ライフサイエンスコーパス: mediastinal

1 ure rates in patients with and those without mediastinal abnormalities at preoperative PET were compa

2 ung parenchymal, airway, pleural, hilar, and mediastinal abnormalities systematically reviewed initia

3 for enteral nutrition (n = 18), drainage of mediastinal abscess (n = 4), gastric decompression (n =

4 thickness oesophageal segment destroyed by a mediastinal abscess and leading to direct communication

5 Mediastinal activity appears to be constant over time an

6 t computed tomography images showed enlarged mediastinal adenopathy with increased [(18)F]fluorodeoxy

7 vealed a 5-cm right upper lobe mass, without mediastinal adenopathy, and a 6-cm cystic mass in the sp

8 ecoming standard of care for the sampling of mediastinal adenopathy.

9 h EBUS-TBNA requires a detailed knowledge of mediastinal anatomy.

10 n they involve either side of the neck, with mediastinal and distant metastases.

11 PV, and accuracy of hyperdense non-calcified mediastinal and hilar lymph nodes, known as "brilliant l

12 jective noise at the level of the trachea on mediastinal and lung parenchymal images (P < .001) and n

13 tion of the pleural cavity rapidly activates mediastinal and pericardial FALCs.

14 scored as lymphatic type 1 (little or no T2 mediastinal and supraclavicular signal) to type 4 (T2 si

15 solidation (63%), pulmonary nodules (31.4%), mediastinal and/or hilar lymphadenopathy (23%), mass-lik

16 encing of precursor, primary (testicular and mediastinal) and chemoresistant metastatic human GCTs, w

17 pectoral, supraclavicular, internal mammary, mediastinal, and abdominal nodes.

18 lysed based on parenchymal, airway, pleural, mediastinal, and vascular sequelae of PTB.

19 Classical Hodgkin lymphoma and primary mediastinal B cell lymphoma (PMBCL) are related lymphoma

20 es: diffuse large B-cell lymphoma or primary mediastinal B-cell lymphoma (DLBCL/PMBCL; n = 28), low-g

21 f Blood, Ceriani et al introduce, in primary mediastinal B-cell lymphoma (PMBCL), a new prognostic fa

22 clerosis Hodgkin lymphoma (NSHL) and primary mediastinal B-cell lymphoma (PMBL) are the common types,

23 Primary mediastinal B-cell lymphoma (PMBL) is a rare but aggress

24 Primary mediastinal B-cell lymphoma (PMBL) is a subtype of diffu

25 79), Burkitt lymphoma (BL; n = 36), primary mediastinal B-cell lymphoma (PMBL; n = 12), B-cell lines

26 btype showed biological overlap with primary mediastinal B-cell lymphoma and conferred excellent prog

27 Primary mediastinal B-cell lymphoma is a distinct subtype of dif

28 d histology (diffuse large B-cell lymphoma v mediastinal B-cell lymphoma v Burkitt lymphoma or Burkit

29 nsformed from any indolent lymphoma, primary mediastinal B-cell lymphoma, and follicular lymphoma gra

30 uding diffuse large B-cell lymphoma, primary mediastinal B-cell lymphoma, and transformed follicular

31 with diffuse large B-cell lymphoma, primary mediastinal B-cell lymphoma, or transformed follicular l

32 herapy in 51 patients with untreated primary mediastinal B-cell lymphoma.

33 ed for radiotherapy in patients with primary mediastinal B-cell lymphoma.

34 cases were molecularly classified as primary mediastinal B-cell lymphoma.

35 uding classical Hodgkin lymphoma and primary mediastinal B-cell lymphoma.

36 th diffuse large B-cell lymphoma and primary mediastinal B-cell lymphoma.

37 cell lymphoma, Hodgkin lymphoma, and primary mediastinal B-cell lymphoma.

38 Mediastinal B-cell lymphomas present in the mediastinum

39 of 6 with gray zone, 1 CR of 6 with primary mediastinal B-cell, and 1 CR of 3 with posttransplant ly

40 Mediastinal biopsy was the most frequent (26 subjects).

41 Mediastinal bleeding is common following pediatric cardi

42 ajor complications and postoperative 24-hour mediastinal blood loss.

43 or with residual [18F]FDG activity below the mediastinal blood pool (MBP) uptake.

44 (68)Ga-DOTATOC uptake was higher in mediastinal blood pool at the 1-h time point (P = 0.018)

45 Patients with stage IA to IIA HL and no mediastinal bulk underwent PET assessment after three cy

46 als involving patients without B symptoms or mediastinal bulk, a score of 5 rather than a positive PE

47 age I and II (<3 nodal sites, no B symptoms, mediastinal bulk, or extranodal extension) enrolled betw

48 Air in all mediastinal compartments was also associated with increa

49 er, CT findings of posterior PNM, air in all mediastinal compartments, and concurrent hemothorax are

50 ertrophy of the bronchial arteries along the mediastinal course, diffuse thickening of the walls of n

51 ratification of early-stage HL patients with mediastinal disease and thus contribute to risk-adapted,

52 LDH level at diagnosis, mediastinal disease, and combined BM-positive/CNS-positi

53 thermore, among the patients with refractory mediastinal disease, our model distinguished those who w

54 ies, such as pulmonary fibrosis, pleural and mediastinal disease, solid lesions, bronchial disease, a

55 vs 21 [14%]), and respiratory, thoracic, and mediastinal disorders (13 [9%] vs 17 [12%]).

56 ssure on the neighboring lung parenchyma and mediastinal displacement.

57 s an overall difference in mean (SD) 24-hour mediastinal drain loss: cohort, 12.6 mL/kg (6.4); FC, 11

58 n, if so, management may be conservative but mediastinal drainage is important if significant extrava

59 D103(+) myeloid dendritic cells migrating to mediastinal draining lymph nodes and bearing migratory a

60 After TAC, enlarged heart mediastinal draining lymph nodes showed a high density o

61 trophils and monocytes and then delivered to mediastinal draining lymph nodes.

62 lated with an increase in donor cells in the mediastinal draining lymph nodes; increased lymphatic ve

63 re complicated by extensive subcutaneous and mediastinal emphysema that occurred without any obvious

64 a previous chest X-ray that showed bilateral mediastinal enlargement; for this purpose, enhanced ches

65 etion was confirmed in arterioles from human mediastinal fat in patients with essential hypertension

66 rticularly helpful in patients with positive mediastinal findings at preoperative PET.

67 rising in this setting genetically resembled mediastinal GCTs rather than de novo myeloid neoplasms.

68 The anterior mediastinal germ cell tumors (GCTs) are the most common

69 ymphoma (PMBL) are the common types, whereas mediastinal gray-zone lymphoma (MGZL) is extremely rare

70 s intermediate between PMBL and NSHL, called mediastinal gray-zone lymphomas, have been described.

71 For patients with stage I or II bulky mediastinal HL, no substantial statistically significant

72 nalysis in patients with stage I or II bulky mediastinal Hodgkin lymphoma (HL).

73 objective noise at the level of the aorta on mediastinal images (P = .507); (b) significantly higher

74 1279 Hodgkin lymphoma patients treated with mediastinal irradiation and quantified the standard inci

75 vors of Hodgkin's lymphoma (HL) who received mediastinal irradiation have an increased risk of corona

76 he excess risk of cardiac interventions from mediastinal irradiation.

77 d survivors of HL who survived 5 years after mediastinal irradiation.

78 Blood and mediastinal isolates were characterized as CA-MRSA by pu

79 Classical Hodgkin lymphoma (cHL) and mediastinal large B-cell lymphoma (MLBCL) are lymphoid m

80 Mediastinal large B-cell lymphoma (MLBL) represents 2% o

81 ssification of children and adolescents with mediastinal large B-cell lymphoma (MLBL), and highlight

82 Cure rates for primary mediastinal large B-cell lymphoma (PMBCL) have improved

83 Primary mediastinal large B-cell lymphoma (PMBCL) is a subtype o

84 The pathogenesis of primary mediastinal large B-cell lymphoma (PMBCL) is incompletel

85 Primary mediastinal large B-cell lymphoma (PMBL) cells depend on

86 Primary mediastinal large B-cell lymphoma (PMBL) represents a cl

87 natures to those of diffuse LBCL and primary mediastinal large B-cell lymphoma (PMBL).

88 ment options for relapsed/refractory primary mediastinal large B-cell lymphoma (rrPMBCL) are limited,

89 Patients with relapsed or refractory primary mediastinal large B-cell lymphoma (rrPMBCL) have a poor

90 Primary mediastinal large B-cell lymphomas (PMBLs) are aggressiv

91 ling molecules are down-regulated in primary mediastinal large B-cell lymphomas and Hodgkin's lymphom

92 ydatid presents as a fluid-density posterior mediastinal lesion on chest radiograph with destruction

93 est radiograph was suggestive of a posterior mediastinal lesion with fluid density and destruction of

94 f an elderly male with incidental finding of mediastinal lesion, which was initially thought to be an

95 In a total of 208 patients, 215 lung/mediastinal lesions (seven patients were biopsied twice)

96 For example, resolution modeling for mediastinal lesions and iterative deconvolution for lung

97 intraperitoneal inoculation, 1 to 8% of the mediastinal LN cells were infected.

98 of CD69+/CD103+ CD8+ T cells to the draining mediastinal LN via the lymphatic vessels, which we term

99 ns, and COX-2 inhibition markedly suppressed mediastinal LNM.

100 to have prognostic implications despite the mediastinal LNs being histologically negative.

101 ctive value for detecting lymph nodes in any mediastinal location and for patients without lymph node

102 sport of live bacteria from the lungs to the mediastinal lymph node (MDLN).

103 cell transfer, the T cells isolated from the mediastinal lymph node (med-LN) of aged animals exhibite

104 Mediastinal lymph node (MLN) enlargement on chest comput

105 atic proliferation were largely found in the mediastinal lymph node (mLN), rather than the airways; h

106 l recruitment mainly occurs in the posterior mediastinal lymph node (pMLN).

107 ium tuberculosis occurs in the lung-draining mediastinal lymph node and requires transport of M. tube

108 After stimulation of mediastinal lymph node and spleen cells with UV-inactiva

109 Mediastinal lymph node and spleen epitope-specific CD8(+

110 significantly enhanced in the lung-draining mediastinal lymph node and spleen, and there is an incre

111 aortic surgery, the patient had undergone a mediastinal lymph node biopsy.

112 d the majority of prion-bearing cells in the mediastinal lymph node by six hours, indicating intranod

113 Mediastinal lymph node cells from Flt3L-treated mice sec

114 The in vitro cytokine secretion of mediastinal lymph node cells was determined using ELISA.

115 igh-affinity ligand CD155 was upregulated in mediastinal lymph node dendritic cells from allergic mic

116 m mass in the right upper lobe with multiple mediastinal lymph node disease ( Fig 1 ).

117 ned in a 1:1 ratio to SABR or lobectomy with mediastinal lymph node dissection or sampling.

118 l-cell lung cancer (NSCLC) is lobectomy with mediastinal lymph node dissection or sampling.

119 He then undergoes right upper lobectomy and mediastinal lymph node dissection, which demonstrate no

120 Analysis of a mediastinal lymph node from one patient highlighted the

121 omography (FDG-PET/CT) imaging for detecting mediastinal lymph node involvement in patients with pote

122 l outcomes, including pathologic evidence of mediastinal lymph node involvement, distant metastasis,

123 Sensitivity for detecting mediastinal lymph node metastases, using pathologic conf

124 with suspected lung cancer, the presence of mediastinal lymph node metastasis is a critical determin

125 The mediastinal lymph node NK cells were activated, expressi

126 h alloantigen-induced expression of IL-10 in mediastinal lymph node or splenic T cells, intragraft ex

127 Lastly, mediastinal lymph node re-stimulation experiments showed

128 mputed tomography screening with and without mediastinal lymph node resection (MLNR) under an Institu

129 as early as 2 days post-IN inoculation; the mediastinal lymph node was an early site of replication

130 Negative regulation of cells in the mediastinal lymph node was minimal compared with that pr

131 airways (bronchoalveolar lavage), lung, and mediastinal lymph node were examined 10 d postinfection

132 t ILC2s accumulated equally in the recipient mediastinal lymph node.

133 lored DC subset present in the lung-draining mediastinal lymph node.

134 lung and enhanced the T(H)2 response in the mediastinal lymph node.

135 d with a decrease in the number of DC in the mediastinal lymph node.

136 sulted in an abolished T(H)2 response in the mediastinal lymph node.

137 berculosis Ag85B-specific CD4 T cells in the mediastinal lymph node.

138 ungs, it decreased trafficking of DCs to the mediastinal lymph node.

139 Unlike examination of mediastinal lymph nodes (LNs), which depends on surgical

140 electively accumulated in the myocardium and mediastinal lymph nodes (med-LN) of infarcted mice, acqu

141 lymphocytes rapidly redistribute to regional mediastinal lymph nodes (MedLNs) during influenza infect

142 nd, to a lesser extent, in the lung-draining mediastinal lymph nodes (medLNs) of virus-infected mice.

143 ent activation and migration to the draining mediastinal lymph nodes (MLNs) during IV infection.

144 0) levels remained elevated in the lungs and mediastinal lymph nodes (mLNs) throughout the acute LCMV

145 ce, by residual antigen in the lung-draining mediastinal lymph nodes (MLNs).

146 g naive CD4 T cells appear to migrate to the mediastinal lymph nodes along a CD62L-independent, CCR7-

147 acterized the DC population in the heart and mediastinal lymph nodes and analyzed long-term cardiac i

148 uppressed the accumulation of T cells in the mediastinal lymph nodes and lung granulomatous regions w

149 regs rapidly accumulate in the lung-draining mediastinal lymph nodes and lungs.

150 tivation of DN1 T cells was initiated in the mediastinal lymph nodes and showed faster kinetics compa

151 arise in mesenteric, axillary/brachial, and mediastinal lymph nodes and spleen based on differential

152 influenza-specific CD8 T cells in lymphoid (mediastinal lymph nodes and spleen) and nonlymphoid tiss

153 T cells in the mediastinal lymph nodes and the intramyocardial endothel

154 oreover, this drainage can occur directly to mediastinal lymph nodes and there is no interlobar lymph

155 failed to proliferate as extensively in the mediastinal lymph nodes as in mice infected only with BC

156 ere is a large right hilar mass and enlarged mediastinal lymph nodes but no pulmonary emboli.

157 ures yet contributed to TH2 expansion in the mediastinal lymph nodes but not in the lungs.

158 are retained in the peritoneum and draining mediastinal lymph nodes for a prolonged period following

159 Granulomas and mediastinal lymph nodes from active-disease but not late

160 Mediastinal lymph nodes from airway-sensitized Abcg1(-/-

161 in vivo and likewise detect mKATE2(+) DCs in mediastinal lymph nodes from infected mice.

162 rformance of MR imaging in staging hilar and mediastinal lymph nodes in NSCLC on both a per-patient a

163 illus but greatly diminishes their egress to mediastinal lymph nodes independent of neutrophil microb

164 ession of interleukin (IL)-17 transcripts in mediastinal lymph nodes induced by effector cells alone.

165 Therefore, mediastinal lymph nodes may be false-positive on (18)F-F

166 was stratified according to the presence of mediastinal lymph nodes measuring 1 cm or more in the sh

167 xamined the T(H)2 cytokine production in the mediastinal lymph nodes of DEP-exposed CCR2 knockout and

168 -specific CD4(+) T-cell proliferation in the mediastinal lymph nodes of mice.

169 ic analysis of CD11c(+) dendritic cells from mediastinal lymph nodes of the infected mice showed that

170 mic sites and morphologic characteristics of mediastinal lymph nodes on spiral computed tomography fo

171 MATERIAL/METHODS: Anatomical distribution of mediastinal lymph nodes on spiral CT was reviewed in 39

172 In contrast, mediastinal lymph nodes remained nonluminescent througho

173 cells and more IFN-gamma from PBMC, BAL, and mediastinal lymph nodes than monkeys with latent infecti

174 f) of naive CD4 T cells appears to enter the mediastinal lymph nodes through a blood-to-lung-to-lymph

175 lumen and did not need to spread through the mediastinal lymph nodes to cause a systemic infection.

176 es in murine hearts, pericardial AT, spleen, mediastinal lymph nodes, and bone marrow were quantified

177 of infection, is initially restricted to the mediastinal lymph nodes, and does not involve other lymp

178 nocyte-derived dendritic cell numbers in the mediastinal lymph nodes, and increased T-helper type 2 (

179 n early (36 h-4 d) expansion of Tregs in the mediastinal lymph nodes, and later (12-16 d) increases i

180 l distribution and morphological patterns of mediastinal lymph nodes, as demonstrated on spiral CT, c

181 ration (EBUS-TBNA) biopsies of the hilar and mediastinal lymph nodes, but the feasibility and usefuln

182 there was a marked expansion of cells within mediastinal lymph nodes, comprised mainly of innate lymp

183 Such granulomas occur in the lung and the mediastinal lymph nodes, in the heart, and in other vita

184 Using this novel approach to study DCs in mediastinal lymph nodes, we observed that most blood-der

185 h2, Th17 cells, and Tregs, in the spleen and mediastinal lymph nodes, with expansion of splenic antig

186 dritic cells (DCs) in lung and lung-draining mediastinal lymph nodes, with lung CD11b(+) DCs displayi

187 hemagglutinin were primarily observed in the mediastinal lymph nodes.

188 h greater regulatory T cell expansion in the mediastinal lymph nodes.

189 ease in the size of the right hilar mass and mediastinal lymph nodes.

190 ptoms unexpectedly showed high FDG uptake in mediastinal lymph nodes.

191 ased C. neoformans-specific Th2 cells in the mediastinal lymph nodes.

192 e number of DCs carrying OVA in the lung and mediastinal lymph nodes.

193 ung conventional dendritic cells to draining mediastinal lymph nodes.

194 res across the lung epithelium into draining mediastinal lymph nodes.

195 s in the cervical lymph nodes but not in the mediastinal lymph nodes.

196 ncreased numbers of NK cells in the lung and mediastinal lymph nodes.

197 uickly out of the lung and into the thoracic/mediastinal lymph nodes.

198 charge translocate rapidly from the lung to mediastinal lymph nodes.

199 production in blood but not in the affected mediastinal lymph nodes.

200 asized to other lobes of the lung and to the mediastinal lymph nodes.

201 the lungs and central memory T cells in the mediastinal lymph nodes.

202 ion of pulmonary dendritic cells (DC) to the mediastinal lymph nodes.

203 RNA expression and impaired DC homing to the mediastinal lymph nodes.

204 ll as several enlarged hilar and ipsilateral mediastinal lymph nodes.

205 In most cases, the mediastinal lymphadenectomy included the low para-esopha

206 nic space-occupying lesions (35%); abdominal/mediastinal lymphadenopathy (20%), ocular disease (18%)

207 nic space-occupying lesions (35%), abdominal/mediastinal lymphadenopathy (20%), ocular disease (18%),

208 Patients with isolated mediastinal lymphadenopathy (IML) are a common presentat

209 ings outside the parenchymal lung, including mediastinal lymphadenopathy and pericardial effusion, sh

210 and more interlobular septal thickening and mediastinal lymphadenopathy on computed tomography of th

211 rdial delayed enhancement of the septum, and mediastinal lymphadenopathy should raise the suspicion f

212 osed in 5 of 77 patients (6.5%), while hilar/mediastinal lymphadenopathy was found in 25 of 76 patien

213 rdial delayed enhancement of the septum, and mediastinal lymphadenopathy were more often see in those

214 (PET) scan confirmed the lung lesion and the mediastinal lymphadenopathy without distant metastases.

215 pital for examination of bilateral hilar and mediastinal lymphadenopathy.

216 ary lesions in the lungs along with necrotic mediastinal lymphadenopathy.

217 ents were divided into four groups: anterior mediastinal lymphoma (group A, n=16), anterior mediastin

218 differences between prechemotherapy SUVt of mediastinal lymphoma and normal thymus and postchemother

219 De novo mediastinal lymphoma was correctly diagnosed by EBUS-TBN

220 n 100 cases of de novo or suspected relapsed mediastinal lymphoma was investigated by comparing EBUS-

221 nd accuracy of EBUS-TBNA in the diagnosis of mediastinal lymphoma were 89%, 97%, 98%, 83%, and 91%, r

222 diastinal lymphoma (group A, n=16), anterior mediastinal lymphoma with subsequent recurrence (group B

223 Vt of 3.4 or higher is a strong predictor of mediastinal lymphoma.

224 tion of normal thymus or thymic rebound from mediastinal lymphoma.

225 an be successful in the diagnosis of de novo mediastinal lymphomas and is ideally suited in distingui

226 Emicro-Myc/DNMT3B7 mediastinal lymphomas have more chromosomal rearrangemen

227 NMT3B7 expression increases the frequency of mediastinal lymphomas in Emicro-Myc animals.

228 Recently, mediastinal lymphomas with features intermediate between

229 ibitor A20, in Hodgkin lymphomas and primary mediastinal lymphomas.

230 images demonstrated the presence of a large mediastinal mass (11x8 cm) located in the anterior media

231 or and transverse dimensions of the anterior mediastinal mass or thymus on axial CT images and measur

232 otic systems, involving the thoracic cavity (mediastinal mass resections, lobectomies, and esophagect

233 ximum standardized uptake values of anterior mediastinal mass, thymus (SUVt), and bone marrow at the

234 3 years (range, 14 to 59 years), and 46% had mediastinal masses >/=10 cm.

235 ssive tumors that typically present as large mediastinal masses in young women.

236 nts are usually young and present with large mediastinal masses.

237 In response to mediastinal nerve stimulation, most IC neurons became ex

238 uisition and optimization, identification of mediastinal nodal and vascular structures, EBUS-TBNA sam

239 and colorectal cancers and of CT in lung and mediastinal nodal disease points to future tailored use

240 non-small-cell lung cancer with ipsilateral mediastinal nodal metastases (N2) have shown the feasibi

241 nths postoperatively and required a negative mediastinal node biopsy, no excessive vitamin intake, no

242 y significant decreases in FDG-avid lung and mediastinal node disease.

243 re biopsies of the lung mass and ipsilateral mediastinal nodes confirmed a poorly differentiated non-

244 F was seen in cases of mildly (18)F-FDG-avid mediastinal nodes in lung cancer and small liver metasta

245 , and cisplatin for relapse in the lungs and mediastinal nodes with a rising AFP level starting in Ja

246 (absence of (18)F-FDG-avid foci in nonhilar mediastinal nodes), symmetry (difference between left an

247 evidence of testicular, retroperitoneal, or mediastinal non-seminomatous germ cell tumours based on

248 e prognostic factors in both groups; primary mediastinal nonseminoma (group A) and elevations of alph

249 nd were particularly prevalent among primary mediastinal nonseminomas (72%).

250 avel the clonal relationship between primary mediastinal nonseminomas (PMNs) and hematologic somatic-

251 Five (24%) of 21 primary mediastinal nonseminomatous GCTs are continuously diseas

252 Mediastinal nonseminomatous GCTs carry a poor prognosis

253 One in every 17 patients with primary mediastinal nonseminomatous GCTs develop an incurable he

254 metastatic disease, nor were there enlarged mediastinal or hilar lymph nodes.

255 perforation or anastomotic leak with limited mediastinal or pleural contamination.

256 safe and effective when performed along with mediastinal or pleural drainage.

257 s; (c) similar visual perception of noise on mediastinal (P = .132) and lung (P = .366) images, mainl

258 scopy and surgery); 13 patients had positive mediastinal PET findings, and 77 had negative mediastina

259 ediastinal PET findings, and 77 had negative mediastinal PET findings.

260 gnosis (benign vs malignant) on the basis of mediastinal pleural thickening (sensitivity, 81%; specif

261 e interpretation, the diagnostic accuracy of mediastinal pleural thickening, shrinking lung (hemithor

262 ations, performing substantially better than mediastinal pleural thickness and shrinking lung, and mi

263 e finding of frequent TP53 alterations among mediastinal primary nonseminomas may explain the more fr

264 Mediastinal primary site and two or more lines of prior

265 later therapy, platinum-refractory disease, mediastinal primary tumor site, nonseminoma histology, i

266 Mediastinal primary, pulmonary metastases, age, or doses

267 llaborative Group (IGCCCG) prognostic group, mediastinal primary, pulmonary metastases, and smoking a

268 se (12 vs 19%) but a higher rate of previous mediastinal radiation (8.4 vs 1.8%) (P < 0.001).

269 Recently, the recognition that mediastinal radiation is associated with significant lon

270 ns for TI included porcelain aorta, previous mediastinal radiation, chest wall deformity, and potenti

271 lude a combination of immunochemotherapy and mediastinal radiation.

272 efficacy and challenge the need for routine mediastinal radiation.

273 flect treatment bias and history of previous mediastinal radiation.

274 D for patients receiving a MHD of 20 Gy from mediastinal radiotherapy, compared with patients not tre

275 e has resulted in routine consolidation with mediastinal radiotherapy, which has potentially serious

276 apy, compared with patients not treated with mediastinal radiotherapy.

277 Global innervation assessed with heart-to-mediastinal ratio and washout rates was preserved in all

278 ons including cardiopulmonary resuscitation, mediastinal reexploration, placement on extracorporeal m

279 t commonly found in sacrococcygeal, gonadal, mediastinal, retroperitoneal, cervicofacial and intracra

280 Mean counts in mediastinal ROI were computed from a fixed volume in 3 d

281 method that incorporates various cardiac and mediastinal segmentation schemes in which upper and lowe

282 Mediastinal sequelae included lymph node calcification (

283 ed inflammatory and atelectatic changes with mediastinal shift to the right.

284 The ratio between myocardial and mediastinal signal at 15 and 125 min and extrapolated at

285 The mediastinal signal was measured and fitted to a linear m

286 On MVA, primary mediastinal site (P < .001), two or more lines of prior

287 Features extracted from mediastinal sites were highly predictive of primary refr

288 aphy-guided transbronchial needle aspiration mediastinal staging (EBUS group) in 62 patients (37.3%)

289 NA) is an established technique for invasive mediastinal staging of non-small cell lung cancer (NSCLC

290 nded as a first-line diagnostic modality for mediastinal staging.

291 th parameters allowing for identification of mediastinal structures and adrenal glands is still much

292 pneumonectomy, proximal to or involved with mediastinal structures, abutting the chest wall, or recu

293 nition of EBUS/computed tomography images of mediastinal structures.

294 pattern with increased (sometimes exclusive) mediastinal thoracic lymph node involvement, indicating

295 graphy (PET/CT) in the management of primary mediastinal (thymic) large B-cell lymphoma (PMBCL).

296 It is within this mediastinal tissue that T cells develop and are extensiv

297 edge of benign conditions that might mimic a mediastinal vascular pathology is important for therapeu

298 PS if a solid portion was detectable in the mediastinal window setting (nonmeasurable, < 50%, or > 5

299 nclusion Detection of a solid portion in the mediastinal window setting allows subsolid nodules to be

300 When a solid portion was measurable in the mediastinal window, the specificity for adenocarcinoma i