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1 5-1.4% for distribution volume, and 0.0% for mean transit time.
2 3% for distribution volume, and 0.2-1.8% for mean transit time.
3 ose groups (45 and 30 mg), except for DCE CT mean transit time.
4 ividuals without CCSVI, without any delay in mean transit time.
5  distal pressure multiplied by the hyperemic mean transit time.
6 od flow, relative cerebral blood volume, and mean transit time.
7 al blood volume (6-33% decrease), and tissue mean transit time (10-54% increase) were observed in the
8 s, p = 0.03, respectively) and a decrease in mean transit time (31 +/- 8 vs 60 +/- 2 s, p = 0.04, res
9                                              Mean transit time and blood volume fraction were compara
10                             Brain perfusion (mean transit time and blood volume fraction) was compara
11 sing quantitative voxel based thresholds for mean transit time and cerebral blood volume (CBV).
12  striatal necrosis in an 8-month-old infant, mean transit time and cerebral blood volume were low rel
13 aminal lesions, striatal flow was normal but mean transit time and cerebral blood volume were low, co
14 lood brain barrier (BBB), reducing capillary mean transit time and GCRs.
15 usion coefficient, tissue oxygen saturation, mean transit time, and blood volume fraction in the cort
16 s per group); local brain oxygen saturation, mean transit time, and blood volume fraction were subseq
17 o calculate tissue blood flow, blood volume, mean transit time, and permeability-surface area product
18 asurement of tumor blood flow, blood volume, mean transit time, and permeability-surface area product
19                          Visual analysis and mean transit time calculation were performed on the rest
20 ime, elevated cerebral blood volume and high mean transit time/cerebral blood flow and cerebral blood
21 gged correlation and compared the results to mean transit time derived using bolus tracking.
22 ured) had low cerebral blood flow, prolonged mean transit time, elevated cerebral blood volume and hi
23                 The inverse of the hyperemic mean transit time has been shown to correlate with absol
24                The MS group showed prolonged mean transit time in the periventricular NAWM, as compar
25 raction in tumors and normal tissue, and for mean transit time in tumors; however, permeability value
26 monstrated by a 3-fold increase in ribosomal mean transit times in cell-free extracts from hibernator
27  (Pcs), right atrial pressure (Pra), and the mean transit time (inverse of blood flow [Tmn]), were me
28 .90) than did those on blood flow and tracer mean transit time maps (r approximately 0.6), likely as
29  as the difference between lesion volumes on mean transit time maps and DW images, divided by DW lesi
30  algorithm identifies hypoperfused tissue in mean transit time maps by simultaneously minimizing the
31 BV), absolute cerebral blood flow (CBF), and mean transit time (MTT) (referenced to an arterial input
32 volume (BV), and lower (>0.30 and >0.39) for mean transit time (MTT) and permeability surface area pr
33                           Parameter maps for mean transit time (MTT) and plasma flow (PF) were evalua
34                              The parenchymal mean transit time (MTT) is theoretically superior to oth
35 ved brain perfusion: hypoperfusion volume on mean transit time (MTT) map decrease >30% from baseline
36 fraction (PV), distribution volume (DV), and mean transit time (MTT) of gadopentetate dimeglumine.
37 the glomerular filtration rate (GFR) and the mean transit time (MTT) of the tracer for the vascular c
38 erive blood flow (BF), blood volume (BV) and mean transit time (MTT) perfusion maps.
39 f mean CBF, cerebral blood volume (CBV), and mean transit time (MTT) were determined between hemisphe
40          Blood flow (BF), blood volume (BV), mean transit time (MTT), and capillary permeability-surf
41          Blood flow (BF), blood volume (BV), mean transit time (MTT), and permeability-surface area p
42          Blood flow (BF), blood volume (BV), mean transit time (MTT), and permeability-surface area p
43                    Distribution volume (DV), mean transit time (MTT), and portal fraction (PF) of blo
44 culate tissue blood flow (BF), blood volume, mean transit time (MTT), and vascular permeability-surfa
45                    Blood flow, blood volume, mean transit time (MTT), permeability-surface area produ
46 ity, blood volume (BV), blood flow (BF), and mean transit time (MTT), were calculated at the primary
47  parameters, pulmonary blood flow (PBF), and mean transit time (MTT).
48 flow (CBF), cerebral blood volume (CBV), and mean transit time (MTT).
49 ncluding Blood Flow (BF), Blood Volume (BV), Mean Transit Time (MTT)] and permeability parameters [in
50 ed software, it is possible to calculate the mean transit time of room-temperature saline injected do
51 farct volume (SE = 80%; SP = 97%), Increased mean transit time on PCT was predictive of the tissue at
52 , total blood flow, distribution volume, and mean transit time) parameters were calculated by placing
53 o determine tissue blood flow, blood volume, mean transit time, permeability, and hepatic arterial fr
54                                  Blood flow, mean transit time, permeability, and hepatic arterial fr
55 sure divided by the inverse of the hyperemic mean transit time provides an index of microcirculatory
56 ebral blood flow, cerebral blood volume, and mean transit time, respectively.
57  Hg; balloon: 79 [75-93] mm Hg; P = .01) and mean transit time (sham: 0.39 [0.23-0.62] s; balloon: 0.
58  lower portal flow, distribution volume, and mean transit time than did the background liver (all P <
59 oronary pressure, whereas the inverse of the mean transit time to room temperature saline was used to
60 , and positive correlation was found between mean transit time values and disability scales in patien
61 volume (CBV), cerebral blood flow (CBF), and mean transit time were assessed with dynamic susceptibil