ライフサイエンスコーパス: meliloti

1 exhibited antimicrobial activity against S. meliloti.

2 open reading frames, podJ1 and podJ2, in S. meliloti.

3 diated regulation of stress adaptation in S. meliloti.

4 ream responses to its symbiont Sinorhizobium meliloti.

5 the closely related bacterium Sinorhizobium meliloti.

6 eted in the incompatible interaction with S. meliloti.

7 d ChvI, and localizes to the periplasm of S. meliloti.

8 odel root nodulating bacterium Sinorhizobium meliloti.

9 em (PTS) have been found in the genome of S. meliloti.

10 of the mechanisms that govern motility in S. meliloti.

11 both free-living and symbiotic states of S. meliloti.

12 osynthesis of exopolysaccharides (EPS) by S. meliloti.

13 codes an LPS sulfotransferase activity in S. meliloti.

14 quorum sensing downregulates motility in S. meliloti.

15 architecture of the biofilm of Sinorhizobium meliloti.

16 y reduced nodulation when inoculated with S. meliloti.

17 with the diazotropic bacterium Sinorhizobium meliloti.

18 les following inoculation with Sinorhizobium meliloti.

19 novel lipid of R. leguminosarum 3841 and S. meliloti.

20 otic nitrogen fixing bacterium Sinorhizobium meliloti.

21 d) genes in the soil bacterium Sinorhizobium meliloti.

22 has not been characterized previously in S. meliloti.

23 an important role in manganese uptake in S. meliloti.

24 )-polymerase activator DctD of Sinorhizobium meliloti.

25 ication in the legume symbiont Sinorhizobium meliloti.

26 tween alfalfa and its symbiont Sinorhizobium meliloti.

27 tages during invasion of M. truncatula by S. meliloti.

28 odulation of alfalfa (Medicago sativa) by S. meliloti.

29 the symbiotic interaction with Sinorhizobium meliloti.

30 influenced the activity of NCR247 against S. meliloti.

31 on of NCR247 lowered its activity against S. meliloti.

32 the nitrogen-fixing bacterium Sinorhizobium meliloti 1021 is needed for an effective symbiosis with

33 ylphosphonate in the bacterium Sinorhizobium meliloti 1021 is proposed based on the analysis of the g

34 e plant intracellular symbiont Sinorhizobium meliloti 1021 is required for conjugal transfer of DNA.

35 Previous studies had shown that S. meliloti 1021 mutants that produce increased levels of s

36 ogen-fixing rhizobial symbiont Sinorhizobium meliloti 1021 produces acidic symbiotic exopolysaccharid

37 otal lipids from R. leguminosarum 3841 or S. meliloti 1021 were added.

38 t, enhances the symbiotic productivity of S. meliloti 1021 with the host plant Medicago truncatula cv

39 tal mating of these genes into Sinorhizobium meliloti 1021, a strain that lacks these particular GalA

40 ologous expression of RgtA in Sinorhizhobium meliloti 1021, a strain that normally lacks GalA modific

41 guanylate (c-di-GMP) levels in Sinorhizobium meliloti 8530, a bacterium that does not carry known cel

42 Sinorhizobium meliloti, a legume symbiont and Brucella abortus, a phyl

43 Sinorhizobium meliloti, a legume symbiont, and Brucella abortus, a phy

44 S. meliloti, a symbiont of leguminous plants, synthesizes m

45 termined that BacA specifically protected S. meliloti against oxidized NCR247.

46 new avenues for understanding the complex S. meliloti-alfalfa interactions which occur during symbios

47 ld more in this interaction compared with S. meliloti alone.

48 S. meliloti also produces sulfated cell surface polysacchar

49 Strikingly, Sinohizobium meliloti, an intracellular symbiont of legume plants, ca

50 e symbiosis that forms between Sinorhizobium meliloti and alfalfa requires biosynthesis of Nod factor

51 In the symbiosis between Sinorhizobium meliloti and alfalfa, mutations in GlnD, the major bacte

52 that have been inoculated with Sinorhizobium meliloti and are developing root nodules.

53 The lipid A molecules of S. meliloti and B. abortus are unusually modified with a ve

54 crucial for the chronic infection of both S. meliloti and B. abortus.

55 le-dependent regulons of CtrA and DnaA in S. meliloti and C. crescentus.

56 o related alphaproteobacteria, Sinorhizobium meliloti and Caulobacter crescentus, serve as models for

57 ere that two of these species, Sinorhizobium meliloti and Caulobacter crescentus, simply lack any ext

58 analyses of the SOE SorT from Sinorhizobium meliloti and its cognate electron acceptor SorU.

59 symbiotic relationship between Sinorhizobium meliloti and its host Medicago sativa (alfalfa) depends

60 he establishment of the symbiosis between S. meliloti and its host Medicago sativa.

61 rogen-fixing symbiosis between Sinorhizobium meliloti and its legume host alfalfa (Medicago sativa) d

62 o study the coordinate differentiation of S. meliloti and its legume partner during nodule developmen

63 rogen-fixing symbiosis between Sinorhizobium meliloti and its leguminous host plant Medicago truncatu

64 Symbiosis between S. meliloti and its plant host alfalfa is dependent on bact

65 nt of a nitrogen-fixing symbiosis between S. meliloti and its plant hosts.

66 Using the Sinorhizobium meliloti and Medicago truncatula symbiotic system, we pr

67 uch as the interaction between Sinorhizobium meliloti and Medicago, bacteroid differentiation is driv

68 In addition, MtFNSII-2 was inducible by S. meliloti and methyl jasmonate treatment, whereas MtFNSII

69 tative proteins, SMc02148 from Sinorhizobium meliloti and PA3455 from Pseudomonas aeruginosa, reveale

70 ommonalities between symbiotic Sinorhizobium meliloti and pathogenic Brucella bacteria in terms of ex

71 s that have been classified as Sinorhizobium meliloti and Sinorhizobium medicae.

72 nvolved in more than carbon regulation in S. meliloti and suggest that the phenotypes observed occur

73 n the requirements for RpoH1 and RpoH2 in S. meliloti and that there must be other crucial targets.

74 truncatula, its symbiosis with Sinorhizobium meliloti, and on soil microbial community structure.

75 Our data indicate that in S. meliloti, and possibly in other Rhizobiaceae species, th

76 s a close relative of both B. abortus and S. meliloti, and this bacterium is the causative agent of c

77 the nitrogen-fixing bacterium Sinorhizobium meliloti appeared to be normal in the npd1 mutant.

78 Coexpression of S. meliloti aqpS and arsC in a strain of E. coli lacking th

79 d the role of aqpS in arsenic resistance, S. meliloti aqpS and arsC were disrupted individually.

80 rter], and arsC (arsenate reductase), the S. meliloti ars operon includes an aquaglyceroporin (aqpS)

81 gest that AqpS is the only protein of the S. meliloti ars operon that facilitates transport of arseni

82 This pathway is likely not limited to S. meliloti as suggested by the presence of homologous gene

83 es and the rhizobial bacterium Sinorhizobium meliloti as well as with the pathogenic oomycete Aphanom

84 osely related model bacterium, Sinorhizobium meliloti, as a heterologous host for expression of a Ca

85 successfully predict sRNAs in Sinorhizobium meliloti, as well as in multiple and poorly annotated sp

86 The Medicago truncatula-Sinorhizobium meliloti association is an excellent model for dissectin

87 to wild-type and succinoglycan-deficient S. meliloti at the early time point of 3 days postinoculati

88 Sinorhizobium meliloti bacA mutants are symbiotically defective, deoxy

89 We show that, when starved, dividing S. meliloti bet hedge by forming two daughter cells with di

90 The S. meliloti bluB mutant is unable to grow in minimal media

91 a greater role in the overall fitness of S. meliloti both during the free-living stage and in its as

92 n the closely related bacteria Sinorhizobium meliloti, Brucella abortus, and Ochrobactrum anthropi.

93 Sinorhizobium meliloti can live as a soil saprophyte and can engage in

94 Sinorhizobium meliloti can live as symbionts inside legume root nodule

95 emonstrate that the YbeY from E. coli and S. meliloti can reciprocally complement the rRNA processing

96 approach, we identified a homolog of the S. meliloti carbohydrate sulfotransferase, LpsS, in Mesorhi

97 In Sinorhizobium meliloti, catabolite repression is influenced by a nonca

98 chanisms governing the alterations of the S. meliloti cell cycle in planta are largely unknown.

99 To achieve polyploidy, the S. meliloti cell cycle program must be altered to uncouple

100 The predicted S. meliloti cell cycle regulon of CtrA, but not that of Dna

101 ll growth that enabled global analysis of S. meliloti cell cycle-regulated gene expression.

102 Only 28% of the 462 S. meliloti cell cycle-regulated genes were also transcript

103 out the small 83 amino acid protein FeuN, S. meliloti cells are unable to grow, and this phenotype is

104 In addition, S. meliloti cells constitutively producing Nod factors were

105 Moreover, adding Nod Factor antibody to S. meliloti cells inhibits biofilm formation, while chitina

106 Sinorhizobium meliloti cells store excess carbon as intracellular poly

107 d lsrB genes are expressed in free-living S. meliloti cells, but they are not required for cell growt

108 ation with the use of flavone pre-treated S. meliloti cells, completely restored nodulation in flavon

109 e HK-two RR motif found in the Sinorhizobium meliloti chemotaxis pathway and measuring the resulting

110 Our discovery of S. meliloti chemotaxis to plant-derived QACs adds another r

111 ipid A is important, but not crucial, for S. meliloti chronic infection and that BacA must have an ad

112 their microscopic nodule phenotype using S. meliloti constitutively expressing lacZ.

113 f three quorum sensing systems present in S. meliloti, controls the production of the symbiotically a

114 We show that S. meliloti CpdR1 has a polar localization pattern and a ro

115 The S. meliloti cpdR1-null mutant can invade the host cytoplasm

116 In Sinorhizobium meliloti, CuxR stimulates transcription of an EPS biosyn

117 Sinorhizobium meliloti dctA encodes a transport protein needed for a s

118 ted selection procedure, four independent S. meliloti dctA mutants were isolated that retained some a

119 6S rRNA precursor that accumulates in the S. meliloti DeltaybeY strain.

120 Within specialized host cells, S. meliloti differentiates into highly polyploid, enlarged

121 ntracellular regulatory pathways to drive S. meliloti endoreduplication and differentiation during sy

122 Deletion of hprK in S. meliloti enhanced catabolite repression caused by succin

123 Sinorhizobium meliloti enters into a symbiotic relationship with legum

124 The alpha-proteobacterium Sinorhizobium meliloti establishes a chronic intracellular infection d

125 The production of the Sinorhizobium meliloti exopolysaccharide, succinoglycan, is required f

126 Sinorhizobium meliloti ExoR regulates the production of succinoglycan

127 In S. meliloti, exoR and the exoS-chvI two-component system re

128 The Sinorhizobium meliloti ExoS/ChvI two-component signaling pathway is re

129 In pathogenic bacteria closely related to S. meliloti, exoS-chvI homologues are required for virulenc

130 In S. meliloti, ExoS/ChvI is a key regulator of gene expressio

131 ula plants inoculated with the Sinorhizobium meliloti exoY mutant, and the M. truncatula vapyrin-2 mu

132 ecule also inhibits the expression of the S. meliloti exp genes, responsible for the production of EP

133 microscopy after staining with X-Gal for S. meliloti expressing a constitutive GUS gene, by confocal

134 When tested for nodulation by Sinorhizobium meliloti, flavonoid-deficient roots had a near complete

135 the alpha-proteobacteria (e.g. Sinorhizobium meliloti), for dephosphorylating CheY-P.

136 Sinorhizobium meliloti forms symbiotic, nitrogen-fixing nodules on the

137 However, in S. meliloti, free-living cells of the cpdR1-null mutant sho

138 erial BacA protein is critical to prevent S. meliloti from being hypersensitive toward NCR AMPs.

139 Recently, we discovered that a Sinorhizobium meliloti gene, bluB, is necessary for DMB biosynthesis.

140 tom Affymetrix GeneChip with the complete S. meliloti genome and approximately 10,000 probe sets for

141 The S. meliloti genome encodes 14 of these alternative sigma fa

142 were identified in the recently sequenced S. meliloti genome.

143 oxide in roots inoculated with Sinorhizobium meliloti greatly increased our understanding of the role

144 Expression profiling of free-living S. meliloti grown with the plant signal molecule luteolin i

145 otic nitrogen-fixing bacterium Sinorhizobium meliloti harbors a gene, SMc02396, which encodes a predi

146 including Brucella; however, its role in S. meliloti has not been investigated.

147 S. meliloti has three quorum-sensing systems (Sin, Tra, and

148 ymology and genetics of PHB metabolism in S. meliloti have been well characterized, phasins have not

149 S. meliloti Hfq is involved in controlling the population d

150 acterizing Ca L. asiaticus regulators, an S. meliloti host can be used for preliminary identification

151 ument complex transcriptional profiles of S. meliloti in different environments.

152 pression in the soil bacterium Sinorhizobium meliloti in response to the plant-secreted flavonoid lut

153 its a reduced transcriptional response to S. meliloti, indicating that the machinery responsible for

154 structure-related genes in both PhiM9 and S. meliloti-infecting T4 superfamily phage PhiM12, which co

155 This phylogenetic and structural study of S. meliloti-infecting T4 superfamily phage PhiM9 provides n

156 on microscopy structure of the Sinorhizobium meliloti-infecting T4 superfamily phage PhiM9.

157 lotillin-like genes (FLOTs) in Sinorhizobium meliloti infection of its host legume Medicago truncatul

158 d that protein exudation in the M. sativa-S. meliloti interaction caused an increase in the secretion

159 This report shows that M. truncatula-S. meliloti interactions involve ecotype-strain specificity

160 ferentiation of the bacterium, Sinorhizobium meliloti into a nitrogen-fixing bacteroid within the leg

161 The S. meliloti iolA (mmsA) gene product seems to be involved i

162 Sinorhizobium meliloti is a gram-negative soil bacterium capable of fo

163 Sinorhizobium meliloti is a gram-negative soil bacterium found either

164 Sinorhizobium meliloti is a gram-negative soil bacterium, capable of e

165 Sinorhizobium meliloti is a member of the Alphaproteobacteria that fix

166 Sinorhizobium meliloti is a nitrogen-fixing bacterial symbiont of alfa

167 The bacterium Sinorhizobium meliloti is attracted to seed exudates of its host plant

168 These results imply that, when S. meliloti is exposed to environmental arsenate, arsenate

169 hat the ExpR/Sin quorum-sensing system in S. meliloti is involved in the regulation of genes responsi

170 Growth of S. meliloti is modelled in three ecological niches (bulk so

171 onally, during early stages of symbiosis, S. meliloti is presented with an oxidative burst that must

172 We show that GroEL1 of Sinorhizobium meliloti is required for efficient infection, terminal d

173 on the lipid A in the free-living form of S. meliloti, is essential for the chronic infection.

174 ferent species of rhizobia in a strain of S. meliloti lacking endogenous NodD activity.

175 Our studies of an S. meliloti loss-of-function hfq mutant have revealed that

176 After inoculation with compatible S. meliloti, LYK3:GFP puncta are relatively stable.

177 l role during key steps of the Sinorhizobium meliloti-M. truncatula symbiotic interaction.

178 studies indicate that, in contrast to the S. meliloti-Medicago model symbiosis, bacteroids in the S.

179 ences early signalling events in the Ensifer meliloti-Medicago truncatula interaction.

180 The Sinorhizobium meliloti megaplasmid pSymA has previously been implicate

181 iate GalA donor substrate is available in S. meliloti membranes.

182 dioxygenases and hydrolases in Sinorhizobium meliloti, Mesorhizobium loti, and Bradyrhizobium japonic

183 The S. meliloti ML beta-glucan participates in bacterial aggreg

184 a inoculated with succinoglycan-deficient S. meliloti more strongly express an unexpectedly large num

185 d with wild-type, succinoglycan-producing S. meliloti more strongly express genes encoding translatio

186 S. meliloti MsbA2 is required for the invasion of nodule ti

187 In planktonic S. meliloti, MucR properly coordinates a diverse set of bac

188 he host plant Medicago sativa, Sinorhizobium meliloti must overcome an oxidative burst produced by th

189 ium meliloti or to the invasion defective S. meliloti mutant, exoA.

190 trans rescue with a Nod factor-deficient S. meliloti mutant.

191 ied the symbiotic infection phenotypes of S. meliloti mutants deficient in succinoglycan production o

192 d this by constructing and characterizing S. meliloti mutants in the lpxXL and acpXL genes, which enc

193 These S. meliloti mutants induced a more severe infection phenoty

194 We have constructed S. meliloti mutants that lack HPrK or that lack key amino a

195 two-part screen, we identified Sinorhizobium meliloti mutants that were both sensitive to hydrogen pe

196 Mapping of S. meliloti mutations conferring resistance to PhiM12, N3,

197 ute to the release of free fatty acids in S. meliloti, neither one can use phospholipids as substrate

198 ensures that a cascade of the Sinorhizobium meliloti nitrogen fixation genes is induced as the conce

199 S. meliloti Nod factor is a lipochitooligosaccharide that u

200 the synthesis of an inducer of Sinorhizobium meliloti nod genes, as well as a gene associated with No

201 , 4'-dihydroxyflavone, a major inducer of S. meliloti nod genes, completely restored nodulation.

202 n sulfated fungal Myc-LCOs and Sinorhizobium meliloti Nod-LCOs, having very similar structures, each

203 D, we assayed the DNA binding activity of S. meliloti NodD1 treated with the flavonoid inducer luteol

204 Sinorhizobium meliloti nodL and nodF mutations additively reduce infec

205 S. meliloti nodulation factor treatments of MtROP9i led to

206 A17, the Fix phenotypes are reversed with S. meliloti NRG185 and NRG34, and there is a correlation be

207 S. meliloti NRG185 produces oligosaccharides that are almos

208 a A17, and the phenotype is reversed with S. meliloti NRG185.

209 Sinorhizobium meliloti NRG247 has a Fix(+) phenotype on Medicago trunc

210 ass spectrometry, and the analysis of the S. meliloti NRG247 oligosaccharides showed that this strain

211 fixing root nodules induced by Sinorhizobium meliloti on the model plant Medicago truncatula, tubules

212 first characterization of the Sinorhizobium meliloti open reading frame SMc01113.

213 plants exposed for 24 h to WT Sinorhizobium meliloti or to the invasion defective S. meliloti mutant

214 etween Medicago truncatula and Sinorhizobium meliloti Our analysis revealed a poor correlation betwee

215 species, R. leguminosarum bv trifolii and E. meliloti, (over)expressing the CelC2 coding gene, celC.

216 Sinorhizobium meliloti participates in a nitrogen-fixing symbiosis wit

217 ExoR(c20) was isolated directly from S. meliloti periplasm to identify its N-terminal amino acid

218 genomic and structural levels, PhiM9 and S. meliloti phage PhiM12 have a small number of open readin

219 he recently characterized cyanophage-like S. meliloti phages of the PhiM12 group.

220 e gram-negative soil bacterium Sinorhizobium meliloti plays an important role in the establishment of

221 As in C. crescentus, the S. meliloti PodJ1 protein appears to act as a polarity beac

222 We leveraged synchronized S. meliloti populations to determine how treatment with a s

223 Sinorhizobium meliloti produces an exopolysaccharide called succinogly

224 discovered that in the absence of VLCFAs, S. meliloti produces novel pentaacylated lipid A species, m

225 ng SMc01113/YbeY expression in Sinorhizobium meliloti produces symbiotic and physiological phenotypes

226 Independent of host choice, E. meliloti rapidly adapted to its local host genotype, and

227 The symbiotic lifestyle of Sinorhizobium meliloti requires a drastic cellular differentiation tha

228 Symbiotic infection by S. meliloti requires an osmosensory two-component system co

229 Sinorhizobium meliloti requires exopolysaccharides in order to form a

230 Sinorhizobium meliloti requires ExoS/ChvI two-component signalling to

231 herefore, like E. coli and other species, S. meliloti requires only one groEL gene for viability, and

232 of the whole-genome expression profile of S. meliloti reveals that the ExpR/Sin quorum-sensing system

233 ysaccharide produced by strain Sinorhizobium meliloti Rm1021 contributes to symbiosis with Medicago s

234 The Sinorhizobium meliloti Rm1021 Delta glnD-sm2 mutant, which is predicte

235 ions of A. tumefaciens C58 and Sinorhizobium meliloti Rm1021 genomes.

236 trogen stress response (NSR) of wild type S. meliloti Rm1021, and isogenic strains missing both PII p

237 st identity to SinI (71%) from Sinorhizobium meliloti Rm1021.

238 As populations of Sinorhizobium meliloti Rm2011 were similar in bulk/dissolved and ENM t

239 ns involve ecotype-strain specificity, as S. meliloti Rm41 and NRG247 are Fix+ (compatible) on M. tru

240 o compatibility; most importantly, (v) an S. meliloti Rm41 derivative, carrying exo genes from an M.

241 whereas the oligosaccharides produced by S. meliloti Rm41 include many nonsuccinylated subunits, as

242 ibility with the microsymbiont Sinorhizobium meliloti Rm41.

243 The inferred S. meliloti RpoH promoter consensus sequences share feature

244 ze the global transcriptional response of S. meliloti rpoH1, rpoH2, and rpoH1 rpoH2 mutants during he

245 r, luteolin, the plant-derived inducer of S. meliloti's nod genes, is not required for mature biofilm

246 Contrary to S. meliloti, S. fredii HH103 showed little or no sensitivit

247 The nitrogen-fixing symbiont Sinorhizobium meliloti senses and responds to constantly changing envi

248 ere we report evidence that in Sinorhizobium meliloti, sensing of AHLs with acyl chains composed of 1

249 Proline utilization A from Sinorhizobium meliloti (SmPutA) is a 1233-residue bifunctional enzyme

250 Sinorhizobium meliloti stores carbon and energy in poly-3-hydroxybutyr

251 biont of one of these species (Sinorhizobium meliloti strain Rm1021) and an opportunistic bacterial p

252 es this type of specificity pertaining to S. meliloti strain Rm41.

253 Nearly all Ensifer meliloti strains completely lack ebpA, tcrA, tcsA and fs

254 On wild-type plants, S. meliloti strains producing no succinoglycan or only unsu

255 eric oligosaccharides (STOs) from the two S. meliloti strains were compared by chromatography and mas

256 Analysis of free-living S. meliloti strains with mutations in genes related to nitr

257 lycan oligosaccharide populations between S. meliloti strains; (iv) the structural nature of the succ

258 one of two exopolysaccharides produced by S. meliloti (succinoglycan or EPS II) is required for nodul

259 acteria with similar ecological niches as S. meliloti, suggesting that the CtrA cell cycle regulatory

260 in mutant nodules, we assayed Sinorhizobium meliloti symbiosis gene promoters (nodF, exoY, bacA, and

261 SMc01113 gene is absolutely required for S. meliloti symbiosis with alfalfa and also for the protect

262 er, in the Medicago truncatula-Sinorhizobium meliloti symbiosis, incompatibility between symbiotic pa

263 (Fix+) in Medicago truncatula-Sinorhizobium meliloti symbiosis.

264 S. meliloti synthesizes an unusual sulfate-modified form of

265 two cpdR homologues, cpdR1 and cpdR2, of S. meliloti that encode single-domain response regulators.

266 c model of the legume symbiont Sinorhizobium meliloti that is integrated with carbon utilization data

267 tic nitrogen-fixing bacterium, Sinorhizobium meliloti The LDSS-P profiles that overlap with the 5' se

268 e study of trehalose uptake by Sinorhizobium meliloti, the demonstrated approach is applicable to man

269 of Sma0114 from the bacterium Sinorhizobium meliloti, the first such characterization of a receiver

270 y do not stimulate nod gene expression in S. meliloti, the flavonoids naringenin, eriodictyol, and da

271 First discovered in Sinorhizobium meliloti, the main response regulator CheY2-P shuttles i

272 Here we identify in Sinorhizobium meliloti, the Medicago symbiont, a cAMP-signaling regula

273 Quorum sensing (QS) in Sinorhizobium meliloti, the N-fixing bacterial symbiont of Medicago ho

274 Sinorhizobium meliloti, the nitrogen-fixing symbiont of alfalfa, has t

275 In the plant symbiont Sinorhizobium meliloti, the ortholog of VtlR, named LsrB, is involved

276 ales order: the plant symbiont Sinorhizobium meliloti, the plant pathogen Agrobacterium tumefaciens,

277 In Sinorhizobium meliloti, the production of exopolysaccharides such as s

278 In particular, in Sinorhizobium meliloti there are four groESL operons and one groEL gen

279 In Sinorhizobium meliloti there are two rpoH genes, four groESL operons,

280 We utilized the genomic sequence of S. meliloti to identify an open reading frame, SMc04267 (wh

281 at ExoR binds to ExoS in the periplasm of S. meliloti to inhibit ExoS/ChvI activity, and that ExoR re

282 obacterial pathogens and might be used by S. meliloti to sense host cues during infection.

283 play an important role in the ability of S. meliloti to successfully invade its host, also helps the

284 rminating seeds, we assayed chemotaxis of S. meliloti towards betonicine, choline, glycine betaine, s

285 This peptide affects Sinorhizobium meliloti transcription, translation, and cell division a

286 A SMc01003-deficient mutant of S. meliloti transiently accumulates diacylglycerol, suggest

287 We recently identified an S. meliloti two-component sensory histidine kinase, CbrA, w

288 S. meliloti unable to produce the exopolysaccharide succino

289 hat a FadD-deficient mutant of Sinorhizobium meliloti, unable to convert free fatty acids to their co

290 ng symbiosis with host plants, Sinorhizobium meliloti undergoes a profound cellular differentiation,

291 When they are available, Sinorhizobium meliloti utilizes C(4)-dicarboxylic acids as preferred c

292 ed inoculum of 68 rhizobial strains (Ensifer meliloti) via a select-and-resequence approach can be us

293 ne sma0113 as needed for strong SMCR when S. meliloti was grown in succinate plus lactose, maltose, o

294 The CbcX orthologue ChoX from Sinorhizobium meliloti was similar to CbcX in these binding properties

295 Searching the genome of S. meliloti, we identified a potential lysophospholipase (S

296 ula and its rhizobial symbiont Sinorhizobium meliloti, which includes more than 23,000 proteins, 20,0

297 We paired the bacterium Ensifer meliloti with one of five Medicago truncatula genotypes

298 ccharides is unclear, although mutants of S. meliloti with reduced LPS sulfation exhibit symbiotic ab

299 Medicago truncatula roots and Sinorhizobium meliloti would identify regulated plant genes that likel

300 previous report, we show that Sinorhizobium meliloti YbeY exhibits endoribonuclease activity on sing