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1 s of this analysis were overall survival and melanoma-specific survival.
2 ant disease control and resulted in enhanced melanoma-specific survival.
3 points were RFS, overall survival (OS), and melanoma-specific survival.
4 The primary end point was melanoma-specific survival.
5 cell invasion are robust predictors of poor melanoma-specific survival.
6 e patterns, including conditional rates, and melanoma-specific survival.
7 comes were local recurrence, metastasis, and melanoma-specific survival.
8 enotypes and assessed their association with melanoma-specific survival.
9 Recurrence, overall survival, and melanoma-specific survival.
10 diagnosis of MIS was evaluated using 15-year melanoma-specific survival, 15-year relative survival (i
11 dissection was not associated with increased melanoma-specific survival among 1934 patients with data
12 prognostic information but did not increase melanoma-specific survival among patients with melanoma
13 , including results for overall survival and melanoma-specific survival, as well as durability of res
14 ults from prior studies.OBJECTIVE To compare melanoma-specific survival between patients with histopa
17 to extend disease free survival and increase melanoma-specific survival for patients with early nodal
18 d prolongs distant disease-free survival and melanoma-specific survival for patients with nodal metas
20 mitotic rate was a significant predictor of melanoma-specific survival (Hazard Ratio [HR] = 1.2, 95%
21 tus was an independent prognostic factor for melanoma-specific survival (hazard ratio [HR] = 1.34; 95
22 tasis, 0.62; P=0.02) and the 10-year rate of melanoma-specific survival (hazard ratio for death from
23 mph-node biopsy is associated with increased melanoma-specific survival (i.e., survival until death f
24 whereas a marker in EBF1 was associated with melanoma-specific survival in advanced-stage patients (r
25 (P<0.0001) and significantly associated with melanoma-specific survival in both stage III (P<0.0001)
26 all six genes were significant in predicting melanoma specific survival (MSS) in a univariate analysi
27 rt to assess recurrence-free survival (RFS), melanoma-specific survival (MSS) and overall survival (O
30 onfirmed objective response rate (cORR), and melanoma-specific survival (MSS) per investigator; overa
31 age III cohort versus the AJCCv8 cohort, the melanoma-specific survival (MSS) rates at 5 years were 6
33 d rates of 10-year overall survival (OS) and melanoma-specific survival (MSS) were 82% and 94%, respe
36 nd clinical outcomes: overall survival (OS), melanoma-specific survival (MSS), and recurrence-free su
37 l lymph node (SLN) had significantly reduced melanoma-specific survival (MSS), DFS, regional node rec
42 sociated with poorer overall (OS; P < .001), melanoma-specific survival (MSS; P = .0025), and disease
46 in the recurrence rate, overall survival, or melanoma-specific survival of patients with MIS treated
49 ntermediate-thickness melanomas, the 10-year melanoma-specific survival rate was 62.1 +/- 4.8% among
50 treatment-related difference in the 10-year melanoma-specific survival rate was seen in the overall
52 in stage IA compared with the AJCCv8 cohort, melanoma-specific survival rates at 10 years were 95.1%-
60 ial, including results for overall survival, melanoma-specific survival, relapse-free survival, and d
62 sults suggest that the MAPK pathway mediates melanoma-specific survival signaling by differentially r
63 patients with a brisk TIL score had improved melanoma-specific survival than those with a nonbrisk sc
65 ive and progression-free at 3 years, 10-year melanoma-specific survival was 96% with nivolumab plus i
70 ol analysis, the mean (+/-SE) 3-year rate of melanoma-specific survival was similar in the dissection
71 (AJCC-8) melanoma staging system focuses on melanoma-specific survival, we developed a multivariable