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1 n to myeloperoxidase-dependent activation of matrix metalloproteinase 7.
2 or repression of Kaiso target genes, such as matrix metalloproteinase-7.
3 tive precursors (pro-Crps) by the convertase matrix metalloproteinase-7.
5 tor [TNF]), TNF-alpha-converting enzyme, and matrix metalloproteinase-7 and -9) were assessed on HCLE
6 splayed similar CCL expression and suggested matrix metalloproteinase-7 and MMP9 as possible extracel
8 s by stimulating expression and secretion of matrix metalloproteinases 7 and 9 (MMP7/9), by which MMP
9 Two proteases that process OPN (thrombin and matrix metalloproteinase 7) and cleaved OPN were increas
10 h as Bcl-2 (B-cell lymphoma 2), c-Myc, MMP7 (matrix metalloproteinase-7), and cyclin D1in vitro and i
11 ls of vascular endothelial growth factor and matrix metalloproteinase 7, and increased invasive behav
12 ing the SARS-CoV-2 main protease, caspase-3, matrix metalloproteinase-7, and cathepsin B-simply by al
13 four serum biomarkers (surfactant protein D, matrix metalloproteinase 7, CA19-9, and CA-125) that wer
15 of the matrix metalloproteinase matrilysin (matrix metalloproteinase-7) in the mouse mammary gland p
19 n (CD44HSPG) recruits proteolytically active matrix metalloproteinase 7 (matrilysin, MMP-7) and hepar
20 eolytic activation of inactive precursors by matrix metalloproteinase-7 (matrilysin, EC, MMP-7(a)).
21 s associated with epithelial barrier repair (matrix metalloproteinase 7, matrix metalloproteinase 3,
22 s, termed cryptdins (Crps), are activated by matrix metalloproteinase-7-mediated proteolysis of inact
23 duced seizures, the proNGF processing enzyme matrix metalloproteinase 7 (MMP-7) and its inhibitor TIM
25 with survival, accounting for the effect of matrix metalloproteinase 7 (MMP-7) blood concentration a
26 vitro acinar transdifferentiation depends on matrix metalloproteinase 7 (MMP-7), a proteinase express
32 converted to bactericidal CRS4C-1 peptide by matrix metalloproteinase-7 (MMP-7) proteolysis of the pr
33 ttaching the consensus peptide substrate for matrix metalloproteinase-7 (MMP-7), an enzyme that is up
34 es, beta-catenin transcriptionally regulates matrix metalloproteinase-7 (MMP-7), but the association
35 the proteolytic activation of precursors by matrix metalloproteinase-7 (MMP-7), prompting an analysi
36 nsin precursors are cleaved and activated by matrix metalloproteinase-7 (MMP-7), we determined if add
37 lung cancer cell invasion and down-regulated matrix metalloproteinase-7 (MMP-7), which promoted lung
39 onstrate that HOCl regulates the activity of matrix metalloproteinase-7 (MMP-7, matrilysin) in vitro,
42 C4-Fc (truncated N-cadherin), or deletion of matrix-metalloproteinase-7 (Mmp-7) reduced VSMC apoptosi
43 riptional responses dependent on matrilysin (matrix metalloproteinase 7 [MMP-7]) and stromelysin-2 (M
46 adenocarcinoma with a concurrent increase of matrix metalloproteinase 7 (MMP7) expression in mouse pr
50 interleukin 25, we show here that epithelial matrix metalloproteinase 7 (MMP7) was expressed during a
55 in compositional analysis with LC-MS/MS, and matrix metalloproteinase-7 (MMP7) were identified as a p
56 fic PCR array assays revealed that WNT5A and matrix metalloproteinase-7 (MMP7) were upregulated by FO
57 lationship between the expression of FXR and matrix metalloproteinase-7 (MMP7), a collagenase and sig
59 rtantly, a combinatorial signature including matrix metalloproteinase 7, pulmonary and activation-reg
60 ), angiopoietin-2 (related to angiogenesis), matrix metalloproteinase-7 (related to extracellular mat
61 , saliva Vascular-endothelium-growth-factor, Matrix-metalloproteinase-7, serum Epithelial-neutrophil-
62 4, killer immunoglobulin-like receptors, and matrix metalloproteinase-7 were also found to be associa