ライフサイエンスコーパス: mean arterial pressure

1 ed with radiotelemetry devices for recording mean arterial pressure.

2 ized this exposure to a 5 mm Hg reduction in mean arterial pressure.

3 were adjusted for age, body mass index, and mean arterial pressure.

4 and evoke a pressor reflex known to increase mean arterial pressure.

5 d provides prognostic utility beyond that of mean arterial pressure.

6 stolic BP, diastolic BP, pulse pressure, and mean arterial pressure.

7 se cardiovascular outcomes, independently of mean arterial pressure.

8 ds and a norepinephrine infusion to maintain mean arterial pressure.

9 hood systolic or diastolic blood pressure or mean arterial pressure.

10 The primary outcome was 24-hour mean arterial pressure.

11 required norepinephrine to maintain adequate mean arterial pressure.

12 Our primary outcome was 24-hour mean arterial pressure.

13 l hypertension but without MetS for the same mean arterial pressure.

14 al patients in whom norepinephrine increased mean arterial pressure.

15 +/- 767 dynes; p < 0.001) with no effects on mean arterial pressure.

16 the echocardiographic cardiac index and the mean arterial pressure.

17 ventricular stroke volume without affecting mean arterial pressure.

18 ty is affected by baseline serum lactate and mean arterial pressure.

19 ual mean arterial pressure was above optimal mean arterial pressure.

20 found this correlated with post-brain death mean arterial pressures.

21 significantly associated with diastolic and mean arterial pressures.

22 , waist circumference (-1.1 to -1.9 cm), and mean arterial pressure (0.0 to -1.1 mm Hg) at 6 months a

23 [95% CI -0.01, 0.03]; p = 0.36; n = 32,961); mean arterial pressure (-0.06 mm Hg [95% CI -0.19, 0.07]

24 it, -8.7, -5.1]; p(group) < 0.0001), similar mean arterial pressure (-1.1 mm Hg [95% confidence limit

25 g adequate intravascular volume and adequate mean arterial pressure (1C); avoiding use of intravenous

26 phrine by 0.04 +/- 0.02 mug.kg.min increased mean arterial pressure 20 mm Hg in all patients.

27 amounts of crystalloid to maintain adequate mean arterial pressure (2C) and the avoidance of hetasta

28 Mice underwent 90 minutes of shock (mean arterial pressure 30 mm Hg) and resuscitation via f

29 essure (23+/-2 vs. 10+/-3 mmHg, p=0.006) and mean arterial pressure (37+/-1 vs. 24+/-2 mmHg, p=0.006)

30 caine, and Mg generated significantly higher mean arterial pressure (48 mm Hg [95% CI, 44-52] vs 33 m

31 e (-17 mm Hg; 95% CI, -25 to -8; p < 0.001), mean arterial pressure (-7 mm Hg; 95% CI, -12 to -1; p =

32 hemodynamic parameters except a decrease in mean arterial pressure (-7 mm Hg; p = 0.041) and in syst

33 dynamic variables were relatively preserved (mean arterial pressure 70 [65-77] mm Hg, cardiac index 3

34 line, patients with RAI presented with lower mean arterial pressure (76 +/- 12 versus 83 +/- 14 mmHg,

35 (23 +/- 8 vs 17 +/- 7; P < .0001) and lower mean arterial pressure (81 +/- 16 vs 85 +/- 15 mm Hg; P

36 CPAP reduced baseline mean arterial pressure (94 +/- 2 vs. 89 +/- 2 mm Hg, P =

37 5% CI: 0.4, 1.7; p = 0.001), 0.8-mmHg higher mean arterial pressure (95% CI: 0.2, 1.4; p = 0.01), and

38 91) and COMACARE (Carbon Dioxide, Oxygen and Mean Arterial Pressure After Cardiac Arrest and Resuscit

39 In contrast, an elevated mean arterial pressure along with increased central and

40 We performed a comprehensive time-weighted mean arterial pressure analysis (time-weighted-average-m

41 iotensin II caused a significant increase in mean arterial pressure and a rapid reduction in catechol

42 d 3) assess the relationship between optimal mean arterial pressure and brain tissue oxygenation.

43 correlation was observed between changes in mean arterial pressure and cardiac index (r = 0.035, p =

44 were assessed for each time-weighted-average-mean arterial pressure and cumulative-time-below mean ar

45 ial pressure analysis (time-weighted-average-mean arterial pressure and cumulative-time-below various

46 gnancy-induced hypertension, ouabain reduced mean arterial pressure and enhanced placental HSP27 phos

47 e EPR and the hypoxia-induced CR (O(2) -CR), mean arterial pressure and heart rate were significantly

48 Cardiac work was estimated as the product of mean arterial pressure and heart rate.

49 DD and without LVDD, had significantly lower mean arterial pressure and higher Model for End-Stage Li

50 he association between time-weighted-average-mean arterial pressure and ICU-mortality for each thresh

51 serin in RN-NSC-grafted rats reduced resting mean arterial pressure and increased heart rate in all b

52 ient variability in the relationship between mean arterial pressure and indices of brain oxygenation.

53 The dynamic information in continuous mean arterial pressure and intracranial pressure monitor

54 eries summary statistics of minute-by-minute mean arterial pressure and intracranial pressure.

55 istic Organ Dysfunction-2 score now includes mean arterial pressure and lactatemia in the cardiovascu

56 An inverse relationship between mean arterial pressure and mortality was identified (p =

57 Low mean arterial pressure and need for high doses of vasopr

58 C) was calculated as laser-Doppler flowmetry/mean arterial pressure and normalized to maximum.

59 on increased cardiac index, whereas reducing mean arterial pressure and peripheral vascular resistanc

60 Mean arterial pressure and PP were continuously recorded

61 re were no differences between groups in the mean arterial pressure and R-R interval responses to non

62 ith increased risk for incident CVD, whereas mean arterial pressure and relative wave reflection (cor

63 Baseline mean arterial pressure and renal blood flow were similar

64 ally inhibited the effects of vasopressin on mean arterial pressure and significantly reduced the eff

65 r (quadratic) association between the lowest mean arterial pressure and the primary outcome of myocar

66 lated the 0- to 6-hour time-weighted average mean arterial pressure and used multivariable logistic r

67 s per minute; P<0.01) despite a reduction in mean arterial pressure and was inversely related to puls

68 ion between increasing time-weighted average mean arterial pressures and good neurologic outcome, def

69 ic and diastolic blood pressure, 22 SNPs for mean arterial pressure, and 10 SNPs for pulse pressure)

70 re reactivity index and identify the optimal mean arterial pressure, and 3) assess the relationship b

71 raction, even after adjustment for sex, age, mean arterial pressure, and BMI.

72 on isolated aortic rings, cardiac function, mean arterial pressure, and both the renal vascular perf

73 These results were adjusted for age, sex, mean arterial pressure, and cardiovascular risk factors.

74 nt CVD with forward pressure wave amplitude, mean arterial pressure, and global reflection coefficien

75 as a percentage of total blood volume (TBV), mean arterial pressure, and heart rate, which were recor

76 ral biomarkers, such as WBC, oxygen content, mean arterial pressure, and heart rate, yielded estimati

77 actice, uses shock index as a substitute for mean arterial pressure, and incorporates serum lactate a

78 ose-dependently decreased PP, did not modify mean arterial pressure, and increased SVR.

79 anial pressure, cerebral perfusion pressure, mean arterial pressure, and jugular venous bulb oxygen s

80 Heart rate, mean arterial pressure, and left ventricular work (2-dim

81 ic blood pressure, diastolic blood pressure, mean arterial pressure, and pulse pressure from the Inte

82 ere performed for systolic BP, diastolic BP, mean arterial pressure, and pulse pressure.

83 = 17) had larger reductions in diastolic BP, mean arterial pressure, and PWV (-2.24 +/- 1.31 mm Hg, -

84 udy is to describe changes in cardiac index, mean arterial pressure, and their relationship to other

85 Heart rate, mean arterial pressure, and total peripheral resistance

86 graphy (EEG) power; (3) a modest decrease in mean arterial pressure; and (4) a progressive shift of t

87 As the patients' actual mean arterial pressure approached optimal mean arterial

88 Every 10 mmHg drop from baseline in mean arterial pressure associated with a 3% increase in

89 sive rats displayed significant reduction in mean arterial pressure associated with attenuation of bo

90 There was no difference in mean arterial pressure at 1, 24, or 48 hours between gro

91 On average, the 24-hour mean arterial pressure at 12 weeks was lower in the grou

92 uent blood removal/retransfusion to maintain mean arterial pressure at 30 mm Hg).

93 of the blood volume, subsequent titration of mean arterial pressure at 35 mm Hg), anesthetized and in

94 Mean arterial pressure at 6 hours was 72.2 mm Hg in the

95 rent guidelines, which recommend maintaining mean arterial pressure at 85 to 90mm Hg for a week after

96 ary end point was a response with respect to mean arterial pressure at hour 3 after the start of infu

97 ) at OGTT, maternal height at OGTT, maternal mean arterial pressure at OGTT, maternal smoking and dri

98 itation, norepinephrine titrated to maintain mean arterial pressure at preshock values, mechanical ve

99 E, blood pressure targets were not achieved (mean arterial pressure at study end: NE: 81 mm Hg [76-85

100 en groups in the median cumulative time with mean arterial pressure below 60 mm Hg (7 vs 7 minutes; d

101 n arterial pressure: for 1 mm Hg decrease in mean arterial pressure below 75, 70, 65, 60, and 55 mm H

102 ynchronized ventilation elicited the highest mean arterial pressure, best oxygenation, and a normal m

103 ower systolic BP (beta=-4.11; P=2.8x10(-4)), mean arterial pressure (beta=-3.50; P=8.9x10(-6)), and r

104 was no significant difference in the 24-hour mean arterial pressure between the control group and the

105 to 10, consisting of age, oxygen saturation, mean arterial pressure, blood urea nitrogen, C-Reactive

106 flow is not attributable to factors such as mean arterial pressure, blood viscosity and pH.

107 al mean arterial pressure approached optimal mean arterial pressure, brain tissue oxygenation increas

108 Advancing age and lower mean arterial pressure, but not the presence of a transt

109 L-NAME increased mean arterial pressure by approximately 17 mm Hg in both

110 Cocaine increased mean arterial pressure (by 14+/-2 mm Hg [mean+/-SE]), he

111 Time-weighted-average-mean arterial pressure captures both the severity and du

112 Mean arterial pressure, cardiac output, cerebral blood f

113 PP, mean arterial pressure, cardiac output, SVR, and ascites

114 ic blood pressure, diastolic blood pressure, mean arterial pressure, carotid intima-media thickness a

115 Although mean arterial pressure, characteristic aortic impedance,

116 ased NOS3 and GUCY1A3 expression and reduced mean arterial pressure, combined them into a genetic sco

117 at the 25th percentile (78 mm Hg) of lowest mean arterial pressure compared with at the median of 87

118 e RDN than the intact group (2-month fall in mean arterial pressure: control-intact, -10 +/- 1 mm Hg;

119 Coronary vascular resistance (CVR; mean arterial pressure/coronary blood velocity) was used

120 Mean arterial pressure decreased similarly during endoto

121 Hg and 170 mm Hg of steady-state changes in mean arterial pressure, defined as static CA.

122 ssation, normalization of serum lactate, and mean arterial pressure did not differ among groups.

123 ment for first trimester body mass index and mean arterial pressure, differences in intima thickness

124 Work rate and mean arterial pressure during exercise were similar in c

125 We additionally collected mean arterial pressure, end-tidal CO2, and temperature.

126 (ASIC) activation and reflexively increases mean arterial pressure; endomorphin release is also incr

127 ociated with hemodynamic instability (higher mean arterial pressure extrema points frequencies were a

128 Episodes of hypotension were common, with mean arterial pressure falling by a median of 22 mmHg (i

129 If, despite fluid management, mean arterial pressure fell by more than 10 mm Hg from b

130 In response to hemorrhage, mean arterial pressure fell in all groups, with the fall

131 , HS/CR, or HS/CR+MC-2 (HS = 40% of baseline mean arterial pressure for 60 minutes; CR = return of sh

132 ists also nominated an appropriate range for mean arterial pressure for each patient during surgery.

133 Time-weighted-average-mean arterial pressure: for 1 mm Hg decrease in mean art

134 V amplitude, and 1 ms pulse width, restoring mean arterial pressure from 0 to 37 mmHg.

135 low from 351 +/- 55 to 182 +/- 67 mL/min and mean arterial pressure from 96.7 +/- 18.2 to 41.5 +/- 4.

136 In 5-year-old female uni-x and sham sheep, mean arterial pressure, glomerular filtration rate, and

137 e Inotropic Score greater than 50 to reach a mean arterial pressure greater than 65 mm Hg despite ade

138 included the following physiologic targets: mean arterial pressure greater than 70 mm Hg, cerebral p

139 th good neurologic outcome at a threshold of mean arterial pressure greater than 70 mm Hg.

140 eater than or equal to 20 mm Hg, decrease in mean arterial pressure greater than or equal to 10 mm Hg

141 ned as the need for vasopressors to maintain mean arterial pressure greater than or equal to 65 mm Hg

142 This threshold (mean arterial pressure > 70 mm Hg) had the strongest ass

143 as the first-choice vasopressor to maintain mean arterial pressure >/= 65 mm Hg (1B); epinephrine wh

144 Mean arterial pressure >/=65 mm Hg and central venous pr

145 ature management at 33 degrees C with target mean arterial pressure >/=65 mm Hg is associated with in

146 sure-responders were defined as CI >=10% and mean arterial pressure >=10%, respectively.

147 on and treatment with vasopressors targeting mean arterial pressure (>/=65 mm Hg) and blood transfusi

148 ts with lower baseline diastolic BP (DBP) or mean arterial pressure had more progression of subcortic

149 3) was associated with incident CVD, whereas mean arterial pressure (hazard ratio, 1.10; 95% confiden

150 Mean arterial pressure, heart rate, and lactate were reg

151 We measured mean arterial pressure, heart rate, central venous press

152 and objective physical variables (including mean arterial pressure, heart rate, respiratory rate, an

153 alization (HR: 23.2; P = 0.01), and baseline mean arterial pressure (HR: 0.92; P = 0.01) were found t

154 An increased mean arterial pressure in addition to resolution of tach

155 ationale: Exogenous angiotensin II increases mean arterial pressure in patients with catecholamine-re

156 ceptor agonist), caused a graded increase in mean arterial pressure in rats with sinoaortic denervati

157 ted basal blood pressure and acute change in mean arterial pressure in response to angiotensin II (An

158 difference in the primary outcome of 6-hour mean arterial pressure in septic shock patients receivin

159 effective arterial elastance despite similar mean arterial pressures in control subjects.

160 At baseline HR, diastolic and mean arterial pressures in IST and POTS were higher vers

161 re observed in CIH and HC rats, although the mean arterial pressure increase was lower after CIH.

162 After infusion of angiotensin II, mean arterial pressure increased by 61.6 mmHg in MD-NOS1

163 After fluid challenge, mean arterial pressure increased from 73 mm Hg (interqua

164 Mean arterial pressure increased significantly in MD-NOS

165 Mean arterial pressure initially decreased further under

166 each eye at each of the 8 time points as 2/3(mean arterial pressure-intraocular pressure [IOP]).

167 t, and perfusion within proximity of optimal mean arterial pressure is associated with increased brai

168 Mean arterial pressure is critically important in patien

169 A significant increase in mean arterial pressure is observed in early adulthood in

170 ot be aggressively treated in this period if mean arterial pressure, lactate clearance, and diuresis

171 , hospital location, era, systolic pressure, mean arterial pressure, lactate, bundle compliance, amou

172 Outcomes included 24-hour survival rates, mean arterial pressure, lactate, hemoglobin, and estimat

173 Exclusion criteria for both groups were mean arterial pressure less than 60 mm Hg, contraindicat

174 ours, when shock was present, animals with a mean arterial pressure less than 65 mm Hg (n = 6) had si

175 , significant associations only remained for mean arterial pressure less than 65 mm Hg (odds ratio, 1

176 o mortality included: a single occurrence of mean arterial pressure less than 65 mm Hg (p = 0.0051) o

177 arterial pressure less than 80 mm Hg and any mean arterial pressure less than 70 mm Hg.

178 sensitivity analyses based on every hour of mean arterial pressure less than 80 mm Hg and any mean a

179 oup in which animals were exsanguinated to a mean arterial pressure level of 40 mm Hg during 30 minut

180 nation from the 30th to the 60th minute to a mean arterial pressure level of 40 mm Hg; or control gro

181 Pressure reactivity index can yield optimal mean arterial pressure, lower and upper limit of autoreg

182 ctivity index-based determination of optimal mean arterial pressure, lower and upper limit of autoreg

183 The mean optimal mean arterial pressure, lower and upper of autoregulatio

184 study was a composite of severe hypotension (mean arterial pressure < 60 mm Hg) and bradycardia (hear

185 sion (systolic blood pressure </=90 mm Hg or mean arterial pressure </=65 mm Hg) presenting to the em

186 ly randomized to strict or usual BP control (mean arterial pressure </=92 mmHg or 102-107 mmHg, respe

187 hemodynamic deterioration with an intrinsic mean arterial pressure <60 mm Hg during a sustained epis

188 uded: albumin <2.5 g/dL, heart rate >90 bpm, mean arterial pressure <60 mmHg, white blood cell count

189 pressor titration suggest a universal target-mean arterial pressure (MAP) >65 mm Hg.

190 nalyses to identify the relationship between mean arterial pressure (MAP) and cerebral blood vessels'

191 pressor reflex (EPR) is defined by a rise in mean arterial pressure (MAP) and heart rate (HR) in resp

192 Mean arterial pressure (MAP) and OPP (systolic, diastoli

193 enome-wide gene-smoking interaction study of mean arterial pressure (MAP) and pulse pressure (PP) in

194 with systolic BP (SBP), diastolic BP (DBP), mean arterial pressure (MAP) and pulse pressure (PP), we

195 Moreover, inflammation increased maternal mean arterial pressure (MAP) and was associated with ren

196 Hypotension was defined as a mean arterial pressure (MAP) below 65 mm Hg for at least

197 ) and diastolic BP (DBP) were measured, with mean arterial pressure (MAP) calculated.

198 used to observe the systolic, diastolic, and mean arterial pressure (MAP) correlations.

199 The measured mean arterial pressure (MAP) exhibited measurable deviat

200 This study aimed to determine the optimal mean arterial pressure (MAP) in patients with AMI and sh

201 ow levels of estradiol-17beta (E2) increases mean arterial pressure (MAP) in young female Sprague-Daw

202 essment of CA during steady-state changes in mean arterial pressure (MAP) induced by intravenous infu

203 nship of Doppler BP to systolic BP (SBP) and mean arterial pressure (MAP) is uncertain and Doppler me

204 us females; however, i.c.v. leptin increased mean arterial pressure (MAP) only in males.

205 The nocturnal mean arterial pressure (MAP) was compared with the dayti

206 Mean arterial pressure (MAP) was defined as diastolic BP

207 re (SBP), diastolic blood pressure (DBP) and mean arterial pressure (MAP) were significantly (P < 0.0

208 Renal blood flow (RBF), mean arterial pressure (MAP), and heart rate (HR) were c

209 IOP, mean arterial pressure (MAP), and HR increased rapidly a

210 for Endstage Liver Disease (MELD) score, low mean arterial pressure (MAP), and non-SBP infections.

211 udied systolic BP (SBP), diastolic BP (DBP), mean arterial pressure (MAP), and pulse pressure (PP) av

212 with systolic BP (SBP), diastolic BP (DBP), mean arterial pressure (MAP), and pulse pressure (PP), w

213 erebral artery blood velocity (MCA V(mean)), mean arterial pressure (MAP), cardiac output (CO) and pa

214 Mean arterial pressure (MAP), heart rate (HR), BT, motor

215 lly low systolic blood pressure (SBP) and/or mean arterial pressure (MAP).

216 ic blood pressure, diastolic blood pressure, mean arterial pressure (MAP)], brachial artery blood flo

217 procedural increase of median SBP (+11%) and mean arterial pressure (MAP, +10%, both p < 0.001), and

218 rst 24-hours after ICU admission (median: 25 mean arterial pressure measurements per-patient).

219 he first 24 hrs of intracranial pressure and mean arterial pressure monitoring to known baseline risk

220 tics of continuous intracranial pressure and mean arterial pressure monitoring.

221 c expression of Kv1.5 channels), we measured mean arterial pressure, myocardial blood flow, myocardia

222 e arterial load parameters did not change in mean arterial pressure-nonresponders.

223 erebroventricular CMT-3 attenuated increased mean arterial pressure, normalized sympathetic activity,

224 ratio, 2.71; 95% CI, 1.67-4.39; p < 0.001), mean arterial pressure (odds ratio, 0.979; 95% CI, 0.963

225 morrhagic shock by blood withdrawn until the mean arterial pressure of 30 mm Hg and maintained at thi

226 igs (35-40 kg) were anesthetized and bled to mean arterial pressure of 35-40 mm Hg for 90 minutes, fo

227 chemic events rose rapidly below an absolute mean arterial pressure of 60 mmHg.

228 d by a vasopressor requirement to maintain a mean arterial pressure of 65 mm Hg or greater and serum

229 4, p = 0.009) compared with animals with an mean arterial pressure of 65-70 mm Hg (n = 4).

230 e mean daily dose of vasopressor to insure a mean arterial pressure of 65-75 mm Hg.

231 .kg.min for more than 24 hours to maintain a mean arterial pressure of 70 mm Hg or greater.

232 Targeting a mean arterial pressure of 80 to 85 mm Hg, as compared wi

233 iving Sepsis Campaign recommends targeting a mean arterial pressure of at least 65 mm Hg during initi

234 These data suggest that maintaining a mean arterial pressure of greater than 65 mm Hg may be a

235 Hypotension was defined as a decrease in the mean arterial pressure of greater than or equal to 15% c

236 sed to assess the association between lowest mean arterial pressure on each intensive care day, consi

237 In multivariate linear regression models for mean arterial pressure or SVRI in patients with severe m

238 m creatinine, bilirubin or albumin, baseline mean arterial pressure, or study design, size or time pe

239 e, in patients with higher baseline BP (DBP, mean arterial pressure, or systolic BP), those with decl

240 uration and magnitude of clinically observed mean arterial pressure outside optimal mean arterial blo

241 rates (p < 0.05), cardiac index (p < 0.05), mean arterial pressure (p < 0.05), PaO2/FIO2 (p < 0.05),

242 those in the crystalloid group, had a higher mean arterial pressure (P=0.03) and lower net fluid bala

243 ng amounts of hypotension (defined by lowest mean arterial pressures per day) were strongly associate

244 indicated that independent of differences in mean arterial pressure, pH and blood viscosity, race acc

245 etion with CHD risk tended to be modified by mean arterial pressure (Pinteraction=0.08) and was modif

246 atin use, blood pressure medication use, and mean arterial pressure, PP quartile was still associated

247 In vehicle-treated sheep, mean arterial pressure progressively declined from 25 to

248 perature were not correlated with changes in mean arterial pressure (r = 0.0002; p = 0.85).

249 ality (p = 0.76) associated with hypotensive mean arterial pressure readings (</=60 mm Hg) were indep

250 is study was to examine the effect of RDN on mean arterial pressure, renal function, and the reflex r

251 f the alpha2-adrenergic agonist clonidine on mean arterial pressure, renal sympathetic nerve activity

252 Cardiac index responders and mean arterial pressure-responders were defined as CI >=1

253 The mean arterial pressure response to fluid bolus in cardia

254 o repeat fluid bolus based solely on lack of mean arterial pressure response to the initial fluid, si

255 Cardiac index-responsiveness and mean arterial pressure-responsiveness rates were 33% and

256 hip between cardiac index-responsiveness and mean arterial pressure-responsiveness.

257 Mean arterial pressure SD score increased with HD only.

258 Factors associated with higher cIMT and mean arterial pressure SD-scores were HD group, higher u

259 norepinephrine required to maintain a target mean arterial pressure; secondary outcomes included hemo

260 ressure and invasive arterial blood pressure mean arterial pressures showed better agreement; acute k

261 Mean arterial pressure, skin blood flow via laser-Dopple

262 e treatment prevented the further decline in mean arterial pressure, substantially reduced heart rate

263 CFH is associated with mean arterial pressure, SVRI, and peripheral perfusion i

264 ease in maximum negative dP/dt (dP/dt(Min)), mean arterial pressure, systolic pressure, diastolic pre

265 on, fungal infection, vasopressor use, and a mean arterial pressure target of 80-85 mm Hg.

266 septic shock to undergo resuscitation with a mean arterial pressure target of either 80 to 85 mm Hg (

267 d trials are needed to determine the optimal mean arterial pressure-targets in this patient populatio

268 ger reduction in systolic blood pressure and mean arterial pressure than did either CPAP or weight lo

269 After the rise in mean arterial pressure, there was a significant increase

270 arterial pressure and cumulative-time-below mean arterial pressure threshold (55, 60, 65, 70, and 75

271 severity and duration of hypotension below a mean arterial pressure threshold and cumulative-time-bel

272 e-time-below is the total time spent below a mean arterial pressure threshold.

273 l pressure and cumulative-time-below various mean arterial pressure-thresholds) during the first 24-h

274 arameters, grafting RN-NSCs restored resting mean arterial pressure to normal levels and remarkably a

275 ce index was then calculated as the ratio of mean arterial pressure to regional cerebral blood flow.

276 n be added to norepinephrine to either raise mean arterial pressure to target or to decrease norepine

277 of proteinuria were repeat transplantation, mean arterial pressure, transplant glomerulopathy, micro

278 , we increased noradrenaline dose to elevate mean arterial pressure up to 85-90 mm Hg before collecti

279 a threshold effect at time-weighted average mean arterial pressure value of 70 mm Hg.

280 (95% CI, 0.01-0.14) and between low and high mean arterial pressure was 0.05% (95% CI, 0.00-0.10).

281 Basal mean arterial pressure was 15 mm Hg higher and glomerula

282 Mean arterial pressure was 3.5 mm Hg (4%) higher (median

283 acetaminophen-induced hypotension, the nadir mean arterial pressure was 64 mm Hg (95% CI, 54-74).

284 relationship did not persist when the actual mean arterial pressure was above optimal mean arterial p

285 We found that time-weighted average mean arterial pressure was associated with good neurolog

286 Mean arterial pressure was reduced to 30 mm Hg for 90 mi

287 methyl-L-thiocitrulline (1 mumol/kg, IV) and mean arterial pressure was registered.

288 Mean arterial pressure was slightly higher in SHR but wi

289 The effect of vasopressin on mean arterial pressure was unaltered and that on renal v

290 Cutaneous vascular conductance (CVC = flux/mean arterial pressure) was expressed as a change from b

291 Changes in mean arterial pressure were closely correlated with decr

292 Leg blood flow and mean arterial pressure were determined, whereas leg vasc

293 diac index, left ventricular dimensions, and mean arterial pressure were measured using bilateral ven

294 The risk scores for blood pressure and mean arterial pressure were not associated with any of t

295 oke volume, cardiac output and reductions in mean arterial pressure were similar between age groups a

296 tibular otolith system and a decrease in the mean arterial pressure when a person stands up.

297 5 mins from a decrease in cardiac output and mean arterial pressure, whereas treated rats survived un

298 significant increases (P < 0.05; n = 7-8) in mean arterial pressure, which were generally accompanied

299 Increasing mean arterial pressure with noradrenaline in septic shoc

300 lepressin was titrated to raise and maintain mean arterial pressure within no less than 10 mm Hg from