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1 dp-ucMGP (dephosphorylated-undercarboxylated matrix Gla-protein).
2 xylase activity and in undercarboxylation of matrix gla protein.
3  may be related to loss of regulation of the matrix Gla protein.
4 osphatase, osteoprotegerin, osteopontin, and matrix Gla protein.
5                    Endothelial cells lacking matrix Gla protein, a BMP inhibitor, underwent epigeneti
6 ar calcification, mice with gene deletion of matrix Gla protein, a bone morphogenetic protein (BMP)-i
7  express bone-associated genes (osteopontin, matrix gla protein, alkaline phosphatase, and the transc
8 inflammatory cascade, including osteopontin, matrix Gla protein and fetuin A (also known as a2-HS-gly
9 s with MS globally expressed lower levels of matrix Gla protein and osteonectin, whereas alkaline pho
10 jury, suggesting a prominent association for matrix Gla protein and osteopontin in ABCC6-related dyst
11 -regulation of the mineralization inhibitors matrix Gla protein and osteopontin.
12 nd calcification inhibitors osteoprotegerin, matrix Gla protein, and osteopontin.
13 ), Pigment epithelium-derived factor (PEDF), Matrix-Gla protein, and Thrombospondin-2.
14 ally expressed in the lymphatic endothelium (matrix Gla protein, apolipoprotein D precursor, and sele
15 f these molecules, including osteopontin and matrix Gla protein, are well known inhibitors of vascula
16  beta-catenin and induction of periostin and matrix gla protein but does not induce genes associated
17 e-specific gene SM22alpha and high levels of matrix Gla protein but little osteopontin mRNA.
18 s of mineralization, such as osteopontin and matrix Gla protein, but not osteocalcin, were concomitan
19 rboxylase activity results in a reduction of matrix gla protein carboxylation, thus allowing peripher
20 yte-like cells in calcified blood vessels of matrix Gla protein deficient (MGP(-/-)) mice.
21                                           In matrix Gla protein-deficient mice, a model of human vasc
22                                    Moreover, matrix Gla protein-depleted human aortic endothelial cel
23              Dephosphorylated-uncarboxylated matrix Gla protein (dp-ucMGP) concentrations were measur
24              Plasma dephospho-uncarboxylated matrix gla protein ((dp)ucMGP), which increases when vit
25                               Enhancement of matrix Gla protein expression in Ins2Akita/+ mice, as me
26 values for six of the propeptides (factor X, matrix Gla protein, factor VII, factor IX, PRGP1, and pr
27  95% CI, 1.23-2.04; P<0.001), uncarboxylated matrix Gla protein (HR, 1.55; 95% CI 1.23-1.95; P<0.001)
28 s, has evolved via animal models such as the matrix gla protein, knock-out, and the targeted overexpr
29 ntial for angiogenesis and is antagonized by matrix Gla protein (MGP) and crossveinless 2 (CV2), both
30 ssion of BMP-2 and BMP-4; the BMP inhibitors matrix Gla protein (MGP) and Noggin; activin-like kinase
31 revious studies suggest an important role of matrix Gla protein (MGP) and thrombospondin 2 (THBS2) in
32                         Osteocalcin (OC) and matrix Gla protein (MGP) are considered evolutionarily r
33 n proposed on the basis of the properties of matrix Gla protein (MGP) as a vitamin K-dependent calcif
34 sts of about 18% mineral, 80% fetuin, and 2% matrix Gla protein (MGP) by weight, and the presence of
35  beta-catenin signaling and/or inhibition of matrix Gla protein (MGP) carboxylation.
36  caused by loss-of-function mutations in the matrix Gla protein (MGP) gene.
37                                              Matrix GLA protein (MGP) has been identified as a calcif
38                                              Matrix GLA protein (MGP) has previously been shown to en
39                                 Mutations in matrix Gla protein (MGP) have been correlated with vascu
40                                              Matrix Gla protein (MGP) is a 14-kD extracellular matrix
41                                              Matrix Gla protein (MGP) is a potent inhibitor of vascul
42                                              Matrix Gla protein (MGP) is a vitamin K-dependent post-t
43                                              Matrix Gla protein (MGP) is an antagonist of BMPs that i
44                                              Matrix Gla protein (MGP) is an inhibitor of vascular cal
45                                              Matrix GLA protein (MGP) is expressed in endothelial cel
46                                              Matrix GLA protein (MGP) is ubiquitously expressed with
47 ification, OPN mutant mice were crossed with matrix Gla protein (MGP) mutant mice.
48                              Inactivation of Matrix gla protein (Mgp) revealed that MGP is an inhibit
49 MP activity due to the loss of BMP inhibitor matrix Gla protein (MGP) shows induction of Hoxd3.
50 onding control tumors and expressed elevated matrix GLA protein (MGP) that mediated enhanced tumor an
51 hosphate mineral and the proteins fetuin and matrix Gla protein (MGP) that was initially discovered i
52             Here, we show that deficiency of matrix Gla protein (MGP), a BMP inhibitor, causes induct
53                      A candidate molecule is matrix GLA protein (Mgp), a mineral-binding ECM protein
54  have observed increased expression of serum matrix Gla protein (MGP), a potent inhibitor of soft tis
55 arget contact also induced the expression of matrix Gla protein (MGP), a regulator of BMP function in
56 nary endothelium, resulting from the loss of matrix Gla protein (MGP), causes ectopic hepatic differe
57 f 5 less critical VKD proteins [osteocalcin, matrix Gla protein (Mgp), growth arrest specific protein
58 ably expressing chimeric coagulation factor, matrix Gla protein (MGP), or osteocalcin as VKD reporter
59  vitro; and a reduction in the expression of matrix Gla protein (MGP), osteopontin (OPN), and vascula
60                     Vitamin K also activates matrix Gla protein (MGP), which protects against pulmona
61  the mineral regulators osteonectin (ON) and matrix Gla protein (MGP).
62 scular disease and is, in part, prevented by matrix Gla protein (MGP).
63 ization capacity due to reduced fetuin-A and matrix gla-protein (MGP) levels.
64 c6-/- serum, and positive immunostaining for matrix-gla-protein (MGP), fetuin-A, and ankylosis protei
65 mostasis (factor IX [FIX]) or calcification (matrix Gla protein [MGP]) were reacted in the presence o
66 calcification in vivo was further studied in matrix GLA protein null (MGP(-/-)) mice whose arteries s
67 ein produced by extrahepatic tissues such as matrix Gla protein or osteocalcin.
68  (i.e. apolipoprotein E, vitamin D receptor, matrix Gla protein, peroxisome proliferator activated re
69      We notably found that undercarboxylated matrix Gla protein precisely colocalized within areas of
70 n, including lipoprotein(a), mineral-binding matrix Gla protein, soluble guanylate cyclase, dipeptidy
71 NA and calcification inhibitors fetuin-A and matrix Gla protein suggests a novel role for EVs in inte
72 hosphate mineral and the proteins fetuin and matrix Gla protein that was initially discovered in the
73                   Regulatory factors include matrix GLA protein, the phosphate cotransporter Pit-1, a
74 on molecule/GA733-2, Kop protease inhibitor, matrix gla protein, tissue inhibitor of metalloproteinas
75 ast growth factor 23 (FGF23), uncarboxylated matrix Gla protein (ucMGP), and fetuin-A are regulators
76 kin by specific antibodies demonstrated that matrix gla protein was found predominantly in undercarbo
77     BMP antagonists follistatin, noggin, and matrix Gla protein were expressed in cultured bovine and
78 hown to have abundant uncarboxylated form of matrix Gla protein, which allowed progressive tissue min
79 of both coagulation factors in the liver and matrix gla protein, which, in fully carboxylated form, i