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1 1, adaptive immunity, alphabeta T cells, and mature B cells.
2 repertoire, and antigen-driven selection of mature B cells.
3 through editing toward lambda light chain in mature B cells.
4 ytic leukaemia (CLL) is a clonal disorder of mature B cells.
5 blocks by expressing bnAbs conditionally in mature B cells.
6 previously described, a 2-fold reduction in mature B cells.
7 hrough Nod1 promotes competitive survival of mature B cells.
8 ment, enabling the expression of VRC26UCA in mature B cells.
9 role for Fbw7 in the survival and fitness of mature B cells.
10 sential regulator of cellular homeostasis in mature B cells.
11 block at the pro-B cell stage and a lack of mature B cells.
12 V-2) strain has the unique ability to infect mature T cells.
13 evealed an intrinsic proliferative defect of mature T cells.
14 eration of a large number of double negative mature T cells.
15 ays a significant role in the homeostasis of mature T cells.
16 ng the specific impact of Notch signaling in mature T cells.
17 stem and progenitor cells (HSPCs) through to mature T cells.
18 h positive and negative selections to become mature T cells.
19 identity and shape the proper generation of mature T cells.
20 red for PT progenitors to differentiate into mature PT cells.
21 necessary for the generation of functionally mature LTi cells.
22 ss central to stimulus-secretion coupling in mature beta cells.
23 this is partially compensated for by longer mature root cells.
24 odulates the differentiation and function of mature blood cells.
25 cell lineage and the effect ceases as cells mature to EE cells.
26 f HSCs, but there were no obvious defects in mature immune cells.
27 o transiently upregulated by high glucose in mature IDG-SW3 cells.
28 in a shift from luminal progenitor cells to mature luminal cells.
29 lveolar epithelial cells and morphologically mature type II cells.
30 eding, migrate into the gonad primordia, and mature into germ cells.
31 tightly regulated process that generates all mature red blood cells.
32 s to ECFCs and can also trigger sprouting of mature endothelial cells.
33 second strain of EBV, EBV type 2, to infect mature peripheral T cells.
34 the cellular protein CD21 to gain entry into mature peripheral T cells.
35 aintenance of the transducing stereocilia in mature cochlear hair cells.
36 ption of how to phenotypically characterize, mature, and freeze the cells.
37 old KO BM are differentiating cells that can mature into functional B cells.
38 structure of the transducing stereocilia in mature mouse cochlear hair cells.
39 B2, significantly enhances the production of mature enucleated red blood cells.
40 rs cannot produce multiple basal bodies, and mature into single ciliated cells.
41 ical NF-kappaB signaling was impaired in all mature naive CVID-derived B cells.
42 med electron tomography (ET) of immature and mature Ty3 particles within cells.
43 uited to germinal centers, and they affinity matured, and formed memory B cells.
44 well as the caspase-1-mediated generation of mature IL-1beta secreted from cells.
45 ion deficits along with dramatic decrease in mature oligodendrocytes and their progenitor cells.
46 ow that deletion of Stim1 and Stim2 genes in mature Treg cells abolishes Ca(2+) signaling and prevent
47 identical grafts, containing high numbers of mature NK cells, according to PT-Cy-based protocols in 2
49 y control Ca2+ fluxes that are essential for mature T-cell activation and differentiation and protect
53 exhibit a transcriptional profile similar to mature alpha cells and although they produce proinsulin
56 s in mice leads to the prolonged survival of mature B cells and expanded B cell compartments in secon
57 e responsible for the lifelong production of mature blood cells and are the rational target for clini
58 mal promoters are densely methylated in less mature CD56(bright) NK cells and are progressively demet
59 rst example of phenotypic continuity between mature CLL cells and their progenitors in the bone marro
61 s, NSG-SGM3 mice supported the population of mature human tissue-resident mast cells and basophils.
62 Transcriptional profiling of normal ER(+) mature luminal mammary epithelial cells and ER(+) breast
63 characterized by the excessive production of mature myeloid cells and arise from the acquisition of s
64 ate that group 1 ILCs consist of circulating mature natural killer (NK) cells and tissue-resident ILC
65 showed myeloid hyperplasia with predominant mature neutrophils, and decreased progenitor cells and l
69 expressing ThPOK compared with wild-type CD4 mature T cells and compromising cytotoxic program, faile
70 esulting in the continued presence of CD8(+) mature T cells and the generation of a large number of d
71 they undergo development from precursors to mature, circulating cells and how our understanding of t
72 1 (a gene-expression pattern associated with mature follicular B cells) and also attained increased c
75 A/C helps distinguish normal from neoplastic mature T cells, and VAMP-7 recapitulates light-cytometri
76 d levels of 36 blood cell traits (platelets, mature/immature red cells, and myeloid/lymphoid/compound
78 In response to T cell-dependent antigens, mature B cells are stimulated to form germinal centers (
81 d IFN-gamma led to a significant increase in mature CD27(-) CD11b(+) NK cells as well as a significan
82 ts suggest that motoneuron properties do not mature by cell autonomous mechanisms alone, but also dep
85 ghtly regulated in thymocytes, as well as in mature T cells both at steady state and upon stimulation
86 en implicated in inhibiting BCR signaling in mature B cells but promoting pre-BCR signaling during ea
88 and its ligand IL-33 are known regulators of mature myeloid cells, but their roles in AML are not kno
89 s from multipotent CPCs to intermediates and mature cardiac cells by population and single-cell RNA-s
90 s the differentiation of PT progenitors into mature PT cells by regulating the expression of genes as
92 al immature thymocytes to differentiate into mature T cells by binding to peptide-MHC ligands togethe
93 equirements for the generation of terminally mature functional innate effector cells can be elucidate
94 established differentiation therapies, some mature leukemia cells can de-differentiate and reacquire
95 D28 signaling results in the accumulation of mature, peripheral TRA-specific T cells capable of media
96 56(bright) NK cells and is confined to fully mature NK cells characterized by the NKG2A(-)KIR(+)CD57(
100 ciated Ifng promoter demethylation, the less mature phenotype of Tyk2(-/-) NK cells correlated with a
101 Es are functionally distinct from their more mature but still naive T cell counterparts, because they
102 variant transcription factor 2 (ETV2)(3) in mature human endothelial cells cultured in a serum-free
103 ls, providing insights toward vaccination of mature B cell-deficient individuals and implications in
104 ctile waves and their synchronization within maturing, unlabeled induced pluripotent stem cell-derive
105 a robust means for expansion of functionally mature stem cell-derived astrocytes for preclinical inve
107 tional, as judged by its capacity to support mature T cell development in vivo after transplantation
110 y modifying the immunoglobulin (Ig) genes of mature B cells directly using genome editing technologie
111 cell depletion in mice, that a population of mature B cells distinguishable by IgD(low/-) expression
113 ymphocytic leukemia (CLL) is a malignancy of mature B cells driven by B-cell receptor (BCR) signaling
115 sion, the accumulation of gamma-tubulin-2 in mature neurons and neuroblastoma cells during oxidative
118 y little is known on the mechanisms by which mature endothelial cells exit from a quiescent state and
119 We find expression of viral receptor ACE2 in mature choroid plexus cells expressing abundant lipoprot
121 eceptor-deficient mice, the thymus generates mature T cells expressing MHC-independent TCRs that reco
125 of peripheral B cell tolerance that restrain mature B cells from mounting inappropriate responses to
126 tantial advances have been made in producing mature beta-cells from human pluripotent stem cells that
127 Tregs continuously constraining one-third of mature CD4(+)Foxp3(-) cells from converting to pathogeni
128 Even though Plerixafor liberates HSPCs and mature immune cells from bone marrow, competitive repopu
130 -like macrophages increase the production of mature, enucleated erythroid cells from umbilical cord b
132 ling, pericytes upregulate genes involved in mature pericyte cell function, together with a remarkabl
133 y diphtheria toxin-based ablation of >50% of mature DG granule cells (GCs) or by prolonged brain-spec
134 characterized events is the acquisition of a mature T-cell gene expression program characterized by t
136 ad entered a new phase of proliferation, and mature MTB cells had begun to move from the main body of
137 However, differentiation of human PSCs into mature, conventional T cells has been challenging with e
139 rlying the high-level expression of GATA1 in maturing erythroid cells have been studied extensively,
142 s, as we have defined a function for CD21 on mature peripheral T cells, i.e., as a receptor for EBV.
143 n-binding protein 1 (LDB1) serve to maintain mature adult beta cell identity, revealing clues as to h
144 nd Cd11c mRNA levels, and the frequencies of mature dendritic cells, IFNgamma-, and IL-17- producing
151 etion of Spi1 and Spib resulted in a lack of mature B cells in the spleen and a block in B cell devel
152 docrine progenitors, and differentiating and mature beta cells in vivo Pdx1(DeltaAREAII/-) mice exhib
153 haracterized by the expansion of monoclonal, mature CD5(+)CD23(+) B cells in the peripheral blood, se
154 uman embryonic stem cells, the generation of mature functional beta cells in vitro has remained elusi
156 ment of CMML and JMML disease-initiating and mature leukemic cells in vivo, allowing creation of gene
157 e and increase differentiation and output of mature myeloid cells in response to stress stimuli to pr
158 d autoreactive new emigrant/transitional and mature naive B cells in NMOSD patients compared to healt
160 lithium can trigger transdifferentiation of mature principal cells to intercalated cells in adult ki
164 LK could restore progenitor-like features in mature CD30+ peripheral CD4+ T cells, in keeping with a
168 n to be affected in schizophrenia (SCZ) when matured-from induced pluripotent stem cells (iPSCs) deri
170 (CH) arises when a substantial proportion of mature blood cells is derived from a single dominant hem
174 alphabeta T cell antigen receptors (TCRs) on mature T cells is selected in the thymus where it is ren
175 lphabeta T cell receptor (TCR) repertoire on mature T cells is selected in the thymus, but the basis
176 f ageing, whereas the function of individual mature thymic epithelial cells is compromised only modes
178 its dependence on POM121, and a reduction of mature NPCs in Torsin-deficient cells lead us to conclud
180 acquires frequent inactivating mutations in mature B cell malignancies, especially in the MYD88(L265
181 lymphoma (PTCL) is a heterogeneous group of mature T-cell malignancies; approximately one-third of c
182 allosum and external capsule, and decline of mature oligodendrocytes and oligodendrocyte precursor ce
183 rogenitor cells, and a subset of these cells mature into mesangial cells (MCs) that continue to expre
185 ervical cancer micromilieu and found CD83(+) mature dendritic cells (mDC) coexpressing IL23 in the st
186 s in 8% of cases the translocation occurs in mature B cells mediated by activation-induced cytidine d
187 puts, abGCs exert monosynaptic inhibition of mature granule cells (mGCs) through group II metabotropi
189 K3 kinase inhibition can greatly enhance the mature character of NK cells most desired for effective
192 Anaplastic large cell lymphoma (ALCL) is a mature T cell neoplasm that often expresses the CD4+ T c
193 l prolymphocytic leukemia (T-PLL) is a rare, mature T-cell neoplasm with a heterogeneous clinical cou
197 as improved survival rates for children with mature B-cell non-Hodgkin lymphoma (NHL); however, assoc
198 younger than 18 years of age with high-risk, mature B-cell non-Hodgkin's lymphoma (stage III with an
199 and adolescents with high-grade, high-risk, mature B-cell non-Hodgkin's lymphoma and was associated
201 S similarly showed reduced myelin thickness, mature oligodendrocyte cell numbers and mRNA levels of m
202 in myelination, and mutant mice had reduced mature oligodendrocyte cell numbers, reduced myelin thic
203 em cells (HSCs) in the bone marrow (BM) form mature blood cells of all lineages through expansion of
204 e click chemistry to fluorescently label the mature PG in whole bacterial cells of Bacillus subtilis.
205 nditions of nutrient starvation and that the mature Red Blood Cells of some RTT patients retain mitoc
206 ansmission under physiological conditions at mature synapses made by Purkinje cells onto neurons in t
207 sence of GW and inflammatory signals, either mature dendritic cells or IL12, also demonstrated enhanc
208 d innate immunity, induced via modulation of mature myeloid cells or their bone marrow progenitors, m
209 (>18 years) with newly diagnosed, untreated mature T-cell or NK lymphomas (WHO 2001 or 2008 classifi
213 tocompatibility complex class II (MHC)-II(+) mature antigen-presenting cell population kinetics durin
219 y detected by measuring the precursor or the mature cytokine of bulk cell populations by techniques s
220 tially infected neural progenitor cells over mature neurons, reduced both cell populations, and cause
221 these findings, viral glycoproteins fail to mature in SPCA1-deficient cells preventing viral spread,
225 associated with patient outcome, and a high mature neutrophil/T-cell ratio resulted in inferior prog
227 t islets are thought to contain functionally mature beta cells, recent analyses of transgenic rodent
228 ferentiating B cells can directly generate a mature B cell receptor (BCR) and bypass the requirement
229 the recovery of bone marrow cellularity and mature blood cell recovery at 21-30 days post-irradiatio
233 ly tolerated or positively selected into the mature B cell repertoire as well as at what stage, to wh
236 n early life determines the clonality of the mature B-1 B cell repertoire and ensuing antibody respon
241 ltiple inhibitors that blocked production of mature glucagon from proglucagon, beta-cells retained th
243 moxifen-inducible genetic lineage tracing of mature adipocytes and single-cell RNA sequencing reveale
244 ere, we profiled >25,000 differentiating and mature mouse neutrophils using single-cell RNA sequencin
246 f miRNAs in RPE cells, we used two different mature RPE cell-specific Cre recombinase drivers to inac
247 and partial deletion at the transitional to mature B cell stage, but become Env(-) upon receptor edi
252 differentiation were consistently present in mature myeloid cells such as neutrophils and monocytes a
253 ion of the CAR-28/zeta led to elimination of mature CAR(+) T and B cells, suggesting that the CAR-med
254 erates has been puzzling and its function in mature T cells suggests promoting rather than repressing
256 s and introduced 24 new residues forming the mature protein, alpha1(Lys374Serfs)*(25) Cell surface ex
257 -1, CD34, VE-Cadherin, FLK1, and TIE2 lacked mature arterial, venal, and lymphatic cell-surface marke
258 ally, we show wild-type Tcrb alleles produce mature alphabeta T cells that express upstream Vbeta pep
259 crucial survival factor for transitional and mature B cells that acts as rheostat for the maturation
260 low level (BD(L)) are a novel population of mature B cells that emerge in the spleen from the transi
261 d thymic Treg [tTreg] cells) or derived from mature conventional CD4(+) T cells that underwent TGF-be
262 into computational models of developing and mature cortical pyramidal cells that express NaV1.2.
264 hey undergo an additional maturation step to mature naive T cells that circulate through secondary ly
265 ctory from primitive progenitor-like to more mature neuronal-like cells, the relative proportions of
266 cursor to immature memory and finally from immature to mature memory cells, the latest being a no-ret
267 microbial products in promoting survival of mature B cells through up-regulated Nod1, providing a po
269 karos expression was deleted specifically in mature T cells, thus avoiding defects observed in germli
270 lative contribution of adult-born (abGC) and mature (mGC) granule cells to epileptiform network event
271 GR deficiency results in impaired homing of mature B cells to bone marrow, whereas migration to othe
272 oral immunity is dependent on the ability of mature B cells to undergo antibody gene diversification
273 ity of its differentiation intermediates and mature effector cells to expand upon demand, thereby pro
274 m human stem cells allow differentiating and mature human retinal cells to be studied in unprecedente
275 myeloid-biased hematopoietic stem cells and mature myeloid cells to levels in young animals, but lym
276 -activation is sufficient to reprogram fully mature supporting cells to proliferate and regenerate ha
277 esent at the tips of the tall stereocilia in mature hair cells, together with PCDH15 isoforms CD1 and
278 tic-myeloid origin and distinguish them from mature lymphatic endothelial cells, tumor-infiltrating l
279 xpression of the IgM and IgD BCR isotypes on mature naive follicular B cells tunes responsiveness to
280 pend on their capacity to differentiate into mature and functional cell types after transplantation.
282 lls can differentiate in culture to generate mature basal and luminal cell types, including ER+ cells
284 vitro with evidence of differentiation into mature epithelial cell types found in native human airwa
286 Using human brain organoids comprised of mature neural cell types as a three-dimensional tissue s
288 differentiation of immature blast cells into mature, functional cell types and lineages of the immune
289 y exercise, and permanently marked activated mature hippocampal dentate granule cells using condition
290 , an assay based on the examination of early mature B (eBm5) cells was able to discriminate OIS patie
291 atients, the CD19(+)CD24(int)CD38(int) naive mature B cells were high in CHC-HCC patients with good p
292 tion profile of bone marrow populations, and mature IgD+ B cells were enriched in sialylated bone mar
293 that plasmablast, plasma, transitional, and mature naive B cells were resistant to VACV infection, w
296 ile other B subsets, including transitional, mature naive, memory, and plasma cells, were highly susc
297 ss II(+)NK1.1(+) population as precursors to mature NK (pre-mNK) cells, which also expressed high lev
299 tor dominant negative mastermind-like within mature T cells with C. neoformans Inhibition of T cell-r