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2 have agenesis of >/=1 third molars and that maxillary agenesis was 36% more likely than mandibular a
3 outcome and clinical performance of anterior maxillary all-ceramic implant crowns (ICs) based either
9 how adult endites, the inner branches of the maxillary and labial appendages, are formed at metamorph
11 During analysis of CT studies we evaluated maxillary and mandibular alveolar processes for presence
13 tions and less precise axonal sorting of the maxillary and mandibular branches within the trigeminal
14 eural crest cells (NCCs) that develop in the maxillary and mandibular buds of pharyngeal arch 1 (PA1)
15 L) injected into the gingival tissue (GT) of maxillary and mandibular first molars and into the inter
16 ange in crestal bone levels (AvBL) for 5 x 8 maxillary and mandibular implants after 5.9 years of fol
18 correlated with the dramatic differences in maxillary and mandibular molar phenotypes in Bmp4(f/f);W
19 ects of loss of activin or Bmp4 signaling on maxillary and mandibular molar tooth morphogenesis are m
21 2, Dlx2, Msx1, Barx1, Foxc2 and Fgf8, in the maxillary and mandibular processes of the mutants, indic
22 sions arise along the sites of fusion of the maxillary and mandibular prominences early in facial dev
26 vent plaque removal during brushing over one maxillary and one mandibular posterior dental sextant fo
27 ome conformation capture assay in developing maxillary and periocular tissues suggest that the DNA re
30 males were, in descending order, mandibular, maxillary, and sphenoid bones, while the sphenoid bone w
32 from four study centers, each with a single maxillary anterior Miller Class I or II recession defect
34 posterior non-pit and fissure surfaces, (C4) maxillary anterior surfaces, and (C5) mid-dentition surf
36 n or absent buccal plate occur frequently at maxillary anterior teeth and necessitate careful treatme
37 Gingival recession was present at 32.9% of maxillary anterior teeth and was most common at canines,
42 ome-wide map of candidate enhancers from the maxillary arch (primordium for the upper jaw) of mouse e
43 essential for the expression of Lhx8 in the maxillary arch cells and that Lhx8_enh1 was a direct tar
44 had higher rates of dental anomalies in the maxillary arch than did controls for primary (21% vs. 4%
46 osition in arch), dental arch (mandibular or maxillary), arch location (anterior or posterior), smoki
48 derlying neuroepithelium, the mandibular and maxillary arches, including both their mesenchymal and e
51 vestigated peripheral axons of the zebrafish maxillary barbel (ZMB), an optically clear sensory appen
53 years; mean age: 58 +/- 9.2 years) requiring maxillary bone reconstruction prior to implant placement
54 revealed that 2 lesions were situated in the maxillary bone, 2 in the frontal, and 1 in the zygoma, a
55 We have used a rat model of injury to the maxillary branch (V2) of the trigeminal nerve to produce
56 e subsequently underwent embolization of the maxillary branch of the left ECA using Embozene(R) Micro
57 oth recession defects, oral sites other than maxillary canine and premolar teeth, and Miller Class II
58 associated with a dental abnormality called maxillary canine-tooth mesioversion (MCM) (P = 1.53 x 10
60 the amount of root coverage achievable, with maxillary canines and incisors being associated with the
62 ooth-bound implant-supported restorations in maxillary central incisor sites tends to exhibit an atro
63 phy-based finite element models comprising a maxillary central incisor socket and 4.5 x 13 mm outer-d
64 ular plane angle, and the inclination of the maxillary central incisor were significantly correlated
66 issing the mandibular second molar and their maxillary central incisors are most susceptible to micro
67 us surface involvement being highest for the maxillary central incisors, followed by maxillary poster
69 able outcomes after regenerative therapy for maxillary Class III furcation defects are limited to cli
70 Evidence supporting regenerative therapy in maxillary Class III furcation defects in maxillary molar
71 ll have a modified piercing-sucking mandible-maxillary complex that allows them to drain fluids from
75 ere characterised in both the mandibular and maxillary dentition by a loss of the permanent canines,
79 pulsed electrical currents to ophthalmic and maxillary divisions of the right trigeminal nerve and ce
81 hibited a significantly higher percentage of maxillary epithelium in contact with mandibular epitheli
86 ogically and clinically for the treatment of maxillary facial or interproximal and mandibular facial
87 ed regenerative therapy for the treatment of maxillary facial, mesial, distal, and mandibular facial
90 e of a lambdoidal junction (formed where the maxillary first arch meets the frontonasal processes) in
91 e, age-dependent expansion of the PDL at the maxillary first molar (M1) furcation area was observed.
92 ex was 89.3% and 100% in the MB roots of the maxillary first molars and in the M roots of the mandibu
93 n canals were located in the MB roots of the maxillary first molars and in the M roots of the mandibu
94 in the distal surface of the MB roots of the maxillary first molars and the M roots of the mandibular
95 tion about the mesiobuccal (MB) roots of the maxillary first molars and the mesial (M) roots of the m
96 us defects were created by extraction of the maxillary first molars in C57BL/6 mice, and the sockets
99 the palatal gingival tissues adjacent to the maxillary first molars three times per week for 4 weeks.
106 We found that, in the turtle, mandibular and maxillary ganglion neuron rostrocaudal segregation and t
107 AF) techniques in the treatment of localized maxillary gingival recession (GR) defects, 1 and 5 years
119 incisive canal and the roots of the central maxillary incisors should be kept in mind during dental-
120 ed to a class V-like lesion, and 1 side of 3 maxillary incisors was adapted with recombinant CPNE7 pr
123 olar bone caused by tooth movement after the maxillary incisors were retracted and intruded during or
126 at FPD treatment in posterior mandibular and maxillary jaws with NDIs was as reliable as with SDIs, a
129 two maxillary left second premolars and one maxillary left first molar occurs after sinus-augmentati
134 is case series, loss of pulp vitality of two maxillary left second premolars and one maxillary left f
135 Among treated patients the CoA segment (the maxillary length) and the ANB angle (the antero-posterio
137 mandibular arch-derived structures into more maxillary-like structures, indicating a loss of NCC iden
141 Bmp4 significantly reduced the penetrance of maxillary malformation in both Six1 (-/-) and Six1 (-/-)
143 n addition to ectopic Dlx5 expression at the maxillary-mandibular junction as recently reported in E1
145 one impressions were taken of mandibular and maxillary master casts, and eight stone replicas of each
146 ar development arrested at the bud stage and maxillary molar development arrested at the bud-to-cap t
151 abundantly expressed in the mandibular than maxillary molar mesenchyme in wild-type embryos and that
153 the mandibular molar tooth germs while their maxillary molar tooth germs completed morphogenesis.
154 Runx2(-/-) mutant mice, both mandibular and maxillary molar tooth germs progressed to the early bell
155 ly higher levels of Dkk2 than the developing maxillary molar tooth mesenchyme, these data indicate th
156 opmental arrest at the bud stage but allowed maxillary molars and incisors to develop to mineralized
157 in maxillary Class III furcation defects in maxillary molars is limited to clinical case reports.
159 successfully induced, healing of extraction maxillary molars was examined in 40 female Sprague Dawle
160 Finite element (FE) models of the first maxillary molars were constructed from muCT scans to cal
166 facial and interproximal Class I defects in maxillary molars; 2) facial and lingual Class I defects
167 interproximal Class II furcation defects in maxillary molars; 4) facial and lingual Class II furcati
168 ar molars; 5) Class III furcation defects in maxillary molars; 6) Class III furcation defects in mand
170 omeres (tritocerebrum, mandibular neuromere, maxillary neuromere, labial neuromere) of the SEZ at all
171 ar ganglion neurons are located rostrally to maxillary neurons, with some intermingling, supporting p
173 Adults scheduled to receive bone grafting in maxillary, non-molar, single-tooth extraction sites were
175 creened the olfactory sensory neurons of the maxillary palp (MP-OSNs) using a large number of natural
180 itors of the CO(2)-neuron were tested on the maxillary palps in these two species by single-sensillum
182 These neurons project from the antennae and maxillary palps to the antennal lobe (AL) and from the l
185 mandibular patterning defect resulting in a maxillary phenotype (i.e., homeotic transformation).
189 Experimental gingivitis was induced in one maxillary posterior sextant in nine healthy individuals.
191 furcation defects were surgically created in maxillary premolar teeth in adult, female, mini-pigs and
195 ateral and medial nasal processes and of the maxillary process of the first branchial arch are integr
197 ficantly retarded outgrowth of the nasal and maxillary processes due to reduced proliferation of mese
198 growth and morphogenesis bring the nasal and maxillary processes into contact, and the epithelia co-m
199 ls (activates beta-catenin pathway) into the maxillary prominence or by knocking down endogenous WNT1
200 RCAS::WNT11 retrovirus was injected into the maxillary prominence, and the majority of embryos develo
201 which EDNRA is ectopically activated in the maxillary prominence, resulting in a maxillary to mandib
202 aled that WNT11 prevented lengthening of the maxillary prominence, which was due in part to decreased
203 shelves from the oral side of the embryonic maxillary prominences, elevation of the initially vertic
204 bilateral palatal shelves (PS), arising from maxillary prominences, fuse at the midline, forming the
205 elop as outgrowths from the medial nasal and maxillary prominences, respectively, remodel and fuse to
207 e percentage of TLR4-positive neurons in the maxillary region and among the neurons innervating infla
208 R4 was detected in 19% of the neurons in the maxillary region of TG and in 29% of neurons innervating
211 A is an important structure in the posterior maxillary region; the clinician should be aware of its l
214 bone thickness at 3 mm from the CEJ for the maxillary right central incisor was 1.41 mm and for the
215 was subperiosteally injected into the murine maxillary right diastema between the incisor and the fir
216 red-breeder rats underwent extraction of the maxillary right first molar and standard surgical defect
217 for 5 min/day on the occlusal surface of the maxillary right first molar at a very low magnitude of l
220 xillary left first molars (PDSG and PDCimG); maxillary right molars were used as non-ligature control
224 gatures were placed subgingivally around the maxillary second molars and remained there for 2 weeks b
228 atures (size 7-0) were tied around the mouse maxillary second molars on day 0, followed by the inject
233 are the thickness of the lateral wall of the maxillary sinus (TLWMS) and the thickness of the Schneid
234 riptions of sinus involvement other than the maxillary sinus add to the variability of presentation.
235 All bioglass and/or allograft placed in the maxillary sinus after the osteotome technique underwent
236 al findings and mucosal abnormalities of the maxillary sinus among dental patients, using cone-beam c
237 distances from lower margin to the floor of maxillary sinus and alveolar crest in the 1(st) molar an
239 ery (PSAA) is located on the lateral wall of maxillary sinus and may become injured during such surgi
240 s sites with <10 mm between the floor of the maxillary sinus and the alveolar crest were selected.
242 e meatus cannot be used as a surrogate for a maxillary sinus aspirate in children with ABS, although
244 ospective record review was performed of all maxillary sinus augmentation cases performed during the
247 rforation is the most common complication of maxillary sinus augmentation procedures and has been ass
249 around implants placed in sites treated with maxillary sinus augmentation using anorganic bovine bone
252 ient with radio- and chemotherapy refractory maxillary sinus carcinoma to gauge the progression of th
256 of the present study, it was concluded that maxillary sinus elevation with 100% ABB gives predictabl
258 resent study is to investigate the effect of maxillary sinus floor augmentation on sinus membrane thi
259 s in membrane thickness were evaluated in 65 maxillary sinus floor augmentation procedures via a late
266 ivo were higher than the results in vitro in maxillary sinus volumes with a ratio of 1.05 +/- 0.01 (m
277 bullosa was connected with larger volume of maxillary sinuses (right sinus: p=0.005; left sinus: p=0
278 mplantitis in implants inserted in augmented maxillary sinuses and to analyze possible risk factors.
280 s) with 315 implants inserted into augmented maxillary sinuses with a follow-up ranging from 1 to 18
282 core-based model including the nasal cavity, maxillary sinuses, ethmoid air cells, sphenoid sinus and
286 There was higher incidence of bilateral maxillary sinusitis in patients with septal deviation (p
291 in the maxillary prominence, resulting in a maxillary to mandibular transformation, suggesting that
293 healthy adults requiring restoration of one maxillary tooth were randomized in a 1:2 ratio to receiv
297 , lateral-, superficial-, deep-palate or the maxillary tuberosity) can affect the graft shape and its
300 /-) mice exhibited partial transformation of maxillary zygomatic bone into a mandibular condyle-like