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1 these proteins on centromere orientation in meiosis I .
2 the pattern of chromosome segregation during meiosis I.
3 s are required for spindle bipolarization in meiosis I.
4 an lead to missegregation of homologs during meiosis I.
5 hat Fest is required for proper execution of meiosis I.
6 s of core structural components important in meiosis I.
7 segregation of homologous chromosomes during meiosis I.
8 mologs to ensure their proper segregation at meiosis I.
9 cumulation of APC/C substrates essential for meiosis I.
10 ultivalents and centromere clustering during meiosis I.
11 s that facilitate homolog segregation during Meiosis I.
12 r its telomere-associated segregation during meiosis I.
13 ate homologous chromosome segregation during meiosis I.
14 atids (PSSC) and reverse segregation (RS) in meiosis I.
15 ein that protects centromeric cohesin during meiosis I.
16 display defects in chromosome segregation at meiosis I.
17 gues that ensure their proper segregation at meiosis I.
18 nd their regulation by Ipl1 and Mps1 through meiosis I.
19 e chromosome pairs for proper disjunction in meiosis I.
20 lting in the cessation of spermatogenesis in meiosis I.
21 of kinetochore attachment to microtubules in meiosis I.
22 s II errors among eggs affected by errors in meiosis I.
23 unctions necessary for proper disjunction at meiosis I.
24 nocytes results in arrest during prophase of meiosis I.
25 paired X chromosome separate prematurely, in meiosis I.
26 ssential for preventing precocious exit from meiosis I.
27 l for faithful chromosome segregation during meiosis I.
28 lock of spermatogenesis at early prophase of meiosis I.
29 er chromatid co-orientation in S. cerevisiae meiosis I.
30 nections necessary for proper segregation at meiosis I.
31 k (MEN) and monopolar homolog orientation in meiosis I.
32 thereby prevent cleavage by separase during meiosis I.
33 sufficient for the persistence of SCC after meiosis I.
34 asun spermatocytes arrest during prophase of meiosis I.
35 e maintenance, and chromosome segregation in meiosis I.
36 for the reductional segregation occurring at meiosis I.
37 otic spindle to ensure proper segregation at meiosis I.
38 that promote the lengthy prophase unique to meiosis I.
39 osomes failed to segregate accurately during meiosis I.
40 sover (CO) formation between homologs during meiosis I.
41 e critical for proper homolog segregation in Meiosis I.
42 ary for proper chromosome disjunction during meiosis I.
43 not play a role in centromere protection in meiosis I.
44 2A involved in chromosome congression during meiosis I.
45 f the SAC to inhibit bivalent segregation in meiosis I.
46 etochore cross-linking and co-orientation in meiosis I.
47 hinery modified to segregate homologs during meiosis I?
50 In multicellular organisms, the entry into meiosis is a complex process characterized by increasing
56 hromosomes are segregated during mitosis and meiosis is a major puzzle of biology and biophysics.
66 e that the slow increase in CDK1 activity in meiosis I acts as a timing mechanism to allow stable K-M
69 information between parental chromosomes in meiosis is an integral process for the creation of gamet
70 the four chromatids resulting from a single meiosis, is an ideal method to study the mechanisms of h
72 ycle phases during male meiosis: prophase of meiosis I and after exit from meiosis II, in spermatids.
73 meiosis II led to accelerated completion of meiosis I and an increase in aneuploidy at metaphase II.
74 ibit an absence of chromosome disjunction in meiosis I and an infrequent chromosome disjunction in me
75 ovel decrease in CID levels after the end of meiosis I and before meiosis II, which correlates tempor
78 ce blocks the removal of cohesin during both meiosis I and II, results in alterations in nonhomologou
79 d co-orientation in Saccharomyces cerevisiae meiosis I and inhibiting merotelic attachment in Schizos
80 0) and APC(Ama1) are prematurely active, and meiosis I and meiosis II events occur in a single meioti
81 otic errors, such as nondisjunction (NDJ) in meiosis I and meiosis II, and premature separation of si
86 ost germ cells were arrested at metaphase of meiosis I and no mature sperm were detected in epididymi
87 pendent chromosome segregation during oocyte meiosis I and nuclear assembly during the transition fro
90 yeast Aurora B localizes at telomeres during meiosis I and promotes telomere separation independently
91 Mastl to show that Mastl-null oocytes resume meiosis I and reach metaphase I normally but that the on
92 However, the majority of oocytes complete meiosis I and the resulting eggs retain the correct numb
93 ster chromatids together, first from arms in meiosis I and then from the centromere region in meiosis
94 er chromatid co-orientation in S. cerevisiae meiosis I and to suppress merotelic attachment in S. pom
97 rminally truncated form as predominant after meiosis I, and demonstrated direct physical interaction
98 nces that place centromeres at the centre of meiosis I, and discuss how these studies affect a variet
99 on of segregation-competent bivalents during meiosis I, and findings suggest that age-dependent deple
100 omatids of the X chromosomes separate during meiosis I, and homologous X chromatids segregate to the
101 nderwent more-frequent nondisjunction during meiosis I, and others showed more meiosis II segregation
102 res, kinetochores and chromosome arms in mid-meiosis I, and that MCAK depletion, or inhibition using
103 involved in spindle function in mouse oocyte meiosis I, and whether MCAK is necessary to prevent chro
107 ant percentage of oocytes failed to complete meiosis I because the spindle assembly checkpoint remain
108 subunit localizes first along chromosomes in meiosis I, becoming restricted to the centromere region
109 tion not only affects homolog segregation at meiosis I but also the fate of sister chromatids at meio
110 eiosis, the MEN is dispensable for exit from meiosis I but contributes to the timely exit from meiosi
111 ase (FEAR) network is required for exit from meiosis I but does not lead to the activation of origins
112 MCAK contributes to chromosome alignment in meiosis I, but is not necessary for preventing chromosom
113 Rim4 inhibits translation of CLB3 during meiosis I by interacting with the 5' untranslated region
115 meric Rec8 is protected from separase during meiosis I by shugoshin/MEI-S332 proteins that bind PP2A
117 s separate before prometaphase I, disrupting meiosis I centromere orientation and causing nondisjunct
118 the centromere is required for protection.In meiosis I centromeric cohesin is protected by Sgo2 from
121 site spindle poles (biorientation), accurate meiosis I chromosome segregation requires that sister ch
122 , we show that Wapl is required for accurate meiosis I chromosome segregation, predominantly releases
126 lling the number of recombination events per meiosis is CO homeostasis, which maintains a stable CO n
127 ons. In budding yeast, the decision to enter meiosis is controlled by nutrient and mating-type signal
128 cers, including Stra8, but how the switch to meiosis is controlled in male germ cells (spermatogonia)
130 Correct centriole organization during male meiosis is critical to guarantee a normal bipolar mitoti
131 nd synapsis of homologous chromosomes during meiosis is crucial for producing genetically normal game
133 f PIWI proteins mostly expressed during male meiosis, is crucial for piRNA biogenesis, post-transcrip
136 hat the centering mechanism is maintained in meiosis I despite chromosome movement in the opposite di
137 homologs separate their sister chromatids at meiosis I; detect selection for higher recombination rat
139 hat the cell cycle timing of CID assembly in meiosis is different from mitosis and that the efficient
140 evel of IFY-1 declined immediately following meiosis I division and remained low during meiosis II an
141 on of healthy gametes requires a reductional meiosis I division in which replicated sister chromatids
146 The proper distribution of crossovers during meiosis I ensures accurate chromosome segregation at the
147 hromatids segregate away from one another in meiosis I (equational division), rather than segregating
148 mplex (SC), or synapsis, between homologs in meiosis is essential for crossing over and chromosome se
152 omosome segregation, the defining feature of meiosis, is established by multifaceted kinase control b
154 mitosis-like division by disrupting two key meiosis I events: coorientation of sister kinetochores a
156 naphase-promoting complex/cyclosome to allow meiosis I exit and for the rapid rise of Cdk1 activity t
157 ERK phosphorylates and inhibits Dicer during meiosis I for oogenesis to proceed normally in Caenorhab
160 ctivated by Cdc5-mediated phosphorylation in meiosis I, generating the crossovers necessary for chrom
168 dition, DNA replication or damage during the meiosis I-II interval fails to arrest meiotic progress,
170 1 arrest as one-cell embryos in metaphase of meiosis I in a manner that is indistinguishable from emb
172 release of sister chromatid cohesion during meiosis I in C. elegans also function to inhibit centrio
173 nd Homer report that the proper execution of meiosis I in mouse oocytes requires the stabilization of
178 sley et al. (2016) use the unique biology of meiosis I, in which the cell can exit the division witho
180 ed oocytes did not replicate their DNA after meiosis I indicating that Geminin does not act as an inh
183 hat, in both the male and female germ lines, meiosis is initiated through retinoic acid induction of
184 s recombination is induced to high levels in meiosis, is initiated by Spo11-catalyzed DNA double-stra
185 we provide evidence that oocyte reentry into meiosis is instead associated with a shift in the patter
187 of kinetochores with microtubules transform meiosis I into a mitosis-like division by disrupting two
189 ly in the transition from prophase arrest to meiosis I is also impaired in Rab5a-depleted oocytes.
192 ich recombinant homologs missegregate during meiosis I is significantly greater in SOD knockdown oocy
195 ster kinetochores to be under tension during meiosis I leads to premature Sgo1 removal and precocious
196 hat proper progression of germ cells through meiosis is licensed by YTHDC2 through post-transcription
197 In mammals, genetic recombination during meiosis is limited to a set of 1- to 2-kb regions termed
198 x chromosome inactivation established during meiosis is maintained into spermatids with the silent co
203 onal activation of a cluster of genes at the meiosis I-meiosis II transition, including the critical
205 cytokinesis are not caused by alterations in meiosis I (MI or meiosis II (MII) chromosome dynamics, b
207 on (G2/M transition) and progression through meiosis I (MI) are two key stages for producing fertiliz
208 es with nondisjoined chromosomes 21 due to a meiosis I (MI) error, recombination is significantly red
210 Homologous chromosome segregation during meiosis I (MI) in mammalian oocytes is carried out by th
212 though an SAC operates in mammalian oocytes, meiosis I (MI) is notoriously error prone and polar-disp
214 hich is established through migration of the meiosis I (MI) spindle/chromosomes from the oocyte inter
224 ing high-resolution imaging, we find that in meiosis I, microtubules initially form a "cage-like" str
225 point protein Mad2 results in an increase in meiosis I nondisjunction, suggesting that Mad2 has a con
230 tion of chromosome segregation during oocyte meiosis is of vital importance to mammalian reproduction
231 ect centromeric cohesin from separase during meiosis I or support the spindle assembly checkpoint in
233 The transcriptional regulation of mammalian meiosis is poorly characterized, owing to few genetic an
235 and Bub1 kinase activities localise Sgo2 in meiosis I preferentially to the centromere and pericentr
238 IKIN, suggests that kinase coordination by a meiosis I regulator may be a general feature in the esta
239 spermatocytes exhibit cytokinetic failure in meiosis I, resulting in bi-nucleated secondary spermatoc
247 ction of sister kinetochores biorient on the meiosis I spindle and association of the monopolin compl
248 nd this is essential for the assembly of the meiosis I spindle but not for chromosomes to separate.
251 s of anastral Drosophila melanogaster oocyte meiosis I spindles as well as spindle fibers, centromere
254 ngression and bi-orientation of bivalents on meiosis I spindles, in facilitating formation of K-fiber
255 s on the X chromosome, initiated during male meiosis, is stably maintained during subsequent spermiog
256 nate from mature oocytes that have completed meiosis I, suggesting that Tgkd teratomas originate from
259 iosis II and does not even phosphorylate its meiosis I targets during the second meiotic division.
260 oocytes lacking FZR1 undergo passage through meiosis I that is accelerated by ~1 h, and this is due t
273 P1 homologs to the chromatin at the onset of meiosis I, thereby antagonizing AIR-2 and cooperating wi
274 main, translationally repress cyclin CLB3 in meiosis I, thereby ensuring homologous chromosome segreg
275 Mouse oocytes carrying DNA damage arrest in meiosis I, thereby preventing creation of embryos with d
276 To date, the role of MAPK in control of meiosis is thought to be restricted to maintaining metap
277 ocytes respond to DNA damage by arresting in meiosis I through activity of the Spindle Assembly Check
278 telomeres of homologues during anaphase A of meiosis I, thus preventing complete disjunction until mi
281 set of APC/C activity at the transition from meiosis I to meiosis II led to accelerated completion of
282 de of spindle bipolarization is required for meiosis I to prevent chromosome segregation errors.
285 a B, but biochemical progression through the meiosis I-to-II transition and arrest at metaphase II we
286 some distribution at anaphase of mitosis and meiosis is triggered by separase, an evolutionarily cons
294 rection mechanisms may function in mammalian meiosis I, we speculate that late establishment of kinet
297 dc55) activity undergo a premature exit from meiosis I which results in a failure to form bipolar spi
298 unique reductional chromosome segregation of meiosis I, which also results in chromosomal exchanges.
299 oned off-center on the spindle in oocytes in meiosis I, while under normal tension, as a result of cr
300 e stage but arrest at the G2/M transition of meiosis I, with lack of protein expression of the key me