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1 re negative in young leaves, and positive in mature leaves.
2 tion, but the transcript is less abundant in mature leaves.
3 eaves but increased transcription in healthy mature leaves.
4 nduced transcription of chloroplast genes in mature leaves.
5 treated, healthy potato organs or in wounded mature leaves.
6 n epidermal, mesophyll and vascular cells of mature leaves.
7 r from B supplied as a foliar application to mature leaves.
8 of the silique, pedicel and stem but not in mature leaves.
9 these processes were largely uncorrelated in mature leaves.
10 ver-expressed in developing primordia and in mature leaves.
11 equired to limit the spread of cell death in mature leaves.
12 ed with the mass-based carboxylation rate of mature leaves.
13 adjust the size and cell type composition of mature leaves.
14 oem cell wall ultrastructure in immature and mature leaves.
15 xhibit a significant cellulose deficiency in mature leaves.
16 hydrates metabolism were highly expressed in mature leaves.
17 he chlorophyll biosynthetic pathway, even in mature leaves.
18 n fruits, flowers and cotyledons, but not in mature leaves.
19 ves to an aliphatic wax-dominated profile in mature leaves.
20 inhibition effects, including the wilting of mature leaves.
21 ction were also significantly upregulated in mature leaves.
22 plete or nearly complete DNA loss already in mature leaves.
23 the upper leaf side was gained in young and mature leaves.
24 increased in the vein and mesophyll of young mature leaves.
25 , but relatively little initiation occurs in mature leaves.
26 t in immature leaves but are greater than in mature leaves.
27 umulated excess starch and soluble sugars in mature leaves.
28 he accumulation of neutral lipid droplets in mature leaves.
29 o high light and induced early senescence of mature leaves.
30 elopmental control and induces telomerase in mature leaves.
31 olated from the oil gland secretory cells of mature leaves.
32 ures on both adaxial and abaxial surfaces of mature leaves.
33 ndant transcript but was absent from healthy mature leaves.
34 the companion cells of the smallest veins of mature leaves.
35 immature leaves and was highly expressed in mature leaves.
36 expanding tissues, but substantially less in mature leaves.
37 ieved the repression of the PCNA promoter in mature leaves.
38 tips, shoots, and floral organs, but not to mature leaves.
39 or the full repression of PCNA expression in mature leaves.
40 important roles in the greater resistance of mature leaves against Xanthomonas citri compared with im
43 as a result of damage, increased >50-fold in mature leaves and decreased >1400-fold in immature leave
44 enerated a large data set of small RNAs from mature leaves and developing roots, shoots, and inflores
47 0-fold above wild type, whereas the level in mature leaves and other tissues is no greater than wild
49 sink-source transition of immature leaves to mature leaves and provide knowledge regarding the differ
50 sink-source transition of immature leaves to mature leaves and provide knowledge regarding the differ
51 ted at 45.3% of the total soluble protein in mature leaves and remained stable even in old bleached l
52 ne, is shown to be specifically expressed in mature leaves and the developing pod walls of Brassica n
54 tants exhibited decreased phloem pressure in mature leaves, and altered phloem cell wall ultrastructu
56 accumulated high levels of carbohydrates in mature leaves, and had a higher shoot biomass, contrasti
58 highly expressed in the collection phloem of mature leaves, and its function in phloem loading is wel
59 , these morphological asymmetries persist in mature leaves, and we observe left-right asymmetries in
62 in gene expression varied between young and mature leaves at the same time point and between the sam
65 ese processes do not occur in the absence of maturing leaves but can be restored by application of C(
66 rol and sulfolipid levels were lower than in mature leaves, consistent with low photosynthetic rates
67 is machinery returned to wild-type levels in mature leaves, consistent with the developmental down-re
70 1b null mutant becomes depleted of CSP41a in mature leaves, correlating with a pale green phenotype a
72 he transcriptional control of acclimation in mature leaves distinct from other photoreceptor-regulate
75 t, which ensures the continual production of mature leaves following juvenile-adult transition, there
76 n nighttime leaf respiration rate (RN) among mature leaves from an Arabidopsis (Arabidopsis thaliana)
79 y of organs to produce AsA from GAL-L showed mature leaves have a 3- to 10-fold higher biosynthetic c
81 Suc) found drastically decreased export from mature leaves in cpd28 and cpd47 mutants relative to wil
82 spectroscopy accurately predicts V(c,max) of mature leaves in Panamanian tropical forests (R(2) = 0.9
85 n increased by more than 2-fold in young and mature leaves, indicating that phosphate stimulates prot
89 ct DNA of any size in most chloroplasts from mature leaves, long before the onset of leaf senescence.
91 synthesis, and immunity between immature and mature leaves may contribute to their different response
92 -off of cytochrome b6f complex biogenesis in mature leaves may represent part of the first dedicated
93 stemically, with the irradiance perceived by mature leaves modulating stomatal development in young l
94 onfers expression only in the minor veins of mature leaves, not in the transport phloem of larger lea
95 synthetic capacity with NSCs accumulation in mature leaves, observed most clearly with hexose, and ev
96 abolites, and key inorganic ions in recently mature leaves of 45 dicotyledonous species at midafterno
97 he nighttime O(2) consumption rate (R(N)) in mature leaves of Arabidopsis (Arabidopsis thaliana).
108 e examined the hydraulic architecture of the mature leaves of the model species Populus tremula x alb
109 o temperature (10 degrees C-40 degrees C) in mature leaves of tobacco (Nicotiana tabacum L. cv W38) w
112 d whether responses differ between young and mature leaves or between morning and the end of the day.
116 n with earlier studies, we show that loss of mature leaves results in decreased sugar levels and incr
119 d increases in the fatty acid content of the mature leaves, senescing leaves, and roots, respectively
124 ilability or a defect in sucrose export from mature leaves, suggesting that isi1 mutant plants do not
125 significant impact on plant metabolism, with mature leaves tending to be more extensively affected th
126 bacco, superpromoter activity was greater in mature leaves than in young leaves, whereas in maize act
127 sing entire shoot tissues, most of which are mature leaves that do not elongate under submergence.
129 te in young tissues and geminivirus-infected mature leaves, the GRIK-SnRK1 cascade may function in a
130 n young leaves of A. bisulcatus, but in more mature leaves, the Se-methylseleno-Cys concentration is
131 titutive in roots and inducible by copper in mature leaves; the reverse pattern was observed for MT2.
132 PetC was highly efficient in both young and mature leaves, these data indicate a lifetime of the cyt
134 slocation of the foliar-applied 10B from the mature leaves to the meristematic tissues verifies that
137 ling pathways that control CHS expression in mature leaves using cryptochrome (cry) and phytochrome (
138 A differential response between young and mature leaves was also found in carbon metabolism, with
139 rformance of mannitol-accumulating calli and mature leaves was due to other stress-protective functio
142 Lipid bodies, not observable in uninfected mature leaves, were found in and external to chloroplast
143 here AthDHS2 is highly expressed, but not in mature leaves, where AthDHS1 is predominantly expressed.
144 the formation of elliptical leaf laminae in mature leaves, whereas overexpression of GTE6 resulted i
145 te to the repression of the PCNA promoter in mature leaves, whereas the E2F1 site counters the repres
146 s, siliques, and roots than in dry seeds and mature leaves, whereas the polyphosphoinositide-dependen
147 endophyte-mediated protection was greater in mature leaves, which bear less intrinsic defense against
148 PFD, in 2% and 21% oxygen, in developing and mature leaves, which differed greatly in R in darkness.
149 mesophyll but not in the epidermis of young mature leaves, while this was reversed for zinc distribu
150 n immature leaves, but slightly expressed in mature leaves, while Vfa4 was active in immature leaves
151 eached rapidly and developed necroses, while mature leaves, whose photosynthetic apparatus was fully
152 and increased phospholipid concentrations in mature leaves, with concomitant changes in the expressio
153 s were notably different between growing and mature leaves, with greater anaplerotic, tricarboxylic a
154 nes by near-infrared spectroscopic screen of mature leaves yielded several dozen lines with heritable