-3 and CMP (matrilin-1) were prominent in equimo
68 ts of the ADAMTS-4 cleavage motif identified matrilin-3 as a new substrate for ADAMTS-4.
69 ombinant ADAMTS-4 effectively cleaved intact matrilin-3 at the predicted motif at Glu435/Ala436 gener
70 hanism of polymeric assembly is unknown, the matrilin-3 chain appears to function in the matrix linke
71 in family of proteins and confirm a role for matrilin-3 in the development and homeostasis of cartila
72 the von Willebrand factor A (vWFA) domain of matrilin-3 in two unrelated families with MED (EDM5).
73                                              Matrilin-3 is an oligomeric protein that is present in t
74                                       Mutant matrilin-3 is retained within the rough endoplasmic reti
75                                              Matrilin-3 knock-out (Matn3 KO) mice exhibit these featu
76                                         Thus matrilin-3 may assemble into both homotypic and heteroty
77                           This suggests that matrilin-3 may self-assemble in the proliferation zone,
78 e a molecular stoichiometry of (matrilin-1)2(matrilin-3)2.
79  A data base search identified the latter as matrilin-3, a molecule recently predicted from human and
80    To understand the molecular properties of matrilin-3, a newly discovered member of the novel extra
81 ork, the interacting complex of collagen IX, matrilin-3, and cartilage oligomeric matrix protein (COM
82 ssociated with dramatically reduced COMP and matrilin-3, consistent with known interactions.
83 can-3 (GPC3), phospholipid transfer protein, matrilin-3, tissue factor pathway inhibitor, fibrinogen-
84 r candidate genes identified MATN3, encoding matrilin-3, within the critical region.
85 e extracellular matrix (ECM) adaptor protein Matrilin-4 (Matn4) as an important negative regulator of
86  chain, nidogen-2, netrin-4, matrilin-2, and matrilin-4 is described in the cornea for the first time
87                                              Matrilin-4 largely disappeared after the age of 3 years.
88 ssion was reduced at the mRNA level, whereas matrilin-4 was verified as a novel collagen IX-binding p
89                                              Matrilin-4, collagen XII, thrombospondin-4, fibronectin,
90                                  Matrilin-1, matrilin-4, epiphycan, and thrombospondin-4 levels were
91 h as cartilage intermediate layer protein 1, matrilin-4, extracellular adipocyte enhancer binding pro
92 ountain red foxes carried indigenous western matrilines (78%) and patrilines (85%), the presence of n
93 mtDNA have shown reciprocal monophyly of the matrilines among seven of the nine assumed subspecies [3
94 vel the mechanisms limiting the dispersal of matrilines and generating genetic differentiation across
95 ndants' mortality rate and life span in both matrilines and patrilines.
96 stimate the age of North American indigenous matrilines and patrilines.
97 ects can interact over 2 generations in both matrilines and patrilines.
98 considerable differentiation among butterfly matrilines and this phenomenon showed a largely determin
99 taining VWA-like domains related to those in matrilins and collagens (AMACO), encoded by the VWA2 gen
100 sed from F1 and F2 families from each of the matrilines, and were found to differ significantly betwe
101 ellular matrix components such as collagens, matrilins, and proteoglycans.
102 MAT-3 and MAT-1 cDNAs results in three major matrilins as follows: (MAT-1)(3), (MAT-3)(4), and (MAT-1
103 ated adhesion mechanism may be applicable to matrilin assembly.
104 ent lead to the formation and persistence of matrilines at habitat sites.
105 d other extracellular matrix proteins (e.g., matrilins, cochlin/vitrin, and von Willebrand factor).
106 lyzing GRC haplotypes across nine castanotis matrilines, estimated to have diverged for up to 250,000
107  identified in any of the genes encoding the matrilin family of proteins and confirm a role for matri
108 MP) is the prototype of the newly discovered matrilin family, all of which contain von Willebrand fac
109                                   Thirty-six matrilines from a single Muscovite sample of Adalia bipu
110                           We established two matrilines from the same population of the linyphiid spi
111 e zebrafish as an alternative model to study matrilin function in vivo.
112 ed in (i) infection of previously uninfected matrilines, (ii) a double infection in a matriline alrea
113 suggest that the varying synthetic levels of matrilins in different zones of a growth plate may resul
114 uencies of PPC were highly consistent within matrilines, indicating that individuals maintained lifel
115                         Decreased fitness in matrilines of four or five was associated with agonistic
116  of a growth plate may result in a change of matrilin oligomeric forms during endochondral ossificati
117 Matrilin-1 is the prototypical member of the matrilin protein family and is highly expressed in carti
118 ncreased when mortality and fission of large matrilines reduced group size and the surviving females
119                     Although the function of matrilins remain unclear, we have shown that, in primary
120                      Both size and number of matrilines varied among sites and survivorship and net r

 
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